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Eyes
binocular vision by 3 to 7 months
visual acuity development by 5 years of age
Ears
hearing intact at birth
short, wide and horizontally placed eustachian tubes in young children increase risk of middle ear infections
HEENT Common Meds
antibiotics
antihistamines
analgesics
Antibiotics (oral, otic, opthalmic)
actions: treatment of bacterial infections of the eyes and ears
indications: acute otitis media, otitis externa, conjunctivitis
nursing implications: teach families to complete the entire course as prescribed, check for drug allergies prior to administer
Antihistamines
actions: block histamine reaction
indications: allergic conjunctivitis
nursing implications: topical drops used, oral agents usually precribed if allergic rhinitis accompanies the conjunctivitis
Analgesics
actions: pain relief
indications: otitis media, otitis externa, after eye or ear surgery
nursing implications: narcotic analgesics may be necessary in some instances
Any Age - HEENT Signs and Symptoms of Visual Impairment
dull, vacant stare
Infant - HEENT Signs and Symptoms of Visual Impairment
does not fix and follow
does not make eye contact
is unaffected by bright light
does not imitate facial expression
Toddler & Older Child - HEENT Signs and Symptoms of Visual Impairment
rubs, shuts and covers eyes
squints and blinks frequently
holds objects close or sits close to television
bumps into objects
displays head tilt or forward thrust
Otitis Media
etiology: bacterial or viral
pathophysiology: dysfunction of eustachian tubes
diagnostic evaluation: assessing the tympanic membrane
therapeutic management:
pharmacologic: antibiotics
surgical: myringotomy
Hearing Acuity
maybe transient or permanent
unilateral or bilateral
language development is dependent on the ability to hear
types: conductive, sensorineural, mixed
Conductive Hearing Loss
transmission of sound through the ear is disrupted
Sensorineural Hearing Loss
damage to the hair cells in the cochlea or along the auditory pathway
Mixed Hearing Loss
attributed to both conductive or sensorineural problem
Sleep Apnea
upper airway obstruction (partial or complete) that disrupts respirations during sleep
symptoms: snoring, breathing pauses, gasping on arousal, disturbed sleep, secondary enuresis, day time sleepiness, day time behavioral problems
diagnosis: sleep study
treatment: adentonsilectomy or CPAP/BIPAP
nursing care: early detection/observation, education, counseling
Respiratory Disease in Children
accounts of 50% of childhood illness:
upper respiratory tract infections
croup
lower respiratory tract infections
asthma
bronchopulmonary dysplasia
cystic fibrosis
ciliary dyskinesia
General Aspects of Respiratory Infections
etiology and characteristics:
infectious agents
age
size
resistance
seasonal variations
Upper Respiratory Tract
oranasopharynx
pharynx
larynx
upper trachea
Lower Respiratory Tract
lower trachea
bronchi
bronchioles
alveoli
Clinical Manifestations of Respiratory Infections
vary with age
generalized signs and symptoms and local manifestations differ in young children
anorexia, vomiting, diarrhea and abdominal pain
cough, sore throat, nasal blockage or discharge
respiratory sounds
Upper Respiratory Tract Infections
acute viral nasopharyngitis “common cold”
therapeutic management: often managed at home
prevention: widespread in the general population, hand washing, no eye and nose touching
care management: hydration, nasal suctioning
family support: education on respiratory complications and prevention
Influenza
commonly known as the flu
caused by orthomyxoviruses (A, B, C)
clinical manifestations: dry cough, hoarseness, fever, chills
therapeutic management: hydration, antipyretics
prevention: vaccine recommendations
Tonsillitis
frequent co-occurence with pharyngitis
etiology: frequent URIs
clinical manifestations: inflammation
therapeutic management: tonsillectomy and adenoidectomy
frequent swallowing
Infectious Mononucleosis
etiology and pathophysiology: herpes-like epstein-barr virus
diagnostic tests: spot test (mono)
Acute Epiglottitis
medical emergency
clinical manifestations:
sore throat
pain
tripod positioning
agitation
drooling
difficulty swallowing
inspiratory stridor
mild hypoxia
distress
no cough
therapeutic management:
prevention of progressive respiratory obstruction
intubation
tracheostomy
DO NOT VISUALIZE EPIGLOTTIS
Croup Syndromes
characterized by hoarseness, barking cough, inspiratory stridor, and varying degrees of respiratory distress
affects larynx, trachea and bronchi
types: epiglottis, laryngitis, laryngotracheobronchitis, tracheitis
most often caused by H. influenzae type B
Acute Laryngotracheobronchitis (LTB)
most common of the croup syndromes
generally affects children <5 years of age
organisms responsible: RSV, parainfluenza virus, M. pneumoniae, influenza A and B, pneumococci, staphylococci
Manifestations of LTB
usually preceded by an upper respiratory tract infection
inspiratory stridor
suprasternal retractions
barking or seal like cough
increasing respiratory distress and hypoxia
can progress to respiratory acidosis and respiratory failure
Therapeutic Management of LTB
maintaining the airway
maintain hydration (orally or IV)
nebulized mist with supplemental O2
nebulizer treatments: epinephrine or steroids
Acute Spasmodic Laryngitis
aka spasmodic croup
paroxysmal attacks of laryngeal obstruction
occurs chiefly at night
mild or absent inflammation
most commonly affects children 1-3
Therapeutic Management of Acute Spasmodic Laryngitis
humidified oxygen
antipyretics
antibiotics
potential for intubation or mechanical ventilation
Infections of the Lower Airways
lower airways: considered the reactive portion of the lower respiratory tract
include bronchi and bronchioles
cartilaginous support not fully developed until adolescence
constriction of airways
Bronchiolitis
description: most common infectious disease of lower airways, maximum obstructive impact at bronchiolar level
age group: usually children 2-12 months old, rare after 2 years
etiologic agents: viruses (RSV primarily, adenoviruses, para-influenza viruses, human metapneumovirus, and mycoplasma pneumoniae)
predominant characteristics: labored respirations, poor feeding, cough, tachypnea, retractions and flaring nares, increased nasal mucus, wheezing, may have fever
treatment: supplemental oxygen if <90%, bronchodilators, suction nasopharynx, adequate fluid intake, adequate oxygenation
Pneumonias
primary disease or complication
causative agents: bloodstream infection or inhaled organisms
treatments oriented towards support and symptoms
Pertusis (Whooping Cough)
caused by bordetella pertussis
in the US, occurs most in unimmunized children
highest incidence in spring and summer
highly contagious
risk to young infants
Aspiration Pneumonia
risk for child with feeding difficulties
interprofessional care management
prevention of aspiration
feeding techniques, positioning
avoidance of aspiration risks
hydrocarbons, lipids
solvents
talcum powder
Asthma
chronic inflammatory disorder of the airways characterized by recurring symptoms, airway obstruction and bronchial hyper responsiveness
genetic susceptibility plus environmental triggers
inflammation causes an increase in bronchial hyper responsiveness to a variety of stimuli
most common chronic disease of childhood
third leading cause of hospitalization in children under 15y
may occur at any age, but over 80% will be diagnosed by 4y
reactive airway disease: 50% of children <1y will wheeze, only 20% will go on wheeze again in early childhood
Cystic Fibrosis
multisystem involvement
autosomal recessive trait
increased viscosity of mucous gland secretions
elevation of sweat electrolytes
increase in enzymatic constituents of saliva
autonomic nervous system abnormalities
defective gene inherited from both parents with an overall incidence of 1:4
Prognosis of Cystic Fibrosis
medial survival is 40 years
progressive and incurable disease
transplantation: increases life expectancy, availability of organs, surgical complications
CF Pathophysiology
increased viscosity of mucus gland secretion
secretion results in mechanical obstruction
thick inspissated mucoprotein accumulates, dilates, precipitates, and coagulated to form concretions in glands and ducts
CF Diagnostic Evaluations
sweat chloride test
universal newborn screening
DNA identification of mutant genes
CF Presentation
wheezing respiration
dry nonproductive cough
patchy atelectasis
cyanosis
clubbing of fingers and toes
repeated bronchitis and pneumonia
meconium ileus
distal intestinal obstruction syndrome
excretion of undigested food in stool; increased bulk, frothiness and foul odor
wasting of tissues
prolapse of the rectum
delayed puberty in girls
sterility in boys
salty tasting skin
dehydration
hyponatremic
hypochloremic alkalosis
hypoalbuminemia
CF complications
complications are present in almost all patients with CF, but onset/extent is variable
stagnation of mucus and bacterial colonization result in destruction of lung tissue
tenacious secretions are difficult to expectorate: they obstruct bronchi and bronchioles
CF Respiratory Progression
gradual progression follows chronic infection
bronchial epithelium is destroyed
infection spreads to peribronchial tissue weakening bronchial
peribronchial fibrosis ensues
O2/CO2 exchange decreases
chronic hypoxemia: causes contraction/hypertrophy of muscle fibers in pulmonary arteries/arterioles
pulmonary hypertension
cor pulmonale
pneumothorax
hemoptysis
CF Management of Respiratory Complications
airway clearance therapies
bronchodilator medication
physical exercise
aggressive treatment of pulmonary infections
aerosolized antibiotics
home IV antibiotic therapy
CF Management of Gastrointestinal Problems
replacement of pancreatic enzymes
high protein, high calorie diet
relief of intestinal obstruction (ileus)
reduction of rectal prolapse
treatment of chronic gastroinestinal reflux
CF Management of Endocrine Problems
eventually pancreatic fibrosis occurs; diabetes mellitus may result
pancreatic enzyme deficiency
sweat gland dysfunction
failure to thrive
increased weight loss despite increased appetite
Accident
an event independent of human will caused by outside forces acting rapidly & resulting in physical or mental injury
Common Injury Locations
homes
yards
schools
childcare settings
playgrounds
sports fields
Risk of Injury & Developmental Stages
injury rates are low for infants and increase with age
most common in kids 2-5 due to curiosity, walking, climbing and running
attempting activities before developmental readiness
line of sight and speed perception
self assertion
challenges rules
desire for peer approval
Common Childhood Injuries
SIDS
brain injury
concussion
burns
fire
choking
strangulation
suffocation
falls
poisoning
drowning
child passenger safety
suicidality
gun violence
SIDS
death of an infant younger than 1 year that is unexplained after thorough investigation, autopsy and review
90% deaths occur before 6 months, peak between months 1-4
suffocation, asphyxia, entrapment and strangulation also common
Back to Sleep
always sleep on back until 1 year
firm mattress
fitted sheet
CPSC standards
no toys, soft objects, bumper pads, etc.
Anaphylaxis
acute response to an allergen that involves may organ systems and may be life threatening
usually happens 5-10 mins after exposure/ingestion
management: assessment and support of ABC
O2, epinephrine, bronchodilators, fluids, antihistamins, maybe corticosteroids
4-6 hr observation
d/c planning: identify allergens, teach to use epi-pen, epipen prescription, allergy action, plan for school, avoid allergens, recognize signs and symptoms
Signs of Anaphylaxis
airway: SOB, breathing difficulties, can’t swallow
skin: hives, redness, itchy rash, swelling
stomach: cramps, diarrhea, nausea and vomiting
heart: drop in BP, increase HR, faintness, weak pulse
TBI & Concussion
type of brain injury that changes he way the brain usually works
830,000 pediatric patients present with it a year in US
mild is about 75% of reported cases in US
recovery: days to months
concussion is most common type
Head Injury
falls, MVA, child abuse, bike + pedestrian accidents
ABCs, neuro function: LOC, pupil response, seizure activity, gait, speech
history: LOC, prior heath status, nature of injury
diagnostics: Xray of head and neck, CT, MRI
Minor Head Injury
no LOC or penetration changes
pt acting normal
healthy status prior
monitor at home
Severe Head Injury
maintain airway
monitor breathing, circulation and neuro status
prevent/cease seizures
assess LOC
signs of ICP?
administer mannitol as ordered
manage pain and sedation
monitor for the development of complications
hemorrhage
infection
cerebral edema
herniation
S/Sx that Require Follow Up
constant headache
slurred speech
dizziness
extreme irritability
vomiting 2+ times
clumsiness or difficulty walking
oozing blood or watery fluid from nose or ears
difficulty waking up
uneven pupils
unusual paleness
seizures
any s/x of increased ICP
Brain Injury Prevention
proper seatbelts/car seating
helmet use
safety gates
safety guards on windows
Burns
scald/thermal (hot objects)
electrical
chemical
friction
cold/frostbite
Burn Prevenion
hot water heater below 120, check bath water temp
install smoke detectors
barriers around fireplaces/hot pipes/radiators
cover electrical outlets
no kids in cooking areas
no bottles in microwave
Choking
greater risk in young kids
explore via mouth
still learning chewing/eating
s/sx:
difficulty speaking/breathing
inability to cough
wheezing sounds
clutching throat/gesturing
cyanosis
confusion and LOC
Choking/Strangulation Prevention
keep away small objects
be aware of older kids toys
cut foods
caution with specific foods
supervised mealtime
choking and cpr education
review toy age requirements
Poisoning
meds
cosmetics
cleaning products
arts/crafts
batteries
lead containing
automotive products
gardening products
pesticides
certain plants/mushrooms
Poisoning Physical Exam
hyper or hypotension and thermia
respiratory distress or hyperventilation
pupilary dilation or constriction
Poisoning Labs/Diagnostics
chem panel
ECG
LFT
urine/blood toxicology
specific drug levels
Poisoning Nursing Management
ABCs
monitor Vs
STAT labs
activated charcol
polyethylene glycol for bowel irrigation
dialysis for blood removal of toxin
naloxone if opiate or narcotic
Drowning
greatest risk for ages 1-4
even a bucket of water with a few inches is a risk
Submersion Injury
aspiration leads to poor oxygenation with retention of carbon dioxide
alveolar surfactant is depleted leading to pulmonary edema
hypoexemia results
risk of renal complications
Submersion History Work-Up
detailed history of the event
where: pool or ocean
length of submersion
witnessed?
cold or warm water?
extenuating circumstances?
LOC when found?
CPR?
AED?
cervical spine injury?
last meal?
ABG, ECG, CXR, serum electrolytes
ABCs":
scene CPR
cervical stabilization
suction airway
100% O2
intubation?
NG or OG
compressions?
hypothermia?
Abuse Assessment
Ten-4-Faces
trunk
ears
neck
4 years and yoinger
frenulum
auricular area
cheek
eyes
sclera
patterned bruising
Adolescent Safety Tips
quest for independence
peer relationships are important
risk taking
body image
experimentation
mental health
Acquired Heart Disease
disease, condition or characteristic that is not congenital but develops after birth
resulting from exposure to something, such as an antigen or antibiotic, virus, bacteria, post surgical, side effect of med, etc
Infective Endocarditis (IE)
rare but lifethreatening inflammation of the inner lining of the heart chambers and valves
commonly caused by microorganisms, usually bacteria or fungi, entering the bloodstream and settling on heart valves
Risk Factors of Infective Endocarditis
CHD (septum or valve defects)
prosthetic valves
central venous catheters
History and Exam of Infective Endocarditis
prolonged, unexplained fever
lethargy
weight loss
flu-like symptoms
petechiae - conjunctiva, oral mucosa, extremities
Diagnostic and Treatment of Infective Endocarditis
blood culture - bacteria, fungi
CBC - anemia, leucocytosis
urinalysis - microscopic hematuria
echocardiogram - cardiomegaly, abnormal valve function, vegetation
ECG - prolonged P-R
4 to 6 weeks of antibiotic or antifungal treatment
Nursing Management of Infective Endocarditis
good oral hygiene
ID card
notify PCP or cardiologist of fever or flu like symptoms
dental prophylaxis for high-risk patients
Acute Rheumatic Fever
a systemic inflammatory disease that acts as a delayed autoimmune reaction (2-4 weeks) after untreated GAS (strep throat) or skin infections
primarily affects 5-15yo
more common in disadvantaged areas
Clinical Manifestations of Acute Rheumatic Fever
carditis (50-80%) - often most serious
polyarthiritis (60-80%)
sydenham chorea (10-30%)
subcutaneous nodules (<10% of patients)
erythema marginatum (<6% of patients)
Diagnosis of Rheumatic Fever
Jones Criteria + previous group A strep
2 major +
Jones Criteria
major:
joint involvement
o looks like a heart = myocaritis
nodules, subcutaneous
erythema marginatum
sydenham chorea
minor:
c - crp increased
a - arthralgia
f - fever
e - elevated ESR
p - prolonged PR interval
a - anamnesis of rheumatism
l - leukocytes
Rheumatic Fever Treatment
antibiotics
steroids
aspirin
Kawasaki Disease
acute vasculitis - 6 months to 5 years
thought to be post-viral
appears to be autoimmune
attacks the coronary arteries - causes thrombosis
requires chronic management
Symptoms
High fever - for > 5 days (39.9C or 103.8F)
Desquamation (peeling) hands or feet, perineal region
Strawberry tongue
Diffuse, erythematous, polymorphous rash
Bilateral conjunctivitis
Kawasaki Disease
Tx - high dose IVIG, aspirin
Nursing Management:
monitor cardiac
promote comfort
chapped lips
achy joints
irritability
fevers
Child and Family Education:
continue to monitor temp
cardiology f/u
report toxic effects of aspirin therapy
no nsaids while on aspirin
ROM exercises for joint pain
Cardiomyopathy
increasing incidence among children
risk factors include genetic or congenital heart defects
also occurs as a result of inflammatory or infectious process
result of chronic hypertension
no cure
management is directed at improving heart function and blood pressure
ace inhibitors
beta blockers ca channel blockers
pacemakers
heart transplantation
Hypertension
increasing prevalence among children and adolescents
important to screen and treat
can lead to cardiomyopathy
weight reduction, appropriate diet, increased physical activity
some children require medications such as antihypertensives & diuretics
managed by nephrologists
Dyslipidemia
high lipid levels are a risk factor for cardiovascular disease
increasing prevalence in kids and teens
asymptomatic (screening and early intervention, 9-11, 18-21)
high LDL, low HDL, high triglycerides
60 mins physical activity
low fat dairy, whole grains, lean protein
Fetal Circulation (Before Birth)
placenta (oxygenated blood enters via umbilical vein)
ductus venosus (shunts some oxygenated blood from umbilical vein directly to the inferior vena cava, bypassing the liver)
foramen ovale - opening between RA and LA, bypasses the non-functioning fetal lungs
ductus arteriosus - connects pulmonary artery to aorta, shunts most blood away from lungs
umbillical arteries - returns deoxygenated blood from fetus to placenta
high pulmonary vascular resistance - keeps most blood out of lungs
Summary: oxygenated blood from the placenta to preferentially goes to brain and heart, bypassing lungs and liver via shunts
Placenta Purpose
the fetus doesn’t breathe air, so the placenta provides oxygen and nutrients
blood flow bypasses the lungs
Summary Transition at Birth
first breath —> lungs expand —> pulmonary resistance drops —> systemic vascular resistance rises after umbilical cord clamping
Key Changes at Birth
lungs inflate —> pulmonary vessels dilate —> increased blood flow
umbillical cord clamp —> higher systemic resistance
foramen ovale closes functionally
ductus arteriosus constricts, closes within hrs to days, becomes ligamentum arteriosum
ductus venosus closes, becomes ligamentum venosum
umbilical vessels obliterate, become ligaments
Neonatal Circulation
Lungs now oxygenate blood, all fetal shunts close
blood flows through pulmonary cirulation for oxygenation
all shunts closed
Congenital
born with it
defects occur before birth that change the flow of blood through the heart
risk factors:
genetic - family history, Down’s syndrome
maternal - infection, diabetes, drug and alcohol use
Hypoxia - Low O2
commonly seen with R-L shunting
tetralogy of fallot, transposition of the great vessels, truncus arteriosus, tricuspid atresia
S/S:
blue babies - cyanosis
poor feeding
poor weight gain
clubbing fingers
dyspnea
tachypnea
polycythemia (hydrate!)
Congestive Heart Failure
commonly seen with L-R shunting:
ASD
VSD
PSA
AVSD
s/s:
weight gain
pale with cool extremities
periorbital edema
reduction in # of wet diapers
grunting during feeding
diaphoresis
dyspnea
Defects
holes in heart septum
push blood from L to R of heart
overloads lungs with too much fluid