1/50
Looks like no tags are added yet.
Name | Mastery | Learn | Test | Matching | Spaced | Call with Kai |
|---|
No analytics yet
Send a link to your students to track their progress
postpartum hemorrhage
a cumulative blood loss of at least 1000ml, or bleeding of any amount accompanied by manifestations of hypovolemia within 24 hr of birth
uterine atony
loss of uterine tone due to ineffective contractions
retained placenta
a placenta that is not expelled within 30 mins of the birth
subinvolution
failure of the uterus to return to its prepregnant state
uterine inversion
collapse of the uterine fundus into the uterine cavity and cervix
hematomas
similar to a bruise but its affects larger vessels leading to swelling and discoloration
prolonged labor
labor lasting longer than 20 hr since the onset of contractions
multiparity
indicated a client who has had two or more viable pregnancies lasting at least 20 weeks
operative vaginal birth
birthing requiring the use of instruments to aid in the birth of the fetus
polyhydramnios
excessive amount of amniotic fluid surrounding the fetus in pregnancy
macrosomia
a fetus or newborn who is larger than average 9-10lbs
Uterine
Atony is the primary cause of PPH
Causes of PPH
uterine atony, retained placenta fragments and lacerations
Antepartum PPH
history of high BP, high BMI (greater than 40) and placenta accreta
Intrapartum PPH
labor inductions, prolonged second stage of labor
Postpartum PPH
uterine atony, retained placement fragments, lacerations & full bladder
Clinical Manifestations of PPH
Early and Late PPH presentations
Vital Signs monitoring blood loss and shock
Nursing Interventions of PPH
Third Stage of Labor: uterotonic medication, cord traction and delayed cord clamping
Quantification of Blood Loss- faciltiy protocols
Immediate Postpartum Period- fundal assessment, uterine massage and oxytocin
PPH Risk Factors
An oxytocin-induced labor that lasts longer than 24 hr, prolonged labor, prolonged second stage of labor,
Four or more previous births;
Hematocrit less than 30%;
Platelet count less than 70,000;
Multiple gestation
Placenta accerate
placenta chorionic villi invade the decidua basalis attaching to the uterine myometrium
von Willebrand disease
hereditary disorder with a noted deficiency of von Willebrand factor which is required to maintain expected hemostatsis
Tone (Uterine Atony):
The uterus fails to contract, making it "boggy" and causing rapid bleeding.
Trauma:
Lacerations, uterine rupture, or inversion during delivery, sometimes requiring surgical repair.
Tissue:
Retained placenta or clots preventing the uterus from contracting fully
Thrombin:
Coagulation disorders (e.g., von Willebrand disease, DIC) preventing proper clotting
Tone Causes
placenta previa, overdistention of the uterus, uterine relaxants, previous PPH
trauma causes
c-section, episiotomy, macrosomia
tissue causes
retained placenta, placenta accreta, retained products of conception
thrombin
pre-e, placental abruption, pyrexia in labor, bleed disorders
early/primary PPH
presentation of heavy vaginal bleeding immediatley after birth up to 24 hr postpartum
late/secondary PPH
presentation of heavy vaginal bleeding 24 hr post birth and up to 6 weeks post partum
PPH: Nursing Interventions
Assessment of fundal placement and uterine tone every 15 min for at least 2 hr
Uterine massage to improve uterine tone
Administration of intravenous oxytocin during the postpartum period per hospital policy and physician prescription
Administration of 10 units of oxytocin intramuscularly (IM) if intravenous access is not available
Quantification of blood loss per facility protocol
Continued assessments using the 4 Ts of PPH
Documentation according to facility protocol
Placement of an indwelling urinary catheter to accurately monitor intake and output
Nonpharmacological interventions for PPH
Bimanual Uterine Massage
Pharmacologic Intervention
Uterotonics
Blood Products
Surgical Interventions
Tamponade intrauterine
balloon to reduce bleeding
PPH Management tx anemia
placenta accreta
the placenta attaches too deeply into the edomentrium and partially invades the myometrium
placenta increta
the placenta readily invade the myometrium
placenta percreta
the placenta penetrates through the endometrium and may attach to nearby organs
Uterine Inversion
Rare but serious postpartum complication
Secondary to PPH and can lead to shock
High maternal mortality rate if not managed promptly
Subinvolution of the Uterus Etiology:
Secondary PPH, typically occurs in the 2nd week postpartum week
Subinvolution of the Uterus
Clinical Manifestations:
Enlarged, boggy uterus with sudden onset of vaginal bleeding postpartum
Subinvolution of the Uterus
Nursing Interventions: Client stabilization, Similar to PPH care, large bore IV catheters, lab testing, VS monitoring, administering uterotonics and potential blood products and client education
Uterine inversion
is an emergency where the uterus turns inside out, often immediately after birth, leading to shock
Subinvolution
is the failure of the uterus to return to its normal, smaller size in the weeks following delivery. Inversion is a structural emergency, while subinvolution is a postpartum recovery issue
Postpartum Shock
Etiology
primary cause is PPH
Postpartum Shock Clinical Manifestations:
BP 85/45 or less, HR 110 or greater, SPO2 less than 95%, excessive thirst, restlessness, fear, decreased urinary output, pallor, ringing in the ears (tinitis)
Nursing Interventions for postpartum shock
use of anti-shock garment to maintain BP
Medical Management for postpartum shock
PRBCs blood products
Infusion of fibrinogen concentrate and platelets
Postpartum Infections Etiology
Endometritis
Mastitis
Wound Infections
Sepsis
Clinical Manifestations of Postpartum Infections
Endometritis: Fever, uterine tenderness, foul-smelling lochia, chills, headache, malaise
Mastitis: Flu-like symptoms, fever, redness, swelling, pain in the affected breast
Wound Infection: Warmth, pain, swelling at incision site, purulent drainage
Sepsis: Hypotension, hypoxia, hypothermia, impaired consciousness, oliguria, tachycardia, tachypnea
Nursing Interventions for postpartum infections
Endometritis, Mastitis, Wound Infection: Frequent assessments, antibiotic therapy, pain management, client education on hygiene and medication adherence
Sepsis: Frequent vital sign checks, broad-spectrum antibiotics, crystalloid solution administration, blood culture collection
Risk Factors for PP infections
age younger than 17 older than 40
bacterial vaginosis
black ethnicity
diabetes
bladder cath
frequent cervical exams
hypertension
internal FHR
manual removal of placenta
PPH
post/preterm birth
smoking