examination of heart

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80 Terms

1
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through palpation what can we determine

Temperature

Pain

Tremors

Heartbeats

2
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how to carry out palpation

We press on the intercostal spaces

We place the hand flat

3
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heart beat best felt in horse

5 intercostal space on the left side

4 intercostal space on the right side

4
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heart beat best felt in cattle/small ru

4 intercostal space on the left side

5
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heart beat best felt in dogs,cat,foxes

4-5 intercostal space on the left side

6
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is the shifting of heart beats pathological or physiological

pathological

7
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shifting heartbeat- up

enlarged hear

8
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shifting heartbeat- back

enlarged left ventricle, whole heart, mediastinal lymph nodes, mediastinal tumors

9
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Cardiac enlargement is

​​enlargement of the area where the heartbeat is felt and increased force of the beat

10
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causes of increased heartbeat

• Fever

• Stress

• Second half of pregnancy

• Hypertension

• Cushing's disease

11
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cause of weakened heart beat

• Significant weakening of the heart

• Fluid in the pericardial sac (hydropericardium)

• Fluid in the pleural cavity (hydropneumothorax)

• Advanced pulmonary emphysema

12
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Cardiac dullness area:- Enlarged

Cardiac dilatation and hypertrophy

Hydroendocardia

Leftward displacement of the heart

13
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Pseudoenlargement of cardiac dullness

pseudoenlargment = false enlargement, is an increase in the size of an organ due to infiltration of a tissue not normally found in that organ

Hydrothorax

Hepatomegaly (carnivorous)

Lung tumors

Fibrinous pleuris

14
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Cardiac dullness area: Reduced

Pulmonary emphysema

Pneumothorax

15
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systolic tone

systolic

16
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diastolic tone

diasoltic

17
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Systolic period

time between I and II tone

18
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Diastolic period

time between II and I tone

19
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tone I- muscular

  • Lower sound than tone II

  • Quieter

  • Less resonant

20
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how is tone I created

created as a result of the tremors of closing valves and the muscular-fibrous apparatus of the valves

21
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tone II- valvular

  • Higher and shorter than I

  • Louder

22
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cause of tone II

caused by the tremors of the closing valves of the aorta and pulmonary artery

23
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tone II in horse (pm) left side

  • I – 3 i.c.s just behind the sternum – projection of the pulmonary valve

  • II – 4 i.c.s 2 fingers' width below the shoulder joint line - projection of the aortic valve

  • III – 5 i.c.s. A hand's width below the shoulder joint line - projection of the mitral valve

24
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valves of heart present on left side

pulmonary

aortic

mitral

25
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projection of pulmonary valve in horse/ cattle

LEFT SIDE

I – 3 i.c.s just behind the sternum

26
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projection of aortic valve in horse/ cattle

LEFT SIDE

II – 4 i.c.s 2 fingers' width below the shoulder joint line

27
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projection of mitral valve in horse/ cattle

LEFT SIDE

III – 5 i.c.s. A hand's width below the shoulder joint line

28
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what valve is present on right side

trispid

29
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projection of the tricuspid valve in horse/ cattle

RIGHT SIDE

3 i.c.s. Just above the sternum

30
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projection of the pulmonary valve dog and cats

LEFT SIDE

I – 3rd p.m. of the heart just behind the sternum

31
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projection of the aortic valve dog and cats

LEFT SIDE

II – 4th p.m. of the heart just below the shoulder joint line

32
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projection of the mitralvalve dog and cats

LEFT SIDE

III – 5th p.m. of the heart in the upper half of the 1/3 of the ches

33
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projection of the tricuspid valve dog and cats

IV - 4th p.m. of the heart Just above the sternum

34
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Auscultatio

Rate – number of beats per minute

Intensity – strength of heart sounds

Rhythm – regularity Intervals between beats

Additional sounds

35
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physiological cause of tachycardia

young age, high ambient temperature, excitement

36
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pathological cause of tachycardia

circulatory failure, anemia, adrenal hyperfunction, hyperthyroidism

37
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Physiological cause of bradycardia

sleep, anesthesia, trained animals

38
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pathological cause of bradycardia

arrhythmias, hypothermia, uremia

39
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Arrhythmia

different time interval between individual heartbeats

  • the interval between the first and second tone is shorter

  • Splitting and splitting of the tone – physiologically at the peak of inspiration, pathologically with some arrhythmias and increased pressure

40
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rythm resembles what

galloping horse

41
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how many tones dose rhythm consist of

It consists of 3 tones

An additional tone in the diastolic phas

42
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persistant arrythmia is longer than

2 days

43
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Parcotic arrythmia

sudden onset and end, periodic

44
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dull heart tone caused by

fur, pleural fluid

45
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heart murmur

knowt flashcard image
46
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Additional examination of heart

• ECG

• X-ray

• Cardiac ultrasonography

• Phonologic cardiology

• Magnetic resonance imaging

• scintigraphy

47
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Clinical signs of heart disease that you may see at home

• weakness

• lethargy

• exercise intolerance

• shortness of breath

• Coughing

48
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More clinical signs of heart diease from clinical examination

• an irregular heartbeat

• a heart murmur (which indicates turbulent blood flow over a valve within the heart)

• irregular pulses

• abnormal lung sound

49
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in ECG what axis volatge on

Y

50
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in ECG what axis time on

Time on X axis

51
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P wave

atrial muscle depolarization

52
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QRS complex

ventricular muscle depola

53
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T wave

ventricular muscle repolarization

54
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artefacts during ECG

• Motion

• Breathing

• purring

55
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cause of artefacts

• Movement

• Poor contact between the electrodes and the patient

• Electrical items (e.g. clippers, imaging equipment, warming equipment, other monitoring equipment)

56
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position of animals during ecg

lying in right lateral recumbency

gently restrain limbs

electrodes/pads should be placed on the limbs appropriately

  • right forelimb

  • left forelimb

  • left hind limb

<p>lying in right lateral recumbency</p><p>gently restrain limbs</p><p>electrodes/pads should be placed on the limbs appropriately</p><ul><li><p>right forelimb</p></li><li><p>left forelimb</p></li><li><p>left hind limb</p></li></ul><p></p>
57
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ecg on respiratory patients

should have their ECG performed in sternal

<p>should have their ECG performed in sternal</p>
58
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AV block

a disorder in the heart's rhythm due to a fault in the natural pacemaker

<p>a disorder in the heart's rhythm due to a fault in the natural pacemaker</p>
59
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types of heart blocks

Sinoatrial nodal blokcs (SA)

Atrioventricular blocks (AV)

60
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SA block- I degree

a lag between the time that the SA node fires and actual depolarization of the atria.

Not recognized by ECG

61
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SA- II degree type 1

aka Wenckebach block

  • rhythm is irregular

  • R-R interval gets progressively smaller, while the P-R interval remains constant, until a QRS segment is dropped.

62
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SA- II degree type 2

aka sinus exit block

  • regular rhythm that may be normal or slow.

  • It is followed by a pause that is a multiple of the P-P interval usually (2-4).

63
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R-R interval gets progressively smaller, while the P-R interval remains constant, until a QRS segment is dropped

is charc for what heart block

SA- II degree type 1

64
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SA block- III degree

caused by failure to conduct them.

  • rhythm is irregular and either normal or slow

  • followed by a long pause that is not a multiple of the P-R interval.

  • The pause ends with a P wave, instead of a junctional escape beat the way a sinus arrest would.

65
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what SA block is not recog by ECG

SA I degree

66
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difference btwn SA block- III degree and sinus arrest

  • The pause ends with a P wave, instead of a junctional escape beat the way a sinus arrest would.

67
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AV block- I degree

occurs when there is a delay, but not disruption, as the electrical signal moves between the atrium and the ventricles through the AV node.

68
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AV block- II degree

occurs when the electrical signal between the atria and ventricles is even more impaired than in a first-degree AV block. In a second-degree AV block, the impairment results in a failure to conduct an impulse, which causes a skipped beat.

69
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AV block- III degree

occurs when the signal between the atria and ventricles is completely blocked

there is no communication between the two.

None of the signals from the upper chambers make it to the lower chambers.

On ECG, there is no relationship between P waves and QRS complexes, meaning the P waves and QRS complexes are not in a 1:1 ratio.

70
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what is the most sevre of the AV blocks

Third-degree AV block is the most severe of the AV blocks

71
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in what heart block is no relationship between P waves and QRS complexes

AV block- III degree

72
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what ratio should P waves and QRS complex be in

1:1

73
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AV block- Mobitz I

  • characterized by a progressive yet reversible block of the AV node.

  • defined by progressive prolongation of the PR interval, with a resulting dropped beat (the PR interval gets longer and longer until a beat is finally dropped, or skipped)

74
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AV block- Mobitz II

caused by a sudden, unexpected failure of the His-Purkinje cells to conduct the electrical impulse

the PR interval is unchanged from beat to beat, but there is a sudden failure to conduct the signal to the ventricles, and resulting in random skipped beat

75
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progressive prolongation of the PR interval is characteristic of

AV block- mobitz I

76
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position of large animal during X ray

Lateral projection, animal standing

77
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position of small animal during X ray

Lateral and dorsoventral projection, animal lying down – small animals

78
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what dose x ray allow acess to

assess the condition of the heart and blood vessels inside the chest

79
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what can be determined in x ray

• Enlargement of the heart

• Tightening/weakening of the blood vessel pattern

• Displacement of the heart

80
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Scyntygraphy

  • Heart activity registration after administration of radioactively labeled contrast

  • The distribution and saturation of the organ by contrast are assessed