homeostasis
the tendency for the human body to keep all systems at a default level
heterostasis
idea that different parts of the body can be maintained deliberately out of sync with each other
body desires to "normalize as quick as possible"
regular exercise
body learns to maintain the normal function values longer in balance and restore function more quickly
adaptation
the body learns to cope with the repeated demands through functional and morphological adaptations
aerobic training adaptations
increased:
cardiac output blood volume hematocrit heart volume blood flow to lungs size/number of mitochondria mitochondrial enzyme activity capillarization fat oxidation enzyme activity blood supply to heart stroke volume left ventricle volume ventricular wall thickness
Decreased:
peripheral resistance
resistance training adaptations
increased
muscle strength muscle power balance and coordination BMR lean tissue mass muscle endurance motor performance insulin sensitivity
Decreased:
% body fat low back pain sarcopenia and osteoporosis insulin concentration/response to glucose challenge
increase of individual cross-section of a muscle fibre
dependent on resistance to muscle contraction and total number of concentrations
myofibril hypertrophy
contractile hypertrophy
sarcoplasmic hypertrophy
non-contractile hypertrophy
increase interfibrillar fluid, ATP, glycogen, phosphocreatine, mitochondria
fast glycolytic
increase with training
high contraction rates
large force
fast oxidative-glycolytic
increase with training
high oxidative and glycolytic enzymes
increased potential for powerful muscle contraction
60
Myosin makes up ____% of the total protein content of a muscle fibre with actin and tropomyosin making the next largest contributions therefore - increased myofibrillar protein
fibre type %
aerobic exercise causes increase in more type 1 fibre (oxidative)
same is true for any other type of activity including resistance training
true
True or False: fibre type is genetically determined
aerobic training (enzymes)
increases activity of oxidative enzymes (citrate synthase, Krebs cycle, presence of mitochondria)
reduction in activity of anaerobic enzymes
increased blood volume
increased mitochondrial and capillary density
neural adaptation
level of the CNS and spinal level
increase strength without increase cross-sectional area
increased neural drive of agonist activation
increased motor unit firing, increased motor cortex signal
decreased antagonist activation
connective tissue adaptations
most animal studies indicate that endurance and resistance training can increase max strength of tendons and ligaments
bone adaptations
PA (stress/strain) causes an increase in BMD
weight bearing / tendon stress through resistance training are indicated
site specific
wolff's law
the densities, and to a lesser extent, the sizes and shapes of bones are determined by the magnitude and direction of the acting forces applied to bone
first 2-6 weeks (resistance training)
rapid increase in strength due to neural adaptations
no difference in rate of progressions between males and females
some cellular / mechanistic adaptations
hypertrophy may occur faster than previously thought
10+ weeks (resistance training)
continued strength gains mostly due to cellular adaptations
greater hypertrophy in males due to presence of androgenic anabolic hormones (testosterone)