1/275
Looks like no tags are added yet.
Name | Mastery | Learn | Test | Matching | Spaced | Call with Kai |
|---|
No analytics yet
Send a link to your students to track their progress
urinary incontinence
-involuntary loss of urine severe enough to cause social or hygienic problems
-causes aren’t part of urinary tract
-NOT a normal part of aging
-NOT a normal post-partum effect
stress incontinence
-inability to retain urine
-common in younger women (preg/birth)
-laughs, coughs, sneezes (STRESS)
-can’t close urethra NATURALLY
urge incontinence
-loss of urine for no reason
-no cues until they NEED to go
-older women (may be d/t nerves)
-no feeling of bladder “fullness”
overflow incontinence
-bladder reaches max capacity causing urine leakage
-d/t bladder muscle NOT CONTRACTING
-does not allow bladder to empty
-trickles out to avoid bursting
functional incontinence
-result of other factors
-don’t REMEMBER/FEEL (dementia, spinal)
-PT has disease/injury not related to urinary system
etiology and genetic risk for urinary incontinence
TEMPORARY: constipation, urinary stones, chemo/radiation, drugs
PERMANENT: spinal cord injury, brain/neuro disorder, surgery
incidence and prevalence
-as many as 60% of women over 65 report this condition (shorter urethra, birth)
risk for urinary incontinence increases with…
-chronic conditions
-vaginal deliveries
-pelvic prolapse
-prostate problems
-diabetes
-heart failure (excess fluids, diuretics)
-obesity
history for urinary incontinence
-often unreported (embarrassing)
-use effective screening methods
physical assessment for urinary incontinence
-assess abdomen (putting pressure/distention, bowel sounds/impaction, constipation=PRESSURE)
-inspect females for prolapse (weakened pelvic floor muscles)
-evaluate force and character of urine stream (trickle vs. strong stream, can/can’t stop stream)
-HCP will perform Digital Rectal Exam (feel for enlarged prostate)
psychosocial assessment for urinary incontinence
embarrassed/ashamed
lab assessment for urinary incontinence
-urinalysis (clean catch)
WBC: UTI
BLOOD: stones, UTI, trauma, cancer
GLUCOSE: DM
PROTEIN: kidney damage
bladder scan for urinary incontinence
-shows how much urine is in bladder
POST-VOID RESIDUAL: PT goes pee, scan for how much is left
voiding cystourethrogram (VCUG)
x-ray of urinary system, looks for urinary BACKFLOW
electromyography (EMG) of pelvic muscles
study of muscles in pelvis (can they contract/relax)
CT of kidneys and ureters
structural issues
nonsurgical management for urinary incontinence
NUTRITION: avoid taking in fluid before bed, going in public, etc (STAY HYDRATED)
DRUG THERAPY:
-anticholinergics: oxybutynin (decrease involuntary contractions, stops spasms, FOR URGE)
-beta-adrenergic agonist: mirabegron (urge incontinence, relaxes muscles, fills bladder all the way)
nonsurgical management for urinary incontinence (devices)
-penile clamps (blocks urine flow)
-pessary devices (inserted UP vag, holds things in place, in for 1mo, wash, put it back)
-vaginal cone therapy (weights, helps with pelvic floor muscle, 15mins 2x/day)
-external catheters (condom catheter, purewick)
behavioral interventions for urinary incontinence
-pelvic muscle therapy (which muscles to contract/relax)
-behavioral interventions (setting timers Q30mins, 45, etc)
surgical management (urinary incontinence)
-sling/suspension: mesh tape around urethra, pulls things UP (good for stress incontinence)
PREOP: educate
POSTOP: catheter @ home for 24hrs, take out
-management of urinary catheter (preventing CAUTI’s)
maintaining tissue integrity
-risk for incontinence associated dermatitis
-avoid feminine pads and depends (hold liquid on skin)
-prefer CHUCK pads
-bed-bound PT’s → supra-pubic catheter
home-care management for urinary incontinence
-no barriers to BR (stairs) → give urinals, commodes, etc
self-management education for urinary incontinence
-ID type of incontinence (stres: PF therapy, etc), diet changes
national association of continence
increase quality of life
american urological association
provide resources monetary resources
cystitis
-inflammatory condition of the bladder (NOT A UTI)
-usually refers to inflammation from infection
-irritants can cause cystitis without infection (chemo, radiation, feminine hygiene spray)
urinary tract infections (UTI’s)
-an infection that occur in any area of the urinary tract and the kidneys
acute UTI
bacteria in urinary tract
recurrent UTI
>2 infections in 6mo
>3 infections in 1yr
uncomplicated UTI
-no functional/anatomical
-not peeing post-sex, wiping wrong
complicated UTI
DM, pregnancy, obstructions
etiology and genetic risk for UTI
-E. coli/candida are the most causative organisms
-infectious vs. non-infectious cystitis (chemical exposure)
-catheters are most common factor associated with new onset UTI in hospital and long-term care (CAUTI)→ clean Q8H, hang below bladder
-interstitial cystitis (separate dx: chronic inflammation/autoimmune, of lower urinary tract)
incidence and prevalence for UTI
-UTI is a leading cause of primary care visits
-5th most common health-care associated infection
health promotion and maintenance for UTI
-sterile technique when inserting catheters
-clean technique when using intermittent catheters at home (single-use catheter recommended for home settings)
-National Patient Safety Goal (NPSG)- CAUTI prevention/HOUDINI
-increase fluid intake (dehydration leads to infection)
HOUDINI
H- hematuria
O- obstruction
U- urinary surgery
D- decubitus ulcer
I- intake/output
N- nursing end of life care
I- immobility
physical assessment for UTI
-assess catheter, VS, lower abdomen, bladder palpation
hallmark symptoms for UTI
dysuria, frequency, and urgency
other symptoms for UTI
fever, chills, nausea (older adults: CONFUSION)
lab assessment for UTI
-clean catch urine specimen
-urinalysis
-culture and sensitivity (before ABX, tells us what type of ABX to use)
-CBC/WBC (SEPSIS)
other diagnostic assessment for UTI
-pelvic ultrasound (obstruction, stones)
-voiding cystourethrography (fill bladder with fluid, watch as bladder empties/structural issues)
-cystoscopy (going in & looking @ bladder/ureters, can place a stent if necessary
drug therapy for cystitis
-fluconazole (fungal infections)
-nitrofurantoin/macrobid, trimethoprim/sulfamethoxazole/bactrim (dual fxn ABX, 7-14days)
-fluid intake
-comfort measures
-sitz baths 2-3x/day for 20 mins
surgical management of cystitis
-treats condition that increases risk for recurrent UTI’s
example: cystoscopy (placing stent, cauterize lesions, etc)
home-care MGMT for cystitis
finish ABX, front-back wiping, pee before/after sex
self-MGMT education
how to take drugs, urine assessments, etc
urolithiasis
-presence of calculi (stones) in urinary tract
-nephrolithiasis: kidney calculi
-ureterolithiasis: ureter calculi
-most common associated condition is DEHYDRATION
(no symptoms until in lower urinary tract)
etiology and genetic risk
-metabolic risk factors (dehydration)
-family history, obesity, diabetes, gout increase risk
-diet (increased sodium)
-higher risk for men (d/t diet/protein intake)
incidence and prevalence for urolithiasis
-varies with geographic location, race, family hx
-about 19% of males and 9% of females will have at least one episode
-increase risk for recurrence if no changes are made
history for urolithiasis
-family or personal hx
-diet hx
physical assessment for urolithiasis
-severe, sudden unbearable pain (renal colic→ urine backup, N/V, light-headed, high risk for infection)
-urinary obstruction=MEDICAL EMERGENCY, can impair urinary function
-oliguria/anuria
lab assessment for urolithiasis
-urinalysis
-CBC/WBC
imaging assessment for urolithiasis
CT of the abdomen/pelvis (will show stone/s)
non-surgical MGMT for urolithiasis
-strain urine
-lithotripsy (laser/sound waves, breaks apart stones for passing)
drug therapy for urolithiasis
-used in the FIRST 24-36 HRS WHEN PAIN IS MOST SEVERE
-NSAID’s
-opioids (SEVERE PAIN)
-spasmolytic (oxybutinin)
MINIMAL surgical MGMT for urolithasis
-ureterscopy (small tube and stent in ureter, repeat stones)
-percutaneous ureterlithotomy or percutaneous nephrolithotomy
POST-OP: monitor for bleeding, nephrostomy tube MGMT
open procedures for urolithiasis
-open ureterolithotomy/pyelolithotomy/nephrolithotomy
POST-OP: monitor bleeding, fluid intake
preventing infection (urolithiasis)
-less red meats (gout/purines)
-increase fluids (3L/day), I/O’s, diuretics (less Na)
preventing obstruction (urolithiasis)
#1 OPTION: DRINK LOTS OF FLUID!!!
-fluid intake
-drug therapy (ABX)
-ambulation
benign prostatic hyperplasia (BPH)
-glandular units in the prostate undergo nodular tissue hyperplasia
-can be benign or cancerous
-prostate grows larger as men age
-bladder outlet obstruction affects elimination CAUSING urinary retention, leakage, overflow incontinence, urinary backup
-hydroureter and hydronephrosis
BPH facts
-occurs in 50% of men between 51-60yrs old
-occurs in 80% of men >70yrs old
-EXACT CAUSE IS UNCLEAR
unmodifiable risk factors for BPH
-race, genetic susceptibility, family cancer hx
-get screened at approximate ages
modifiable risk factors for BPH
-obesity/metabolic syndrome, beverage consumption, physical activity
-caffeine, alcohol
history for BPH
-international prostate symptom score (I-PSS)
-elimination patterns
-hematuria
physical assessment for BPH
-digital rectal exam (DRE)
-completed by HCP
lab assessments for BPH
-prostate specific antigen (PSA), GOLD STANDARD DX, presents in blood (should NOT be in there)
-CBC (sepsis)
-BUN/creatinine (urinary backup)
-urinalysis and culture
psychosocial assessment for BPH
-frustration/depression
-embarrassment (dribbling/incontinence)
-libido affected
dx assessment for BPH
-transrectal ultrasound (US probe, view prostate through back)
-cystoscopy
-bladder ultrasound
improving urinary elimination (NON-SURGICAL)
-behavioral modification (decrease alcohol consumption)
-drug therapy (can have erectile effects)
-prostate artery embolization (decreased blood flow to prostate to keep it from getting bigger)
drugs for BPH
-alpha 1 andrenergic antagonists- TAMSULOSIN (flomax, relax smooth muscle in bladder)
-5-alpha-reductase inhibitors- FINASTERIDE (decrease size of prostate
MOSTLY GIVEN A COMBO DRUG!!
improving urinary elimination (surgical management)
-holmium laser enucleation of prostate (HoLEP)
-transurethral resection of the prostate (TURP)
-transurethral incision of the prostate (TUIP)
holmium laser enucleation of prostate (HoLEP)
scope in urethra and remove tissue that’s blocking flow
transurethral resection of the prostate (TURP)
-most common
-remove extra prostate tissue
POST-OP: continuous bladder irrigation (urine will be BRIGHT RED)
transurethral incision of the prostate (TUIP)
small cuts in prostate to decrease pressure on the urethra
home care MGMT for BPH
-possible urinary catheter at discharge
-increase fluid intake (2-2.5L/day)
self-MGMT for BPH
-TURP may cause temporary dribbling (let everything heal, symptoms should resolve)
-kegel exercises
healthcare resources for BPH
follow up with HCP as recommended
pyelonephritis
-bacterial infection→ starts in bladder and moves upward to infect kidneys
-acute (stones) vs. chronic (obstruction from injury, cancer)
-abscesses may develop (may need surgery)
common causes of pyelonephritis
-E. Coli
-enterococcus faecalis
-BACKFLOW OF URINE
process of pyelonephritis
-microbial invasion of renal pelvis
-inflammatory response
-resulting fibrosis (scar tissue)
-decreased tubular resorption and secretion
-impaired kidney function
history for pyelonephritis
ask about prior conditions (pregnancy: treatment is different)
physical assessment for pyelonephritis
ACUTE: flank pain, costovertebral angle tenderness (between ribs and vertebrae)
CHRONIC: less dramatic presentation, but similar symptoms (body gets used to it, kidneys are scarred)
psychosocial assessment for pyelonephritis
embarrassment and shame
lab assessment for pyelonephritis
-urinalysis
-blood cultures
-WBC/ESR/CRP (immune and inflammatory response)
-BUN/Cr/GFR (kidney function)
imaging assessment for pyelonephritis
-KUB (kidneys, ureter, bladder scan)
-CT (shows any defects)
other dx assessments for pyelonephritis
-antibody-coated bacteria in urine
-radionuclide renal scan (put dye inside kidneys, shows how long it takes for them to filter)
non-surgical MGMT for pyelonephritis
-drug therapy-acetaminophen preferred (NSAIDs are nephrotoxic!!)
-catheter replacement (treat infection, no new catheter for around 2-3wks)
-nutrition therapy (adequate fluid)
surgical MGMT for pyelonephritis
-pyelolithotomy (removing stone/s)
-ureteroplasty (backup of urine, repair ureters)
-nephrectomy (kidney REMOVAL, last resort!!)
preventing chronic kidney disease (pyeloneph)
-ABX therapy (specific to condition/bacteria)
-BP control (HTN can damage BV’s)
-hydration
self-MGMT for pyelonephritis
post-op, nutrition, catheter care, symptoms of recurrence
healthcare resources for pyelonephritis
home-health nurse available
hydronephrosis and hydroureter
-problems of urine elimination with outflow obstruction
-hydronephrosis (kidneys enlarge)
-hydroureter (ureter enlarges)
-COMMON CAUSES: stones, tumors, etc)
-can cause permanent damage to kidneys
history for pyelonephritis
-kidney disorders
-urological disorders
physical assessment for pyelonephritis
-flank/abdominal pain, chills, fever, malaise
-inspect flanks
-palpate abdomen
-bladder scan
diagnostic testing for pyelonephritis
-urinalysis
-BUN/creatinine
-US/CT of the abdomen
urologic solutions for pyelonephritis
-cystoscopy
-retrograde urogram (dye in kidneys)
radiologic solutions for pyelonephritis
-nephrostomy (percutaneous/via incision)
-post-operative (educate, monitoring, etc)
acute kidney injury
rapid reduction in kidney function resulting in:
-failure to maintain waste elimination
-fluid and electrolyte imbalance (can’t excrete fluids)
-acid-base imbalance (poor perfusion d/t heart disease, HTN, drugs, etc)
acute kidney injury is defined by: MUST MEET 1 of 3
-increase in serum creatinine by ±0.3mg/dL or more within 24hrs (easiest, most commonly used, takes a long time)
-increase in serum creatinine 1.5x or more than baseline
-urine output less than 0.5mL/kg/hr for 6hrs (straight)
AKI risk factors
-older adults (some can bounce back to baseline)
-adults with chronic illnesses (HTN/DM→ microvascular damage)
-20% of hospitalized patients and 60% of patients in ICU develop AKI
-decreased perfusion (shock, hypotension, sepsis)
-damage to kidney tissue (pyelonephritis, nephrotoxic drugs)
-obstruction of urine outflow (BPH, UTI)
prerenal AKI
-caused by source outside of kidney
-perfusion issue, HF, HTN, shock)
intrarenal AKI
-occurs inside kidney
-HTN EMERGENCY, glomerular disease/nephritis, nephrotoxic drugs)