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health promotion
empower clients to control their health habits
goal: help clients to make informed decisions for healthier lives
National: Policies, campaigns, funding (e.g., taxing tobacco, creating parks).
Individual: Education, guidance, disease management, lifestyle counseling.
wellness
individual actions to achieve holistic health and fullest potential
spiritual, mental, mindfulness, physical, environmental
client centered, based on personal choices and needs
disease prevention
measures to limit exposure to or effects of illness/disease
ex) immunizations, hand hygiene, safe sec practices
four levels:
primary: prevent disease (vaccines, seat belts, health counseling)
secondary: screening (BP checks, mammograms, Pap checks)
tertiary: treatments (rehab, diabetes self care, support groups)
quaternary: prevent unnecessary/ excessive medical interventions (avoid over screening)
Illness-Wellness continuum
right side: high wellness/ optimal health
left side: illness, poor health, death
middle: neutral state (neither wellness nor illness)
health communication and strategies
mass media and campaigns to educate on health issues
ex) CDCs anti smoking campaign with real time stories -> reduced smoking rates
Effective education:
Start at first client encounter.
Use simple, clear language.
Provide small bits of information, reviewed often.
Include printed resources for reinforcement.
healthy people initiative
established by DHHS
new goals every 10 yrs
Provides:
Framework, history, and principles for national health goals.
Public access to 10+ years of health data.
Tools for health professionals and communities.
Interactive data access for individuals/groups to create personal health goals.
Goal: Make the U.S. a leader in population health.
self care
clients promote and maintain own health
seven pillars:
Knowledge & health literacy.
Mental well-being & self-awareness.
Physical activity.
Healthy eating.
Risk avoidance (vaccines, safe sex, avoid tobacco/alcohol).
Good hygiene.
Rational/responsible use of products & services (medications, supplements, preventive care).
nurse self care
healthy meals, exercise, stress reduction, adequate sleep, social support
risk factors
Definition: A Condition that increases the likelihood of disease/altered health.
Modifiable (can change): High BP, tobacco, high-fat diet, alcohol, sedentary lifestyle, uncontrolled diabetes.
Nonmodifiable (cannot change): Age, gender, race/ethnicity, family history.
Nurses: focus on screenings, lifestyle changes, and prevention.
environmental factors
Health affected by air/water quality, pollutants, waste, safety of homes/work/schools.
Risks: Poor air → asthma, strokes, heart disease.
Unclean water → outbreaks of waterborne diseases.
Indoor hazards: Mold, radon, cockroaches, lead paint → developmental/respiratory issues.
socioeconomic factors
Poverty = poor health outcomes, shorter life expectancy.
Contributing issues: Lack of insurance, limited access to care, poor nutrition, sedentary lifestyle, unsafe neighborhoods, stress.
Lower-income groups report higher rates of chronic disease and mental health issues.
health literacy
Ability to understand and apply health information.
Low health literacy → poor medication adherence, poor follow-up, difficulty with self-care.
cultural beliefs
Affects how clients perceive health, illness, and treatment.
Can influence: Medication adherence, end-of-life decisions, pain expression, treatment choices.
Nurses must:
Assess cultural values/beliefs.
Develop cultural competence (awareness, communication, adaptation).
Provide culturally competent care → reduces disparities & improves outcomes.
family dynamics
Family = biologically, legally, or emotionally related individuals.
Families influence health decisions, coping, stress, and compliance.
Patient- and family-centered care (PFCC): Involves both client and family in health planning.
Nurses should include families in education, address barriers, and respect family input.
organs
esophagus → Connects the mouth to the stomach
Stomach → Holds food for 2 to 8 hours
Liver → Produces bile
Gallbladder → Stores and releases bile
Pancreas → Helps metabolize sugars through insulin
Small intestine → Where most of the nutrients from food are absorbed
Large intestine → Turns food into feces (absorbs water/electrolytes)
Rectum → Temporary storage area for feces
Anus → Where feces leave the body as waste
glycemic index
a high glycemic index causes a rapid rise in blood glucose.
food like potatoes have high glycemic indexes
vegetables, legumes, whole grains have low glycemic index -> maintain stable energy and blood sugar levels
calories calculation
servings x calories/serving
ex) Q: Client ate 2 servings of chips (150 cal/serving) + ½ serving of turkey sandwich (600 cal/serving). How many calories total?
Chips: 2 × 150 = 300 calories
Sandwich: 0.5 × 600 = 300 calories
Total = 600 calories
net carbohydrates
required for calculating net carbohydrates
total carbohydrates per serving
dietary fiber
alcohol sugars
sodium
Client eats 1.5 servings of product. How much sodium?
Sodium per serving = 572 mg
1.5 × 572 = 858 mg sodium
Q: If client’s daily sodium limit is 1,800 mg, what %DV is 858 mg?
858 ÷ 1,800 = 48% of daily sodium
physiology of digestion
Digestion = breakdown of food into simple substances → absorbed as nutrients or eliminated as waste.
Provides energy and nourishment for cells, tissues, and organs.
Begins with food consumption → saliva starts breakdown.
Peristalsis = involuntary muscular contractions that move food through digestive tract.
organs of digestion
Mouth → chewing & saliva (enzymes start breakdown).
Esophagus → hollow tube, propels food to stomach.
Stomach → holds food (2–8 hrs); acids & enzymes digest food.
Liver → filters toxins; produces bile (breaks down fats, proteins, carbs).
Gallbladder → stores & releases bile when needed.
Pancreas → produces insulin (regulates sugar metabolism).
Small intestine → final breakdown of food; absorbs nutrients into bloodstream.
Large intestine → absorbs water & electrolytes; converts remainder into feces; contains beneficial bacteria.
Rectum → stores feces until elimination.
Anus → external opening for expelling feces.
common digestive issues
Diarrhea, constipation, nausea, vomiting, abdominal pain, gas, bloating.
Nurses should assess for GI discomfort regularly.
healthy eating patterns
Balanced diet: fruits, vegetables, legumes, whole grains, lean proteins.
Limit unhealthy fats, salt, added sugars, refined grains.
USDA daily recommendations:
2 cups fruit, 2½ cups vegetables, 6 oz grains, 5½ oz protein, 3 cups dairy.
“MyPlate” → visual guide for balanced meals.
nutrients
Macronutrients (needed in large amounts): protein, carbs, fats.
Micronutrients (small amounts): vitamins, minerals.
protein
Major building block → repairs, builds, maintains tissue.
Sources: beans, soy, nuts, meats, eggs, fish.
Needs vary by age, sex, and activity level.
carbohydrates
Primary fuel source (45–65% daily calories).
Simple carbs → sugars (fructose, sucrose, lactose).
Complex carbs → starch & fiber.
Glycemic Index: high GI foods = rapid glucose spike (potatoes, white bread). Low GI = steady glucose (legumes, whole grains).
fats
Provide energy, absorb vitamins, affect cholesterol levels.
Healthy fats = monounsaturated (avocados, nuts, olive oil).
Unhealthy fats = trans fats (processed foods), saturated fats (full-fat dairy, butter).
vitamins
Water-soluble (not stored): B vitamins, vitamin C.
Fat-soluble (stored in liver/fat): A, D, E, K.
minerals
Macrominerals: calcium, phosphorus, magnesium, sodium, chloride, potassium, sulfur.
Trace minerals: iron, zinc, iodine, copper, selenium, fluoride, manganese, chromium.
water
Essential nutrient → more than half body weight.
Functions: brain activity, hydration, waste removal, temperature regulation, lubrication.
Daily intake:
Women → 2.7–3 L/day.
Men → 3.5–3.7 L/day.
nutrition and health
Empty calories = low nutritional value (chips, cookies, soda).
Calorie density: low (fruits/veggies) vs. high (fried/processed foods).
Fiber: improves digestion, lowers cholesterol, stabilizes blood sugar.
reading food labels
Serving size = basis for all nutrient values.
Calories → per serving.
Watch for high sodium, added sugars, saturated fat.
% Daily Value:
<5% = low in nutrient.
≥20% = high in nutrient.
Net carbs = total carbs – fiber – sugar alcohols.
aspiration
entry of food, liquid, or other material into the lungs
overt aspiration
visible signs like coughing, wheezing, choking, congestion, heartburn, throat clearing, or chest discomfort
silent aspiration
no obvious symptoms, making it harder to detect
risk factors for aspiration
dysphagia, poor swallowing reflex, stroke, reflux, mouth sores, dental issues
diet modifications for aspirations
thicken liquids (nectar, honey, pudding consistency) to reduce risk
tube feeding precautions
Verify tube placement with X-ray or capnography.
Keep HOB at 30–45° during feeding.
Monitor for signs of intolerance (vomiting, gastric distention, diarrhea).
Stop feeding if aspiration signs occur (wheezing, coughing, fever).
assisting with eating with risk of aspiration
Nursing responsibilities: Assess swallowing, assist as needed, promote independence, use assistive devices if necessary.
Client positioning: Sit upright at 90°, or elevate HOB to 90° with pillows if in bed.
Pre-meal checklist: Restroom, hand hygiene, dentures, hearing aids, free of clutter, food within reach
A nurse is caring for a client who is at risk for aspiration. Which of the following actions should the nurse take to prevent aspiration during meals? (Select all that apply.)
Cut food, such as meat, in half for easier chewing
Allow extra time for the client to chew and swallow each bite of food
Sit the client up at 90° prior to providing the meal
swallowing techniques for dysphagia
Chin-tuck position
Hold chin to chest while swallowing to narrow airway
Rotation to affected side
Turn head to weak side to direct food to strong side
Tilting to strong side
Tilt head to strong side to push food down
Supraglottic swallow
Swallow while holding breath, then cough to clear residual
Mendelsohn maneuver
Hold swallow 2–3 sec, then relax to improve swallowing efficiency
glucose monitoring
Purpose: Evaluate blood glucose changes, monitor diabetes management, assess diet or stress impact.
Fasting BG: 70–110 mg/dL for non-diabetic client.
Postprandial BG (2 hr after meal): <140 mg/dL.
Hypoglycemia: BG <70 mg/dL. Treat with 15 g carbohydrates (adjusted for age: infants 6 g, toddlers 8 g, small children 10 g). Recheck in 15 min.
insulin basics
Secreted by beta cells, lowers blood glucose
Alpha cells secrete glucagon (raises glucose)
Administered subcutaneously (U-100 most common)
Injection sites: avoid bruised, tender, lumpy areas; 45° for thin, 90° for adequate tissue
Types: rapid, short, intermediate, long, ultra-long acting (differences in onset, peak, duration)
enteral vs parenteral nutrition
Enteral: Nutrition via GI tract (tube feeding). Safer, associated with lower infection, preferred over parenteral. Contraindications: GI bleeding, obstruction, ischemia.
Parenteral: IV nutrition. Used when GI tract cannot absorb nutrients. Can be partial or total. Monitor labs and glucose. Insulin may be added for hyperglycemia.
gastronomy tube feeding
G-tube: Surgically placed (PEG, laparoscopic, or open) into the stomach. Provides nutrition for clients who cannot eat enough orally.
NG tube: Temporary, through nose into stomach. Confirm placement via X-ray before use.
NJ tube: Through nose into jejunum. Used when gastric feeding not appropriate. Confirm with X-ray.
Tube feeding precautions: HOB elevated, monitor residuals, observe for intolerance or aspiration signs.
Bolus NG feeding question:
pH 6.5 → indicates possible correct gastric placement, but verify protocol. Often nurse would recheck pH to confirm placement before feeding.
infection control in pediatric nursing
Advocate, educator, and implementer of infection control measures.
Promote healthy lifestyles: nutrition, sleep, hygiene.
Educate families about vaccines and immunizations.
Ensure children are up-to-date with immunization schedules.
Screen, assess, and monitor for infection.
benefits of client education include
Improves health status and outcomes.
Encourages autonomy and decision-making.
Promotes adherence to treatment.
Reduces anxiety and enhances safety.
hand hygiene
Fundamental in infection prevention.
Applies to health care workers, patients, families, and visitors.
immunizations
Nurses educate parents about importance, risks of vaccine-preventable diseases, and recommended schedules.
Maintain accurate records and employ reminders for upcoming or missed vaccines.
standard precautions
Used for all clients.
Treat all blood, body secretions (except sweat), damaged skin, and mucous membranes as potentially infectious.
Include:
Hand hygiene
PPE (gloves, gowns, masks, eye protection)
Safe injection practices
transmission based precautions
Contact: Direct/indirect contact with child or environment.
Single room preferred
Gown and gloves
Examples: MRSA, VRE, C. difficile, wound drainage
Droplet: Spread through close respiratory contact (<3 ft).
Standard mask for close contact
Single room preferred
Child wears mask if transported
Examples: pertussis, influenza, meningitis
Airborne: Spread through small infectious particles in the air; travel long distances.
Negative pressure room
N95 or respirator required
Examples: TB, SARS-CoV, measles (rubeola), varicella
PPE donning
hand hygiene
gown
mask
goggles/face shield
gloves
PPE doffing
gloves
goggles/face shield
gown
mask
hand hygiene
vaccine preventable vs non vaccine preventable diseases
Vaccine-Preventable:
Measles (Rubeola)
Pertussis
Mumps
Varicella
Non-Vaccine-Preventable:
Erythema infectiosum (Fifth disease)
Roseola
Hand, Foot, and Mouth Disease
Eryhtema infectiosum
Etiology: Caused by parvovirus B19.
Transmission: Respiratory secretions.
Clinical Presentation: “Slapped cheek” rash on face, mild fever, malaise.
Complications: Rare, but can include anemia in immunocompromised clients or fetal hydrops in pregnancy.
Vaccine: Non-vaccine-preventable.
Hand foot and mouth disease
Pathophysiology:
Caused by human enterovirus or coxsackievirus.
Virus enters via mouth → reproduces in intestinal & pharyngeal lymph tissue → spreads to lymph nodes.
It can affect the skin, CNS, heart, and liver.
Virus sheds in stool ~6 weeks, oropharynx ~4 weeks.
Transmission:
Oral-fecal route (diaper changes)
Contact with contaminated surfaces
Close contact (sharing utensils)
Respiratory droplets
Contact with vesicle fluid
Risk Factors: Children <5 years; outbreaks in late spring/summer; highly contagious first 7 days; incubation 3–6 days.
Clinical Presentation:
Low-grade fever, sore throat, malaise
Painful mouth ulcers with red borders, white/gray-yellow base
Maculopapular or vesicular rash on hands, feet, buttocks, arms, legs
Lab Testing: Usually clinical diagnosis; stool or vesicle scraping if needed.
Treatment: Supportive care
Hydration, soft/cold foods, pain management (acetaminophen/NSAIDs
Nursing Interventions:
Comfort measures: hydration, soft foods, analgesics
Limit contact during contagious phase
Avoid acidic or hot beverages
Prevention: Hand hygiene, clean environment; no vaccine available.
impetigo
Etiology:
Bacterial: Group A Streptococcus or Staphylococcus aureus
Primary (unbroken skin) vs Secondary (existing lesion)
Clinical Presentation:
Nonbullous: honey-crusted lesions, face/other exposed areas
Bullous: large blisters, clear/yellow → purulent fluid
Fever and lymphadenopathy possible in bullous
Lab Testing: Clinical evaluation; bacterial culture or biopsy if needed.
Treatment:
Nonbullous: topical mupirocin, sometimes oral antibiotics if multiple lesions
Bullous: oral antibiotics (e.g., cephalexin)
Nursing Interventions:
Education: medication adherence, hygiene, avoid spread
Comfort: gentle skin care, avoid scratching
Prevention: Handwashing, clean clothing/linens, cover lesions, avoid contact during outbreak.
conjunctivitis
Bacterial Conjunctivitis
Etiology: Streptococcus pneumoniae, Moraxella catarrhalis, Haemophilus influenzae
Transmission: Close contact, contaminated surfaces
Clinical: Redness, purulent drainage, matted eyelids
Treatment: Topical antibiotics (ciprofloxacin drops)
Nursing: Hygiene, apply medication, cool/warm compresses
Viral Conjunctivitis
Etiology: Adenovirus, HSV, enteroviruses
Clinical: Redness, watery discharge, often bilateral, may follow URI
Treatment: Supportive care (cool compresses, artificial tears)
Nursing: Avoid touching eyes, hand hygiene, isolation until asymtomatic
Allergic Conjunctivitis
Etiology: Allergens → IgE-mediated inflammation
Clinical: Redness, itching, tearing, both eyes, no discharge
Treatment: Antihistamine/mast-cell stabilizer drops, avoid allergens
Nursing: Environmental control, cool compresses
4. Foreign Body Conjunctivitis
Etiology: Irritation from dust, sand, chemicals
Clinical: Sudden redness, tearing, gritty sensation
Treatment: Remove object, flush with water or saline
Nursing: Prevent rubbing, monitor for complications
stomatitis
Herpetic Gingivostomatitis
Etiology: HSV-1 (sometimes HSV-2)
Clinical: Painful vesicles/ulcers on gums, tongue, cheeks; fever, irritability, difficulty eating
Lab: Viral culture of ulcer
Treatment: Oral or topical antivirals (acyclovir)
Nursing: Hydration, pain management, hygiene, infection control
Aphthous Stomatitis (Canker Sores)
Etiology: Non-infectious, inflammatory, T-cell mediated
Clinical: Painful round/oval ulcers, white-gray/yellow center with red border; typically inner lips/cheeks/tongue
Lab: Clinical diagnosis
Treatment: Topical analgesics, corticosteroids (triamcinolone), avoid triggers
Nursing: Oral hygiene, soft foods, hydration, stress management, avoid spicy/acidic foods
hepatitis
Hepatitis
Definition: Inflammatory condition of the liver caused by viral infections.
Types & Transmission
Hepatitis A: Fecal-oral (contaminated food/water)
Hepatitis B: Bloodborne, sexual contact, body fluids
Hepatitis C: Bloodborne (IV drug use, transfusions)
Hepatitis D: Requires hepatitis B co-infection
Hepatitis E: Fecal-oral, contaminated water
Pathophysiology
Hep A & B: <1% lead to liver failure; incubation 4–12 weeks
Hep C: Chronic infection >50% of cases; incubation ~8 weeks
Risk Factors
Unsafe sexual practices
Intravenous drug use
Poor sanitation
Exposure to infected blood/body fluids
Clinical Presentation
Fatigue, fever, jaundice, anorexia, abdominal pain, nausea, dark urine
Hep A: GI symptoms predominant (nausea, vomiting, diarrhea)
Hep B:
Prodromal phase: malaise, decreased appetite, RUQ pain
Icteric phase: jaundice, hepatomegaly, pale stool, dark urine
Hep C: Mostly asymptomatic; malaise, decreased appetite if symptomatic
Diagnostics
Blood tests for viral markers and liver function
Hep A: IgM antibodies
Hep B: HBsAg, anti-HBs, Ig tests
Hep C: Anti-HCV, liver biopsy
Treatment
Hep A: Supportive care; hydration, manage complications
Hep B:
Acute: supportive, lamivudine if severe
Chronic: antiretroviral therapy
Hep C: Interferon alfa-2b, antivirals for chronic infection
Nursing Interventions
Monitor vitals
Promote hydration & nutrition
Encourage rest
Medication adherence for chronic cases
Educate family about transmission and preventive measures
Prevention
Hep A & B: Vaccination available
Hep C: Avoid blood exposure, sterile needles
Infectious Mononucleosis (EBV)
Definition: Viral infection caused by Epstein-Barr virus; affects B lymphocytes.
Transmission
Saliva (kissing disease)
Close contact
Clinical Presentation
Fever, pharyngitis, fatigue, lymphadenopathy, headache, malaise
Tonsillar exudates may be present
Splenomegaly (50%) or hepatosplenomegaly
Rare: airway obstruction, splenic rupture, encephalitis, Guillain-Barre syndrome
Diagnostics
Heterophile antibody (Monospot) test
Increased lymphocyte count
Treatment
Supportive care: rest, hydration, NSAIDs for pain/fever
Nursing Interventions
Encourage rest and hydration
Avoid contact sports for 4–6 weeks (risk of splenic rupture)
Educate on manifestations of complications
Emotional support and monitoring
Prevention
Avoid sharing utensils, cups, or saliva
Hand hygiene
roseola Infantum (exanthem subitum)
Definition: Viral infection, mainly by HHV-6, affecting infants 6–12 months
Transmission
Saliva, respiratory droplets
Clinical Presentation
Sudden high fever (up to 104°F) for 3–5 days
Irritability, periorbital edema, otalgia, mild upper respiratory symptoms
After fever: rose-pink, maculopapular, non-pruritic rash starting on trunk, spreading to neck, face, extremities
Risk: febrile seizures
Treatment
Supportive: hydration, fever management
Nursing Interventions
Ensure hydration
Dress child in lightweight clothing
Educate parents about febrile seizures
Limit contact with vulnerable individuals until 24h after fever subsides
Prevention
Difficult due to asymptomatic contagious phase
Immunity develops after infection
No vaccine currently available
varicella (chickenpox)
Pathophysiology:
Caused by varicella-zoster virus (VZV).
Primary infection = chickenpox; latent virus can reactivate as shingles (herpes zoster).
Infection occurs in respiratory epithelium, spreads via bloodstream (viremia), forming vesicles.
Transmission:
Airborne droplets or direct contact with vesicle fluid.
Incubation: 2–6 days to primary viremia, 10–12 days to secondary viremia.
Clinical Manifestations:
Pruritic vesicular rash → scabs (chest, back, face → rest of body, mouth, eyelids, genitals, scalp).
Fever, fatigue, sore throat, headache (5–7 days).
Diagnostics:
Clinical evaluation; fluid from vesicles or scab scraping.
Blood tests for acute immunologic response (IgM, IgG).
Treatment:
Supportive: acetaminophen for fever, analgesics for pain, calamine lotion, oatmeal baths, antihistamines (e.g., diphenhydramine).
Avoid aspirin (risk of Reye syndrome).
Hydration essential; monitor for secondary bacterial infection.
Nursing Interventions:
Comfort measures: lukewarm baths, light clothing, nail trimming.
Airborne + contact precautions in hospital; keep at home until all lesions crusted (~1 week).
Prevention:
Varicella vaccine (2 doses); hand hygiene and avoiding exposure.
diphtheria
Pathophysiology:
Caused by Corynebacterium diphtheriae.
Produces exotoxins → gray pseudomembrane, airway obstruction, myocarditis, neuritis.
Transmission: Airborne droplets.
Risk Factors:
Non-immunized children, crowded living, travel to endemic areas (Southeast Asia, Africa).
Clinical Manifestations:
Flu-like symptoms: sore throat, fever, malaise, cervical lymphadenopathy.
Gray pseudomembrane over throat/tonsils (hallmark).
Diagnostics:
Throat swab for culture, PCR, toxin testing.
Treatment:
Diphtheria antitoxin (DAT), antibiotics (penicillin G, erythromycin), supportive care.
Nursing Interventions:
Manage fever (acetaminophen), soft diet, fluids, calm environment.
Infection control; isolation to prevent spread.
Prevention:
DTaP vaccine; immunization of close contacts if case occurs.
mumps
Pathophysiology:
Caused by Paramyxoviridae virus; replicates in upper airway mucosa → viremia → salivary glands, testes/ovaries, CNS (rare).
Transmission: Respiratory droplets, saliva, fomites.
Incubation: 7–21 days; contagious 1–2 days before symptoms, 5 days after onset.
Clinical Manifestations:
Parotitis (swelling near jawline), fever, headache, myalgia, fatigue.
Possible complications: orchitis, meningitis, encephalitis.
Diagnostics: RT-PCR (buccal swab), IgM serology.
Treatment:
Supportive care: rest, hydration, analgesics, hot/cold compresses, soft diet.
Prevention:
MMR vaccine; isolation during contagious period.
measles (rubeola)
Pathophysiology:
Caused by Morbillivirus; enters via nasopharyngeal/conjunctival mucosa → lymph nodes → blood → systemic spread.
Transmission: Respiratory droplets; highly contagious.
Incubation: 10–14 days.
Clinical Manifestations:
Fever, cough, rhinorrhea, conjunctivitis.
Koplik spots: small white spots in mouth.
Rash: hairline → body.
Diagnostics: Clinical + serology or PCR confirmation.
Treatment: Supportive: hydration, nutrition, vitamin A supplementation, manage fever and respiratory symptoms.
Prevention: MMR vaccine; herd immunity important.
Precautions: Airborne isolation if hospitalized.
poliomyelitis (polio)
Pathophysiology:
Caused by poliovirus (enterovirus); replicates in oropharynx and GI tract.
Primarily affects nervous system, may cause paralysis.
Transmission: Fecal-oral, nasopharyngeal secretions.
Clinical Manifestations:
Mild: flu-like (2–5 days).
Severe: paralysis, usually legs; can involve respiratory muscles.
Diagnostics: Throat swab, stool, CSF PCR; MRI/EMG for nerve damage.
Treatment: Supportive: hydration, nutrition, physical therapy, respiratory support, mobility aids.
Prevention: IPV vaccine (inactivated polio vaccine); isolation if infected.
COVID-19 (SARS-CoV-2)
Pathophysiology:
Viral infection affecting multiple systems; overactive cytokine response in severe cases.
Transmission: Respiratory droplets, direct contact; possible fecal-oral.
Clinical Manifestations in children:
Often mild/asymptomatic; fever, cough, respiratory symptoms.
Rare: MIS-C (inflammation in heart, lungs, kidneys, GI).
Diagnostics: PCR or rapid antigen tests; antibody testing for prior infection.
Treatment:
Supportive; severe cases may require oxygen, remdesivir (≥28 days old, ≥3 kg), ritonavir-boosted nirmatrelvir (≥12 yrs, ≥40 kg).
Nursing Interventions: Rest, hydration, fever management, isolation, emotional support.
Prevention: Vaccination ≥6 months old, mask-wearing, hand hygiene, social distancing.
different factors and extra questions
Viral Meningitis risk factors
Close contact with infected individuals
Being in crowded settings
Having a weakened immune system
Seasonality: more common in summer and fall
viral meningitis 4 month old manifestations
Hypothermia (in newborns/young infants)
Poor feeding
Irritability (difficult to console)
Lethargy
Full or bulging anterior fontanel
Viral meningitis- infant interventions
Prepare the infant for a lumbar puncture
Prepare to administer an antibiotic (empiric until bacterial ruled out)
Maintain a quiet environment for the infant
Monitor the infant for seizures
Bacterial meningitis adolescent manifestations
Fever
Headache
Nausea and vomiting
Photophobia
Nuchal rigidity
Irritability
Lethargy
Diplopia or blurred vision
Positive Brudzinski and Kernig signs
Petechial rash (if meningococcal infection)
Bacterial meningitis first intervention
1. Administer empirical antibiotics (priority to prevent morbidity/mortality)
(Lumbar puncture, corticosteroids, and prophylaxis for contacts follow as indicated.)
Bacterial Meningitis adolescent interventions
Administer analgesia
Maintain a dark environment
Hep B most likely mode of transmission
Acquired by coming into contact with infected blood or body fluids
Hep B icteric phase manifestations
Yellow sclera
Dark urine
Right upper quadrant pain
Hep A preschooler complications
Dehydration
Hepatic failure (rare)
Electrolyte imbalances
Jaundice
Nutritional deficits
Hep B nursing interventions
Monitor liver function tests and vital signs
Encourage rest and adequate hydration
Provide small, frequent meals to support nutrition
Administer antiviral medications if prescribed
Educate family on preventing transmission
Mononucleosis indications
Spleen palpable 2–3 cm below costal margin
Temperature 38.6° C (101.5° F)
Cervical lymph nodes 2 cm
Mononucleosis Home management
You should gargle with warm salt water
Roseola Infantum parent teaching
Offer the infant drinks of electrolyte solutions
The child can return to daycare 24 hours after the fever subsides
Monitor the infant for febrile seizures
Do not dress in warm clothing
there is no vaccine for roseola.
extra measures with communicable disease
Isolation Precautions:
Airborne + contact: Chickenpox, measles.
Contact: Polio, COVID-19.
Comfort Measures:
Fever management: acetaminophen (avoid aspirin in viral infections).
Rash/itching: calamine, antihistamines, oatmeal baths.
Hydration and nutrition support.
Prevention:
Vaccination is the cornerstone: Varicella, DTaP (diphtheria), MMR (mumps, measles), IPV (polio), COVID-19.
Family Education:
Stay home until no longer contagious, monitor for complications, hygiene measures, and vaccine adherence.
immunotherapy terms
Antibody (Immunoglobulin)
Y-shaped proteins composed of two heavy and two light chains.
Function:
Neutralize pathogens.
Tag pathogens for destruction (opsonization).
Activate complement system.
Stimulate immune cells (ADCC).
Types:
IgG: Most abundant; long-term immunity.
IgM: First antibody produced during infection.
IgA: Found in bodily secretions; localized immunity.
IgE: Allergic responses; defense against parasites.
IgD: Found on B cells; function less understood.
Role in vaccines: Stimulated to provide immunity against specific pathogens.
Role in diagnostics: Detected to determine current or past infections.
Role in therapy: Monoclonal antibodies target specific diseases (e.g., COVID-19, cancer).
2. Antigen
Substance that provokes an immune response.
Can be:
Exogenous (from outside the body).
Autoantigens (originating inside the body; may cause autoimmune disorders).
Stimulates:
Antibody production.
T-cell activation to destroy infected cells.
Vaccines contain harmless antigens to produce memory cells.
3. Antitoxin
Antibody targeting toxins from bacteria, plants, or animals.
Neutralizes toxins, preventing cell/tissue damage.
Uses:
Treatment of botulism, tetanus, snake bites.
Vaccine development (toxoid vaccines).
4. Attenuate
Process of weakening a pathogen while retaining its ability to stimulate immune response.
Used in live attenuated vaccines (e.g., varicella, MMR).
Careful balance: effective immunity without causing disease.
5. Cocooning
Public health strategy protecting vulnerable individuals (e.g., newborns) by immunizing close contacts.
Reduces disease transmission to high-risk individuals
immunity types
1. Natural Immunity (Innate)
Present from birth.
Rapid, nonspecific defense:
Physical barriers: skin, mucous membranes.
Immune cells: macrophages, neutrophils, NK cells.
Inflammatory response.
Does not provide long-term pathogen-specific memory.
2. Acquired Immunity (Adaptive)
Develops after exposure to specific pathogens.
Two forms:
Active Immunity: Body produces antibodies (natural infection or vaccination).
Passive Immunity: Antibodies transferred from another person (maternal antibodies, immunoglobulin therapy).
Provides targeted defense and memory (active) or immediate temporary protection (passive).
3. Herd Immunity
Indirect protection of non-immune individuals when a large portion of the population is immune.
Threshold varies with disease contagiousness (e.g., 95% for measles).
Supports community protection and disease prevention.
immunobiologics
1. Vaccines
Contain weakened, killed, or components of pathogens.
Stimulate antibody and memory cell production.
Types:
Combination: Multiple antigens in one injection.
Conjugate: Polysaccharide antigen linked to carrier protein (e.g., Hib, pneumococcal).
Monovalent: Targets one pathogen.
Polyvalent: Targets multiple strains or related pathogens.
Considerations: Contraindications vs. precautions; mild illness usually not a reason to delay.
2. Immunomodulators
Medications that enhance or suppress immune responses.
Used for autoimmune diseases or immunodeficiency.
3. Monoclonal Antibodies
Lab-produced antibodies targeting specific proteins or cells.
Treat cancer, autoimmune disorders, infectious diseases.
4. T-cell Therapy
Uses patient’s own immune cells or genetically modified cells to target diseases (cancer, infections).
5. Immunoglobulin Therapy
Pre-formed antibodies from donors (IVIG or injections like HBIG).
Supports patients with immune deficiencies (e.g., SCID, Kawasaki disease).
6. Toxoids
Inactivated bacterial toxins in vaccines (e.g., diphtheria, tetanus).
Stimulate antibody production without causing disease.
vaccine considerations
Contraindication: Permanent exclusion due to high risk (e.g., severe allergic reaction).
Precaution: Elevated risk but can still vaccinate with monitoring.
Mild illness does not delay vaccination.
Administration:
IM injections (vastus lateralis for infants, deltoid for older children/adults).
example vaccine Hib
Protects against Haemophilus influenzae type B.
Series: 2, 4, (6), and 12–15 months.
Effective in preventing meningitis and epiglottitis in children under 5.
extra vaccine questions
Where to insert HiB vaccine for 4-week-old infant
Administer the injection in the infant’s vastus lateralis
Hepatitis A and B teaching
“The common reactions to the vaccine are pain at the injection site and a mild fever.”
Poliovirus vaccine for a febrile 2-month-old
Delay the administration of the poliovirus vaccine
Pneumococcal vaccine education
The child should be monitored for 15 minutes following administration.
Rotavirus vaccine teaching
Common adverse effects of the vaccine include mild diarrhea and vomiting.
Varicella vaccine education
The varicella vaccine protects against chickenpox. It is usually given in two doses: the first at 12–15 months, and the second at 4–6 years
Vaccine for an 8-year-old requiring whooping cough immunization
Tdap
8. MMR vaccine concerns
“I understand your concerns. Extensive research shows the MMR vaccine is safe and is not linked to autism. It protects against measles, mumps, and rubella
HPV vaccine schedule for an 11-year-old
Two doses administered 6 to 12 months apart
Rationale: Children 9–14 years old receive 2 doses; 3 doses are for ages 15–45 or immunocompromised.
Meningococcal vaccine education for an 18-year-old
The adolescent should receive MenACWY if not previously vaccinated. The first dose is typically given at 16–18 years if missed earlier. The vaccine is IM, commonly in the deltoid, and mild side effects may include soreness at the injection site, fatigue, or headache.
Influenza vaccine for a 9-month-old who has never had it
Administer the vaccine and inform the parent that the child will need a second injection in 1 month.
Refer to the manufacturer’s instructions to determine the dose of the vaccine for the child.
Rationale: Children <9 years old receiving the flu vaccine for the first time need two doses 1 month apart. It is IM, not subcutaneous. Annual vaccination is required, not every other year, and it can be given before 12 months.
Influenza vaccine for a 6-year-old with egg allergy
Use special precautions when administering the influenza vaccine to the child.
Rationale: Egg allergy is not a contraindication, but monitoring is recommended.
COVID-19 vaccine for a 3-year-old moderately immunocompromised child
“Your child should receive an annual booster once the original series is completed.”
COVID-19 vaccine teaching for a newly licensed nurse
Monitor adolescents for 15 minutes for syncope following administration.
There is a risk of myocarditis in children 12 years old and older following immunization.
Avoid further immunization in children who experience pericarditis following a prior COVID-19 immunization.
parasitic disease overview
Definition: Illnesses caused by parasites (protozoa, helminths, arthropods) that thrive at the host's expense.
Transmission Routes:
Contaminated food/water
Insect bites
Contact with infected individuals
Contaminated soil
Common Parasitic Diseases:
Malaria: Plasmodium via mosquito
Amoebiasis: Entamoeba histolytica via contaminated food/water
Giardiasis: Giardia lamblia via contaminated water
Helminthic infections: Pinworm, hookworm, roundworm, tapeworm via soil/food/contact
Clinical Presentation:
Mild: GI discomfort, fever, skin rashes
Severe: Multi-organ involvement
Diagnosis: Lab tests (blood, stool, tissue)
Management:
Prevention: Sanitation, clean water, vector control, health education
Treatment: Antiparasitic medications, supportive care
Nursing Role: Prevention education, hygiene promotion, proper sanitation, monitoring, and follow-up
scabies (integumentary)
Cause: Sarcoptes scabiei mite burrowing under skin
Transmission: Direct skin-to-skin contact or contaminated linens (~10 minutes sufficient)
Pathophysiology:
Mite burrows under skin (up to 10 mm)
Eggs hatch in 2–3 weeks
Mites survive 1–2 months
Clinical Presentation:
Intense itching (especially at night)
Linear burrows, erythematous papules
Common sites: wrists, genitals, interdigital spaces
Complications: secondary bacterial infection (cellulitis, impetigo)
Diagnosis: Clinical exam; skin scrapings/microscopy
Treatment:
Topical permethrin cream (head to toes, removed after 8–14 hrs)
Treat close contacts
Nursing Interventions:
Educate on hygiene, handwashing, laundering linens in hot water, isolation if in institutions
Comfort Measures: Cool baths/compresses, trim fingernails, avoid scratching
Lice (integumentary) (pediculosis)
Types:
Head lice (Pediculus humanus capitis)
Body lice (Pediculus humanus corporis)
Pubic lice (Pthirus pubis)
Transmission: Close contact, sharing personal items (combs, hats, bedding)
Clinical Presentation:
Pruritis from hypersensitivity reaction
Small red bumps, sores from scratching
Visible nits (eggs) attached to hair shafts
Diagnosis: Clinical exam, visual identification of lice/nits
Treatment:
Permethrin shampoo or lotion (repeat in 1 week)
Environmental cleaning (launder clothing/bedding, vacuum furniture)
Caution with lindane or ivermectin in children (neurotoxicity)
Nursing Interventions:
Hygiene education, combing to remove nits, check household contacts
Prevention: Avoid sharing personal items; regular inspection of close contacts
Pinworms (enterobiasis) (GI system(
Cause: Enterobius vermicularis
Transmission: Ingestion of eggs via contaminated hands, bedding, clothing, or surfaces
Pathophysiology:
Eggs hatch in GI tract → adult worms migrate to anal area to lay eggs
Lifecycle: 4–8 weeks
Clinical Presentation:
Nocturnal perianal itching
Irritability, disturbed sleep, occasional abdominal discomfort
Diagnosis: Tape test or pinworm paddle early morning
Treatment:
Mebendazole (repeat dose after 2 weeks)
Treat all household members
Nursing Interventions:
Hand hygiene, trimming nails, morning bathing, laundering bedding/clothing in hot water
Comfort Measures: Avoid scratching, manage irritation
Prevention: Treat all contacts, maintain hygiene, clean living spaces
Giardiasis (GI system)
Cause: Giardia duodenalis protozoan
Transmission: Fecal-oral route via contaminated water or food
Pathophysiology:
Parasite alters intestinal epithelial cells → diarrhea, malabsorption
Clinical Presentation:
Diarrhea (watery), abdominal cramps, bloating, gas, fatigue, weight loss
Pediatric: mild diarrhea, abdominal discomfort
Diagnosis: Stool samples (3 separate samples; microscopy, DFA, EIA)
Treatment:
Metronidazole (may take with food to reduce stomach upset)
Hydration and nutritional support as needed
Nursing Interventions:
Handwashing, proper sanitation, avoid untreated water, safe food handling
Comfort Measures: Maintain hydration, limit dairy if sensitive
Prevention: Ensure clean water, educate community on hygiene and safe drinking practices
communicable diseases in pediatrics
Children are highly susceptible to communicable diseases due to immature immune systems and close-contact environments (schools, daycares).
Rapid transmission in communities can lead to severe illness, hospitalization, or long-term complications.
Nursing role: prevention, early recognition, education, and implementation of infection control.
Assessment of communicable diseases
History: onset and progression of illness, fever, cough, rash, malaise, myalgia, anorexia.
Exposure history: contacts with similar symptoms, recent travel.
Immunization status: determines risk for vaccine-preventable diseases.
Physical exam: hydration, respiratory status, skin lesions, lymph nodes, neurological assessment.
Rash evaluation: characteristic appearances aid diagnosis (e.g., “slapped-cheek” for fifth disease, maculopapular for measles).
Diagnostic testing: throat swabs, blood samples, cultures as indicated.
Clinical example: measles
Clinical presentation: fever, maculopapular rash, red spots with white centers in the mouth, not itchy.
Important questions:
Recent travel
School/daycare attendance
Immunization status
Nutrition and hydration
Key concerns: unvaccinated status → high risk for measles.
Expected interventions: IV fluids, supportive care, infection control (airborne precautions), parent education.
Education points:
Measles is viral.
Primarily affects unvaccinated children.
Incubation ~10–14 days.
Public health involvement for contact tracing.
Vitamin A supplements and hydration support recovery.
Avoid NSAIDs for fever; warm mist humidifier can ease respiratory symptoms.
parasitic infestations and infections
children are vulnerable to parasitic infections due to hygiene practices and exposure in schools.
Common examples: scabies, pinworms, lice (pediculosis), giardiasis.
Assessment:
Rash, burrows, itching (worse at night for scabies).
Vital signs, hydration, pain assessment.
Exposure history, travel, recent outbreaks at school.
Reporting findings: temperature, skin changes, pain, heart rate.
Diagnosis examples:
Scabies → linear burrows, itching
Pinworms → nocturnal perianal itching
Pediculosis → lice and nits visible in hair
Giardiasis → gastrointestinal symptoms after contaminated water exposure
Management
Priority interventions: infection control (contact precautions), trimming fingernails, treating infected individuals, and notifying close contacts.
Medications:
Topical permethrin cream → scabies, lice
Anthelmintic medication → pinworms
Antifungals and antivirals → as indicated
Parent education:
Wash bedding/clothing in hot water; dry on high heat.
Treat all family members if necessary.
Vacuum furniture.
Avoid unnecessary bathing that can interfere with topical treatments.
Monitor for adverse reactions (e.g., difficulty breathing).
vaccines in pediatrics
Assessment before administration:
Allergy history and prior vaccine reactions.
Immunization records to identify missed doses.
Underlying health conditions (e.g., immunocompromised status).
Child’s fear or anxiety.
Analysis & Prioritization:
Consider risks vs. benefits (e.g., immunocompromised child may require postponement).
Identify contraindications and precautions.
Planning & Solutions:
Follow ACIP immunization schedule.
Use age-appropriate comfort measures:
Infants: sucrose solution, breastfeeding
Older children: topical anesthetics, distraction techniques (bubbles, parental involvement)
Ensure correct vaccine storage, preparation, and aseptic technique.
Implementation:
Verify VIS provided to parents.
Administer using proper technique.
Document: date, time, route, dose, site, lot number, manufacturer, VIS acknowledgment.
Evaluation:
Monitor for reactions: allergic (respiratory distress, hives) or syncopal episodes.
Observe high-risk patients for 15 minutes.
Report significant adverse events to VAERS.
Provide post-vaccine education for parents on supportive care and signs of delayed reactions.