BU - PS371
4 Ds
deviance, danger, distress, dysfunction
morbidity
state of having a specific illness/condition
comorbidity
state of having 2 or more illnesses/conditions
incidence
percentage of new expression of given condition within target period
prevalence
any expression of given condition
genotype
genetic information
phenotype
what's physically expressed
allele
one or more alternate forms of a gene that arise by mutation, found at same place on chromosome
heritability
proportion of presence within a given population (believed to be due to genetic variation)
validity
extent to which what's being measured is measuring what it's supposed to - is it accurate?
internal validity
confidence that variables of interest (independent variables) produce specific outcome of interest (dependent variables)
external validity
how well findings relate to real world conditions
reliability
consistency of a measure - can same results be reproduced? is measure consistent?
internal reliability
extent to which measure is consistent within itself, split-half method
split-half method
measures extent to which all parts of test contribute equally to what's being measured
external reliability
extent to which a measure varies from one use to another
test-retest
measures stability of test over time
inter-rater
degree to which different raters give consistent estimates of the same behavior
nomothetic
based on peer-based norms (most common in present)
idiographic
assessments based on individual (more common in past)
sign
indicator of condition as observed by others, not individual, objective, estimated visually or with instruments
symptom
indicator of condition as perceived by individual, subjective, cannot be estimated/quantified
syndrome
condition characterized by specific set of symptoms
correlation
extent to which two variables are connected
causation
extent to which one variable causes another
does correlation equal causation?
NO!
morbidity
relating to state of being ill or diseased
comorbidity
more often than not simultaneous presence of one or mother additional illnesses/disorders
incidence
percentage of new expression of given disease within target period
prevalence
percentage within certain population of any expression of given disorder within target period
presumed casual factors
nature, nurture, integration
emphasis on independent and interrelated biological, social, and psychological factors & the way they interact with one another
developmental factors
high-risk periods, at-risk populations, critical periods? age-related factors that influence possibility of disorders?
treatment-related factors
options/treatments of choice, psychological vs. biological vs. other treatment options, what's best for whom & when?
paradigm
model or framework for which to view a phenomenon
philippe pinel
humane treatment of mentally ill during french revolution
william tuke
founder of the retreat at york, humane treatment of mentally ill, "moral treatment"
dr. benjamin rush
among first of americans to believe that mental illness was "disease of the mind" and not demonic possession, "father of american psychiatry"
dorothea dix
advocated for humane treatment of people with mental illness in asylums in america
emil kraepelin
classification system for identification of mental illness, credited with first record-keeping system in mental hospitals
key figures - psychodynamic
freud, jung, adler, horney
characteristics - psychodynamic
maladaptive behavior is the result of unconscious unresolved conflict. pathology results as net observable effect of unmet needs
theoretical foundation - psychodynamic
case studies, correlational research
temporal focus - psychodynamic
historical
research support - psychodynamic
moderate
key figures - behavioral
skinner, pavlov, watson, thorndike
characteristics - behavioral
all behavior is learned, pathology results as maladaptive ways of coping with experience
theoretical foundation - behavioral
experimental-based
temporal focus - behavioral
ahistorical
research support - behavioral
strong
key figures - cognitive-behavioral
beck, seligman, bandura, maultsby, linehan
characteristics - cognitive-behavioral
maladaptive thoughts lead to maladaptive behavior - cognitive errors can be unmasked and modified
theoretical foundation - cognitive-behavioral
animal research, case studies, experimental human research
temporal focus - cognitive-behavioral
historical and contemporary
research support - cognitive-behavioral
strong
key figures - humanistic/existential
maslow, rogers, satir
characteristics - humanistic/existential
pathology results from difficulty finding pleasure/fulfillment
theoretical foundation - humanistic/existential
subjective, mostly self-report
temporal focus - humanistic/existential
present, future
research support - humanistic/existential
weak
key figures - multicultural/sociocultural
wing sue, sue, linton, parham, white, comas-diaz, landrine
characteristics - multicultural/sociocultural
maladaptive responses to conflicts between dominant and "minority" cultural norms
theoretical foundation - multicultural/sociocultural
interpersonal dynamics, sociology, anthropology, political science
temporal focus - multicultural/sociocultural
historical and contemporary
research support - multicultural/sociocultural
modest
historical perspective disease model
each disorder has clear underlying physiological cause
genome-wide associated studies (GWAS)
identify specific genetic markers believed to be associated with psychiatric disorders
common disease common variant (CDCV)
many genes contribute to association with disorder (most likely)
multiple rare variant (MRV)
few genes (multiple rare variances) contribute to association with disorder
genetic linkage analyses
examine traits with known genetic coding as potential markers of disorder
twin studies
prevalence of schizophrenia in identical vs. fraternal twins
adoption studies
twins raised separately w/o biological parents instead of together
family studies
expression of traits among relatives as a function of degree of relatedness
diathesis-stress model
diathesis = physiological vulnerability to expression of disorder
risk for expression determined by stress, but stress not constant throughout life
reciprocal gene-environment
more dynamic than diathesis-stress model, takes genetically encoded traits into consideration (increase/decrease probability of encountering stressful experiences)
cross-sectional
study of effects/relationships over time by recruiting groups of individuals (eg. grouping by age)
longitudinal
study of effects/relationships over time, across multiple assessment sections
case studies
detailed exploration of rare occurrences, help generate hypotheses
single-subject experiments
allows testing of causal hypotheses
single-person study flaws
susceptible to bias, difficult to generalize
population studies
get incidence, prevalence across population, eg. kessler et al
group experiments
existing or created for experiment, correlational and comparative
multiple person experiments pros and cons
easy to repeat, enhance external validity, but don't address causal factors
observational data
purely raw observation, nothing in manipulated
experimental data
intentional manipulation of variables in experimental environment
clinical trial phase i
evaluation of safety, open label (no deception), may represent first human use, very small samples
clinical trial phase ii
evaluate usefulness, risk, side effects, dose - placebo controlled, somewhat larger sample group, typically blinded - always at least single blinded, sometimes double blinded - compare study drug to placebo
clinical trial to phase iii
establish efficacy, compare to other treatments - blinded, random assignment to treatment groups
independent variable
variable that's being manipulated
dependent variable
variable that changes as outcome of manipulation
single blind study
one party (subject/experimenter) is unaware whether they are in the experimental or control group
double-blind study
neither party is aware which is the control group and which is the experimental group
statistical significance
results are reliable/statistically significant (p=.05)
clinical significance
contextualization of results - overall effect on well-being, health, functionality
purpose of assessment
background, diagnosis, track treatment goals and change if needed, select research subjects
concurrent validity
ability for a test to predict event in the present - does it line up with another measures known to measure outcome?
predictive validity
ability for a test to predict event in the future - does measure accurately predict outcomes that we would expect?
standardization
process in which procedures used in research are kept the same
bias
we want none of it. get that shit outta here