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Grading Pulses (4-point scale)
+4 = bounding
+3 = full, increased
+2 = normal
+1 = diminished, barely palpable
0 = absent
Edema Pitting Scale
0+ No pitting edema
1+ Mild pitting edema (2mm)
2+ Moderate pitting edema (4mm, usually disappears in 10-15 seconds)
3+ Moderately severe pitting edema (6mm, more than 1 min)
4+ Severe pitting edema (8mm, lasts more than 2 mins)
S1 (“lub”)
Ventricular contraction = increased pressure
Tricuspid and Mitral valves close
Systole
S2 (“dub”)
Ventricular relaxation = decreased pressure
Aortic and Pulmonic valves close
Diastole
S3 (“Ken-TUCK-y”)
Extra heart sound resulting from vibration of ventricles in diastole
Increased atrial pressure
CHF, cardiomyopathy, children, young adults
S4 (“TENN-e-see”)
Active LV filling when atrial contraction forces blood into a noncompliant LV
HTN, pulmonary HTN, elderly
Splits
Occurs when tricuspid/mitral valves (S1) or the pulmonic/aortic valves (S2) do not close simultaneously
Young
Snaps/Clicks
Cardiac valves are generally open without noise unless thickened, roughened, or altered because of disease
Valvular stenosis (mitral valve snap)
Semilunar valves (ejection systolic clicks)
Mitral Valve Prolapse (late non-ejection systolic click)
Heart Murmurs
A gentle, blowing, swooshing sound
Turbulent blood flow
Children, adolescents, young athletes
Systolic Murmurs
May indicate heart disease, but often occurs when heart is normal
Diastolic murmurs
Usually indicates heart disease
Causes of Heart Murmurs
Velocity of blood increases (ex: exercise)
Viscosity of blood decreases (ex: anemia)
Structural defects in the valves or unusual openings in the chambers
Grades (Intensity) of Murmurs
Grade I: Very faint
Grade II: Quiet but clearly audible
Grade III: Moderately loud
Grade IV: Loud, may be associated with a thrill
Grade V: Very loud (thrill present)
Grade VI: Loudest (thrill present)
Valvular Heart Disease
Acquired congenital disorder of:
Stenotic valve (does not open completely)
Incompetent valve (does not close completely)
Endocarditis
Infection of endothelial layer of the heart, including cardiac valves
Causes of Endocarditis
Valvular heart disease
Congenital lesions
Direct injury
Previous heart damage
Clinical Findings of Endocarditis
Heart sounds normal during early infection. In late infection, murmur is heard if valve damage occurs
Pericarditis
Inflammation of layers of pericardium and myocardium. Infection of the sac encasing and protecting the heart
Causes of Pericarditis
MI
Cancer
Trauma
Infections
Cardiac surgery
Clinical Findings of Pericarditis
Pericardial friction rub
Angina
Venous Thrombosis
Thrombus (clot) develops within a vein
A DVT could present with pain, edema, erythema, and decreased pulses in the leg
Thrombophlebitis
Inflammation of vein with or without a clot
Aneurysm
Dilation of artery from weakness in arterial wall
Clinical Findings of an Aneurysm
Abdominal aortic aneurysm
Chest pain
Hoarseness or dysphagia from pressure on esophagus
Thrill or Bruit
Abdominal Aortic Dissection (AAA)
Treatment based on >6cm size prior to rupture