2.23 Developmental Abnormalities of Tendons

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What is the term for a condition where the limb deviates from normal vertical alignment?

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1

What is the term for a condition where the limb deviates from normal vertical alignment?

Flexural deformity.

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2

What are the two types of flexural deformities in horses?

Laxity (lengthening) and "contracted tendons" (shortening).

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3

What are the two categories of flexural deformities?

Congenital and acquired.

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4

What are examples of congenital flexural deformities?

  1. Flexural laxity

  2. Hyperflexion

  3. Rupture/elongation of the common digital extensor tendon

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5

What are examples of acquired flexural deformities?

  1. Club foot

  2. Flexural deformity in metatarso/carpophalangeal joint

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6

What is the cause of flexural laxity?

  • Quite frequent problem in premature foals. Intrauterine infection, mother infection.

  • In older horses, previous injuries or diseases, nutritional deficiencies, lack of exercise, or normal ageing.

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7

What are the common signs of congenital flexural laxity in foals?

Overextension, toe elevation, and contact of the fetlock joint with the ground.

<p>Overextension, toe elevation, and contact of the fetlock joint with the ground.</p>
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8

What causes congenital flexural laxity in foals?

Intrauterine infection, mother infection, and premature birth.

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9

What is the diagnosis of flexural laxity?

X-ray: Check skeletal maturity – shows insufficient mineralization and delayed long bone development.

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10

What is the recommended treatment for flexural laxity?

Box stay, moderate exercise, light bandaging, and shoeing with heel elongation.

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11

When is euthanasia indicated for flexural laxity?

Severe insufficient mineralisation.

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12

Why is surgery contraindicated in most cases of flexural laxity?

Surgery is usually not needed, and severe cases with insufficient mineralization may require euthanasia.

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13

How can flexural laxity be prevented?

  • Prevent premature delivery of foal by proper care of the mare. Premature foals should not be allowed to stand up.

  • With older horses, prompt attention to tendon injuries, good nutrition, and sufficient exercise will help prevent development of weak or lax flexor tendons.

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14

What is the main clinical sign of hyperflexion in foals?

Flexion of the carpal (preying mantis) or fetlock joints (knuckling), with an inability to stand or nurse.

<p>Flexion of the carpal (preying mantis) or fetlock joints (knuckling), with an inability to stand or nurse.</p>
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15

What are possible causes of hyperflexion in foals?

Infection of the mother, intrauterine mispositioning, and unknown factors.

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16

How is hyperflexion diagnosed?

CS, x-ray.

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17

How is hyperflexion treated in foals?

Reshaping, moderate exercise, NSAIDs, oxytetracycline, and surgery to cut flexor tendons (flexor carpi ulnaris/ulnaris lateralis).

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18

What is the effect of oxytetracycline in the tendons?

Binds calcium and causes lengthening.

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19

What occurs when there is a rupture or elongation of the common digital extensor tendon?

Inability to extend the digit and step on the hoof, with over-knuckling and swelling.

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20

What is the diagnosis for rupture or elongation of the common digital extensor tendon?

CS, USG. Free ends of tendon palpated.

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21

What is the best treatment for ruptured or elongated common digital extensor tendon in foals?

Casting or splinting for 2-3 weeks and allowing standing to improve function.

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22

What is club foot in horses?

A condition where the deep digital flexor tendon (DDFT) shortens, causing the hoof to rotate downward.

<p>A condition where the deep digital flexor tendon (DDFT) shortens, causing the hoof to rotate downward.</p>
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23

What age group is most affected by club foot?

Suckling foals between 4 weeks to 6 months.

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24

What is the main cause of club foot in foals?

Overfeeding, mineral imbalances, sudden nutritional changes, hyperparathyroidism, exercise, or trauma.

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25

How can overfeeding lead to club foot?

High energy and protein diet → bone grows faster than tendon.

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26

What are the clinical signs of club foot?

High heels, walking on toes, and lack of contact between the hoof's solar surface and the ground.

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27

What are the stages of club foot?

Stage I: hoof wall <90°; Stage II: >90° - bad prognosis.

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28

How is club foot diagnosed?

X-ray and observation of sole elevation.

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29

What is the treatment for club foot?

Hoof correction, shoeing (e.g., Dallmer shoe), and surgery such as desmotomy of ALDDFT or elongation of the DDFT.

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30

How is club foot prevented?

Take off pasture, reduce energy/protein consumption. Keep on hard ground, hoof trimming.

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31

What is flexural deformity in the metacarpophalangeal joint (MTCP)?

A condition in older foals (10-18 months) involving shortening of the suspensory apparatus and flexor tendons.

<p>A condition in older foals (10-18 months) involving shortening of the suspensory apparatus and flexor tendons.</p>
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32

What are the main causes of flexural deformity in the MTCP joint?

Poor ossification, overfeeding, nutritional changes, mineral imbalances, and trauma.

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33

What are the clinical signs of flexural deformity in MTCP?

Hoof has normal appearance, but straight, upright appearance of pastern → over knuckling

<p>Hoof has normal appearance, but straight, upright appearance of pastern → over knuckling</p>
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34

How is flexural deformity in MTCP diagnosed?

CS.

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35

How is flexural deformity in the MTCP joint treated?

  • Heel support with a wedge

  • Transecting accessory ligaments of the DDFT and SDFT.

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36

What tendon also originates in the same place as the long digital extensor tendon?

Lateral digital extensor tendon.

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37

For flexural laxity, which bones do you look at on x-ray for mineralisation?

Cuboidal bones.

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38

What type of support bandage can you use?

Robert-Jones.

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39

What is the problem with bandaging?

Pressure necrosis, so have to change regularly.

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