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maintenance period definition
period of time following anesthetic induction in which a stable level of anesthesia is achieved
why is an animal getting too deep
individual variation
ventilating too frequently
improper vaporizer function
technician error
why is an animal getting too light
innapropriate O2 flow rate
is there gas in the vaporizer?
are there leaks in the system?
is your et tube placed properly?
over usage of O2 flush valve
3 body systems we monitor
cardiovascular system
respiratory system
CNS
blood pressure normals
systolic- 80-140 mmHg
diastolic- 50-80 mmHg
MAP- 70-100 mmHg
3 things that contribute to blood pressure
cardiac output
blood volume
vascular resistance
cardiac output equation
CO= SV x HR
causes of hypotension
hypovolemia
overdose of anesthetic
decrease in CO
causes of decrease in CO
bradycardia
intense tachycardia
v tach
decreased SV
drugs
decrease in venous return to heart
positioning of the pt
managing hypotension
± decrease anesthetic gas
bolus IV fluids
give pos inotropic drugs
pos inotropic drugs
ephedrine
dopamine
dobutamine (equine)
ca gluconate
3 ways to measure BP
doppler
oscillometric
direct arterial catheter
values doppler gives you
BP (systolic)
pulse rate
values pulse oximeter gives you
Spo2 (or Pao2)
pulse rate
locations pulse ox can be placed
tongue
pinna of ear
webbing of toe
vulva
prepuce
rectal probe
causes of desaturation
V/Q mismatch
disconnect of breathing system
blocked airways
inadequate o2 flow rate
erroneous readings
probe placement, motion of pt, cautery
hypovolemia
general anesthesia leads to a decreased what?, it can also lead to?
tidal volume of 25%
hypercapnia or atelectasis
4 ways to monitor respiratory system
capnograph
res bag
flutter valves, et tube fogging, esophageal stethoscope, pulse ox
rate and character
capnograph gives what values
RR
etco2
inco2
2 types of capnographs
mainstream
sidestream
controlled ventillation
controlling the volume of air, rate of respiration and pressure of air being introduced into the animal
normally ventilate every 5 min, why?
prevent hypercapnia
prevent atelectasis
3 steps to ventilating
close pop off
squeeze res bag to 20 cmH2O
open pop off
why provide pos pressure ventilation
prevent hypercapnia
precent atelectasis
helps mitigate hypoventilation
help counteract low tidal volume
prevent hypoxemia
if surgical procedure requires it
how to completely take over respirations
ventilate pt at rate of 12-16 rpm to gain control of pt breathing
after approx 3-5 min pt will not be breathing on their own
decrease ventilations to 8-12 rpm for remainder of surgery
after surgery must gradually decrease ventilations to wean pt off
mechanical ventilators
machine will always be hooked up to the res bag port
good for long procedures
mechanical ventilators control what 4 things
inspiratory pressure
inspiratory time
tidal volume
respiratory rate
3 types of mechanical ventilators
pressure cycle ventilator
volume cycle ventilator
time cycle ventilator
values ecg gives you
HR
heart rhythm
most common artifacts on ecg
movement
cautery
60 cycle interference
drying of the electrodes
PVC’s (premature ventricular contractions)
some ectopic source causing ventricles to fire prematurely
have widened QRS complex
see occasionally, no tx needed
2 types of PVC’s
unifocal- originates from same place in ventricle
multifocal- originates from multiple places
generalized causes of PVC
electrolyte imbalance
GDV
excess circulating catecholemines
drugs
tx of PVC’s
find underlying cause
increase ventilations (and then decrease gas)
notify dr
can turn life threatening very quickly
ventricular tachycardia
multiple vpc’s in arow
v- tach causes
same as pvc
very bad cardiac output
v-tach tx
lidocaine drip/cri
v-fib
usually a terminal rhythm
v-fib tx
defibrillation
cpr
asystole
animal must be dead for 3-5 min before you will ever see this
sinus tachycardia
increased heart rate
rhythm is regular
yes to all systemic approach questions
results in increased workload of heart muscle
sinus tachycardia tx
deepen animal
sinus bradycardia
decreased heart rate
rhythm is regular
yes to all systemic approach questions
results in decreased CO
sinus bradycardia tx
± change anesthetic plane
anticholinergic
atrial ventricular blocks
either a delay in or block of conduction between SA node and AV node
1st degree AV block
delay in conduction between atrium and ventricle
be aware of them, but are not a significant problem
2nd degree AV block
some impulses between atrium and ventricle are blocked
not life threatening yet
3rd degree AV block
have more than one lone p wave in consecutive sessions
life threatening condition
t waves normal
should be no more than 25% height of r wave
abnormalities with t waves
spiked = hyperkalemia
broadened + heightened = myocardial hypoxiaabno
abnormal t wave interventions
ventilate pt
± decrease anesthetic gasmali
malignant hyperthermia
not very common
a genetic disorder will have increased muscle activity leading to hyperthermia (under anesthesia)
causes of hypothermia
decreased muscle activity
decrease in overall metabolic rate
introduction of cold anesthetic gas into alveolar sacs
surgical preparation of pt
opening up body cavity
ways to prevent hypothermia
always keep something between pt and table
use a circulating water blanket
use bair hugger
warm IV fluids
consequences of hypothermia > 96 F
no physiologic damage
consequences of hypothermia 90-94 F
decrease in anesthetic requirements
consequences of hypothermia 82-86 F
medically induced coma- no anesthetic requirements