EMT Crash Course: Chapter 9: Airway, Respiration, and Artificial Ventilation

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130 Terms

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Ventilation
moving of air in & out of the lungs; required for effective oxygenation & respiration
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Inhalation
active part of ventilation; energy is required
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During inhalation
diaphragm & intercostal muscles contract → intrathoracic pressure decreases → vacuum is created → thorax enlarges → air passes through upper airway to lower airway → gets to alveoli
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Exhalation
passive part of ventilation; no energy is required
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During exhalation
diaphragm & intercostal muscles relax → thorax decreases → air is compressed out of the lungs
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Air obstruction
blockage of an airway structure leading to the alveoli; will prevent effective ventilation
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Causes of airway obstruction include
tongue (\#1 common cause), fluid (saliva, blood, mucus, vomit), swelling, & foreign bodies
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Methods of controlling oxygen delivery
increasing/decreasing rate of breathing & increasing/decreasing the tidal volume of breaths
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Hypoxia
inadequate delivery of O2 to the cells
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Early indications of hypoxia
restlessness, anxiety, irritability, dyspnea, cyanosis, and tachycardia
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Dyspnea
shortness of breath
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Late indications of hypoxia
altered/decreased level consciousness, severe dyspnea, cyanosis, and bradycardia
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Bradycardia
slow heart rate
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Carbon dioxide drive
body's primary system for monitoring breathing status; body monitors CO2 leels in the blood & cerebrospinal fluid
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Hypoxic drive
backup system CO2 drive, monitors oxygen levels in plasma, may be used by end-stage chronic obstructive pulmonary disease patients who have high levels of CO2
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Prolonged exposure to high concentrations of oxygen in hypoxic drive patients
may depress spotaneous ventilations
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Do not \_______ from acutely ill/injured patients
withhold oxygen
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Oxygenation
delivery of oxygen to the blood; ventilation is required
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Oxygenation is required for respiration BUT
does not ensure respiration
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Surrounding air
contains about 21% O2
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Expired air
contains about 16% O2
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Respiration
exchange oxygen & CO2
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Heart and brain become irritable due to
lack of oxygen almost immediately
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Brain damage begins within
about 4 minutes
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Permanent brain damage likely within
6 minutes
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Irrecoverable injury is likely within
10 minutes
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Airway/Breathing Assessment
- Look for chest rise/fall
- Listen for breathing, ability to speak, & lung sounds
- Feel for air movement & chest rise/fall (Put ear near patient or hand on patient's chest)
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Adequate Breathing
normal RR & rhythm, nonlabored breathing, adequate tidal volume, & clear bilateral lung sounds
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Inadequate breathing
- Abnormal RR or breathing pattern
- Nasal flaring (enlargement of nostrils)
- Abnormal, diminished, or absent lung sounds
- Paradoxical motion (flail chest segment moves in opposite direction of the thorax
- Unequal chest rise/fall
- Dyspnea, accessory muscle use, retractons
- Cyanosis
- Agonal respiration (dying gasps)
- Apnea (no breathing)
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Auscultation
use of stethoscope to listen for lung sounds; left lung field is always compared to right lung field (side to side never top to bottom)
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Anterior auscultation
- Place the stethoscope at the midclavicular line about the second intercostal space (2 inches below clavicle but above nipple line)
- Auscultate bilaterally (on both sides of the chest)
- Place the stethoscope at about the 4th intercostal space at midaxillary line
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Posterior auscultation
Place the stethoscope at about the midclavicular line & below the scapula bilaterally; lung sounds are often easier to access/hear posteriorly
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Normal lungs sounds
clear & equal bilaterally
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Absent/diminished lung sounds
indicates little or no air exchange
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Wheezing lung sounds
high pitched sounds usually heard during exhalation (lower airway)
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Rales
"wet" or "crackling" sounds (upper airway) caused by pneumonia. bronchitis. respiratory infections that cause mucus buildup)
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Stridor
high-pitched sounds indicating partial upper airway obstruction (auscultated in upper airway/neck)
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Rhonchi
low-pitched sounds resemble snoring (upper airway)
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Pulse Oximetry
considered the "sixth vital sign."; Monitoring of oxygen saturation (SaO2) is now part of the standard of care for EMS; often a function provided on cardiac monitor/ defibrillators
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SaO2
measures % of hemoglobin (RBC) that are saturated with O2; does not identify definitively how much oxygen is in the blood, but it's an indication of respiratory efficiency
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Normal SaO2
\>98%, but
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Advantages of pulse oximetry
fast, easy, noninvasive assessment tool
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Limitations of pulse oximetry
- Indication of respiratory efficiency, not confirmation
- Cannot measure the amount of hemoglobin, only the oxygen saturation of the hemoglobin that is present
- Other clinical assessments must be performed as well
- Measurement may be difficult to obtain on some patients due to hypovolemia, hypothermia, anemia, nail polish, carbon monoxide poisoning
- Measures saturation of hemoglobin; it can- not distinguish between oxygen saturation and carbon monoxide saturation.
- Time delay between patient's pulse oximeter reading & current RR
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Head-tilt-chin left
preferred manual method of opening the airway
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Indications for Head-tilt-chin lift
altered/decreased LOC, suspected airway obstruction, requiring suctioning
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Contraindications for Head-tilt-chin lift
Suspected c-spine injury
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Indications for Jaw-thrust maneuver
patients with altered/decreased LOC AND suspected c-spine injury
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Contraindications for Jaw-thrust maneuver
conscious patients
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Oropharyngeal airway (OPA)
used to prevent the tongue from obstructing the airway; failure to size or insert OPA correctly can cause the tongue to block the airway; remove OPA if patient gags
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Indication for OPA
unresponsive patients without a gag reflex
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Contraindications for OPA
conscious patients or any patient with intact gag flex
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Sizing OPA
measure from the corner of mouth to earlobe
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Inserting OPA in Adults
1. Manually open airway
2. Suction if needed
3. Insert OPA upside down with distal end pointing toward roof of mouth
4. Rotate 180° while advancing OPA until flat
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Inserting OPA in Pediatrics
1. Manually open airway
2. Suction if needed
3. Depress tongue with depressing
4. Insert directly or insert sideways and rotate
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Nasopharyngeal Airway (NPA)
used to prevent tongue from obstructing airway in patients that can't protect their own airway
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Indications for NPA
unresponsive patient w/o gag reflex & patients with decreased LOC but with intact gag preventing use the OPA
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Contraindications for NPA
conscious patients with gag reflex that can protect their airway, severe head/facial trauma/injury; resistance to insertion in both nostrils; NPAs are not typically used for patients
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Sizing NPA
measure from tip of nose to earlobe
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Inserting NPA
1. Lubricate NPA with water-solube lubricant
2. Always insert NPa with bevel toward septum
3. Try larger nostril first & switch if needed
4. Advance gently, rotating as necessary, DO NOT force
5. Remove immediately if patient gags
6. Suction if needed
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Aspiration
entry of matter into lungs, drastically increases risk of death
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Indications for Suction
secretions (blood, vomit, mucus, oral secretions, etc.) in the airway that could be aspirated, obstruct the airway, or interfere with ventilations or insertion of a mechanical airway adjunct
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Suction should generally be performed
after the airway is opened manually and before insertion of mechanical airway adjunct
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Suction units
should be able to generate a vacuum of 300 mmHg when tubing is clamped
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Fixed suction
suction unit permanently mounted in a vehicle, hospital
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Hand-powered suction
manually powered portable suction unit
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Suction catheter
attaches to the suction unit & is inserted into the patient's airway to remove secretions; single-patient use only (tubing & disposable canisters as well)
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Rigid suction catheter (tonsil tip or Yankauer)
best suited for suctioning the oral airway
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French catheter (whistle-tip)
flexible catheter that comes in several sizes, best suited for suctioning nose, stoma, or inside of advanced airway device
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Suctioning increases the risk of
hypoxia
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Adults Max Suction time
15 seconds
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Pediatric Max Suction time
10 seconds
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Infant Max Suction time
5 seconds
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Recovery position (patient on his side)
reduces sick of aspiration; unresponsive patients with adequate breathing and no c-spine should be in this position
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Supplemental Oxygen
goal → maintain pulse ox of ≥94%, not needed if no signs or resp. Distress or ≥94% pulse ox
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Indications for Supp O2
- Cardiac arrest
- Receiving artificial ventilation
- Suspected hypoxia
- Signs of shock (hypoperfusion)
- Pulse O2 < 94%
- Altered/decreased LOC
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Oxygen cylinders
seamless steal or aluminum cylinder of various sizes, usually green, can never be left standing unattended, amount of O2 measured in psi (full cylinder has about 2000 psi)
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Cylinder should be taken out of service & refilled if below
200 psi
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Pin indexing system
safety feature that prevents a CO2 cylinder from being connected to O2 regulator
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Flow meters/pressure regulators
Flow meters are connected to pressure regulators. In combination, they re- duce the pressure coming from the tank to safe levels and allow a specific flow rate. The flow rate is measured in liters per minute (lpm or L/ min)
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Nonrebreathers (NRB) masks
preferred method O2 admin in prehospital, referred to as "high-flow" O2 admin, available in adult and pediatric sizes
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Flow rate of NRB
10-15 LPM
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Oxygen delivered in NRB
up to 90%
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Cautions of NRB
prolonged use can dry & irritate nasal passage if oxygen is not humidified
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Simple face mask
similar to nonrebreather, but w/o oxygen reservoir; flow rate → 6-10 lpm, 40-60% oxygen
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Venturi mask
mask delivers precise concentration of low-flow oxygen, rarely used in the prehospital environment
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Tracheostomy
surgical procedure that creates an opening through the neck & into the trachea
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Humidification of oxygen
increases the moisture of supplemental O2 by flowing it through water prior to inhalation by the patient
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Oxygen toxicity
alveoli can collapse due to long-term exposure to high concentrations of oxygen; rarely occurs in prehospital environment
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Respiratory depression
risk for COPD patients on the hypoxic drive however, it typically requires long- term exposure to high-concentration oxygen; retinal damage can occur in newborns with lon-term exposure to high concent. O2
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Assisted Ventilation (also artificial ventilation or positive pressure ventilation (PPV))
includes mouth to mask, flow-restricted, oxygen-powered ventilation device & automatic transport ventilators and BVM; indicated for any patient with inadequate spontaneous breathing leading to severe respiratory distress or failure
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Bronchoconstriction
narrowing of the airways
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Pulmonary edema
condition caused by too much fluid in the lungs; This fluid collects in the many air sacs in the lungs, making it difficult to breathe
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Apnea
no spontaneous breathing
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Agonal breaths
shallow, ineffective gasps
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Bradypnea
slow breathing
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Tachypnea
fast breathing
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Hypoventilation
breathing too slow or shallow
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Any unresponsive patient receiving artificial ventilations should have an airway adjust in place to
prevent the tongue from obstructing the airway
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Complications of PPV
- Increased intrathoracic pressure, which reduces circulatory efficiency
- Gastric distention, which increases the risk of vomiting & can compromise ventilatory efficiency
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Hyperventilation
common mistake; occurs when ventilations are provided too fast, too deep, or both