Central chemoreceptors in the medulla sense
pH
Peripheral chemoreceptors in the aorta sense
oxygen
Lesion to the medulla can cause this abnormal breathing pattern
Biots/ataxic breathing
Biots/ataxic breathing
irregular pattern of deep and shallow breaths
Heart failure or TBI can cause this abnormal breathing pattern
Cheyne-Stokes
Cheyne-Stokes
repeated cycles of deep breathing followed by shallow breathing and a short period of apnea
Metabolic acidosis or kidney failure can cause this abnormal breathing pattern
Kussmaul’s Respiration
Kussmaul’s Respiration
RR > 20, increased depth and dyspnea
Stroke and other NM conditions can cause this abnormal breathing pattern
asymmetrical/lateral-costal breathing
Stroke, spasticity, SCI, flail chest can cause this abnormal breathing pattern
chest falls with inspiration and abdomen rises with expiration
Periodic breathing
rapid breathing followed by up to 10 second pause
Periodic breathing is NORMAL for
1st 2 weeks to 5-6 months
Injury above T1 may have these cardiopulm implications
orthostatic hypotension, temp regulation
Components contributing to proper diaphragm pressures
glottis, diaphragm, pelvic floor, postural support
Exercise increases level of this good cholesterol
HDL
How to assess asymmetrical breathing pattern
tape measure
Asymmetrical breathing and postural abnormalities put pt at risk for
under-ventilation of alveoli
Muscles to assess in person post CVA
hamstrings, pecs, rhomboids, QL, middle traps
Interventions for decreased breath sounds, crackles, low pulse ox
posture
stacking
ACB
lateral costal expansion
segmental breathing
Pair this with stand to sit movement
speech (eccentric breathing)
Pair isometric activity with this
breath hold
Pair concentric activity with this
inspiration
A stroke has this effect on muscle fibers
decrease type 2 fibers
This LDL is considered high
100 mg/dL
Decreased power output, lower VO2 max, and shift of muscle fiber types can be caused by
stroke
Things to consider about 6MWT post stroke
limited by balance
poor coordination
low correlation to peak VO2
_% of stroke survivors have co-existing cardiac disease
75
_% of stroke survivors have silent ischemia
40
Aerobic exercise post stroke has shown to improve walking speed by _
0.7 m/s
Pt interventions should address these 4 outcomes
brain repair
neuroplasticity
cardiorespiratory fitness
metabolic health
Aerobic exercises should begin _ hours post stroke
24-48
_ intensity and _ frequency at start of aerobic exercise
low, high
20-30 mins of low intensity walking had this effect in animal models
reduced lesion volume
This trial found that mobilizing before 24 hours since stroke had poor outcomes
AVERT
Optimal neuroplasticity window
within 12 weeks
Within first 12 weeks post stroke, levels of _ are higher allowing for better neuromotor recovery
BDNF
FITTI correlated with increased BDNF levels
F: 5-7 days
I: min-mod
T: 30 mins
T: walking
Initiation: 1-7 days post CVA
Peak VO2 post stroke
8-15 ml/kg/min
Peak VO2 post stroke is about _% of aged matched controls
40-50
ADL’s are _ mets for stroke population
3
To perform ADLs post stroke, need a max MET level of _ and ability to sustain a MET level of _
5.7
3.7
Strokes are correlated with _ regarding metabolic effects
poor glucose tolerance
dyslipidemia
Aerobic exercise recommendations 1-3 weeks post stroke
low intensity
high frequency, 1-3x/day
5-10 mins bouts
Aerobic exercise recommendations 4-7 weeks post stroke
4-6 sessions/week
30-60 mins bouts
low to mod intensity
Aerobic exercise recommendations 8+ weeks post stroke
3-5 sessions/week
20-60 min bouts
55-80% HRR or RPE 11-14/20
Resistance training recommendations post stroke
2-3 sessions/week
1-3 sets
10-15 reps
50-80% 1RM
Age estimated HR equation may _ HR max in those with stroke
overestimate
max HR prediction equation for those with stroke
206.9 - (0.67 x age)
max HR prediction equation for those on beta blocker
164 - (0.7 x age)
Most common cause of mortality following SCI/MS
respiratory complication
Pts most vulnerable to respiratory illness within _ after SCI
1 year
Risk of respiratory failure directly associated with _
injury level
Injury at C1-C3
vent dependent
diaphragm paralyzed
Injury at C3-C4
periods of unassisted ventilation
need vent at night
Injury at C5
diaphragm intact
intercostals and abdominals paralyzed
decreased lung volume
Injury at C6-C8
independent breathing
below C7 can cough with pecs
Injury at T1-T4
intercostals drive breathing
reduced cough efficiency due to abdominal weakness
Injury at T5-T11
minimal disruption to cardiovascular system below T6
Injury at T12
respiratory function comparable to normal person
Spinal shock
loss of reflexes, motor function, and sensation below lesion
Spinal shock can cause this to occur at chest wall
paradoxical depression of ribs
Significant increase of vital capacity is seen within _ weeks of injury
5
These increase post spinal shock
vital capacity, total lung capacity, inspiratory capacity
This decreases post spinal shock
functional residual capacity
Injury upper 6 thoracic nerves can result in
reduced FEV1
Partial loss of sympathetic tone with injury above this level
T4
Arms at 90, full abduction and ER forces most excursion at _ when breathing
chest
Arms at side forces most excursion at _ when breathing
diaphragm
Position for optimal diaphragmatic breathing
supine, bolster under knees, neck flexed, arms IR
Ways to facilitate diaphragmatic breathing
sniffing
lateral costal expansion
upper chest inhibition
Voice should be _ during diaphragmatic breathing
soft, slow
What will inhibit diaphragmatic breathing in those with cerebral palsy
LE contractures causing poor pelvic alignment
What will inhibit diaphragmatic breathing in those with CVA
one sided weakness, neglect
What will inhibit diaphragmatic breathing in those with SCI
poor resting diaphragm positon
lack of innervation
TLSO
Position to facilitate accessory muscle breathing
anterior pelvic tilt
no pillows
arms ER
towel along spine
Voice should be _ during accessory muscle facilitation
loud, demanding
Ways to facilitate accessory muscle breathing
diaphragm inhibition
pec stretch
lateral-costal expansion
butterfly maneuver
What will inhibit accessory muscle breathing in those with cerebral palsy
forward head
torticollis
UE contracture
What will inhibit accessory muscle breathing in those with CVA
muscle imbalance, neglect
What will inhibit accessory muscle breathing in those with Guillian Barre
tracheostomy
muscle weakness
What will inhibit accessory muscle breathing in those with Parkinson’s
stiff trunk
What will inhibit accessory muscle breathing in those with SCI
cervical collar
lack of muscle innervation
bad wheelchair prescription
Abdominal binder can improve these
voice
FVC
orthostatic hypotension
Abdominal binder must be placed below _
ribs
Accessory muscles of inspiration
SCM
scalenes
pec minor
Primary muscles of inspiration
external intercostals
diaphragm
Muscles of quiet expiration
passive recoil of lungs, ribs, and diaphragm
Muscles of active expiration
internal intercostals
abs
QL
Desired outcomes of IMT in SCI
increase strength
increase aerobic capacity
reduce dyspnea
Desired outcomes of EMT in SCI
increase strength
improve cough force
improve speech
Non-targeted inspiratory resistance trainer
P-Flex
Targeted inspiratory trainer
Threshold trainer, provides visual target
What position does pt have to be in to use P-Flex
upright
RR has litte effect on resistance provided with this device
threshold
resistance dependent on _ and _ for P-Flex
RR and size of hole
Expiratory muscle trainer
The Breather
Only use airway suctioning if pt can’t
expectorate on their own
other techniques not working
How does constipation impact breathing?
less space for lungs to expand
What can cause constipation in NM population
sphincter spasticity
T6 injury and above experience chronotropic incompetence resulting in
blunter HR response