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Gestational Conditions
Disorders that did not exist before pregnancy
Occurrence puts woman and fetus at risk
Hypertension in pregnancy
Chronic Hypertension
Preeclampsia
HELLP Syndrome
Eclampsia
Gestational diabetes mellitus
Hyperemesis gravidarum
Hemorrhagic complications (PPH)
Surgery during pregnancy
Trauma
Infections during pregnancy
Hypertension in Pregnancy
Systolic blood pressure (BP) >140 mm Hg and diastolic BP > 90 mm Hg
Severe hypertension: systolic BP >160 mm Hg and diastolic BP >110 mm Hg
Hypertensions in Pregnancy (Significance and Incidence)
Hypertensive disorders of pregnancy are increasingly common, involving 5 to 10% of pregnancies.
Women over 40 years of age are at highest risk.
Hypertension is the leading cause of maternal and perinatal morbidity and mortality worldwide.
SOGC (Society of Obstetricians and Gynecologists of Canada) has recommendations regarding prevention of pre-eclampsia and its associated complications
Hypertensive States of Pregnancy
Chronic Hypertension
Superimposed preeclampsia
Gestational Hypertension
Preeclampsia
Severe Preclampsia
Eclampsia
Chronic hypertension
Hypertension present before pregnancy
Superimposed preeclampsia
Chronic hypertension in association with pre-eclampsia (after 20 weeks of gestation)
Gestational hypertension
Hypertension develops at or after 20 weeks of gestation in previously normotensive woman without proteinuria.
Preclampsia
Hypertension develops at or after 20 weeks of gestation in previously normotensive woman with proteinuria.
The main pathogenic factor is not an increase in BP but poor perfusion resulting from vasospasm.
Pathologic changes caused by disruptions in placental perfusion and endothelial cell dysfunction
Believed to be an interaction between cardiovascular and uteroplacental responses to pregnancy.
Arteriolar vasospasm diminishes the diameter of blood vessels, which impedes blood flow to all organs and increases BP.
Signs and symptoms of preeclampsia
Swelling of face or hands
Fluid retention (decreased urine output)
Sudden weight gain (>2lbs/week / > 6 lbs/mos)
Persistent headache
Seeing spots or changes in eyesight
Pain in the upper abdomen or shoulder
Nausea and vomiting (in the second half of pregnancy)
Difficulty breathing (dyspnea)
Tachycardia
Diagnostic Tests for Preeclampsia
Hematocrit - >35 & rising
Kidney involvement – Increased BUN & Uric acid
Coagulopathy – decreased platelet count
Bleeding time prolonged – decreased fibrinogen
Endothelial cell dysfunction – placental ischemia
Hepatic – decreased liver perfusion
Elevated liver enzymes AST & ALT indicate inflammation
Severe preeclampsia
Hypertension
Proteinuria
Cerebral disturbances
Epigastric pain
Eclampsia
Seizure activity or coma in woman diagnosed
with preeclampsia (no other reason)
Hematocrit
volume of blood made up of RBC’s
BUN (Blood urea nitrogen)
amount of nitrogen in waste product urea
Uric acid
waste product
Endothelial Cells
line all blood vessels and regulate exchanges between bloodstream and tissues
Signs of preeclampsia
First Sign - Elevated BP
Second Sign - Proteinuria
3 Questions for preeclampsia
Do you have a headache?
Visual disturbance?
RUQ epigastric pain?
Fetal health surveillance for preeclampsia
nonstress test [NST]
contraction stress test [CST],
Laboratory tests
contraction stress test [CST],
mild contraction stimulation via IV oxytocin (minimal) or nipple stimulation
Plan of Care for Preeclampsia
ensure maternal safety & healthy baby
Plan of Care for Preeclampsia (Mild)
Fetal health surveillance
Activity restriction
Diet
Plan of Care for Preeclampsia (Severe and HELLP Syndrome)
Hospital care (O2 for perfusion)
Magnesium Sulphate (1-1 care)
Control of BP (< 160/100)
No NSAIDS (elevate liver enzymes)
Plan of Care for Eclampsia
Immediate care
Postpartum nursing care
Future health care
Magnesium Sulphate
a medication used in pregnancy, primarily to prevent and treat seizures in women with preeclampsia or eclampsia, and for fetal neuroprotection before anticipated early preterm delivery
Magnesium Sulphate: Vital Signs to take for baseline
BP
Pulse
Resp Rate
Temp
DTRs (Deep tendon reflexes)
O2 Sat
LOC
Minimum Monitoring done for Mom’s on Magnesium Sulphate
Continuous pulse oximetry
Q1H: BP, HR, RR
Q4H: DTRs, LOC, temp
Foley Catheter
I&O Q1H
Max IV intake 80 ml/hr
Magnesium Sulphate Side Effects
Facial Flushing
Hypotension
Metallic Taste
N&V
Palpitations
Sweating
Magnesium Sulphate Toxicity
Muscle weakness
Loss of DTRs
Hypothermia
Resp. depression
Altered cardiac condition
Circulatory collapse
How to fix HDP (hypertensive disorders in pregnancy)
The only way to fix HDP (hypertensive disorders in pregnancy) is to deliver the baby and remove the placenta.
The method of delivery will depend on severity and condition of fetal well-being
Gestational Diabetes Mellitus (GDM)
Hyperglycemia that is first recognized in pregnancy.
Prevalence: 3.8 to 6.5%
Higher among Aboriginal, Latin American, South Asian, Asian, and African women.
Maternal and Fetal Risks of Gestational Diabetes Mellitus
2 times the risk of developing hypertensive disorders
Uncontrolled GDM can impact fetus development (LGA infants)
Screening for GDM
Risk factors: age, hx, obesity, polycystic ovarian syndrome
Identify hyperglycemia early
GDM Antepartum Interventions
Diet
Exercise
Monitoring blood glucose levels
Insulin therapy
Fetal surveillance
GDM Intrapartum Interventions
monitor q1h
FHR
no glucose IV bolus
GDM Postpartum Interventions
returns to N
reoccurs = risk of Type 2 later
Nursing Care of Infants of Mother with Diabetes
Critical to stabilize the newborn’s blood glucose
In the first two hours of life, even a healthy term newborn’s blood glucose falls
Early skin-to-skin and initiation of BF
Frequent BF
Blood sugar levels monitored as per protocol and intervention to maintain blood glucose in target range
May be a medical indication for supplementation with expressed breast milk or, if EBM not avail., formula
May need to be admitted for NICU for IV D10W
GDM Symptomatic Infant
Jittery
Temp. Instability
Apnea/Bradycardia
Sweating
Irritability
Tachypnea
Pallor
Poor Feeding
Weak Cry
Hypotonia
Seizures
Loss of Consciousness
Hyperemesis Gravidarum
Nausea and vomiting during pregnancy is the most common medical condition affecting 50% - 90% of women at some point in their pregnancy.
HG is protracted vomiting, retching, severe dehydration, and weight loss requiring hospitalization
Usually begins during the first 10 weeks of pregnancy
Etiology - not well understood, increased levels of Estrogen / hCG, assoc. with transient hypothyroidism
Clinical Manifestations of Hyperemesis Gravidarum
wt. loss
dehydration
low BP
increased pulse
poor skin turgor
Assessment for Hyperemesis Gravidum
Severity (freq, duration, diarrhea, wt. fluid/electrolytes, ketonuria – dehydration, CBC, psychosocial
Nursing Care for Hyperemesis Gravidum
Initial care: Clear fluids to diet with protein, IV, antiemetics, I & O,
Follow-up care – small freq high protein/bland meals, Mg, K, decrease odors
compassionate, sympathetic care
Hemorrhagic Disorders
Hemorrhagic disorders in pregnancy are medical emergencies (Hypolvolemia)
50% of bleeding in the third trimester is placenta previa or placental abruption.
Incidence of Miscarriage - Spontaneous Abortion (Early Pregnancy Bleeding)
15 – 20%
Clinical Manifestations of Miscarriage - Spontaneous Abortion (Early Pregnancy Bleeding)
back pain, cramping, bleeding
Early Pregnancy 1-6 weeks: heavy menstrual flow
Early Pregnancy 6-12 weeks: mod discomfort, blood loss
Late Pregnancy 12 – 20 weeks: age, parity, infection, cervical dilation,
Types of Miscarriage - Spontaneous Abortion (Early Pregnancy Bleeding)
threatened
inevitable
incomplete
complete
missed
Assessment of Miscarriage - Spontaneous Abortion (Early Pregnancy Bleeding)
ultrasound
lab tests
infection
loss
Nursing Care of Miscarriage - Spontaneous Abortion (Early Pregnancy Bleeding)
Medical-surgical management (rest, supportive care, cx dilation, D & C (oxytocin)
Home care – iron supp, bleeding, sexual activity, family planning, support groups, F/U phone call, loss
Premature Dilation of the Cervix
Incompetent cervix, cervical insufficiency
Passive and painless dilation of the cervix without contractions or labour
Etiology – past trauma, D& C (dilation and curettage), short cervix
Nursing Care of Premature Dilation of the Cervix
Medical-surgical management
Conservative management of restricted activity and hydration
Prophylactic cerclage (24 weeks) – removed at 35 – 37 wks.
Nursing care – monitor for contractions, PROM, infection
Home Care – decrease activity, bedrest, bleeding, emerg phone numbers
Ectopic Pregnancy
occurs when a fertilized egg implants and grows outside the main cavity of the uterus
Incidence of Ectopic Pregnancy
(1 in 7,000 pregnancies) with diagnostics
Fertilized ovum implanted outside uterine cavity
95% occur in uterine (fallopian) tube
Other sites: Ovary (0.5%),Abdominal cavity (1.5%), Cervix (0.3%)
Clinical manifestations of Ectopic Pregnancy
Missed menstrual period
Abdominal pain
Mild-to-moderate dark red or brown intermittent vaginal bleeding occurs in up to 80% of women
Nursing Care of Ectopic Pregnancy
Treatment options: salpingostomy, salpingectomy
Hospital care – pre-op/post-op, V/S, discuss future fertility
Home care – loss & grief / infertility support groups
Salpingostomy is surgical opening of fallopian tube
Salpingectomy is removal of fallopian tube
Late Pregnancy Bleeding - Placenta Previa
Placenta implanted in lower uterine segment near or over cervical os
Classification based on degree cervical os is covered by placenta
Cervical OS
refers to the openings of the cervix, specifically the internal os (opening into the uterus) and the external os (opening into the vagina).
Types of placental previa
Complete placenta previa – covers internal cervical os completely
Marginal placenta previa – 2.5 cm or closer to the cervical os
Low-lying placenta – no measurement of edge of placenta to cervical os
Incidence and Risk factors of Placenta Previa
0.5% of births
Risk Factors:
previous placenta previa
C/S
endometrial scarring
Diagnosis of Placenta Previa
transabdominal ultrasound examination
transvaginal ultrasound
Clinical Manifestations of placenta previa
diagnosed by ultrasound, painless bright red vag. bleeding (2nd or 3rd trimester)
Maternal and fetal Outcomes of placenta previa
Complications: hemorrhage, abnormal placental attachment, poor placental exchange, hysterectomy
Fetal risks: malpresentation, stillbirth, and fetal anemia preterm birth
Late Pregnancy Bleeding: Abruption
Placental abruption: premature separation of placenta
Incidence and Etiology of Abruption
1 in 75 – 226 pregnancies. Hypertension, trauma (MVA), cocaine use, smoking, PROM, twin pregnancies
Grades of Abruption
mild, moderate, severe
Clinical Manifestations of Abruption
partial or complete separation
Dark, vaginal non-clotting bleeding,
Abdominal back pain,
Port-wine stained amniotic fluid
Uterine contractions
Uterine tenderness
Fetus and Maternal outcomes of abruption
Maternal: hemorrhage, hypovolemic shock, thrombocytopenia (low platelets), renal failure
Fetus: Abnormal FHR patterns, neurological defects, IUGR, death, > incidence of SIDS
Late Pregnancy Bleeding: Placental Variations
Placenta accrete
Placenta increta
Placenta Percreta
Velamentous insertion of cord/ vasa previa
Cord insertion and placental variations
Battledore (marginal) insertion of cord
Placenta accrete
(placenta previa) trophoblastic invasion extends beyond normal endometrial barrier.
Placenta increta
extends into the myometrium
Placenta percreta
extends beyond the uterine serosa
Velamentous insertion of cord/ vasa previa
rare anomalies
Higher incidence in multiple gestations and pregnancies from assisted reproductive technology
Cord vessels branch at membranes and course onto placenta
Rupture of membranes or traction on cord may tear one or more fetal vessels
Fetus may rapidly bleed to death as a result
Battledore (marginal) insertion of cord
Increases risk of fetal hemorrhage, especially after marginal separation of placenta
Infections Acquired During Pregnancy
Sexually transmitted infections
Impact morbidity rates, infertility
Urinary tract infections (common – 20%)
Responsible for 10% of hospitalizations
Asymptomatic bacteriuria
Cystitis (bladder infection)
Pyelonephritis
Asymptomatic bacteriuria
antibiotics
repeat urine culture
Cystitis (bladder infection)
dysuria
urgency
suprapubic pain
3-day course of antibiotics which may stain urine orange
Pyelonephritis
common, 2nd trimester
fever
chills
pain in lumbar area
N & V
admitted with IV antibiotics (10–14 d)
monitor for sepsis
regular urine cultures
Physical trauma during pregnancy
MVA, falls, burns, Violence
Significance - (6 - 7% of pregnancies) & (80% fetal deaths)
Effect of trauma on pregnancy is influenced by the following:
Length of gestation
Type and severity of the trauma
Degree of disruption of uterine and fetal physiological features - fetal death
Special considerations for mother and fetus:
Physiological alterations of pregnancy
Fetal survival depends on maternal survival.
Pregnant woman must receive immediate stabilization and care for optimal fetal outcome
Fetal physiological characteristics
Careful monitoring of fetal status
Fetal monitor tracing works as an “oximeter” for fetal well-being.
Perimortem Caesarean Birth
Poor fetal survival with C/S > 20 minutes after maternal death.
Consider Caesarean birth after 4 minutes of resusc. with no response in the mother.
Rarely successful
Postpartum Hemorrhage (PPH)
Leading cause of maternal death worldwide, preventable
Postpartum hemorrhage (PPH) traditionally defined as:
Loss of >500 mL of blood after vaginal birth
Loss of >1000 mL after Caesarean birth
Any blood loss that has the potential to cause hemodynamic instability
Among leading cause of maternal mortality worldwide
Life-threatening, with little warning
Stages of PPH
Early/ Primary – within 24 hrs of birth
Late/secondary - > 24 hrs – 6 weeks post birth
Due to retained placenta fragments /infection
Subinvolution of uterus (retained frag, pelvic infection)
Discharge teaching – signs of normal involution, complications
4 T’s of PPH
tone
Uterine atony
Marked hypotonia of uterus
Leading cause of early PPH
tissue
Retained placental fragments
Placenta accreta, increta, percreta
trauma
Lacerations of birth canal
Uterine inversion
Hematomas
thrombin
Various clotting disorders
Correction of underlying cause
Assessment for PPH
Evaluation of contractility of uterus.
Firm massage of fundus
Administer intravenous fluids and medication to manage bleeding.
Active management of 3rd stage to prevent PPH
Oxytocin admin after delivery of fetal shoulder
Immediate fundal massage after complete birth
Plan of Care and Implementation for PPH
Hypotonic uterus – massage, elimination of bladder distention, IV
Bleeding with a contracted uterus (manual exploration of uterine cavity for placenta
Pharmacologic remedies – homeostatic actions or oxytocin age
Puerperal infection (Postpartum Infection)
any infection of genital canal within 28 days after abortion or birth
Most common infecting agents are the numerous streptococcal and anaerobic organisms.
Endometritis
Wound infections
Urinary tract infection
Mastitis
Structural Disorder related to Child-bearing
Disorders of uterus and vagina related to pelvic relaxation and urinary incontinence are often the result of child-bearing.
Uterine displacement and prolapse
Posterior displacement, or retroversion
Uterine prolapse:
more serious displacement.
Cystocele and rectocele
Cystocele: protrusion of bladder downward into vagina when support structures are injured
Rectocele is herniation of the anterior rectal wall through relaxed or ruptured vaginal fascia and rectovaginal septum.
Urinary incontinence
Postpartum psychological complications
Psychosocial complications in PP period: implications for mother, newborn, and entire family.
Interfere with attachment
May threaten safety and well-being of mother, newborn, and other children
Perinatal mood disorders (PMD)
Up to 80% - mild depression or “baby blues.”
10 to 15% - more serious depression (PPD)
Can eventually incapacitate women to point of being unable to care for themselves and their babies
Paternal mood disorder
Predictor is having a partner with PMD
Postpartum anxiety disorder
Generalized anxiety disorder
Obsessive-compulsive disorder
Panic disorder and panic attacks
Specific phobias
Social anxiety disorder
Post-traumatic stress disorder
Postpartum depression without psychotic features
Characterized by low mood and lack of interest in activities, can be mild to severe
Treatment options
Antidepressants, antianxiety medications, and electroconvulsive therapy
Psychotherapy focuses on fears and concerns of new responsibilities and roles; monitoring for suicidal or homicidal thoughts
Postpartum psychosis
Syndrome characterized by depression, hallucinations, delusions, and thoughts of harming either infant or self
Psychiatric emergency; may require psychiatric hospitalization
Antipsychotics and mood stabilizers such as lithium are treatments of choice
Psychological Nursing Care
Screening for perinatal mood disorder
On postpartum unit
In the home and community
Referral
Providing safety
Psychiatric hospitalization
Psychotropic medications
Psychotropic medications and lactation
Other treatments for perinatal mood disorders
Loss and Grief
Any perception of loss of control during the birthing experience, hopes and dreams
Birth of child with complex condition
Maternal death
Fetal or neonatal death
Grief is a process of recovering from a loss.
Many emotional, cognitive, behavioural, and physical responses
Overlapping phases in grief process
Acute distress
Searching and yearning
Disorientation
Reorganization and resolution
Family aspects of grief
Grandparents and siblings
Communicating and care techniques
Help mother, father/partner, and siblings actualize their loss
Help parents with decision making
Help bereaved to acknowledge and express their feelings
Communicating and care techniques
Normalize grief process and facilitate positive coping
Meet the physical needs of postpartum bereaved mother
Create memories for parents to take home
Cultural and spiritual needs of parents
Provide sensitive care at and after discharge
Provide postmortem care respecting parents’ wishes
Documentation