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what are social norms and how does it link to abnormality
unwritten behavioural expectations that vary on culture, time and context
social deviants are seen as abnormal as they dont abide by social norms
AO3 for deviation from social norms as an explanation for abnormality
✅ real world application - used to diagnose antisocial personality disorder
❌cultural differences - e.g. inappropriate to diagnose Afro-Caribbean people as abnormal as they are 7x more likely to be diagnosed w/ schizoprenia
what is FTFA
failure tp function adequately means someone cant cope with everyday life, e.g. they cant shower or get out of bed
AO3 for FTFA
✅takes into account individual lived experiences - using GAF scale 1 (normal) to 6 (FTFA), uses holistic view and acknowledges struggles
❌only takes those who cant function into account - e.g. psychopaths are abnormal but can still function
what is statistical infrequency
behaviours we see frequently are normal, behaviours we see infrequently are abnormal
uses normal distribution (bell curve), normal behaviour is in the middle and abnormal behaviour is shown on the left and right
AO3 for statistical infrequency
✅objective , uses quantitative data (easy to interpret)
❌abnormal behaviour ≠ negative , for example high IQ
what is deviation from ideal mental health
(Jahoda) individual is abnormal if they deviate from having:
Positive perception of self
Autonomy
Resistance to stress
Mastery of environment
Accurate perception of reality
Self actualisation
AO3 for DfIMH
✅holistic, suggests personal development
❌too strict, many people would be seen as abnormal
what is a phobia
irrational fear of an object or situation
characteristics of phobias
behavioural → Panic, Endurance, Avoidance
emotional → Anxiety, Fear, Unreasonable emotional response
cognitive → Selective attention to phobic stimulus, Irrational beliefs, Cognitive distortions
characteristics of depression
behavioural → Reduced energy levels, A change in eating behaviour, Aggression
emotional → Sadness, Guilt
cognitive → Negative schema, Poor concentration
characteristics of ocd
behavioural → Compulsions, Avoidance
emotional → Anxiety, Depression
cognitive → Obsessive thoughts, Hypervigilance (a state of high alert)
AO1 behaviourist explanation of phobias
acquired through classical conditioning - the phobic stimulus (NS) is paired with UCS that naturally causes a fear response & becomes CS
maintained via operant conditioning - negative reinforcement when fear is removed from avoiding situation
positive reinforcement when relief is added
AO3 for behaviourist explanation for phobias
✅real world application - exposure therapies
❌doesn’t take cognitive aspects into account
✅shows link between bad experiences and phobias (Little Albert)
❌not all phobias are due to bad experiences
AO1 behaviourist approach to treating phobias
Systematic desensitisation → (gradual exposure) 1. anxiety hierarchy, 2. relaxation techniques, e.g. breathing exercise, 3. exposure due to reciprocal inhibition
Flooding → immediate and full exposure to phobic stimulus, temporary panic leads to extinction as person learns phobic stimulus is harmless due to exhaustion from fear response
AO3 for behaviourist approach to treating phobias
SD → ✅effective (was compared to control group with no SD) - but not effective for social phobias (anxiety hierarchy)
✅acceptable to patients
✅benefits those with learning disabilities
Flooding → ✅cost effective (2-3 hrs)
❌traumatic
❌doesnt work with all phobias (e.g. social)
AO1 cognitive explanation depression
Beck - depression is due to irrational thoughts from maladaptive internal mental processes
negative self schema + faulty informational processing (viewing negative aspects of a situation) = negative triad (negative view of self, world and future)
Ellis’ ABC model - bad mental health stems from irrational thoughts which interfere with happiness and being free from pain
A - activating thoughts
B - irrational beliefs
C - consequence
what is mustabatory thinking
thinking the world should be a certain way ultimately leads to dissappointment
AO3 depression
✅research support for negative triad - showing cognitive vulnerability can help predict later depression
✅real world application for negative triad - young people who show negative triad are monitored for treatment
❌Ellis’ ABC model only explains reactive depression, not internal/endogenous
❌unethical as it blames depressed people for their thought process
AO1 for cognitive approach to treat depression
Beck’s CBT and Ellis’ REBT change negative schemas and challenge irrational thoughts
Beck’s CBT - patient acts as a scientist by creating and testing hypotheses on validity of their irrational thoughts, and when they see its not valid, negative schema and irrational thoughts are discarded
thought checking - irrational thoughts are identified from negative schema by setting hw tasks like a diary
Ellis’ REBT - ABCDE model (dispute and effect added)
dispute - vigorously argue with client using empirical and logical arguments to challenge irrational beliefs
empirical → asking for evidence
logical → show that beliefs dont make sense
AO3 for cognitive approach to treating depression
✅effective (compared to group with drugs and both had 86% improvement)
✅cost effective (6-12 sessions)
❌doesnt work with those who have learning difficulties
❌CBT has high relapse rate (42%)
AO1 for biological approach to explaining OCD
genetic → OCD is inherited, caused by 230 candidate genes which increase vulnerability for OCD so its polygenic as its caused by a combination of genetic variations that increase vulnerability for OCD
diathesis stress model also explains OCD → genetic predisposition of OCD and a traumatic event
neural → malfunctioning neurotransmitters - low serotonin causes OCD when its removed too quickly from synapses before it transmits signals, causing low moods and obsessive thoughts
frontal lobe is responsible for decision making and can become dysfunctional which can lead to OCD (e.g. hoarding)
Para hippocampal gyrus → processing unpleasant emotions → can become faulty and cause OCD, resulting in anxiety and depression
AO3 for biological explanation of OCD
✅research support - twin studies (genetic)
❌conflicting research - environmental factors cause OCD
✅research support (neural) - SSRIs help OCD so serotonin must be involved
✅real world application - SSRIs treatment
how do SSRIs work
presynaptic nerve releases serotonin to postsynaptic nerve
in OCD patients, serotonin is quickly reabsorbed by presynaptic nerve before it transmits signals
SSRIs inhibit uptake of serotonin so it stays in synapses longer
this reduces anxiety and elevates mood
AO1 for treatment of OCD
drug therapies - SSRIs are given to patients where they take 20mg of fluoxetine for 3-4 months daily
Alternatives to SSRIs → tricyclics and SNRIs (when SSRIs fail, have more side effects)
AO3 for treatment of OCD
✅effective
✅cost effective
❌side effects - nausea, headaches
❌patients prefer CBT - they dont want to be reliant on drugs or CBT is more effective to them