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from book manual + doc's ppt / discussion
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phase I periodontal therapy
required regardless of the extent of the disease
aimed at stopping the progression of gum disease by addressing its root microbial etiology
the long-term success of periodontal treatment depends predominantly on maintaining the results achieved during this phase.
allows the dentist to evaluate the tissue response and gauge patient motivation, which are crucial elements for overall treatment success.
included in phase I
correcting restorative defects
treating occlusal trauma, splinting
antibiotic therapy, furcation involvement
calculus removal, intensive plaque control
tissue reevaluation, caries management
treating gingival and periodontal infections
restoration correction & prosthetic devices
—all procedures that doesn’t require scalpel
reminders for phase I therapy
deep pockets and furcations → make SRP difficult, and surgical access can improve results.
younger patients with extensive attachment loss → more likely to have aggressive forms of disease requiring extensive therapy
if inflammation persists after scaling and planning, further therapy is often necessary.
surgical treatment → only advisable if phase I therapy resulted in the gingiva being free of overt inflammation and the patient has established effective daily plaque control (e.g., 20% or less of tooth surfaces free of plaque).
other terms for phase I nonsurgical phase
initial therapy
cause-related therapy
etiotropic phase of therapy
nonsurgical periodontal therapy
5-mm
primary guideline for referral
This standard relates to clinical attachment loss present at the reevaluation appointment
—If the typical root length is about 13 mm, 5 mm of clinical attachment loss means only about half the bony support remains.
9 mm or greater
Success rates diminish when probing depths are __
primary objective of phase I
to alter or eliminate the microbial etiology and contributing factors associated with gingival and periodontal diseases.
this aims to halt disease progression and restore the dentition to a state of health and comfort.
major goal of phase I
to control the factors responsible for periodontal inflammation, especially the removal of subgingival bacterial deposits and subsequent patient control of plaque levels.
management of local contributing factors in phase I
treatment of food impaction areas and occlusal trauma
orthodontic tooth movement, extraction of hopeless teeth
complete removal of calculus, possible use of antimicrobial agents
restoration or temporization of carious lesions to remove bacterial reservoirs
correction or replacement of poorly fitting restorations and prosthetic devices (these can be plaque traps)
Sequence of Procedures in phase I
Step 1: Plaque Control Instruction
Step 2: Removal of Calculus (Scaling and Root Planing)
Step 3: Recontouring Defective Restorations and Crowns
Step 4: Management of Carious Lesions
Step 5: Tissue Reevaluation (Critical Post-Therapy Step)
step 1: plaque control instruction
essential component for successful therapy
instruction should begin in the first appointment, teaching the patient to brush correctly (focusing on the gingival third) and use interdental aids
step 2: removal of calculus (scaling and root planing)
accomplished using scalers, curettes, ultrasonic instrumentation, or a combination of devices
laser technology may also be used
Step 3: Recontouring Defective Restorations and Crowns
Defects like overhangs are plaque traps and must be corrected by smoothing surfaces/overhangs or replacing the restorations.
step 4: management of carious lesions
removal of carious tissue and placement of temporary or permanent restorations is indicated
removes the reservoir of bacteria, maximizing the healing of periodontal tissues.
step 5: tissue reevaluation
tissue require approximately 4 weeks to heal sufficiently before accurate probing can occur.
at this appointment, periodontal tissues are probed and conditions are evaluated to determine if further treatment, such as periodontal surgery, is indicated.
scaling and root planing
extensively studied, effective, and reliable
80% — reduction in bleeding on probing
2-3 mm — mean probing depth reductions
percentage of periodontal pockets 4mm or greater in depth can be reduced by 50% to 80%
Healing of the gingival epithelium
heals over approximately 4 weeks
results in the formation of a long junctional epithelium rather than new connective tissue attachment
1 to 2 weeks
weeks after therapy the attachment epithelium reappears
—Reduced inflammation leads to decreased clinical signs of inflammation (less redness and swelling)
1-2mm
amount of recession (tissue shrinkage) often apparent after S&P
—transient root sensitivity frequently accompanies healing but can be diminished by good plaque removal
—patients should be warned about the appearance of longer teeth (due to recession) and tooth root sensitivity to maintain trust and motivation.
factors requiring consideration for referral:
extent of disease:
extensive bone loss, even localized, may require specialized reconstructive techniques.
root length:
short roots are more seriously jeopardized by 5 mm of attachment loss than long roots.
hypermobility:
suggests contributing factors; more guarded prognosis
factors considered before referring
extent of disease
root length
generalized / localized deep involvement
hypermobility
difficulty of scaling & root planing