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atresia
a congenital absence or closure of an orifice in the body
common sites of atresia in the GI tract
esophageal
small bowel
duodenal
colonic
esophageal atresia
esophagus fails to develop past some point resulting in discontinuation
symptoms are visible soon after birth
requires immediate surgery to treat
radiographic appearance of esophageal atresia
lack of GI air below the diaphragm
OG tube blocked
what is common with esophageal atresia
tracheoesophageal fistula
most common type of tracheoesophageal fistulas
the lower portion of the esophagus forms a fistula with the trachea
small bowel atresia
congenital discontinuation of a portion of the small bowel
most common in the duodenum
duodenal atresia
dilated stomach and duodenum above obstruction with no other abdominal gas
requires immediate surgery
duodenal atresia is common with
downs syndrome
radiographic sign of duodenal atresia
double-bubble sign
colonic atresia is AKA
imperforated anus
colonic atresia
congenital failure of the development of the anus
usually forms a fistula with the bladder
treated with surgeries
colostomy
then a creation o fa rectum and opening
pyloric stenosis
congenital abnormality of the stomach in which the pyloric canal muscle is greatly thickened narrowing the opening
treated with surgery
pyloromyotomy
cuts muscle to widen the opening
visualized in via ultrasound or UGI
radiographic sign of puloric stenosis
apple core or string sign
hirschsptung’s disease is AKA
congenital megacolon
Hirschsprung’s disease common site
sigmoid colon
hirschsprung’s disease
caused by a lack of nerve cells in the muscle layers of the distal colon that normally trigger contractions
causes no peristalsis
can lead to bacterial infections from not excreting stool
radiographic appearance of Hirschsprung’s disease
abdomen XR
gross dilation
no air/fluid in the distal colon
BE
dilated proximal colon
constriction
gastroesophageal reflux disease (GERD) is AKA
esophagitis
GERD
results from weak cardiac sphincter allowing backflow of gastric acid and contents into the esophagus
diagnosed via UGI
Valsalva maneuver
Trendelenburg
can lead to precancerous tissue development
primary cause of esophageal inflammation
GERD
esophageal strictures
narrowing or tightening of the esophagus
treated with repeated dilations to remain open
Barrett’s esophagus
caused by severe reflux
normal lining of the lower esophagus is destroyed and replaced by abnormal tissue
precancerous
treated with radiofrequency ablation
radiographic appearance of Barrett’s esophagus
granular appearance
reticular mucosal pattern
strictures are common
types of hiatal hernias
sliding
paraesophageal
sliding hiatal hernia
type I
90% of hiatal hernias
schatzki’s ring
paraesophageal hiatal hernia
type II
10% of hiatal hernias
treatment of hiatal hernias
sutures and surgiacal mesh to reinforce after surgery
gastroenteritis is AKA
gastritis
gastroenteritis
thickening of the gastric rugae with superficial erosions
caused by consumption of NSAIDs, alcohol, tobacco, stress, and bacterial
diagnosed via double contrast UGI
treated with anti-biotics, steroids, and acid-blocking meds
peptic/gastric ulcer disease
inflammatory process involving the stomach and duodenum
occurs more frequently in duodenum and lesser curvature
complications with peptic/gastric ulcers
GI bleeds
perforation
outlet obstruction
radiographic appearance of peptic ulcers
halo sign
appears as a persistent collection of barium surrounded by a halo of edema
Crohn’s disease is AKA
regional enteritis
Crohn’s disease is most common in
terminal ileum
Crohn’s disease
1st major chronic inflammatory disease
continued inflammation results in severely narrowed, rigid segment of the small bowel in which the mucosal pattern is lost
no cure
treated with immunosuppressants and steroids
common result of Crohn’s disease
enterocolic fistulas
found in about 50% of patients
ulcerative colitis
2nd major inflammatory disease
inflammatory lesion of the colon
primarily only effects colon
tends to start in rectosigmoid area
appearance of ulcerative colitis on XR
thumb printing of colon
when chronic
progressive loss of haustra
colon appears smooth
ulcerative colitis vs Crohn’s
ulcerative colitis
inner lining
in the colon primarily
Crohn’s
all layers
in all areas of bowel
appendicitis
inflamed vermiform
develops when the neck of the appendix becomes blocked by an appendicolith
RLQ pain, fever, increased WBC count
diagnosed with CT or US
treated with an appendectomy
esophageal varices
dilated veins in the wall of the esophagus caused by increased pressure of the portal venous system
diagnosed with esophagram in supine position with thin barium
treated by banding the engorged veins to prevent further enlargement/rupture
radiographic appearance of esophageal varices
cobblestone appearance