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cardiovascular system
in mediastinum
left of midline
above diaphragm
includes heart and extremities
pericardium
outermost layer
epicardium
thin outermost layer
myocardium
thick muscular middle layer
endocardium
the innermost
two upper chambers
right and left atria at the base of heart, thin walled returning blood from the veins
two lower chambers
right and left ventricles at the apex
thick walled and pumps the blood to the lungs throughout the body
arteries
carries oxygenated blood from heart to body carries blood away from heart
veins
carries deoxygenated blood towards the heart
pulmonary circulation
right heart pumps deoxygenated blood to the lungs
systematic circulation
left heart pumps oxygenated blood to rest of the body
cardiac cycle
to help ensure proper circulation the heart contracts and relaxes rhythmically creating a 2 phase cardiac cycle
systole
diastole
systole
ventricles contract- blood ejected from left ventricle into aorta and from RV into pulmonary artery
mitral and tricuspid valves close
pressure rises
aortic and pulmonic valved open
blood ejected into artiers
pressure falls
diastole
mitral and tricuspid valves open
blood moves from atria to ventricles
ventricles dilate an energy requiring effort that draws blood into the ventricles as the atria contract, thereby moving blood from atria to ventricles
atria contract as ventricles almost filled
causes complete emptying of atria
electrical conduction of the heart
intrinsic electrical conduction system enables the heart to contract within itself
coordinates sequence of muscular contractions taking place during cardiac cycle
sinoatrial node
atrioventrical node
bundle of His
purkinje fibers
conductivity
current flows in orderly sequence first across atria to AV node low in atrial septum → delayed slightly so atria have time to contract b4 ventricles are stimulated → impulse travels to bundle of His right and left bundle branches and then through ventricles
systole (contraction)
pulmonary /aortic valves open
mitral /tricuspid closed
S1 best heard at APEX “lub”
diastole-(relaxation)
diastole blood fills the ventricles
AV valves are open
aortic valve closed
s2 best heard at BASE “dub”
cardiovascular health history
complaints of
pain
dyspnea
palpitations
dizziness
medications
edema
nocturia
past health history
HTN (hypertension)
blood clots
surgeries
illnesses
congenital heart defects/murmur
rheumatic fever
diabetes
EKG
lipid profile
physical exam
inspection 1st- neck vessels pain discomfort cough fatigue SOB
3 positions- sitting supine left lateral recumbent
no percussion due to potential for complications
inspection
aortic pulmonic erbs point tricuspid mitral
symmetry
deformities of the thoracic cage
visible pulsations or heaves
may see visible impulse at 5th intercostal or medial space
apical impulse or point of maximal impulse
palpatiion
focus on areas found on inspection
feel for pulsations vibrations heaves thrills apical impulse
palpate areas
auscultation -Lub
S1 closure of the AV valves beginning of ventricular systole best heard at the APEX the mitral and tricuspid valves
auscultation- dub
s2 closure of semilunar valves after systole complete beginning of ventricular diastole best heard at base aortic an pulmonic valves
auscultation
note rate rhythm strength of beats
apical pulse count for 1 full minute
focus on S1 and S2 sounds
listen foe extra sounds or deviations from normal atrial fibrillation murmurs rubs
heart murmurs
turbulent flow with a swooshing or blowing sound
conditions that contribute to heart murmurs
increased blood velocity
structural valve defects
valve malfunction
abnormal chamber openings
neck vessels
carotid artery, jugular veins
carotid artery pulse→ ventricular systole
level of jugular venous pressure reflects right atrial pressure
neck vessels assessment
inspec- observe juglar venous pulse, evaluate pressure
auscult and palp - carotid arteries
hypotension
low BP may not perfuse organ systems asequately
hypertension
BP> 130/80
murmurs
blowing sounds turbulence in blood flow
myocardial infarction
occlusion of arterial blood flow causing tissue damage
congestive heart failure
congestion in pulmonary or systemic circulation related to inadequate pumping
thrombus
blood clot
fluid volume deficit hypovolemia
isotonic fluid loss
fluid becomes hypertonic drawing fluid from the cells into the interstitial space leaving cells depleted of fluid and unable to function properly
fluid volume excess hypervolemia
excess isotonic fluid common cases kidney malfunction and ineffective heart pumping resulting in the accumulation of fluid in lungs and dependent parts of the body