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upper leg lameness is more common in
beef cattle
where in upper leg is commonly lame in cattle
stifle
what can happen in the stifle
cranial cruciate ligament lesions
associated with falling, slipping, and/or mounting
what are clinical signs of cranial cruciate ligament lesions
joint effusion/swelling, pain, crepitus
positive cranial drawer test
clicking sound
what causes arthrogryposis
genetics (angus, charolois, Holstein)
something toxic in utero
dam with viral disease (BVD, Bluetongue, etc)
what is arthrogryposis
congenital limb contracture
what is crooked calf syndrome
lupine induced arthrogryposis
induces fetal sedation so growth is deformed
what is chondrodysplasia
genetic disorder affecting cartilage development, results in dwarfism
can be caused by manganese deficiency
what causes contracted tendons
inherited factors
in utero malpositioning
contracted tendons is
common congenital problem in cattle
usually at carpus or fetlock
how to treat mild to moderate contracted tendons
PT
manual stretching
how to treat moderate contracted tendons
bandage, splint, cast
analgesia
how to treat severe contracted tendons
surgery (tenotomy)
what are hygromas
localized swelling of tissue
capsule filled with fluid surrounded by layer of fibrous tissues
what causes hygromas
intermittent trauma
lack of bedding
what causes osteochondrosis
blood supply failure → focal ischemia → subchondral bone cyst
what is ostochondrosis
affects metaphyseal growth plate and immature joint cartilage
affects beef breeds and young bulls
what are clinical signs of osteochondrosis
joint effusion
shifting weight lameness, pain at joint flexion
subchondral bone defect, cyst like lesions, and loose bone fragments on radiograph
osteochondrosis often progresses to
OCD and osteoarthritis
how is osteochondrosis treated
surgical debridement of lesion
what is upward patellar fixation
patella fixed on proximal aspect of medial trochlear ridge of femur
hind limb forced to remain rigidly extended
upward patellar fixation is more common in
horses
brahman cattle
how is upward patellar fixation treated
medial patellar desmotomy
what can cause gastrocnemius tear
intense struggling to stand
underlying selenium deficiency
usually tears at muscle/ tendon junction
what are clinical signs of gastrocnemius tear
dropped, severely flexed hock joint
hoc touches ground when attempt to stand
usually unilateral
how is gastrocnemius tear treated
provide support if standing
guarded prognosis is tear at muscle belly or complete transection of tendon
what is the function of peronius tertius
hock flexion and tarsal flexion
what causes peronius tertius tear/ rupture
excessive effort trying to rise on slippery surface
trauma with hyperextension of limb
mounting/ being mounted
what are clinical signs of peronius tertius tear/rupture
toe touching lame but not painful
inability to flex hock
limb pulled backward with little effort
what is elso heel (soastic paresis)
heritable neuromuscular disease in cattle
causes severe, spastic contraction of hind leg muscles
leads to permanently straight, overextended hind leg
what are clinical signs of elso heel
starts between 2-10 months of age
stiff pendulous leg
one or two limbs affected
gastrocnemius hard and rigid
how is elso heel treated
salvage
tenotomy of gastrocnemius and ½ of SDFT
tibial neurectomy
what is a common and serious cause of stifle lameness
cranial cruciate rupture/ tear
what causes CCL rupture
trauma (falling, poor footing)
secondary to DJD
post legged conformation (too straight at stifle)
what are clinical signs of CCL rupture/tear
very lame
outward rotation of leg
stifle joint effusion
clicking sound at walk or manipulation
how to treat CCL rupture/ tear
stall rest
NSAIDs
surgical repair by extracapsular imbrication and ligament replacement
if no treatment, poor prognosis
what is coxofemoral luxation
head of femur slips out of pelvic socket
what is a common orthopedic injury in cattle that is more prevalent in dairy
coxofemoral luxation
what direction luxation is most common for coxofemoral
craniodorsal
what causes coxofemoral luxation
trauma
periparturient hypocalcemia and dystocia trauma
what contributed to coxofemoral joint stability
joint capsule
femoral head ligament
gluteal muscles
deep hip muscles
what are clinical signs of coxofemoral luxation
NWB lameness
sudden onset
shortening of limb, dropped hip
crepitus on manipulation
how is coxofemoral luxation diagnosed
abduct leg while palpating
severe gluteal swelling
rectal palpation
how is coxofemoral luxation treated
closed reduction- difficult and high recurrence
open reduction (expensive)
FHO (expensive)
what causes spinal fractures/ compression
dystocia, falling, slipping
spinal abscess
lymphoma (BLV)
what are clinical signs of spinal fractures/ compression
downer
sitting dog
how to treat spinal fractures/ compression
supportive care and NSAIDs
what is obturator paralysis/ down cows
syndrome with variety of causes
unable to rise, primarily in hind end
more common in females
what can cause obturator paralysis in dairy cows
hypocalcemia***
accidents
dystocia
what can cause obturator paralysis in beef cattle
dystocia***
metabolic conditions with Ca and Mg
what causes obturator paralysis in small ruminants
hypocalcemia
ketosis
dystocia
what are the stages of obturator paralysis
inciting event
recumbency
further injuries from death of struggling
with downer cows
immediately determine if euthanize or receive additional care
what to remember when working with downer cow
no dragging
no lifting with chains
provide feed and water where they are
when to euthanize down cattle within 24-36 hours
unable to sit up unaided
refuse to drink or eat
no method to physically manage them
what PT/ support to give to downer cow
place in well bedded area
provide good footing
rolling, slings, hobbles
what is nutritional mypopathy
white muscle disease, stiff lamb disease
acute to subacute myodegeneration of cardiac and/or skeletal muscle
what can cause nutritional myopathy
dietary deficiency of selenium and/or vitamin E
what are clinical signs of severe selenium deficiency
white muscle disease
muscle stiffness
sudden death due to cardiac failure with no signs of sickness
what are signs are marginal selenium deficiency
abortions, retained placentas
reduced fertility
weak, stillborn, lethargic neonates
reduced growth rate and immune response
what is seen in WMD cardiac form
exercise associated, acute respiratory distress
pulmonary edema with pink foam
weakness, irregular HB
concurrent skeletal form
what is seen in skeletal form of WMD
stiff gait, bunny hopping
difficulty standing, muscles hard
dyspnea
lesions are symmetrical!!
weak calves at birth
how is WMD treated
supportive care and rest
prevent secondary infections
vitamin E (no toxicity) and selenium (toxicity)
how to prevent WMD
provide selenium!