1/184
Looks like no tags are added yet.
Name | Mastery | Learn | Test | Matching | Spaced | Call with Kai |
|---|
No analytics yet
Send a link to your students to track their progress
PTSD: Defining Features
-Trauma and Response
-Reexperiencing
-Avoidance
-Emotional numbing problems
(Many trauma survivors do not develop PTSD
PTSD Diagnosis
Reaction persists > one month
Events that typically elicit PTSD
Combat and sexual assault most common
Events in life that tend to trigger vulnerabilities in life
-Invalidation
-Trivialization
-Loss of control
Acute Stress Disorder: Onset
Immediate
-0 to 28 days after the trauma
Acute Stress Disorder: Duration
Three days and four weeks
Acute Stress Disorder: Symptoms
Depersonalization
Depersonalization
When we feel disconnected from our body/person
Derealization
Feeling detached from reality- time person space (sensorium; orientation times 3)
Acute Stress Disorder: Treatment
Short term psychotherapy and antidepressant medication
Post Traumatic Stress Disorder: Onset
At least one months after the trauma occurs
Post Traumatic Stress Disorder: Duration
Lasts at least one month and can persist for several years
Post Traumatic Stress Disorder: Symptoms
Avoidance, heightened awareness and changes in mood or cognition
Post Traumatic Stress Disorder: Treatment
Long term psychotherapy, medication, and EMDR therapy
Acute PTSD
Diagnosis 1-3 months post trauma
Chronic PTSD
Diagnosis > 3 months post trauma
Delayed onset PTSD
Onset 6+ months post trauma
-May not see symptoms until 6+ months
PTSD: Causes
-Intensity of trauma
-Learned alarms (direct conditioning; observational learning)
-Biological vulnerability
-Uncontrollability and unpredictability
-Social support post trauma reduces risk
PTSD vs. c-PTSD: Both
-Reexperiencing
-Avoidance
-Sense of threat
c-PTSD
-Reexperiencing
-Avoidance
-Sense of threat
-Affect dysregulation
-Negative self concept
-Interpersonal disturbances
Silent PTSD/c-PTSD
-High sensitivity
-Hidden mistrust
-Dissociation/maladaptive daydreaming
-Over thinking
-Chronic worry
-Mood swings
-Hyper vigilance and fawn response
Fawn Response
long term nervous system response; response to stress and make sure that other people are okay
Trauma: Attachment Disorder
-Disturbed and developmentally inappropriate behaviors in children
-Unable or unwilling to form normal attachment relationships with caregiving adults
-Result of inadequate or neglectful care in early childhood
(RAD/DSED)
RAD: Reactive Attachment Disorder
-Abnormally withdrawn and inhibited behavior
-Less receptive to support from caregivers
DSED: Disinhibited Social Engagement Disorder
-Overly friendly with unknown adults
-Children might wander off often, approach strangers with no hesitation; hug or touch
-Treat adults as parents just for attention
Non-Organic Failure to Thrive
-Infancy
-Malnourished
-Even if the infant is fed, etc. they still die
Psychosocial Dwarfism
Toddler—>preschool
-Even if toddler/preschooler is fed, etc. they still die
Trauma can involve
-Loss of self
-Impediment of intimate relationships
-Shame
-Emotional Distance from self and others
(Can retire our nervous system; become reactive)
Trauma and Irrational Brain: Three Brains
3-Neocortex Brain
2-Limbic Brain
1-Reptilian Brain
Neocortex Brain
-Metaphorical
-Rational/thinking
Limbic Brain
-Social/emotional
-Feeling
-Amygdala
Reptilian Brain
-Basic life support
-Fight/Flight/Freeze
Amygdala vs. PreFrontal Lobe
Low road vs. High road
-Emotional brain takes control, may lead to impulsive and irrational actions
Vs.
-When taking in stimuli (misinterpretation, impulse control, irrational actions outside of person's control), helps with interpretation and impulse control
Trauma changes the Nervous System
-Highly reactive
-Emotional numbness
Highly reactive
Energy dedicated to suppressing inner chaos
Emotional numbness
-When trauma is ongoing and/or inescapable; next level is DVC
-Autonomic Nervous System regulates the vagus nerves; connects the brain to major systems in the body supporting
DVC: Dorsal Vagal Complex
Dissociation
Polyvagal Chart
Down—>Up
-Ventral Vagal
-Fight Flight
-Freeze
Ventral Vagal
Safe
-Parasympathetic Nervous System
(Calm, grounded, settled)
Fight Flight
-Sympathetic Nervous System
(Denver, hyperarousal, rage, panic, anger, fear)
Freeze
-Dorsal Vagal
(Hyperarousal, collapse, immobility, dissociation, numbness)
Child Abuse and Domestic Violence
-Affiliation Motivation
-Learned helplessness (freeze; dorsal Vagal)
-Reduced executive function (reptilian Brain and Limbic system, stuck in FFF)
-fMRI (Broca's area limited during acute and/or flashbacks)
Stuck in Freeze/Fight/Flight
-Oversensitivity; faulty alarm system
-Mirror neurons + faulty alarms system = popsicle misinterpretation
(Detachment, anxiety, rage, impulse control)
-"Out of Sync"
-Ignore gut feelings
-Alexithymia
Oversensitivity: Faulty alarm system
-Stuck in survival mode
-Energy for social bonds, attention, learning, strategic thinking
Alexithymia
Inability to feel and describe emotions
Flashbacks and living in the past
Flashbacks don't have a 'story' (beginning, middle, end)
-Brief images, no conclusion/closure
-Broca's area reduced
-Visual cortex highly activated
-Left hemisphere Uc activity reduced
-Right hemisphere highly activated
Broca's area
Responsible for speech articulation
-When experiencing flashbacks, it is vivid
-Body responds as if they are back in that scnario
Left hemisphere vs. right hemisphere
Speech (Activity Reduced) vs. Image processing (Highly Activated)
-Brain believed and responds as if still in event
Insight Therapies
Don't work
-Brain organization (reptilian, Limbic, cortex)
-Few issues result of defects in understanding and identification: no amount of insight can overrule older parts of brain
(They know something is going on with them)
-Understanding≠control
Attention Deficit Hyperactivity Disorder (ADHD)
Associated with numerous impairment
Behavioral
Cognitive
Social and Academic problems
ADHD: Cognitive
Less goal directed behavior
Harder to get on/start task
Overwhelmed with all -arts, they freeze
Inability to filter irrelevant stimuli (attend to everything)
Poor working memory
Difficulty with delayed gratification
Appear 2-3 years younger than their peers
DSM-5 Symptom Types
Predominantly inattentive
Hyperactive/impulsivity
Inattentive + Hyperactive/Impulsivity Combined
Diminished regulations of emotions
Substance use
Delinquency
Interrupting others
ADHD: Facts and statistics
Approx. 11% of school aged children
S/S ~3-4 years
Similar s/s childhood, adulthood
ADHD: Gender Differences (Boys:Girls)
3:1
ADHD: Cultural Factors
Most commonly diagnosed in US; prevalence fairly worldwide
ADHD Causes: Genetic Contributions
Heritability
DAT-DA transport gene implication
Dopamine (how it’s being used)
ADHD Causes: Neurobiological correlations
Smaller brain volume
Inactivity of Frontal Cortex and Basal Ganglia
Abnormal frontal lobe development and functioning
Maybe the baby didn’t grow enough; correlation
Reduced function in the Prefrontal Cortex
More impulsive and focus and rational thinking is harder
Reduced function of Basal Ganglia
Hyperactivity
ADHD Causes: Biological Contributions
Role of toxins
Food additives (altered gut microbiota)
Maternal smoking
Imbalance of Omega 3 and 6
Need good prebiotic and probiotic diet
Imbalance of Omega 3
(too) Calming
Imbalance of Omega 6
Chronic inflammation
ADHD Causes: Psychosocial Contributions
Kids tend to be popular during early elementary but viewed negatively later school years
Low self esteem
Peer rejection results in social isolation
RSD - Rejection Sensitivity Dysphoria
ADHD Causes: Trauma
Trauma more common than previously thought
Doesn’t cause but there is an association
Lots of misdiagnosis of Trauma and ADHD
Biological Treatment of ADHD: Goal
Reduce impulsivity and hyperactivity
Improve attention
Biological Treatment of ADHD: Stimulant Medications
Approx. 4 million American children
Low doses improve focusing abilities
Ritalin, Dexedrine, Adderall
Problem: May increase risk for later substance use
Biological Treatment of ADHD: Genes affect response to meds
Trial and Error
Autism Spectrum Disorder
Problems in language, socialization, cognition (to some degree)
Pervasive
Autism: Defining feature
Self centered thinking (focused inward)
Cognitive energy directed inward; it can be challenging to notice social cues
Autism: Main areas of impairment (Communication)
Communication and social interaction
About 25% don’t acquire effective speech
Mute or extremely limited
Autism: Main areas of impairment (Behavior)
Restricted, repetitive patterns of behavior, interests, or activities
Need to maintain “sameness”
High level of consistency in activities or experiences (routine)
Autism Starts
Infancy and toddlerhood
Autism in infancy and toddlerhood
Don’t interact with caregivers
Maintain eye contact
Collections of things
Designs
No physical touch (cuddling)
Solitary play
Savant
Meet criteria of autism but is a genius in a specific area
Not very common
New label in DSM-5TR
Encompasses previously classified “pervasive developmental disorders”
Autistic disorder
Asperger’s disorder
Childhood disintegrative disorder
Rhett syndrome
Only about 30% on the spectrum have severely noticeable s/s
Spectrum
Pragmatic Language
Social akwardness
Monotropic Mindset
Information Processing
Sensory Processing
Repetitive Behavior
Neuro-motor Differences
Autism Spectrum Disorder: Most common
Deficits in social interaction and communication
Restrictive Repetitive Patterns of Interests
Restrictive in Social Interaction and Communication
Learn everything about it, until get into another thing
Drawing to sowing to yellowstone
Sign and Symptom: Masking
Mimicking
Identity crisis
When they mask in a conversation, they don’t usually know what the other person is saying in conversations
Particularly women
Exhausting
Sign and Symptom: High sensitivity to environment
Lights, noises, scents
Sign and Symptom: Eye contact
May look different than expected
Sign and Symptom: Splitting
Looking at things dichotomously (either good or bad)
Sign and Symptom: Emotional dysregulation
Rage, sadness, etc.
BDP
Sign and Symptom: Alexithymia
Inability to identify and describe feelings
Comorbidity with
ADHD/Anxiety/Depression
Distinct but can be comorbid with ADHD
Psychological and Social Dimensions: Historical views
Failed parenting
High SES
High IQ
Lack of self awareness
Behavioral correlates (echolalia, self injury)
Best predictors of Autism
IQ
Amount of language development by age 6
A larger head and brain size by age 6
Biological Dimensions
Significant genetic component
Polygenic
Oxytocin lessened
Older parents (fathers)
Polygenic
Numerous genes on several chromosomes
Oxytocin
Bonding hormone
Lessened: not wanting to bond with others
Neurobiological Influences
Amygdala
Oxytocin
Neurobiological Influences: Amygdala
Larger size at birth - higher anxiety, fear
Elevated cortisol
Neuronal damage: results from high stress, which may affect processing of social situations
Causes of Autism Spectrum Disorder: Biological
Neurobiological influences
Vaccines do not increase risk of autism
See autism ages 2-3
Treatments of Autism Spectrum Disorder: Psychosocial Treatment
Behavioral approaches
Skill building
Reduce problem behaviors
Communication and language training
Increase socialization
Naturalistic teaching strategies
Early intervention critical
Treatments of Autism Spectrum Disorder: Biological Treatments
Medical interventions has little positive impact on core dysfunction
Except highly agitated, repetitive behavior
Some drugs decrease agitation
Tranquilizers, SSRIs
Indicators of good prognosis: High IQ, good language ability,
Learning Disorders
Reading Impairment (Dyslexia)
Impairment in written expression (Dysgraphia)
Impairment in numbers of mathematical reasoning (Dyscalculia)
Schizophrenia
Ability to function is impaired by severely distorted beliefs, emotions, perception, and thought processes
1 in 10 people develop this (24 million worldwide)
Pervasive type of diagnosis
Schizophrenia: Myths
Split personality (don’t have this)
Dissociative state
Violence (more likely victims)
Psychosis
Descriptor (not a diagnosis)
Hallucinations and Delusions