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Autonomic dysreflexia symptoms are
Severe hypertension, flushing and sweating above the level of injury
Definitive management of a SUFE is
Internal fixation across the growth plate
A contraindication for a nasopharyngeal airway is
A base of skull fracture
Medical management for phaechromocytoma is (two drugs)
alpha blocker - phenoxybenzamine
then beta blocker - propranolol
Pain is osteoarthritis is typically
Worse after exercise and better after rest, tends to get worse through the day
A dermatofibroma is a
Raised, brown papule usually precipitated by an injury or insect bite
Definitive management for atypical endometrial hyperplasia is
Total hysterectomy with bilateral salpingo-oopherectomy
The way to work out AKI stage from creatinine is
New creatinine divided by old creatinine
Answer 1.X = stage 1
Answer 2.X = stage 2
Answer 3.X = stage 3
Management of severe lithium toxicity is
Haemodialysis
The length of time of anticoagulation after a PE is (provoked and unprovoked)
Provoked - 3 months
Unprovoked - 6 months
An obstructive picture on spirometry is
FEV1 reduced, FVC normal or less reduced
The paediatric BLS algorithm calls for
15:2 ratio at 100-120bpm
Drugs that must be stopped in AKI
NSAIDs
Aminoglycosides
ACE inhibitors
Angiotensin II receptor antagonists
Diuretics
Drugs that may accumulate and cause toxicity in AKI
Metformin
Lithium
Digoxin
Rhabdomyolitis causes (kidney pathology)
Acute tubular necrosis
Risk factors for ectopics are (6)
Damage to tubes (pelvic inflammatory disease, surgery)
Previous ectopic
Endometriosis
IUCD
Progesterone only pill
IVF (3% of pregnancies are ectopic)
Immunology findings in primary biliary cholangitis are
Raised serum IgM and antimitochondrial antibodies
Lithium toxicity may be precipitated by (3)
Dehydration
Renal failure
Drugs: diuretics, ACE inhibitors/angiotensin II receptor blockers, NSAIDs and metronidazole.
Clinical features of central retinal vein occlusion are
Sudden painless monocular loss of visual acuity, dense central scotoma, ipsilateral relative afferent pupil defect, retinal haemorrhage, oedema and dilated veins giving a blood and thunder appearance
First line management for refractory ascites is
Large volume paracentesis over 6 hours with an albumin infusion