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postterm newborn
inability of placenta to provide adequate oxygen and nutrients to fetus after 42 weeks
nursing assessment: typical characteristics
dry, cracked, wrinkled skin; possible meconium stained
long, thin extremities; long nails; creases cover entire soles of feet
wide-eyed, alert expression
abundant hair on scalp
thin umbilical cord
limited vernix and lanugo
perinatal asphyxia - postterm
living in hypoxic environment prior to birth due to placental insufficiency, leaving little to no oxygen reserves available to withstand stress of labor
poor tolerance to stress of labor, frequently leading to acidosis
placental deprivation or oligohydraminios, leading to cord compression and subsequent reduction in perfusion to fetus
complications associated with postterm newborn
perinatal asphyxia (caused by placental aging or o
oligohydraminios)
hypoglycemia (caused by acute episodes of hypoxia related to cord compression which exhausts carbohydrate reserves)
hypothermia (caused by loss of subcutaneous fat)
polycythemia (caused by an increased production of rbcs to compensate for a reduced oxygen environment)
hypogylcemia - postterm
hypoxia secondary to depleted glycogen reserves
placental insufficiency secondary to placental aging contributing to chronic fetal nutritional deficiency further depleting glycogen stores
hypothermia - postterm
associated with depleted glycogen stores, poor subcutaneous fat stores, and disturbances in cns thermoregulation due to hypoxia
increased risk for acidosis and hypoglycemia secondary to metabolic stress
loss of subcutaneous fat secondary to placental insufficiency
use of stored nutrients for nutrition due to lost ability of placenta to nourish fetus
subsequent wasting of subcutaneous fat, muscle, or both
loss of natural insulation (subcutaneous fat) important in temperature regulation
nursing implications for hypothermia - postterm
maintain a neutral thermal environment to promote stabilization of newborn’s temperature
assess skin temperature and respiration characteristics
monitor abgs and blood glucose levels
eliminate sources of heat loss
dry newborn thoroughly
wrap in warmed blanket with stockinette cap on head
use radiant heat source
nursing implications for hypothermia
monitor blood glucose levels, initially on arrival to nursery and hourly thereafter
maintain fluid and electrolyte balance
watch for subtle changes
initiate early oral feedings if possible; if not, administer iv infusion with 10% dextrose in water
polycythemia - postterm
intrauterine hypoxia triggers increased rbc production to compensate for lower oxygen levels
nursing implications for polycythemia
ensure adequate hydration (orally or iv)
monitor hematocrit levels (goal is 60%)
administer partial exchange transfusion, albumin, or normal saline iv to reduce rbc volume and increase fluid volume (controversial)
meconium aspiration - postterm
commonly associated with chronic intrauterine hypoxia
struggling by fetus makes respiratory efforts and bearing down with abdominal muscles, leading to expulsion of meconium into amniotic fluid
normal sucking and swallowing by fetus elads to meconium filling airways
nursing implications for meconium aspiration - postterm
initiate resuscitation measures as necessary
suction airways and support ventilation
hypoglycemia overview
with the loss of placenta at birth, the newborn now must assume control of glucose homeostasis through intermittent oral feedings
initiate breastfeeding as soon as possible; otherwise, formula feed the infant on demand
if oral feedings are not accepted, a buccal dextrose gel or an iv infusion with 10% dextrose in water may be needed to maintain the glucose level above 40 mg/dL
polycythemia overview
hyperviscosity results from the increased number of rbcs
poor perfusion to organs occurs with hyperviscosity and is associated with long-term motor and cognitive neurodevelopmental disorders
observe for clinical signs of polycythemia
ruddy skin
abdominal distention, vomiting, and poor feeding
cyanosis and tachycardia (less common)
tachypnea, weak suck reflex, jaundice, lethargy, hypotonia, irritability, jittery
monitor hematocrit as ordered
newborns with symptoms and with a hematocrit of 70% or less may simply be supported with adequate fluid intake, close observation, and a repeat hematocrit level in 12 hours
if the newborn shows symptoms, a partial plasma exchange transfusion may be needed to decrease the viscosity of blood and relieve symptoms
signs and symptoms of hypoglycemia
lethargy
tachycardia
respiratory distress
poor feeding
diaphoresis
hypothermia
weak cry
hypotonia
preterm newborn
body system immaturity affecting transition to extrauterine life, increasing risk for complication
respiratory system
cardiovascular system
gi system
renal system
immune system
central nervous system
effect of prematurity on body systems
cns
hypothermia due to a relatively larger body surface area and heat production instability
unable to shiver
lack flexion and muscle tone
flaccid
immune system
deficiencies in igg
impaired ability to develop antibodies
thin and fragile skin
weak blood vessels
immature immune system
infection
glucose abnormalities
retinopathy of prematurity related to immature retinal vascularization
respiratory
respiratory issues due to surfactant deficiency
apnea as a consequence of immature respiratory control
cardiovascular
patent ductus arteriosis
hypotension
intraventricular hemorrhage related to the preterm infant’s fragile germinal matrix
anemia (preterm infants have lower levels of circulating hemoglobin at birth as compared with term infants)
gastrointestinal
blood is shunted to brain and heart which can lead to gastrointestinal ischemia and damage to intestines
small stomach capacity, limited absorption in the gut
feeding difficulties, lack the ability to suck and swallow
necrotizing enterocolitis
slow growth
renal
less able to concentrate urine
slow gfr
retain fluid
risk for electrolyte imbalance
increased risk for drug toxicitity
preterm characteristics
weight < 5.5 lb
scrawny appearance
poor muscle tone
minimal subq fat
undescended testes
plentiful lanugo
poorly formed ear pinna
fused eyelids
soft spongy skull bones
matted scalp hair
absent to few creases in soles and palms
minimal scrotal rugae; prominent labia and clitoris
thin transparent skin
abundant vernix
promoting oxygenation in the preterm infant
preterm infant lacks surfactant which lowers surface tension in the alveoli and stabilizes them to prevent their collapse
inability to initiate and establish respirations leads to hypoxemia, acidosis, and hypercapnia
failure to initiate extrauterine breathing or failure to breathe well after birth leads to hypoxia
the heart rate falls, cyanosis develops, temperature decreases, blood pressure decreases, and respirations are altered (apnea, tachycardia, retractions, grunting, and nasal flaring)
if the newborn does to not respond to tactile stimulation with effective respirations or vigorous crying, position and clear the airway, then immediately provide positive-pressure ventilation
promoting optimal nutrition
obtain blood glucose measurements upon admission to the nursery and every 1 to 2 hours to evaluate for changes
initiate early oral feedings or gavage feedings
if oral or gavage feedings aren’t tolerated, initiate an iv glucose infusion
assess skin for pallor and sweating
assess neurologic status for tremors, seizures, jitterness, and lethargy
monitor weight daily
maintain temperature using warmed blankets, radiant warmer, or warmed isolette
monitor temperature
cluster care and provide rest periods
maintaining thermal regulation
frequently assess temperature every hour
encourage immediate skin-to-skin contact after birth
observe for signs of cold stress: respiratory distress, central cyanosis, weak cry, abdominal distention, apnea, bradycardia, and acidosis
clinical manifestations of infection
increase in apnea
respiratory distress
increased need for respiratory support
hypotonia
lethargy
poor feeding
temperature instability
hypotension
tachycardia
interventions to prevent infection
monitor for changes in vital signs such as temperature instability, tachycardia, tachypnea, or decreased oxygen saturation
assess for feeding intolerance, which can be an early sign
encourage skin-to-skin contact
avoid using tape
use equipment that can be discarded
adhere to standard precautions
use sterile gloves in invasive procedures
avoid coming to work ill
remove all jewelry on your hands prior to washing
manifestations of pain in the newborn
sudden high-pitched cry
facial grimace with furrowing of brow and quivering chin
increased muscle tone
oxygen desaturation
increase in heart rate
body posturing, such as kicking, squirming, and arching
limb withdrawal and thrashing movements
increase in heart rate, blood pressure, pulse, and respirations
fussiness and irritability
cries tool
evaluates cry, oxygen requirement, increased vital signs, expression, and sleeplessness
non-pharmcological techniques for pain
gentle handling, rocking, caressing, cuddling, and massaging
rest periods before and after painful procedure
kangaroo care (skin to skin) during procedure
breastfeeding
use of a facilitated tuck (holding arms and legs in a flexed position)
application of topical anesthetics prior to venipuncture or lumbar puncture
nonnutritive sucking (pacifier dipped in sucrose) prior to procedure
minimal use of tape with gentle removal
warm blankets for wrapping
reduction of environmental stimuli
distractions such as with colored objects or mobiles
meconium aspiration syndrome
serious condition that occurs when newborn inhales particulate meconium mixed with amniotic fluid into the lungs while still in utero or when taking the first birth after birth
induces airway obstruction, surfactant dysfunction, hypoxia, and chemical pneumonia with inflammation of pulmonary tissues
nursing assessment for meconium aspiration syndrome
assess the amniotic fluid for meconium staining when the maternal membranes rupture
green-stained amniotic fluid suggests the presence of meconium in the amniotic fluid and should be reported immediately
after birth, note any yellowish-green staining of the umbilical cord, nails, and skin
this staining indicates meconium has been present for some time
symptoms
barrel-shaped chest
progressive respiratory distress including cyanosis
marked tachypnea which progresses to significant respiratory distress
intercostal and subxiphoid retractions
end-expiratory grunting
coarse crackles and rhonchi
meconium aspiration syndrome diagnostic findings
chest x-ray shows streaky linear densities progressing to patchy infiltrates with a flattened diaphragm and marked hyperaeration
metabolic acidosis
atelectasis
nursing management for meconium aspiration
suctioning at birth
supplemental oxygen to maintain 95% to 98%
neutral thermal environment
ensure adequate nutrition and fluid balance
provide continuous reassurance and support to the parents
hyperbilirubemia
a total serum bilirubin level above 5 mg/dL resulting in jaundice
jaundice is a yellowish discoloration of the skin and sclera of the eyes caused by the deposition of bilirubin in those areas when increased levels of unconjugated bilirubin exist in the newborn’s circulation
newborns produce large quantities of bilirubin which is a byproduct of the breakdown of rbcs
processed in the liver and normally excreted out of the body in the urine and stools
physiologic jaundice
bilirubin levels reach a high enough level after 24 hours of age to manifest as jaundice, peaking on the third to fourth day life
early onset breast feeding jaundice
not breastfeeding enough
late onset breast feeding jaundice
related to changes in composition of breast milk, may need to supplement with formula
early frequent feedings can provide the newborn with adequate calories and fluid volume (via colostrum) to stimulate peristalsis and passage of meconium to eliminate bilirubin
pathologic jaundice
refers to the development of jaundice within the first 24 hours of life, regardless of gestational age
hemolysis due to rh isoimmunization or abo incompatibility, metabolic or respiratory acidosis, and congenital inherited defects of enzyme
kernicterus results when free bilirubin crosses the blood brain barrier adn binds to brain tissue, resulting in selective brain damage
chronic bilirubin encephalopathy
nursing assessment for jaundice
neonatal jaundice first becomes visible in the face and forehead, identified by gentle pressure on the skin, since blanching reveals the underlying color
spreads in a cephalocaudal manner
nursing management for jaundice
documentation of the timing of onset of jaundice is essential to differentiate between benign (later than 24 hours) and significant (earlier than 24 hours) hyperbilirubemia
ensure newborn feeding (breast milk or formula) occur every 2 to 3 hours to prompt emptying of bilirubin from the bowel
encourage breastfeeding (at least eight feedings per day)
check for at least six wet diapers daily and a transition to at least four yellow, seedy stools
phototherapy
cover the newborn’s genitals and shield the eyes
turn every two hours
remove for feedings
assess temperature every FOUR hours
monitor fluid intake and intake/output
encourage breast or bottle feeding every 2-3 hours
check skin turgor for evidence of dehydration
monitor stool for consistency and frequency
providing parent teaching about jaundice
seek treatment
lethargy, sleepiness, poor muscle tone, floppiness
poor sucking, lack of interest in feeding
high-pitched cry
teaching guidelines - caring for a newborn with phototherapy
inspect nb’s skin, eyes, and mucous membranes
home health nurse will come to set up light system
keep the lights about 12-30 in above newborn
cover your newborn’s eyes with patches or cotton balls and gauze
keep the newborn undressed, except for the diaper area; fold the diaper down below the newborn’s navel in the front and as far as possible in the back
turn every two hours
remove for feedings
remove the eye patches during feedings
record temperature, weight, and fluid intake daily
document frequency, color, and consistency of all stools; should be loose and green as the bilirubin is broken down
keep the skin clean and dry
risk factors for jaundice
bruising at birth
prematurity
history of a sibling with jaundice
inadequate breastfeeding
hemolytic disease
birth injury such as cephalohematoma
polycythemia
down syndrome
family history of hemolytic disorder
maternal diabetes
male sex