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hypercapnia
increase of carbon dioxide in the blood
hypoxemia
decrease of oxygen in the blood
increased respirations as well, but less effective than hypercapnia
white/clear mucoid
colds
bronchitis
viral infections
Yellow or green sputum
bacterial infections
rust colored sputum
TB
pneumococcal PNA
Pink, frothy sputum
pulmonary edema
some sympathomimetic medications have a side effect of pink tinged mucus
Mycoplasma PNA cough characteristic
hacking
early heart failure characteristic cough
dry
Croup cough characteristic
barking
colds, bronchitis, PNA cough characteristics
congested
Orthopnea
difficulty breathing when supine
state number of pillows needed to achieve comfort
i.e. two pillow orthopnea
Pack years formula
years of smoking x ppd
AP (anteroposterior) to Transverse Diameter
normal chest
transverse diameter (side-to-side) is greater than AP diameter (front-to-back)
normal ratio: 1:2
in COPD (esp. emphysema):
Lungs are chronically overinflated with air
chest expands in all directions
AP diameter increases until it almost equals transverse diameter
ratio becomes closer to 1:1
Barrel Chest
used to describe when chest ratio of AP to transverse diameter is 1:1
rounded, bulging chest
ribs more horizontal
seen in COPD
esp. emphysema
crepitus assessment and findings
crackling/grating sensation on palpation
subcutaneous crepitus causes
trapped air
trauma
pneumothorax
Joint crepitus cause
rough joint surfaces
arthritis
fracture
assessment findings for pulmonary embolism
sudden SOB
chest pain
tachypnea
tachycardia
hemoptysis
coughing up blood
hypoxemia
assessment findings for PNA
sudden onset of fevers/chills
productive cough
rust-colored, purulent
pleuritic chest pain
SOB
fatigue
urgent findings from thorax palpation
asymmetric chest expansion
surface anomalies
lumps
masses
tenderness
crepitus
Assessment findings of pleural friction rub
superficial, coarse, grating, low-pitched sound
like leather rubbing together
heard on both inspiration and expiration
pleurae are inflamed and lose their normal lubricating fluid
surfaces rub together
Silent chest
Absence of breath sounds
Indicates no air is moving in or out of the lungs.
Considered an ominous sign
severe airway obstruction
status asthmaticus
end-stage respiratory compromise
Stridor
High-pitched, monophonic inspiratory crowing sound, louder in the neck than over the chest wall.
Upper airway obstruction, due to:
swollen or inflamed tissues
lodged foreign body.
Associated with:
croup, acute epiglottitis in children
foreign body aspiration
Airway obstruction may be life-threatening
Absent or Decreased Breath Sounds with Distress
Obstruction of bronchial tree, air, or fluid (pleural effusion) in pleural space.
May indicate:
pneumothorax
large pleural effusion
tension pneumothorax
absent sounds are accompanied by:
tracheal shift
unstable vital signs
requires immediate intervention
Widespread, Severe Wheezing
Diffuse, high-pitched, musical squeaking sounds (polyphonic).
Airflow obstruction
often found in:
acute asthma
severe chronic emphysema
Emergent Concern
Inability to move air effectively combined with respiratory distress or silent chest indicates respiratory failure risk
Chronic Bronchitis
a type of COPD with proliferation of mucus glands
excessive mucus
inflamed bronchi
partial obstruction
Inspection findings of Chronic Bronchitis
Hacking
rasping productive cough
dyspnea
fatigue
cyanosis
possible clubbing of fingers.
palpitation findings of Chronic Illness
Tactile fremitus normal
palpable vibrations
percussion findings for Chronic bronchitis
Resonant
Auscultation findings for chronic bronchitis
Normal vesicular sounds
prolonged expiration.
Adventitious sounds of Chronic Bronchitis
Crackles over deflated areas
wheezing during exacerbations
Emphysema
a type of COPD caused by destruction of pulmonary connective tissue
permanent enlargement of air sacs distal to terminal bronchioles
Inspection findings for emphysema
Barrel chest
accessory muscle use
tripod position
shortness of breath
especially on exertion
tachypnea
Palpation findings for Emphysema
Decreased tactile fremitus and chest expansion.
Percussion findings for Emphysema
Hyperresonant
Auscultation findings for Emphysema
Decreased breath and voice sounds
prolonged expiration
Adventitious sounds in Emphysema
Usually none, but occasional wheeze
Asthma
(reactive airway disease)
Allergic hypersensitivity to inhaled allergens
bronchospasm
inflammation
edema
mucus production
Inspection findings of Asthma
Increased respiratory rate
audible wheeze
accessory muscle use
cyanosis
retraction of intercostal spaces
Palpation findings of Asthma
Tactile fremitus decreased
tachycardia
Percussion findings of Asthma
Resonant
may be hyperresonant during attack
Adventitious sounds of Asthma
BL wheezing on expiration
sometimes both inspiratory and expiratory
Normal Respiratory Pattern
Rate: 10–20 breaths per minute
Depth: 500–800 mL
Pattern: Even, with occasional sighs that expand alveoli
Sigh (respiratory pattern)
Occasional sighs are normal
frequent sighs may indicate emotional dysfunction
can lead to hyperventilation
Tachypnea
Rapid, shallow breathing (>24/min).
Normal with fever, fear, or exercise.
Also occurs with respiratory insufficiency, pneumonia, pleurisy, alkalosis, or lesions in the pons
Hyperventilation
Increased rate and depth
Can be a normal response to exertion, fear, or anxiety.
Leads to CO₂ loss (alkalosis)
-
Seen in:
diabetic ketoacidosis
Kussmaul respirations
salicylate overdose
lactic acidosis
hepatic coma
midbrain lesions
Bradypnea
Slow, regular breathing
<10/min
Associated with:
drug-induced depression of the medulla
increased intracranial pressure
diabetic coma
Hypoventilation
Irregular, shallow breathing.
Caused by:
narcotic or anesthetic overdose
prolonged bed rest
chest splinting to avoid pain
Cheyne-Stokes Respiration
Cycle of gradually increasing and then decreasing respirations
followed by 20 seconds of apnea
Normal in infants and older adults during sleep
-
Seen in:
severe heart failure
renal failure
meningitis
drug overdose
increased intracranial pressure
Biot Respiration
Similar to Cheyne-Stokes, but is irregular
Normal respirations (3–4) followed by apnea.
-
Seen with:
head trauma
brain abscess
heat stroke
spinal meningitis
encephalitis
Chronic Obstructive Breathing (Air Trapping)
Normal inspiration with prolonged expiration due to increased airway resistance.
-
Seen in:
COPD, especially with exertion, leading to dyspnea because of incomplete expiration
Bronchial (tracheal)
normal breath sounds
Pitch:
high
Amplitude:
loud
Location:
Trachea and larynx
Quality:
Harsh, hollow, tubular.
Duration:
Expiration longer than inspiration.
Bronchovesicular
normal breath sounds
Pitch
moderate
Amplitude:
moderate
Location
Major bronchi
between scapulae posteriorly
between upper sternum anteriorly
Quality:
Mixed
Duration
Inspiration and expiration equal
Vesicular
normal breath sounds
Pitch:
low
Amplitude:
soft
Location:
Peripheral lung fields where air flows through smaller bronchioles and alveoli.
Quality:
Rustling
like the sound of wind in trees.
Duration:
Inspiration longer and louder than expiration
Crackles (Rales)
Discontinuous, popping sounds heard mostly during inspiration.
Caused by:
air colliding with secretions
sudden airway opening.
Fine crackles
high-pitched, short
Coarse crackles
louder
lower-pitched
longer.
Atelectatic crackles
short-lived in sleeping or elderly patients when alveoli are partially collapsed
not pathologic
Wheezes (Rhonchi)
Continuous musical sounds
usually more prominent during expiration
High-pitched wheezes
(sibilant)
polyphonic, squeaky
Low-pitched wheezes (sonorous)
Snoring, moaning quality
Stridor
High-pitched, crowing inspiratory sound.
Louder in the neck than chest wall.
Caused by upper airway obstruction (croup, epiglottitis, foreign body).
Emergent finding
can be life-threatening
Assessment findings for meningitis
severe HA
n/v
light sensitivity
nuchal rigidity
neck stiffness
fever
AMS
irritability
confusion
lethargy
loss of balance
blurred vision
Auscultation process for the thyroid
Indication: If the thyroid gland is palpably enlarged during assessment
Technique:
Place the bell of the stethoscope lightly over the thyroid gland.
Listen carefully for abnormal vascular sounds.
Normal Finding: No sound should be heard.
Abnormal Finding:
A bruit may be heard.
indicates accelerated or turbulent blood flow, often associated with hyperplasia of the thyroid, such as in hyperthyroidism
Tension Headaches
BL
dull
bandlike
stress-related
milder
Migraine Headaches
usually unilateral
throbbing
moderate-to-severe
prodrome/aura phases
triggered by:
hormones
foods
stress
cluster headache
strictly unilateral
excruciating
short but recurrent in clusters
autonomic symptoms and agitation
function of cranial nerve 11
Moves head and shoulders via sternomastoid and trapezius
function of cranial nerve 7
Controls facial expressions and maintains symmetry
assessment finding differences between Stroke and Bell's Palsy
Stroke
Lower face paralysis only
can wrinkle forehead
Bell’s Palsy
Complete half-face paralysis
cannot wrinkle forehead
Red Flag Sx for HA
Thunderclap headache – sudden, severe pain reaching maximum intensity within minutes.
New severe headache
particularly in individuals over 50 years of age.
Headache triggered by Valsalva maneuvers
occurs with coughing, sneezing, straining, or sexual intercourse.
Headache triggered by positional changes
e.g., moving from lying to standing.
Significant change in prior headache pattern
worsening severity or persistence (“never goes away”).
Headache with systemic symptoms
such as fever, rash, neck stiffness, weight loss, personality changes.
First severe headache in an adult or child with no prior history
worst HA in my life
Temporomandibular Joint
articulation of the mandible and temporal bone, located just anterior to the tragus of the ear
enables chewing and speech through hinge and gliding movements
Normal findings include:
smooth, pain-free motion
Inspection of TMJ
Observe the area for swelling or asymmetry.
Muscle Testing: Evaluate strength by asking pt to move jaw against resistance
testing integrity of cranial nerve V
trigeminal
Palpitation of TMJ
Place fingertips just anterior to the tragus and ask the patient to open and close the mouth.
Normal finding: smooth motion without limitation or tenderness.
Abnormal TMJ findings
Crepitus or Clicking
May suggest joint derangement.
Pain or Tenderness
Could indicate arthritis, infection, or TMJ disorder.
Limited ROM
Seen in TMJ syndrome or with trauma.
ROM testing of TMJ
Ask pt to open the mouth maximally
normal is 3–6 cm between upper and lower teeth
Protrude and retract the jaw
Move jaw side to side
normal is1–2 cm
Myxedema (Hypothyroidism)
Slow metabolism
fatigue
weight gain
constipation
cold intolerance
puffy face
dry skin
slow reflexes.
Graves’ Disease (Hyperthyroidism)
Fast metabolism
weight loss
heat intolerance
tachycardia
tremors
exophthalmos
sweating
hyperreflexia
Facial Assessment finding for Cushing syndrome
round, swollen, moonlike face
often plethoric (red-tinged)
sometimes with acne
accompanied by systemic features
truncal obesity
muscle wasting
clinical presentation of Acromegaly due to excessive growth hormone
bony overgrowth of the skull, jaw, and facial structures
elongated head
prominent forehead and jaw
enlarged nose and lips
coarse facial features
Nerve that control facial muscles
cranial nerve 7
controls facial expressions
Vertigo
true rotational spinning sensation
Often results from labyrinthine-vestibular disorders in the inner ear
linked to vestibular dysfunction rather than cerebral perfusion issues
objective vertigo
person perceives the room spinning
subjective vertigo
person feels as though they are spinning
Presyncope
light-headed, swimming sensation or feeling of impending fainting.
Caused by:
decreased blood flow to the brain
cardiac irregularity that lowers cardiac output
“I feel like I’m going to faint”
Disequilibrium
reflects an issue with balance or coordination
not necessarily perfusion or vestibular spinning
associated with musculoskeletal or neurologic disorders affecting gait and posture
“I feel like I’m going to fall down.”
Sinoatrial (SA) Node landmark location
Located near the superior vena cava
at the top of the right atrium.
has intrinsic rhythm
known as the pacemaker of the heart
Atrioventricular (AV) Node landmark location
Positioned low in the atrial septum
between the atria and ventricles
Impulse is delayed slightly here to allow the atria to contract and empty before ventricular contraction
Bundle of His (Atrioventricular Bundle) landmark location
Extends from the AV node into the interventricular septum.
Divides into the right and left bundle branches that travel down either side of the septum
Purkinje Fibers landmark location
Spread out from the bundle branches through the ventricular walls.
Carry impulses that stimulate the ventricular myocardium to contract
assessment findings for heart failure
Subjective
fatigue
dyspnea
chest pain
Objectively
jugular venous distention (JVD)
displaced or sustained apical pulse
S3 gallop
edema
cool extremities
weak pulses
irregular tachycardia.
significance of murmurs
provide clues to valvular heart disease and other abnormalities.
Midsystolic (Ejection) Murmurs
Caused by forward flow through semilunar valves.
aortic stenosis
pulmonic stenosis
Pansystolic (Regurgitant) Murmurs
Caused by backward flow of blood from higher to lower pressure
mitral regurgitation
Diastolic Rumbles (AV Valve Disease)
Caused by abnormal filling
mitral stenosis
Atrioventricular (AV) Valves function
Open during diastole
allows ventricles to fill with blood.
Close during systole
prevents regurgitation of blood back into the atria.
Types of AV valves
Tricuspid valve
between the right atrium and right ventricle
Mitral (bicuspid) valve
between the left atrium and left ventricle
AV valve support structures
Their leaflets are anchored by chordae tendineae attached to papillary muscles
contract to ensure the valves close securely