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discuss bio and/or cog etiologies of one disorder
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PTSD defined
mental disorder triggered by experiencing or witnessing a traumatic event
symptoms include - affective (emotion), behavioural (repetitive), cognitive (disruptions to memory/learning), somatic (physical symptoms)
Classification is called DSM 5 criteria (survey) which discusses symptoms of PTSD
Biological etiology - Neurochemical
neuropeptide Y (NPY)
amino acid that maintains homeostasis, specifically the stress response
pp with PTSD have lower levels of NPY
Cognitive etiology - Locus of control (LOC)
concept about whether people believe they have control over their own lives (internal LOC) or if external forces (luck fate) dictate life outcomes (external LOC)
pp with internal LOC take responsibility for actions, less influenced by others, and have strong self-efficacy
pp with external LOC blame others for success or failure, credit luck/chance for success, and are more prone to experiencing learned helplessness
pp with external LOC are more likely to develop PTSD
Morgan et al
discusses neuropeptide Y (NPY) and PTSD
aim of morgan
to investigate why some military. personnel experience severe PTSD as a result of combat situations, while others do not
method of morgan
70 healthy pp from US army
two conditions; survival training experience or prisoner of war experience (potential PTSD)
levels of NPY were assessed before the start of the experiment and 24 hours after
results of morgan
NPY levels went up in soldiers blood within hours of the prisoner of war experience
correlation between soldiers responses to the two conditions and their level of NPY, those who responded to the prisoner of war experience negatively were those with lower levels of NPY
conclusion of morgan
people with lower NPY have a higher chance of developing PTSD from a traumatic experience because the soldiers with lower NPY had a more negative experience with the prisoner of war condition compared to those that did the survival training condition
morgan eval
analog observation instead of naturalistic observation - allowed researchers to stimulate realities of combat without live combat
analog observation could also decrease ecological validity as combat experiences may lack emotional realities of true combat
sampling bias as soldiers voluntarily do their job as men could potentially have personality or physical traits that may explain their level of resistance
NPY examined before and after experiment which allowed for the data to be analysed with same individuals for potential change
Ratzer et al
examined locus of control and PTSD
Aim of ratzer
to see if the type of LOC is a predictor of PTSD
method of ratzer
52 pp who were admitted to intensive care unit following traumatic injury
longitudinal study of six months
pp given surveys to measure both the level of PTSD and to find their LOC
results of ratzer
19.2% of pp developed PTSD symptoms over the six months
Additionally, there was a significant correlation between those with external LOC and the onset of these symptoms
conclusion of ratzer
external locus of control is a predictor of PTSD because there was a significant correlation between the patients who had an external locus of control and the development of PTSD
eval of ratzer
pp were not actually diagnosed with PTSD but instead researchers relied on the results of a questionnaire, meaning it is possible that other traumas experienced by the individuals prior to the accident may have played a role in the onset of PTSD symptoms.
it was a longitudinal study of six months which is the same as the amount of time that is required in the PTSD DSM 5 criteria before diagnosing PTSD
This meant that the researchers were able to follow the participants for long enough to get a good understanding of whether they had onset PTSD symptoms or not
bio etiology eval
animal research has been found to support these theories. This is a strength because animal research is experimental and thus has better control of the environment which shows a stronger cause and effect relationship
animal studies not generalizable to humans due to the difference in cognition between animals and humans and that humans have culture which could also cause a lack of generalizability with animals.
biological explanations cannot explain a variety of PTSD symptoms which means that biology alone cannot be the reason that PTSD is developed by certain people and not others.
cognitive etiology eval
high ecological validity as the research tends to be on pp who have already experienced a traumatic event
generalizability as most traumas experienced are individual and impossible to replicate
correlational meaning they cannot establish causation so there is no cause and effect relationship which means there could be other factors that influence the development of PTSD
ow internal validity as there is no control over pp daily activity and experiences so some pp could discuss their trauma more often than others which may increase or decrease the likelihood of PTSD
conclusion of essay
Thus, two etiologies of PTSD are locus of control (cognitive) and neurochemicals, specifically neuropeptide Y (biology). An individual with an external locus of control and an individual's with less neuropeptide Y are more likely to develop PTSD after experiencing a traumatic event, as supported by Morgan et al and Ratzer et al.