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mood
state of emotionality that is longer and more consistent than emotions
hypomania
low-level mania, less intense but still positive
euthymia
natural mood state
dysthymia
low levels of depression, less intense but still negative
mania
the highest level of mood
unipolar mood disorder structure
mood remains at one side of the mood chart (either depression or mania)
bipolar mood disorder structure
mood changes between areas of the mood chart
mixed mood disorder structure
mixed symptoms from all areas of the mood chart
recurrent major depression
at least two MDD episodes separated by at least two months between each episode
persistant depressive disorder
depressed mood for more days than not for the past two years
pure dysthymic syndrome
full criteria for major depressive epidodes have not been met in at least the past two years
persistent major depressive episode
full criteria for major depressive episodes have been met for at least a 2 year period
Premenstrul dysphoric disorder (PMDD)
symptoms are present the week before your period, improve within your period, and go away after your period is over
disruptive mood dysregulation disorder
severe recurrent outbursts that are not developmentally appropriate and out of proportion to an event. Only diagnosed between 6-18 years and symptoms must have been present since 10 years old
manic episode
distinct period of abnormal and persistent elevated, expansive, or irritable mood and increased activity/energy lasting for at least 1 week, most of the day almost every day
hypomanic episode
distinct period of abnormal and persistent elevated, expansive, or irritable mood and increased activity/energy lasting for at least 4 days. No marked impairment, hospitalization, or psychotic features are present
bipolar 1
criteria has been met for at least one manic episode
bipolar 2
criteria has been met for at least 1 hypomanic, and 1 major depressive episode
mixed features
major depressive or manic episode having some symptoms of the opposite polarity (ex: depressive episode with some manic symptoms)
rapid cycling
moving quickly in and out of depressive and manic episodes with at least 4 episodes in a year
mood disorders in youth
expression and presentation is different in this population than in adults
increased irritability and aggression
increased focus on behavioural aspects of presentation
mood disorders in older adults
can be more difficult to diagnose than other populations and no difference in rates between men and women. Contributes to physical and mental decline
genetic factors of mood disorders
rates of development are about 2-3x higher if you have family with this diagnosis
37% heritability
permissive hypothesis
argues that lower serotonin levels allow other neurotransmitter systems to become dysregulated
serotonin
regulates moods and behaviours
dopamine
regulates reward processing and anticipation of rewards
anhedonia
inability to experience pleasure/enjoyment in activities that were once pleasurable. accompanied by a loss of interest or motivation.
norepinephrine
regulates energy, sleep, and stress reactions
endocrine system
excessive cortisol release → depression
assessment for mood disorders
file review
intake interview
clinical interview
objective measures (PHQ-9, DASS-21, BDI, HAM-D)
anti depressant medication
SSRIs
tricyclics
monoamine oxidase inhibitors (MAOIs)
mood stabilizer medications
lithium loften for BPD
electrocompulsive therapy (ECT)
electric shock to brain → mini seizure → increased serotonin → blocked stress hormones → neurogenesis promotion in hippocampus
controversial usage but safer now than was in the past
used for severe mood disorders that have been unresponsive to other treatments
trancranial magnetic stimulation (TMS)
electric coil placed over head to generate a localized electromagnetic pulse rather than a shock
newer and more precise
effective for depression treatment
behavioural activation therapy for depression
encourages people to do more of what matters to them, build motivation → feeling better
CBT or ACT for depression
targets depressive thoughts/behaviours, core beliefs, behavioural goals, exposures are replaced with behavioural activation
interpersonal therapy for depression
works on resolving problems in relationships and developing new relationships
CBT for BPD
psychoeducation, identifying and targeting unhelpful thoughts, coping and stress reduction skills, intervention and prevention of future episodes
interpersonal and social rhythm therapy (IPSRT) for BPD
focus to regulate circadian rhythm
regulating sleeping and eating schedules
maintaining a daily schedule
building stress coping skills
family focused therapy for BPD
psychoeducaiton, identification and response to warning signs, family coping responses, communication and problem solving
psychosis
a mental state characterized by severe impairment or distortion in the experience of reality
delusions
beliefs that are fixed/rigid even though they are not accurate. often misinterpret reality
persecution
belief they will be harmed or harassed by someone
referential
beliefs that gestures, environmental cues, comments, etc. are directed at them specifically
somatic
preoccupation with health, organ functioning, body changes, and the meaning of these
grandiosity
belief they have exceptional abilities, wealth, are invincible, etc.
loss of control of mind/body
believes that thoughts are being removed/inserted into their mind. belief that body actions are being controlled by an outside force
bizzare delusions
completely impossible (ex: their organ has been taken by aliens)
non-bizzare delusions
plausible but very unlikely to be true (ex: they are being stalked by the government/police)
positive symptoms
add something to ones functioning (includes delusions, hallucinations, disorganized thoughts/speech, disorganized behaviours)
hallucinations
sensory experiences without external stimulus
disorganized symptoms
severe disruptions in speech, behaviour, emotions
disorganized thoughts/speech
loose associations between ideas
tangentiality
responses are unrelated to topic
derailment
jumping from topic to topic
cognitive slippage (word salad)
speech is incoherent and words are put together into sentences that do not make sense
innapropriate affect
mismatched affect for the situation (ex: crying and distraught when happy)
disorganized behaviours
unusual and erratic behaviours, difficulty with goal-directed behaviours
catatonia
reduction in motor functioning → random and erratic movements
waxy flexibility
tendency to keep limits in a position placed by someone else
negative symptoms
absence/insufficiency of normal behaviours
abolition
unable to initiate/participate in activities (can be seen by apathy)
anhedonia
lack of pleasure
alogia
relative absence of speech
asociality
severe deficits in social functioning
affective flattening
lack of expected emotional responses
schizophrenia development
premorbid
prodromal
onset/deterioration
chronic/residual
brief psychotic disorder
at least one key feature of psychotic disorders but no negative symptoms are present. episode duration is at least one day but less than one month with a full return to premorbid level of functioning
schizophreniform disorder
duration of at least 1 month and less than 6 months
delusional disorder
1 or more delusions with a duration of at least 1 month, never met criteria for schizophrenia, no significant impairment in functioning
schizoaffective disorder
major mood episode with positive symptoms that present for at least 2 weeks when not in a mood episode
dopamine hypothesis
the early view that overactive dopamine systems are found in schizophrenia
prefrontal cortex
has D1 dopamine receptors. people w schizophrenia have decreased stimulation of D1 receptors → negative symptoms
basal ganglia
has D2 receptors. people w schizophrenia have increased stimulation of D2 receptors
hypofrontality
decreased activity in the frontal lobe
sociogenic hypothesis
shows that people w schizophrenia are often in lower social classes
social selection hypothesis
believes that people w schizophrenia may experience a downward spiral that causes them to drift into lower social classes
conventional neuroleptics
lead to an overall decrease in dopamine, effective to treat positive symptoms but has side effects
psuedoparkinsonism
stooped posture, shuffled gait, rigidity, tremors, etc
akathisia
restlessness, trouble standing still, pacing, rocking feet
acute dystonia
facial grimacing, involuntary upwards eye movement, muscle spasms, laryngeal spasms
tardive dyskinesia
protrusion and rolling of tongue, facial dyskinesia, involuntary body movements
atypical neuroleptics
targets dopamine but less motor control, more effective for negative symptoms, may help to increase cognitive functioning
CBT for schizophrenia
targets delusions and depression increases flexibility in thinking
personality
patterns of thinking, feeling, and acting that characterize a given individual
personality traits
reflect behaviours and remain mostly consistent across settings and situations
dimensional model of personality disorders
personality traits are viewed as a continuum and have a degree of expression
personality pathology
an extreme variation of a certain personality trait
categorical model of personality disorders
personality disorders are defined and discrete with specific symptoms
personality disorder
meeting a specific criteria for diagnosis
five factor model of personality disorders
neuroticism
extraversion
openness to experience
conscientiousness
agreeableness
neuroticism
how prone someone is to experiencing unpleasant emotions
extraversion
how much someone prefers being social to being alone
openness to experience
how curious and receptive to new things someone is
conscientiousness
how much someone displays things like organization, punctuation, achievement, motivation
agreeableness
how much someone gets along with others and wants to cooperate
cluster A personality disorders
odd/eccentric (paranoid, schizoid, schizotypical)
cluster B personality disorders
dramatic, erratic, emotional (antisocial, histrionic, borderline, narcissistic)
cluster C personality disorders
anxious/fearful (avoidant, dependent, obsessive, compulsive)
assessment of personality disorders
file review
clinical review
self-reported measures/questionaires
informant reports (if applicable)