1/23
Looks like no tags are added yet.
Name | Mastery | Learn | Test | Matching | Spaced |
|---|
No study sessions yet.
Endotracheal Tube & Endotracheal Intubation
Indications
A tube is inserted through pt's nose/mouth into the trachea
Allows for emergency airway management
Oral intubation - easiest & quickest form of intubation
Usually in ER
Nasal Intubation - has facial or oral trauma
Is not used if pt has clotting problem
Endotracheal Tube & Endotracheal Intubation
Considerations
PLACEMENT
Intubation
usually performed by nurse anesthetist, anesthesiologist, critical care or emergency physician, pulmonologist
Chest x-ray verifies placement of ET tube
Can be cuffed or uncuffed
Cuff of tracheal end - inflated to ensure proper placement & formation of a seal bet cuff & tracheal wall (prevents air leaking)
Seal ensures adequate amount of tidal volume is delivered by mech vent when attached to external end of ET tube
PT is unable to talk when cuff is inflated
NURSING ACTIONS:
Have resuscitation equip to include manual resuscitation bag w/ face mask at bedside at all times
Ensure intubation attempts last no longer than 30 secs & reoxygenate before another attempt
Monitor VS
Verify tube placement
by checking end-tidal carbon dioxide levels & chest x-ray
Auscultate for breath sounds
Observe symmetric chest movement
Stabilize ET tube w/ tube-holding device or secure tape
Monitor hypoxemia, dysrhythmias, aspiration
Mechanical Ventilation
provides breathing support until lung function is restored, delivering 100% oxygen that is warmed (body temp 37*C (98.6*F) & humidified at FiO2 levels between 21-100%
can be delivered via ET tube or tracheaostomy tube
can be cycled based on pressure, volume, time and/or flow
to maintain patent airway & adequate oxygen saturation of greater than 95%
Positive-pressure ventilators deliver air to lungs under pressure throughout inspiration to keep alveoli open & to prevent alveolar collapse during expiration.
Benefits:
forced/enhances lung expansion
improved gas exchange (oxygenation)
decreased work for breathing
Mechanical Ventilation
Indications
Hypoxemia, hypoventilation w/ respiratory acidosis
Airway trauma
Exacerbation of COPD
Acute pulmonary edema due to MI or HF
Asthma attack
Head injuries, cerebrovascular accident, coma
Neurological Disorders (MS, Myasthenia Gravis, Guillain-Barre)
Obstructive Sleep Apnea
Respiratory support following surgery (decreased overload)
Respiratory support while under general anesthesia or heavy sedation
Mechanical Ventilation
Considerations
PREPARATION
Explain procedure
Establish method for pt to communicate, provide writing materials, use dry/erase board
ONGOING CARE
Maintain patent airway
Assess position & placement of tube
Keep tube clear of pooled water & empty as needed
Document tube placement in cm at pt's teeth or lips
Reposition & resecure tube
Use caution when moving pt
Suction oral & tracheal secretions
Support vent tube
Have resuscitation bag w/ face mask
Assess resp status q1/2hr
breath sounds, reduced/absent breath sounds, resp effort, spont breaths
Verify provider prescription each shift. Monitor/doc vent settings qh
Rate, FiO2, Tidal Volume
Mode of Vent
Use adjuncts (PEEP, CPAP)
Plateau or peak inspiratory pressure (PIP)
Alarm settings
Monitor vent alarms (signal if incorrect vent)
Never turn of alarms
Types:
Volume (low pressure) Alarms→indicate a low exhaled volume due to a disconnection, cuff leak, tube displacement
Pressure (high pressure) Alarms→indicate excess secretions, pt biting the tube, kinks in tube, pt coughing, PE, bronchospasm, pneumothorax
Apnea Alarms→indicate the vent does not detect spontaneous respiration in preset time period
Maintain adequate volume in the cuff of ET tube
Asses cuff pressure at least q8h
Maintain pressure < 20 mmHg (or 20-30 cm H2O)
reduce risk of tracheal necrosis
Assess for an air leak (pt speaking, air hissing, <SaO2)
can result in inadequate oxygenation or accidental extubation
Administer meds as prescribed
Analgesics―morphine, fentanyl
Sedatives―propofol, diazepam, lorazepam, midazolam, haloperidol
can require sedation or paralytic agents to prevent competition between extrinsic & intrinsic breathing & resulting effects of hyperventilation
Neuromuscular Blocking Agents―pancuronium, atracurium, vecuronium
used in clinical setting due to long half-life
paralyze muscles but do not sedate or relieve pain
in conjunction w/ analgesic or sedative
Reposition the oral ET tube q24h
Asses for skin breakdown
Perform oral care at least q12h
Provide adequate nutrition
Assess GI functioning q8h
Monitor bowel habits
Admin enteral/parenteral feedings
Monitor during weaning process & watch for signs of weaning intolerance
Respirations > 30/min or < 8/min
BP or HR changes more than 20% baseline
SaO2 <90%
Dysrhythmias, elevated ST segment
Significant decrease in tidal volume
Labored respirations, >use of accessory muscles, diaphoresis
Restlessness, anxiety, <LOC
Have manual resuscitation bag w/ face mask & oxygen readily available
Have reintubation equipment
Suction oropharynx & trachea
Deflate cuff on ET tube & remove tube during peak inspiration
Following extubation:
Monitor signs of respiratory distress
Airway obstruction (ineffective cough, dyspnea, stridor)
Assess SpO2 q 5mins
Encourage deep breathing & coughing, incentive spirometer
Reposition pt to promote mobility of secretions
Older adults:
< respiratory muscle strength & chest wall compliance
more susceptible to aspiration, atelectasis, pulmonary infections
Mechanical Ventilation
Complications
Trauma
Fluid Retention
Oxygen Toxicity
Hemodynamic Compromise
Aspiration
GI Ulceration (GI Ulcer)
Infection
TRAUMA
Barotrauma
damage to the lungs by positive pressure
can occur due to pneumothorax, subq emphysema, pneumomediastinum
Volutrauma
damage to the lungs by volume delivered from one lung to the other
Barotrauma
damage to the lungs by positive pressure
can occur due to pneumothorax, subq emphysema, pneumomediastinum
Volutrauma
damage to the lungs by volume delivered from one lung to the other
FLUID RETENTION
(w/ mech vent) is due to decreased cardiac output, activation of renin-angiotensin-aldosterone system, ventilator humidification
NURSING ACTIONS:
Monitor I&O, weight, breath sounds, ET secretions
OXYGEN TOXICITY
can result from high concentrations of oxygen (typ >50%), long durations of oxygen therapy (typ >24-48hrs), degree of lung disease
NURSING ACTIONS:
Monitor for fatigue, restlessness, severe dyspnea, tachycardia, tachypnea, crackles, cyanosis
HEMODYNAMIC COMPROMISE
mech vent has a risk of > thoracic pressure (positive pressure) = < venous return
NURSING ACTIONS:
Monitor for tachycardia, hypotension, urine output <30 mL/hr, cool, clammy extremities, < peripheral pulses, < LOC
ASPIRATION
keep HOB >30* at all times to < risk -
NURSING ACTIONS:
Check residuals q4h if pt is receiving enteral feedings
GASTROINTESTINAL ULCERATION (STRESS ULCER)
can be evident in pts receiving mech vent
NURSING ACTIONS:
Monitor GI drainage & stools for occult blood
Admin ulcer prevention meds (sucralfate, histamine2 blockers)
INFECTION
can be related to vent intubation or suctioning
NURSING ACTIONS:
Monitor pt for fever, changes in sputum color, consistency, quantity, crackles, rhonchi
Monitor WBCs
Use aseptic technique
ASSIST-CONTROL (AC)
Preset rate and tidal volume.
Client initiates breath and ventilator takes over for the intubated client.
Hyperventilation can result in respiratory alkalosis.
Client can require sedation to decrease respiratory rate.
SYNCHRONIZED INTERMITTENT MANDATORY VENTILATION (SIMV)
Preset rate and tidal volume for machine breaths.
Client initiates breath and tidal volume will depend upon client's effort.
Ventilator initiated breaths are synchronized to reduce competition between ventilator and client.
Used as a regular mode of ventilation or a weaning mode (rate decreased to allow more spontaneous ventilation) for the intubated client.
Can increase work of breathing, causing respiratory muscle fatigue.
INVERSE RATIO VENTILATION (IRV)
Lengthens inspiratory phase to maximize oxygenation in the intubated client.
Used for hypoxemia refractory to PEEP.
Uncomfortable for clients and requires sedation and/or neuromuscular blocking agents.
High risk of volutrauma and decreased cardiac output due to air trapping.
AIRWAY PRESSURE RELEASE VENTILATION (APRV)
Allows alveolar gas to be expelled by the lungs own natural recoil
Time-triggered and pressure-limited
Breaths can be initiated spontaneously or by the ventilator
Causes less ventilator-induced lung injury and fewer adverse effects on the cardiovascular system
INDEPENDENT LUNG VENTILATION (ILV)
Double-lumen ET tube allows ventilation of each lung separately.
Used for clients who have unilateral lung disease.
Requires two ventilators, sedation and/or use of neuromuscular blocking agents.
HIGH-FREQUENCY VENTILATION
Delivers small amount of gas at rates of 60 to 3,000 cycles/min.
High frequency ventilation often used in children.
Client must be sedated and/or receiving neuromuscular blocking agents.
Breath sounds difficult to assess.
POSITIVE END EXPIRATORY PRESSURE (PEEP)
Preset pressure delivered during expiration.
Added to prescribed ventilatorsettings to treat persistent hypoxemia.
Improves oxygenation by enhancing gas exchange and preventing atelectasis.
Amount of - added is typically 5 to 15 cm H20.
PRESSURE SUPPORT VENTILATION (PSV)
Works to keep the alveoli from collapsing during expiration.
Allows for greater oxygenation and makes the work of breathing easier.
Allows for lower levels of FiOz to be used.
Can be used with IMV or AC modes to treat or prevent atelectasis.
Settings 5 to 20 cm H20 (greater than 20 cm H20 can cause lung damage).
CONTINUOUS POSITIVE AIRWAY PRESSURE (CPAP)
Positive pressure supplied during spontaneous breathing. No ventilator breaths delivered unless in conjunction with SIMV.
Risks include volutrauma, decreased cardiac output and ICP.