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Precordium
The area on the anterior chest overlying the heart and great vessels
Mediastinum
Area between the lungs in the middle third of the thoracic cage. Cavity that houses the heart
Base
“top” broader part of the heart
Apex
“bottom” of the heart points down and to the left
superior and inferior vena cava
pulmonary artery
pulmonary veins
aorta
The great vessels [4]
Superior and inferior vena cava
Returns deoxygenated venous blood to the right side of the heart
Pulmonary artery
;eaves the right ventricle, bifurcates, and carries venous blood to the lungs
Pulmonary veins
Returns freshly oxygenated blood to the left side of the heart
Aorta
Carries blood out to the body
Pericardium
Myocardium
Endocardium
Layers of the heart wall [3]
Myocardium
Muscular wall of the heart, does the pumping
Endocardium
Thin layer of endothelial tissue that lines the inner surface of the heart chambers and valves
Pericardium
Tough, fibrous, double-walled sack that surrounds and protects the heart
Atria
Thin-walled reservoir for holding blood
Ventricles
Muscular pumping chambers
Atrioventricular valves
Valves separating atria and ventricles
Tricuspid valve
Right AV valve
Bicuspid/Mitral valve
Left AV valve
Diastole
Ventricles relax and fill with blood
Systole
Heart’s contraction, blood is pumped from the ventricles and fills the pulmonary and systemic arteries
Cardiac cycle
Rhythmic movement of blood through the heart
First heart sound (S1)
Sound occurs with the closure of the AV valves and thus signals the beginning of systole.
Second heart sound (S2)
Sound occurs with closure of the semilunar valves and signals the end of systole.
Third heart sound (S3)
Vibrations heard over the chest during diastole. Sound occurs immediately after S2, when AV valves open and atrial blood first pours into the ventricles.
Fourth heart sound (S4)
Sound occurs at the end of diastole, when the ventricle is resistant to filling. The atria contract and push blood into noncompliant ventricle. Creates vibrations just before S1.
Murmur
Turbulence in blood flow and collision currents. Noise as blood circulates through normal cardiac chambers and valves, gentle swooshing can be heard on the chest wall
1. Increases in velocity of blood flow (flow murmur; e.g., in exercise, thyrotoxicosis)
2. Decreases in viscosity of blood (e.g., in anemia)
3. Structural defects in the valves (narrowed valve, incompetent valve) or unusual openings in the chambers (dilated chamber, septal defect)
Conditions that result in a heart murmur: [3]
frequency (pitch)
intensity (loudness)
Duration
Timing (diastole or systole)
How heart sounds are described: [4]
Depolarization of atria
What happens during P wave?
the interval from the beginning of the P wave to the beginning of the QRS complex (the time necessary for atrial depolarization plus time for the impulse to travel
What happens during P-R interval
Depolarization of ventricals
wat happens during QRS complex?
Repolarization of ventricles
What happens during T wave?
cardiac output
Volume of blood in each systole times beats per minute
Preload
Venous return that builds during diastole. Length to which ventricular muscle is stretched a the end of diastole just before contraction
Afterload
Opposing pressure that the ventricle must generate to open the aortic valve against the higher aortic pressure. Resistance against which the ventricle pumps blood.
Carotid artery
Jugular veins
Vascular structures in the neck that reflect the efficiency of cardiac function: [2]
Carotid artery
Central artery located in the groove between the trachea and the sternomastoid muscle, medial to and alongside the muscle.
Jugular veins
Empties deoxygenated blood directly into the superior vena cava
Internal jugular
External jugular
Two jugular veins present in each side of the neck:
Internal jugular vein
Larger vein that lies deep and medial to the sternomastoid muscle. Usually not visible, although its diffuse pulsations may be seen in the sternal notch when the person is supine.
External jugular vein
More superficial vein, lies lateral to the sternomastoid muscle, above the clavicle.
Foramen ovale
Opening in the atrial septum, normally closes within the first hour after birth
Ductus arteriosus
Oxygenated blood in fetal circulation is pumped through the pulmonary artery but is detoured through this to the aorta. Closes within 10-15 hours after birth
Increased blood volume (30-40%)
Increased stroke volume and cardiac output
Increased pulse rate by 10-15 bpm
Pregnancy considerations for cardiovascular system: [3]
smoking
diet
alcohol
exercise patterns
stress
Older adult lifestyle considerations for cardiovascular system: [5]
Increase systolic BP
increase supraventricular and ventricular arrythmias
Decreased tolerance for tachyarrhythmias
Decreased ability of heart to augment CO with exercise
Older adult developmental considerations for cardiovascular system:
Lower income households
What households in Canada are more likely to report living with cardiovascular disease?
Incidence of high BP has increased with no significant diferences based on sex. Atlantic provinces have the highest rate, western provinces have the lowest
Influence of socioeconomic factors on high BP
Overweight
BMI of 25 - 29.9
Obesity
BMI of 30 or greater
High blood pressure
– Smoking
– Alcohol
– Serum cholesterol
– Obesity
– Diabetes
– Stress
Social and cultural considerations for cardiovascular system [7]
Chest pain
Dyspnea
Orthopnea
Cough
Fatigue
Cyanosis or ashen or pallor
Edema
Nocturia
Past cardiac history
Family cardiac history
Personal habits (risk factors
Subjective data to collect during cardiovascular health history? [11]
Angina
When heart’s vascular supply cannot keep up with metabolic demand. (chest pain)
Dyspnea
“Shortness of breath” on exertion, constant, or intermittent
Orthopnea
Shortness of breath when lying down, goes away when standing. Needing to assume a more upright posture to breathe (note number of pillows)
Nocturia
Feeling of needing to pee at night
Cyanosis or pallor
Skin tint with myocardial infarction or low cardiac output states as a result of decreased perfusion:
elevated cholesterol level,
elevated blood pressure,
random plasma glucose level value in excess of 11.1mmol/L or
known diabetes mellitus,
obesity,
cigarette smoking,
low activity level,
and, for postmenopausal females, length of any hormone replacement therapy for postmenopausal females.
Risk factors for CAD: [8]
Patient suping with head and chest elevated 30 to 40 degrees
patient position for jugular vein assessment:
Patient sitting up
Patient position for carotid artery assessment:
Warm and quiet (so pt is comfortable and you can hear)
Room prep for physical cardio exam:
Pulse and blood pressure
extremities
neck vessels
Precordium
Order for regional cardiovascular assessment:
Marking pen
Small centimetre ruler
Stethoscope with diaphragm and bell endpieces
Alcohol wipe
Equipment needed for cardio assessment:
Bruit
Noise indicates turbulence with a local vascular cause, such as atherosclerotic narrowing. Blowing, whooshing sound indicating blood flow turbulence
Avoid vagal stimulation
Why palpate carotid arteries one at a time?
inspect the anterior chest
Palpate the apical pulse
palpate across the precordium
How to assess precordium:
4th or 5th intercostal space
Where is apical pulse located?
Supine position or midway of left side
How should patient be positioned to feel apical pulse:
Palpate with palmar aspect of four fingers, over apex, left sternal border, and base for palpations
How to palpate precordium:
Thrill
Palpable vibration, feels like the throat of a purring cat. Signifies turbulent blood flow and accompanies loud murmrs.
aortic area
pulmonic area
tricuspid area
erb’s point
mitral area
Auscultatory areas: [5]
Apex
where is s1 louder than s2:
extra heart sounds
murmurs
s1 and s2 separately
Note rate and rhythm
Things to listen for when ausciltating the heart:
S1 is louder than S2 at the apex; S2 is louder than S1 at the base.
S1 coincides with the carotid artery pulse. Feel the carotid pulse gently as you auscultate at the apex; the sound you hear as you feel each pulse is S1
How to distinguish S1 from S2: [2]
S1 split
Hearing the mitral and tricuspid components separately. Audible in the tricuspid area. Is very rapid, with the two components only 0.03 seconds apart. Rare but normal findinig
Base
Where is S2 loudest?
S2 split
Occurs near the end of inspiration in some people. Inspiration separates the timing of closure of the semilunar valves (pulmonic closes 0.06 seconds after aortic). Only heart in pulmonic valve area.
may be caused by congenital defects and acquired valvular defects.
Systolic murmur may occur with a normal heart or with heart disease
Diastolic murmur always indicates heart disease
Causes of heart murmurs: [3]
120-140
Normal infant heart rate:
Because of fetal shunt closure.
Why are murmurs normal within few days of birth?
Apical impulse is sometimes visible in children with thin chest walls.
Describe child position of the apical pulse:
Venous hum
Common in healthy children and no pathological significance. Represents turbulence of blood flow in the jugular system.
increase 10-15 bpm
Change in pulse rate for pregnancy
Lower than normal. Lowest during second trimester and slowly rises during third.
Blood pressure difference in pregancy:
usually lowest in left lateral recumbent position
a bit higher in supine
highest in sitting
How does pregnant woman’s BP vary with position:
Systolic pressure rises gradually, diastolic stays fairly constant. Some experience orthostatic hypotension
Age-related change in BP:
Orthostatic hypotension
A sudden drop in blood pressure when rising to sit or stand
Could cause a reflex slowing of the heart rate. Also, could compromise circulation if the artery is already narrowed by atherosclerosis
Reason to avoid pressure on carotid artery of older adult:
dilated pupils
pallor, cyanosis
Dysnpea
Orthopnea
Crackles, wheezy breath sounds
Decreased BP
N&V
Ascites (fluid in peritoneal cavity)
Dependent, pitting edema
Weak pulse
cool, moist skin
decreased urine output
enlarged spleen and liver
S2 gallop, tachycardia
Fatigue
Infarct, may be cause of decreased CO
Jugular vein distension
Falling O2 sat.
Anxiety
Signs and symptoms of CHF and heart failure: [19]
Patent Ductus Arteriosus
Persistence of the channel joining left pulmonary artery to aorta. This is normal in the fetus and usually closes spontaneously within hours of birth.
Arterial septal defect
Abnormal opening in the atrial septum, resulting usually in left-to-right shunting of blood and causing a large increase in pulmonary blood flow
Ventricular Septal Defect (VSD)
Abnormal opening in septum between the ventricles, usually in subaortic area. The size and exact position vary considerably.
Tetralogy of Fallot
Four components: (a) right ventricular outflow stenosis, (b) VSD, (c) right ventricular hypertrophy, and (d) overriding aorta. The result is a large amount of venous blood shunted directly into aorta away from pulmonary system, so that blood is never oxygenated.
• Aortic stenosis
• Pulmonic stenosis
• Mitral stenosis
• Tricuspid stenosis
• Aortic regurgitation
• Pulmonic regurgitation
• Mitral regurgitation
• Tricuspid regurgitation
Abnormal Findings – Murmurs
Murmurs caused by valvular defects: [8]