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high risk pregnancy
Jeopardy to mother, fetus, or birth
Condition due to pregnancy or result of condition present before pregnancy
Higher morbidity and mortality
Risk assessment with 1st antepartal visit ongoing
High risk pregnancy risk factors
Diabetes
Cardiac and respiratory disorders
anemia
specific infections
Gestational diabetes
Diagnosis with GTT (glucose tolerance test) in pregnancy
Maternal hyperglycemia= fetal hyperinsulinemia
Insulin is a growth factor
Macrosomia
Decreased surfactant
GD risk factors
Obesity
Family hx
Advancing age over 25
Past hx
Hx stillbirth or large baby
Hx abnormal GTT
GD diagnosis
Urine testing
HbA1C??
Screening with 1 hour Glucose tolerance test (GTT)
Random- non scheduled
24-28 weeks
50g oral glucose load (glucola), <140
Diagnostic with 3 hour GTT
100g load
Fasting (<92)
1 hr, 2 hr, 3 hr blood tesing
1 hour: <180
2 hour <153
3 hour: <140
GD TX
Diet
Nutritional consult
Meds
Oral
is an option bur most as good as insulin
Glyburide
Metformin
Insulin
Post Prandial BG of <120
Short acting
Insulin pump
Will have insulin pump intrapartum
Postpartum will have 2hr PP BG with insulin or nothing
GD complications
Mom
HTN
Postpartum hemorrhage
C/S birth
Fetus
Polyhydramnios
Congenital disorders
Macrosomia
Asphyxia/Stillbirth
Neonate
Prematurity
RDS
Hypoglycemia
cardiac and acquired HD
Pathophysiology
Hemodynamic changes/increased cardiac workload overstressing woman’s cardiovascular system
Therapeutic management
Risk assessment, prenatal counseling, increased prenatal visits, Perinatologist
Nursing assessment
Vital signs, heart sounds, weight, fetal activity, lifestyle
Assess for cardiac decompensation
Intrapartum: delivery in side-lying position, FHR, ABG, no Terb!
Postpartum: 24-48 they are still not stable, side lying, HOB
congenital, Acquired, and ischemic HD
Congenital Heart Disease
Septal defects
CoArc
TOF
Acquired Heart Disease
Mitral valve prolapse
Mitral valve stenosis
Aortic stenosis
Ischemic Heart Disease
Myocardial infarction
Anemia
Iron deficiency
Usually due to inadequate dietary intake
Therapeutic management: eliminate symptoms, correct deficiency, replenish iron stores
Nursing assessment
Fatigue, weakness, malaise, anorexia, susceptibility to infection (frequent colds), pale mucous membranes, tachycardia, pallor
Abnormal lab results
Low hemoglobin, low hematocrit, low serum ferritin <12
Compliance with drug therapy: prenatal vitamin and iron supplement
Dietary instruction and counseling
specific infections
TORCH
T toxoplasmosis
O other
R rubella
C
H herpes 1
Toxoplasmosis
Protozoa the infects warm blooded animals – causing toxoplasmosis
Cats are hosts/reservoirs, found cat feces, litter boxes, gardens
Human transmission:
Ingestion of undercooked eggs, red meats containing protozoa cysts/oocytes
Hand to mouth transmission with infected hand
Complications: embryonic damage, or congenital transmission: LBW, liver damage
Prevention is key
Nutrition education
Pet cat: other people change little box or gloves
Rubella
German measles
Worse effects in first 12 weeks of pregnancy
Congenital rubella:
Cataracts
Glaucoma
Hearing loss
Cardiac defects
Neuro effects
Prevention:
Screening
Education
Vaccination – PP
Hepatitis B virus
Transmission through blood and bodily fluids
100 times more infectious than HIV ( can live outside body for >week)
Congenital HBV worse in 3rd trimester
40% of infants of moms with HBV will have Chronic HBV by 6 months of age
Screening for all moms
HBV+ moms
Infant will get HBV vaccine AND HBIG within 12 hours
Breastfeeding??
Parvovirus 19
Very common viral infection of children (AKA Fifth’s disease)
Erythema “slapped face” appearance
Most people have been exposed then immune
Greatest risk to fetus within 20 weeks
Complications to fetus:
Hydrops
IUFD
Cardiac anomalies
Prevention:
School, preschool, daycare workers- education!
Herpes simplex
HSV-1, HSV-2: both can cause genital infections
HSV-1 now main cause of genital infections
Greatest risk of transmission is new active infection near time of birth
Ascending infections after ROM
Direct contact during birth
SAB, IUGR, Neuro damage, Congenital HSV, fetal death
Method of birth
Absence of active infection= vaginal birth
Avoid all procedures (AROM, FSE, instrument delivery)
Active infection: C/S
Prevention
Prophylaxis at 36 weeks with antiviral
Effect on fetus depends on timing of infection
Group B strep
Naturally found in GI tract in 50% of women
25% pregnant women have GBS in the rectal/vaginal area
Most common cause of meningitis/sepsis in newborn
Tested at 36 weeks via vaginal/rectal swab or from urine
Fetal risk also with previous pregnancy with + GBS
+GBS moms will get prophylaxis with PCN intrapartum
Every 4 hours until delivery (at least 1 dose 4 hours before birth)
Clindamycin or Cephalosporin if allergic to PCN
Unknown GBS moms- infants to be closely monitored
Spontaneous abortion
Loss before 20 weeks: spontaneous/induced
Cause unknown and highly variable
First trimester commonly due to fetal genetic abnormalities
20% of pregnant women have vaginal bleeding in 1st trim, ½ of these are having a spontaneous abortion
Second trimester is more likely related to maternal conditions
Cervical insufficiency
Uterine anomalies
Diabetes
Cocaine
HTN
TORCH
Spontaneous abortion risk factors
Up to 50% of all fertilized eggs die and are lost (aborted)
Usually before women know they are pregnant
Usually happens between week 7-12
Higher risks with:
AMA (>35)
Diabetes or Thyroid issues
Hx of 3 or more spontaneous abortions
Spontaneous abortion assessment
Pt will report bleeding, cramping, products of conception
Vaginal bleeding
Color: bright red is significant
Amount: 1 pad/hr is significant
Clots/tissue: Save them!
Vital signs, pain level
Intensity of cramping/contractions
Client’s understanding/emotional needs
Proper history and assessment will let us know what type of AB
Spontaneous abortion types
Threatened
Slight bleeding, no dilation, cervical change or loss of fetal tissue
Confirm viability of fetus with u/s
Supportive treatment: rest, nutrition, hydration
Incomplete
some products passed, D/C
Complete-
all products passed, f/u with MD office, HCG levels
Missed
nonviable embryo retained, D/C or induction
Spontaneous abortion nursing management
Continued monitoring:
vaginal bleeding, pad count, passage of products of conception
pain level
preparation for procedures
medications
Misoprostol (Cytotec)
Mifepristone(RU-486) – prostaglandin to stimulate contractions/sloughing
Support: physical and emotional; stress that woman is not the cause of the loss; verbalization of feelings, grief support, referral to community support group
Ectopic pregnancy
AKA- “tubal pregnancy”
Ovum implantation other than the endometrium of uterus (see Figure 19.1)
Most common cause of maternal death in 1st trimester!
Obstruction to or slowing passage of ovum through tube to uterus
Causes:
Tubal damage/scarring from PID
Altered tubal motility, chromosomal abnormalities
Douching??
ectopic pregnancy management
Therapeutic management
Hallmark sign: abdominal pain with spotting within 6 to 8 weeks after missed menses
HCG test, U/S
Medical: to remove tube or just POC to preserve and no scarring
Surgery if ruptured or implantation other than adnexa
Rh immunoglobulin if woman Rh negative
Medication:
Methotrexate – chemo drug
IM, single dose, 90% success
Folic acid antagonist- inhibits cell division
Pt must be hemodynamically stable
Preparation for treatment
Analgesics for pain
Medications for medical treatment
Teaching about signs and symptoms of rupture
Surgery
Emotional support
Education
cervical insufficiency
Premature dilatation of cervix
Causes:
Lacerations from previous births
Cervical procedures
Congenital issues
Nursing assessment
Pink-tinged vaginal discharge or pelvic pressure
Cervical shortening via transvaginal ultrasound
Therapeutic management
Bed rest, pelvic rest, avoidance of heavy lifting
Cervical cerclage
16 weeks, removed at 37
Preeclampsia
Patho
Most common HTN in pregnancy
HTN with proteinuria after 20 wks
Mild or severe
Vasopasm and hypoperfusion -> decreased placental perfusion-> vasoconstriction, activation of coagulation cascade, fluid redistribution -> degreased organ perfusion
Only cure is delivery
preeclampsia risks
Extreme age >40, <19
Nulliparity
Sperm!
AA
Preexisting HTN, renal dx, Diabetes
Multifetal pregnancy
History
Previous Hx
Family Hx
FOB Family Hx
BMI >29
preeclampsia symptoms
BP
Mild: >140/90 after 20wks
Severe: >160/110
Proteinuria
Mild: 300mg/ 24 he >1+ protein DS
Severe: 500mg/ 24hr > 3+ protien DS
Hyperreflexia (severe) 4+ DTR eith or without clonus
Mild: Hand/facial edema, wt gain
Severe: HA, blurred vision, pulmonary edema, thrombocytopenia, epigastric pain HELLP
preeclampsia nursing management
Labs:
CBC, BUN, AST/ALT, UA
Nursing management
Quiet environment, sedatives, seizure precautions
Antihypertensives
DTR testing
Assessing for magnesium toxicity and labor
Seizure management for eclampsia
Preparation for birth
Mild preeclampsia management (no renal/hepatic/coagulation issues)
Home: Bed rest, daily BP monitoring, and fetal kick counts
Hospitalization
Hypertensive medication/MgSO4
Severe preeclampsia management
Hospitalization
oxytocin and magnesium sulfate
preparation for birth
preeclampsia TX
Magnesium Sulfate (MGSO4)
Treatment at least 24 hr after delivery or longer
increased CNS irritability
SE:
flushed, warm, “gorked”
Toxic SSX:
No DTRs, resp dispression, increased lethargy
4 gm bolus then 2gm/hr maintenance
have a calcium antidote to prevent OD
Antihypertensives
Hydralizine
Labetalol
Nifedipine
Aldomet
Eclampsia
Patho
Preeclamspia with seizures
Risks
Symptoms
BP
PROTEINURIA
SEIZURE/COMA
Hyperreflexia
HA, blurred vision, general edema, thrombocytopenia, epigastric pain HELLP, cerebral hemorrhage
Nursing management
Eclampsia management
Preventable if preeclampsia is caught early
Seizure management, MgSO4, HTN meds; birth (c/s) once seizures controlled
HELLP
Hemolysis: RBC, plt, #Bilirubin
Destruction d/t traveling through constricted vessels
Eevated Liver enzymes
Vasospasms $blood flow in liver = tissue damage
Low Platelets
Plts collect at site of all endothelial damage= all used up = DIC
Occurs in 3rd trimester or w/in 48hrs postpartum
High risk for renal failure, DIC, abruption, liver failure, stroke
Nursing assessment: similar to that for severe preeclampsia; laboratory test results
Nursing management
same as for severe preeclampsia
Blood products to correct hemolysis
PROM
SROM before labor women beyond 37 wks gestation ( at term)
Risk factors
Weakened membranes
Inflammation, UTI
Contractions
increased intrauterine pressure
SS
BP
Mild: >140/90 after 20 wks
severe: >160/110
Proteinuria
mild: 300mg/ 24 hr > 1+ protein DS
severe: > 500mg/24 hr > 3+ protein DS
Hyperreflexia (only severe)
HA
Blurred vision
pulmonary edema
thrombocytopenia
epigastric pain
HELLP
PROM management
Management
Identification of uterine contractions
Infection prevention
Chorioamniotis- infection of amniotic cavity
GBS, Ecoli, BV ( long labor, PROM, Multiple vag exams)
Maternal tachy, fever, uterine tenderness, foul odor!
Antibiotic therapy (3) intra/pp
Mild preeclampsia management (no renal/hepatic/coagulation issues)
Home: Bed rest, daily BP monitoring, and fetal kick counts
Hospitalization
Hypertensive medication/MgSO4
Severe preeclampsia management
Hospitalization
oxytocin and magnesium sulfate
preparation for birth
C/S- dystocia or Fetal distress
TX
dependent on gestational age
no unsterile digital cervical exams until woman is in active labor
expectant management if fetal lungs immature
PPROM
water broke before labor in preterm mom, (preterm) women less than 37 weeks’ gestation
SS
BP: >160/110
Marked Proteinuria
Proteinuria
seizure/Coma
Hyperreflexia
HA
Blurred vision
general edema
cerebral hemorrhage
renal failure
thrombocytopenia
epigastric pain
HELLP
Management
Preventable if preeclampsia is caught early
Seizure management, MgSO4, HTN meds; birth (c/s) once seizures controlled
overall goal for a pregnant client with CAD
Goal is to keep them stable
For congenital and acquired heart disease risk assessment, screening for conditions during prenatal care
To reduce cardiac workload have them turned to lt side, monitor ABGs and FHR, don’t give terbutaline
plan of care for a client and newborn with HIV.
GA has high numbers of HIV- similar to 3rd world country
Today, if a woman takes HIV medicine daily as prescribed throughout pregnancy, labor, and delivery and gives HIV medicine to her baby for 4-6 weeks after delivery, the risk of transmitting HIV to the baby can be as low as 1% or less. 35% if no treatment
NO BREASTFEEDING
Prenatally all women are given the choice of screening
Complications:
Pregnancy: preterm, IUGR, PROM, infection, poor wound healing
Newborn: transmission (2% increase in transmission every hour after ROM)
Therapeutic management: oral antiretroviral drugs BID from 14 weeks until birth; IV administration during labor; oral syrup for newborn for first 6 weeks of life; decision for birthing method
Birthing method: depends on viral load (viral shedding)
>1000- C/S
<1000- may attempt vaginal delivery
Nursing management: history and assessment; HIV antibody testing;
Intrapartum: medication, prevention of invasive procedures (episiotomy)
PP: bath infant immediately- before meds
nurses role in care of pregnant teen
11% of births are in girls 15-19
1 in 4 teen mothers have another baby within 2 years
Realistic role models; emotional support
Nursing management:
Assessing knowledge of health and nutrition for self and child
Challenges of parenting role/future planning
EDUCATION!
Care coordination/social services
risk factors and outcomes of a pregnant client over the age of 35.
risks
gestational diabetes
preeclampsia
labor dystocia
C-section delivery
preterm birth
postpartum hemorrhage
low birth weight of the baby
severe issues with the baby
indicating a stay in the NICU
obesity
Outcomes
higher risk for APGARs below 7 after 5 minutes
asphyxia at birth, congenital malformations
being LGA
aspiration of meconium,
jaundice
transient tachypnea
brachial plexus injury
seizures
sepsis
intrauterine death.
substance abuse during pregnancy
True stats of substance abuse in pregnancy unknown
Maybe 7% of all pregnancies (illicit, smoking, alcohol
5% use illicit drugs
Types:
Cocaine – preterm labor, abruption
Heroin
Marijuana
Methadone- treat as an opioid (for morphine/heroine addiction)
Tobacco- IUGR, LBW
Alcohol - FAS
Effects on fetus: growth restrictions, increased risk of SIDS
Worse in 1st trimester – neuro development issues
Alcohol
30% of pregnant women continue to drink alcohol
Fetus gets same alcohol concentration
Fetal Alcohol Spectrum Disorder
Structural birth defect anomalies
Behavioral/neurocognitive disorders
1 in 100 infants/ yr
substance abuse nursing management
Nursing assessment: urine toxicology (mom and baby)
Nursing management
Nonjudgmental approach
State protection agency investigation for positive newborn drug screen
Neonatal Abstinence Syndrome
WITHDRAWAL (symptoms)
Nursing interventions
metabolic changes in pregnancy
Normal pregnancy:
1st half: increased E+P, insulin, response to insulin = hypoglycemia
2nd half: increased human placental lactogen, cortisol, glycogen, insulin resistance
Fetus removes glucose from maternal blood, but not insulin
Mom uses fat liposis for energy (watch for ketones)
= hyperglycemia- protection for fetus to have enough glucose
Altered insulin requirements
1st half: insulin needs may decrease
2nd half: insulin needs may double or more
After delivery- may abruptly decrease insulin needs
Breastfeeding may also decrease insulin needs
7-10 days after breastfeeding/or no breastfeeding will return to normal insulin needs.