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most common cause of hospitalization in children ages 1-9 yrs
resp conditions
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what needs to be maintained in respiratory disorders?
adequate hydration and nutrition
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child’s upper airway is
shorter and narrower than adults

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more easily occluded when secretions, edema, or foreign bodies enter upper airway

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nose breathers until 3-4 months
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HEENT & respiratory assessment
head

eyes

ears

nose

throat/mouth

work of breathing

cyanosis

clubbing

breath sounds
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retraction locations
supraclavicular

intercostal

suprasternal

substernal

subcoastal
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respiratory distress
severe difficulty in achieving adequate oxygenation despite significant efforts to breathe
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signs of resp distress
restlessness

tachycardia

tachypnea

diaphoresis

cyanosis: late sign
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respiratory failure
body can no longer maintain effective gas exchange

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either o2 demand outweighs o2 supply (hypoxic) or co2 levels rise due to hypoventilation

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**most common cause of cardiopulmonary arrest in children**
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viral: common cold
**cause:** RSV, rhinovirus, parainfluenza, metapneumovirus, COVID, others

**fever:** low

**body aches:** mild

**runny nose:** yes

**cough:** mild to mod

**n&v:** rare

**onset:** gradual

**itchy:** rare

**sore throat:** yes

**headache:** rare
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viral: flu
causes: influ A & influ B virus

fever: yes

body aches: yes

runny nose: sometimes

cough: yes

n&v: sometimes, more common in children

onset: abrupt

itchy: no

sore throat: sometimes

headache: yes
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viral: allergic rhinitis
causes: pollen, dust, mold, animal dander

fever: no

body aches: rare

runny nose: yes

cough: sometimes mild to mod

n&v: no

onset: gradual or abrupt

itchy: yes

sore throat: sometimes

headache: sometimes
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viral: COVID
causes: Sar-CoV-2 virus

fever: yes

body aches: yes

runny nose: sometimes

cough: yes

n&v: sometimes

onset: gradual or abrupt

itchy: no

sore throat: yes

headache: sometimes
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influenza
\
droplet precautions

mild, mod, severe
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flu signs
onset of fever, chills

dry cough

dry throat & nasal mucosa

flushed face

myalgia

fatigue
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flu labs
nasal swab for rapid flu
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flu meds
flu vaccine (>6months)

oseltamivir

* start w/i 48 hrs of symp
* PO for 5 days >1 yrs)

**NO ASPIRIN**
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reye’s syndrome
rare but serious

unknown cause

can occur when recovering from viral inf. if taking aspirin

affects all organs, esp. brain and liver

causes inc ICP & fatty liver
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reye’s signs
rapid breathing

vomiting

severe fatigue

confusion

seizures

LOC
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reye’s treatment/prognosis
NO CURE

early diag to protect brain damage by dec ICP

\
prognosis:

depends on severity of swelling
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bacterial pharyngitis: strep throat causes and signs
causes: group a strep

signs:

* abrupt onset of pharyngitis, headache, fever, abdominal pain
* tonsils & pharynx can be inflamed & covered with exudate (day 2 of illness)
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bacterial pharyngitis: strep throat labs/nursing interventions
labs:

* rapid antigen testing & throat culture

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nursing interventions:

* antibiotics (PO penicillin as least 10 days OR penicillin shot)
* treat pain
* encourage hydration
* return to school 12 hrs after 1st dose IF FEVER GONE
* new toothbrush after 3 days on antibiotics
* wash pillowcase
* follow up if not improved after 2-3 days
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viral pharyngitis: mononucleosis causes/manifestations
cause: epstein-barr virus spread thru oral secretions

aka kissing disease
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viral pharyngitis: mononucleosis manifestations/nursing interventions
manifestations:

* fever
* pharyngitis
* lymphadenopathy
* extreme fatigue

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nursing interventions:

* pain control
* hydration
* education: acute phase last up to 2 wks with prolonged symp. lasting several wks
* no contact sports
* can lead to enlarged spleen or rupture

\
break out with rash treat with amoxicillin
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tonsillectomy nursing considerations
**positioning**

* place in postion to facilitate drainage
* EHOB when awake

**assess for bleeding**

* most common at 1 wk after surg.
* tonsils: highly vascular (protect against inf.)
* evid. of bleeding
* frequent swallowing
* clearing of throat
* restlessness
* tachycardia
* pallor
* monitor for difficulty breathing related to oral secretions, edema, bleeding

**pharm and non-pharm interventions for pain**

* liquid analgesics or tetracaine lollipops
* admin. pain meds on regular schedule

**clear diet (NO REDS, NO ACID)**

* wait until gag-reflex returns
* advance diet as tolerated

**protect surg. site**

* limit activity
* 14 days to recover
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ear infection: otitis media
inner ear infection

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infl. middle ear and middle ear effusion (fluid)

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**with acute infection**: fluid in middle ear is purulent causing tympanic membrane to appear erythematous, bulging, yellow

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usually accompanied by UTI
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ear infection: otitis media nursing interventions
nursing interventions

* monitor airway
* antibiotic (azithromycin)
* o2 support
* feeding & hydration support
* report to health department
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ear infection: otitis externa
inflammation of the external ear canal

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symp: ear pain, itchiness in the ear canal, discharge of liquid for ear

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itchy, redness

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ear drops antibiotics

inflam. steroids are added with ear drops
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recurrent otitis media
3 epi in 6 months OR 4 epi in 1 yr

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tympanostomy can dec freq., severity, duration

* hole in ear, drain pressure and fluid out, and prevent recurrent
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pertiusis
aka whooping cough

highly contagious: droplet

\
cause: bordetella pertussis

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paroxymal cough

* start 2 wks after onset
* last several months
* difficult breathing during coughing fit
* cause post-tussive emesis
* cough fit last several minutes

\
most at risk:
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ear infection: otitis media s&s/meds/compli.
s&s: tugging ear

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need antibiotics with: **PO**

* severe pain
* bilateral OM in children
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first DTaP
2 months
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viral URI
* No antibiotics


* Cold, flu, COVID
* hydration loosens secretions and help get them out
* no cough suppressant in children
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croup
Inflammation of the epiglottis, larynx, trachea, and possibly even the bronchi

\
bc of inflammatory nature of croup & small diameter of child’s airway, croup can be life threatening, especially in younger children

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Classified as either viral or bacterial

* viral: spasmodic laryngitis or laryngotracheobronchitis
* bacterial: epiglottitis and bacterial tracheitis
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Laryngotracheobronchitis
**barky cough with this one but not others**

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usually viral (parainfluenza)

Most common between ages 3 months & 8 years

\
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Laryngotracheobronchitis signs
* low grade fever
* restlessness
* hoarseness
* barky cough
* dyspnea
* inspiratory stridor
* retractions
* infants and toddlers: nasal flaring, intercostal retractions, tachypnea, continuous stridor
* can have stridor at rest & respiratory distress present in severe cases
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Laryngotracheobronchitis treatment
* Mild: oral steroid – usually dexamethasone
* Severe: systemic corticosteroid & nebulized racemic epinephrine
* oxygen if needed
* systemic: methylpred, but normally just dex: epi helps decrease inflammation in airway (vasoconstrictor)

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if a child has stridor and suddenly stop hearing it = bad sign that airway has potentially completely closed
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Epiglottitis
a hinged piece of cartilage found at back of tongue

* Job: close entry to trachea when swallowing 

\
Epiglottitis : spread by infectious droplets

Most common cause: haemophilus influenza & strep pneumoniae

\
Most preventable due to Hib vaccines

Is a MEDICAL EMERGENCY

Rapid onset, usually a few hours

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if this is swollen: have trouble breathing air in and swallowing

^^BIG THING IS DROOLING - NOT ABLE TO SWALLOW SECRETIONS, tripod position^^ 

muffled voice due to proximity of vocal cords
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epiglottis findings/signs
\*\*do not try to assess throat\*\*

Findings: ADD AIR NURSE

* A: abnormal position (tripod)
* D: dysphagia
* D: difficulty speaking
* A: apprehension
* I: increased temp – high fever
* R: rapid onset
* N: nasal flaring 
* U: using accessory muscles
* R: retractions
* S: stridor
* E: enlarged epiglottis – visible on x-ray or on exam
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epiglottis nursing interventions
never leave alone!!

* protect airway
* prepare for intubation
* never anything in their mouth – no oral temps, no tongue depressors, no throat cultures
* do not leave patient alone
* keep child calm
* NPO
* humidified oxygen
* IV corticosteroids & fluids (in OR)
* antibiotics & antipyretics
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tracheitis
no drooling, will hear stridor, will have sudden onset and acute decompensation

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bacterial= secondary infection of upper trachea

* usually caused by staph aureus, staph pneumoniae, pseudomonas

viral laryngotracheitis leads to bacterial tracheitis

* most common viral causes: influenza a & b

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* rare, only about 2% of cases
* bacteria invade tracheal mucosa & cause inflammatory response

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* kids usually appear ill/toxic
* if untreated can lead to airway obstruction & respiratory arrest
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tracheitis symp
usually have URI symptoms the week before then acute decompensation

* change in voice, stridor


* usually sudden decompensation with noisy breathing

high fevers

* drooling= absent, able to swallow secretions unlike epiglottitis
* thick purulent secretions
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tracheitis treatment
need for intubation = common, should be done in OR

* if less severe, oxygen, racemic epi
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lower: pneumonia
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Inflammation or infection of the lower airways (bronchioles and alveolar spaces)

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* Viral: both lungs, starts with cold symptoms
* CAP usually caused by RSV, parainfluenza, influenza
* Bacterial: usually unilateral, usually a bit sicker
* Bacterial pneumonia= more common in those over age 5
* PCV vaccine
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lower: pneumonia risk factors
Risk factors:

* children exposed to cigarette or wood stove smoke
* chronic conditions such as asthma, CF, sickle cell disease, congenital heart disease or immunodeficiency
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lower: pneumonia signs
signs:

* fever
* tachypnea
* cough
* poor oral intake
* vomiting
* abdominal pain
* cough
* may see retractions
* nasal flaring
* chest pain
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lower: pneumonia diag/treatment
Diagnostic: chest x-ray

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Treatment: depends on cause

* anyone with tachypnea and nothing else will do CXR → tachypnea in kids with nothing else is often pneumonia
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lower: bronchiolitis
Viral infection, affects bronchi & bronchioles

* Bronchial mucosa swells & fills with mucus & exudate

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occurs at the bronchiolar level

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primarily caused by Respiratory Syncytial Virus (RSV), but also can be caused by adenoviruses and parainfluenza

* The younger the infant = more severe
* Day 2= more severe symptoms, respiratory distress
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lower: bronchiolitis assessment
__Initially__: rhinorrhea, intermittent fevers, pharyngitis, coughing, sneezing, wheezing, possible ear or eye infection

__With illness progression__: increased coughing and sneezing, fever, tachypnea and retractions, refusal to nurse or bottle feed, copious secretions

__Severe illness__: tachypnea (>70 resp/min), listlessness, apneic spells, poor air exchange, poor breath sounds, cyanosis 
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lower: bronchiolitis labs/interventions/treatment
Lab testing: nasal swab, can test for different types of respiratory viruses

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Nursing Care:

* supplemental oxygen to maintain oxygen saturation >90% (over 4 = high flow)
* encourage fluid intake if able to tolerate, if not IV fluids until acute phase passed
* suction nasopharynx as needed

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Treatment:

* Supplemental oxygen (anything over 4L= high flow for peds)
* ^^Usually no steroids^^
* **Ribavirin**: inhaled antiviral medication for those hospitalized
* fluid hydration
* fever meds: tylenol/moltrin
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bronchitis
inflammation of the trachea, bronchi, and bronchioles

* Usually occurs with viral respiratory tract infection
* Characterized by cough due to inflammation without evidence of pneumonia
* Inflammatory response in mucus membranes of bronchial passages produces hacking cough & thick phlegm
* Rarefy ever bacterial; always viral
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bronchitis symp
Classic symptoms:

* coarse, hacking cough that worsens at night
* chest pain may develop due to cough

\

May have vomiting at night due to swallowing of sputum

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^^No cough meds, honey can be given in children over 1 year of age^^
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asthma
* Chronic lung disease causing narrowing and inflammation of airways
* One of leading causes of hospitalizations in children
* Issue = AIR TRAPPING
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asthma risk factors
* family hx of asthma
* family hx of allergies
* exposure to smoke
* low birth weight
* being overweight
* pollution
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asthma classifications
* Intermittent
* mild persistent
* moderate persistent
* severe persistent
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asthma exam findings
* Dyspnea
* Cough
* audible wheezing
* mucus production
* use of accessory muscles
* sweating, anxiety
* decreased oxygen saturation
* tripod positioning
* inaudible breath sounds or crackles
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asthma diag
pulmonary function tests: most accurate test for diagnosing asthma & severity 

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peak flow meter (done every 1-2 years, blow twice and take higher of two)

* Blow into device to measure amount of air coming out
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asthma treatment
* Inflammation: Corticosteroids
* Methylprednisolone or Dexamethasone IV
* Prednisone PO
* fluticasone
* Bronchospasm: Beta 2 Agonists
* Albuterol (nebulizer or inhaler)
* Increased Mucus Production: Hydration/ mobilization
* Exaggerated Immune Response: Leukotriene Inhibitors
* Montelukast (Singulair)
* Oxygen as needed

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ER/PICU:

* Theophylline (high toxicity risk)
* Magnesium sulfate (potent vasodilator)
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albuterol
short acting

emergent only
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steroids
increases risk of infections

long acting

for severe
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asthma assessment/education
* Assess airway patency, respiratory rate, symmetry effort and use of accessory muscles
* position child to maximize ventilation
* Education
* Trigger Identification
* Use of medications at home
* Steroids
* Aerosol Medications (Beta Agonists)
* Correct use of spacer
* Peak Flow Meters
* Home Management Plan
* Asthma action plan
* Treatments depending on zone
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status asthmaticus
* Temporarily Irreversible Bronchospasm
* Life-threatening episode of airway obstruction, often unresponsive to common treatment
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status asthmaticus signs
* wheezing


* labored breathing
* nasal flaring
* lack of air movement
* use of accessory muscles
* distended neck veins
* tachycardia
* tachypnea
* hypoxia
* diaphoresis

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\*risk for cardiac & respiratory arrest
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status asthmaticus nursing actions
* Monitor oxygen saturation & cardiorespiratory monitor continuously
* Nebulizer X 3 or continuous, add ipratropium to increase bronchodilation
* IV access
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cystic fibrosis
* Genetic disorder that causes exocrine glands to work incorrectly
* Basic Flaw in Chloride Exchange across cell membrane causing exocrine
* Genetics: 1 in 4 chance of transmission if both parents are carriers
* Autosomal recessive (must get gene from each parent)
* Discovered on prenatal screen or newborn screen, or sweat chloride test (
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cystic fibrosis diag.
Pulmonary function testing

Sweat chloride test: measures amt of salt in sweat

* done on arms with gauze


* 39 mmol/L= negative
* 40-49 mmol/L= needs further testing
* 60 mmol/L= positive

\
Meaning? Poor sodium exchange = thick mucus
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cystic fibrosis expected findings
Respiratory:

* Stasis of mucus increases risk for infection
* Early manifestations: wheezing, rhonchi, dry, non-productive cough
* Increased involvement: dyspnea, paroxysmal cough, obstructive emphysema and atelectasis on chest x-ray
* Advanced involvement: cyanosis, barrel-shaped chest, clubbing of fingers and toes, multiple episodes of bronchitis or bronchopneumonia

Gastrointestinal:

* Large, frothy, bulky, greasy, foul-smelling stools
* Failure to gain weight or weight loss
* Delayed growth patterns
* Distended abdomen & thin arms and legs (infant)
* Reflux
* Prolapse of rectum (infant & child)

Integumentary:

* Sweat, tears, and saliva have excessively high content of sodium & chloride

Endocrine & Reproductive:

* Viscous cervical mucus, decreased or absent sperm, decreased insulin production
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cystic fibrosis compl.
Respiratory

* Infections, colonization, bronchial cysts, emphysema, pneumothorax, nasal polyps

Gastrointestinal

* Meconium ileus, prolapse of rectum, intestinal obstruction, GERD

Endocrine

* Diabetes Mellitus

Worrisome Infections

* Pseudomonas Aeruginosa
* Burkholderia Cepacia
* Staph Aureus
* Hemophilus influenza
* Escherichia coli
* Klebsiella Pneumoniae
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cystic fibrosis interventions
* Nutrition: need well balanced diet high in protein and calories
* Formula supplements in addition to breast feeding or through NG tube
* Add salt to food during hot weather / excess sweating
* Should receive regular nutrition evaluations with nutritionist
* Daily supplements of fat soluble vitamins (ADEK)
* Enzymes before meals (within 30 minutes)
* Encourage fluid intake
* Laxatives or stool softeners for constipation
* Exercise
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respiratory treatments
* HFCC Vest
* Flutter Device
* “Huffing”
* Aerosol therapy
* Postural drainage
* Need aggressive antibiotic therapy - High dose antibiotics:, IV or inhaled
* Dornase Alfa (pulmozyme/recombinant human deoxyribonuclease)
* Can cause laryngitis (administer 1-2 times a day)
* Oxygen: as needed but monitor for carbon dioxide retention
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resp assessments
* Lung sounds & respiratory status 
* Growth and development


* Obtain IV access – may have peripherally inserted central line or IV port if getting at home antibiotics
* Sputum for culture & sensitivity
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resp education
* Stay up to date on vaccines including annual influenza and pneumonia vaccine
* Perform regular physical activity
* Palliative care for those in terminal stages
* May need transplant of heart, lungs, pancreas and liver for adolescents who have advanced disease
* Provide support to family and patient
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acute non-infectious resp disorders
* foreign object asp
* pneumothorax
* congenital abnormalities of upper airway
* diaphragmatic hernia
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foreign body asp
* Need to determine if cause of stridor 
* If foreign body, need to determine what it is 
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pneumothorax
accumulation of air in the pleural space; air accumulates between visceral and parietal pleura increases intrapleural pressure making it difficult to expand affected lung

\
* Peak incidence= 16 – 24 y/o
* Will have chest tube inserted
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pneumothorax types
spontaneous, traumatic, tension

* primary spontaneous pneumo occurs in children with no hx of lung disease
* secondary occurs as complication of chronic lung disease such as Cystic fibrosis or asthma
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pneumothorax assessment
* Dyspnea, tachypnea
* Tachycardia
* Diminished breath sounds
* Unequal chest expansion
* Chest pain
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congenital abnormalities of upper airway
* **Laryngomalacia**: larynx = floppy
* Most common cause of stridor in newborns
* Not-life threatening, most outgrow by 18-20 months
* **Tracheomalacia**: excessive collapsibility of the trachea
* Depends on severity, may resolve on own or cause persistent resp distress
* **Tracheoesophageal fistula**: abnormal connection between these two tubes
* As a result, swallowed liquids or food can be aspirated into lungs
* Symptoms: difficulty breathing while feeding, coughing or choking while feeding, rounded abdomen, failure to gain weight
* **Subglottic stenosis:** narrowing of the airway below the vocal cords (subglottis) and above the trachea
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Diaphragmatic Hernia
* Hole or opening in the baby’s diaphragm, the muscle that separates the abdomen from the chest
* Defect allows abdominal organs to move into chest cavity
* Causes crowding of lungs & improper development
* Breathing problems at birth
* Rare