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air conduction portion
delivers air to the lungs
gas exchanging portion
allows oxygen to enter blood and carbon dioxide to be removed
What are gas exchanging structures?
lungs, alveoli, & capillaries
bronchiole Function
smooth muscle allows dilation (increase airflow) and constriction (decreased airflow)
What happens with excessive bronchiole constriction?
dangerous reduction in airflow and difficulty breathing
What can excessive bronchiole constriction cause?
respiratory distress or failure
What are air containing particles?
bacteria, viruses, fungi, dust, pollen & pollutants
mucus function?
traps particles like bacteria, dust and pollutants
cilia function?
moves mucus upward to be expelled
What happens if cilia is damaged?
mucus build up in lungs, increased risks of respiratory infections, reduced lung function
effect of smoking
reduces mucus and destroys cilia
effect of alcohol on cilia
paralyzes cilia
primary site of gas exchange
alveoli
alveolar surfactant
lipoprotein that reduces surface tension and prevents alveolar collapse
what does alveolar surfactant improve?
pulmonary compliance
atelactasis
collapse of alveoli due to lack of surfactant or other casues
who is at risks for low surfactant?
premature infants and smokers
hering-breuer reflex
prevents lung overinflation by stopping inspiration when lungs are stretched
primary regulator of breathing
carbon dioxide
how do lungs regulate pH?
by adjusting breathing rate to control carbon dioxide levels
What happens when breathing increases(hyperventilation)?
carbon dioxide dcreases —> blood is less acidic
what happens when breathing decreases(hypoventilation)?
carbon dioxide increases —> blood is more acidic
Tidal volume(TV)
normal breathing (500 ml)
Inspiratory Reserve Volume(IRV)
extra air inhaled beyond normal breath
Expiratory Reserve Volume (ERV)
extra air exhaled beyond normal exhalation
vital capacity (vc)
tv + irv + erv
residual volume (rv)
air left after forceful exhalation
when does surfactant fully develop?
around 36 weeks gestation
new born characteristics?
nasal breather
when do lungs fully mature?
around 25 years
aging effects on lungs
decreases elasticity, weaker diaphragm, decreased gas exchange
effect of aging on immune system
increased infections
impaired gas exchange(nursing diagnosis)
reduced oxygen/carbon dioxide exchange —> asthma
ineffective airway clearance
blocked airflow due to secretions—> pneumonia
Upper Respiratory Tract Structures
nose, mouth, sinus, pharynx, larynx
Lower Respiratory Tract Structures
trachea, bronchi, bronchioles, alveoli
Cause of infection rhinitis(common cold)
usually viruses (rhinovirus)
incubation period for common cold
1-3 days(usually) ~ 7
Incubation period
time between exposure and symptoms
pnemonia
inflammation of lungs affecting alveoli, alveolar ducts and bronchioles
how does common cold spread?
hand to hand, hand to mucous membrane, contaminated surfaces
infectious agents(pneumonia)
bacteria, virus, fungi
non-infectious agents(pneumonia)
aspiration, smoke, toxins
most common bacterial cause(pneumonia)
streptococcus pneumonia
aspiration pneumonia
inhalation of gastic content intto lungs
risk for aspiration pneumonia
impaired gag reflex
Asthma
chronic pulmonary disease with intermittent, reversible airway obstruction
intermittent
occasional pain
effects on ventilation
airway inflammation—> swelling narrow airways
increased mucus production—> blocked airways
bronchospasm—> muscle tightening around airways
asthma symptoms
dyspnea, wheezing, cough, chest tightness, fatigue
status asthmaticus
life threatening, prolonged asthma attack unresponsive to usual treatment
risk factors for atelectasis
surgery and immobility
pathophysiology of atelectasis
collapsed alveoli—>poor ventilation—>impaired blood flow—> reduced gas exchange
clinical manifestation of atelectasis
dyspnea, tachypnea, cyanosis(severe), decreased breath sounds, hypoxemia
prevention of atelctasis
frequent mobility, coughing, deep breathing, incentive spirometry