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biological contributing factors of specific phobia
-GABA dysfunction
-Long term potentiation
GABA
primary inhibitory neurotransmitter in the CNS and works throughout the brain to make postsynaptic neurons less likely to fire
GABA dysfunction
due to a low level of GABA or an insufficient reception or transmission of GABA across the synapse
GABA dysfunction in contributing to the development of phobia
cause the fight/flight/freeze or anxiety response to be activated more easily, means that the stress response is more easily triggered
effects of low levels of GABA
-genetic vulnerability (biological factors)
-exposure to prolonged stress or environmental toxins, nutritional deficiencies or high caffeine intake (sociocultural factors)
Long term potentiation
long-lasting strengthening of synapses that are repeatedly activated together
Long term potentiation in contributing to the development of phobia
exposure to the phobic stimulus will therefore strengthen the memory circuit and via communication with the amygdala, activate multiple brain regions that then produce a variety of symptoms of fear and anxiety
psychological contributing factors
-role of behavioural models (precipiation by classical conditioning and perpetuation by operant conditioning)
-the role of cognitive bias (memory bias and catastrophic thinking)
precipitation
development origin trigger
perpetuation
maintained persisted
cognitive bias
systematic error in thinking that affects the decisions and judgements that people make
memory bias
a kind of cognitive bias caused by inaccurate or exaggerated memory. each time the person remembers the situation, object or event it is usually more catastrophic or threatening
catastrophic thinking
when an individual repeatedly overestimates the potential dangers of an object or event and assumes the worst
social contributing factors
-specific environment triggers
-stigma around seeking treatment
specific environmental trigger
a specific object or situation in the environment that triggered an extreme fear response
stigma
a mark of disgrace that labels a person as different and separates them from others, individuals are less likely to seek or accept help and go untreated for years
differences in developing a phobia
-individuals prior experience
-individuals subsequent exposure to the object or situation
agonists
imitating and therefore stimulating a neurotransmitter’s activity
antagonists
inhibiting a neurotransmitter’s activity