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What is apraxia of speech?
neurogenic speech sound disorder due to sensorimotor problems in positioning and sequentially moving muscles for the volitional production or speech.
theres a slower rate of speech, distorted speech sound subs, syllable segmentation, articulatory groping, false starts, prosodic impairments, increased errors with longer utterances
unimpaired reflex and automatic acts. no muscle weakness.
different from nonverbal oral apraxia, which is a disorder of nonverbal movement involving oral muscles.
What are some causes of apraxia of speech?
lesions to brocas area, primary motor areas, or supplementary motor area. primary progressive apraxia of speech (caused by degenerative disease, like alzheimers, MS), left hemisphere trauma, surgical trauma, tumors and seizures
What are communication deficits seen in patients with AOS?
Initiation may be slowed or delayed
May use compensatory strategy of reduced rate
Problems in volitional sequencing of movements required for speech
Variability of speech errors
Substitutions more commonly seen than distortions and omissions
Anticipatory substitutions
Post positioning errors (dred for dress)
Metathetic errors (tefalone)
Increased frequency of errors on longer words
Groping and struggling
Attempts at self correction (unsuccessful)
Slower rate of speech
Silent pauses between words
Impaired intonation
Are pts who have AOS aware of their deficits?
Yes
What's the difference between apraxia of speech and primary progressive apraxia of speech?
PPAOS is considered "pure" AOS, where it's the only symptom and the onset is insidious.
How do you assess AOS?
Detailed case history
-Careful examination of medical records
-Interview client/family
-Detailed observation of speech production
Tape record patients speech samples and transcribe responses taking note of groping, self correction, etc
Getting them to repeat speech sounds and patakas
Getting them to repeat progressively longer words
Getting them to repeat words and phrases and sentences
Getting them to count
Picture descriptions
Oral reading
Oral test
Assess limb apraxia
Standardized test such as the APRAXIA BATTERY FOR ADULTS
What do you target in AOS treatment?
Behavioral treatment is most effective
-Initially target easily produced words
-Focus on speech movements
-Drill a variety of sounds and sound targets
-Include articulation, slow rate, prosody
-Emphasize total communication (verbal expressions, gestures, writing, AAC)
-Self-monitoring skills
-Techniques used for aritic and phono
What should treatment procedures include in AOS?
Instruction
Demonstration
Modeling
Shaping
Phonetic placement
Frequent cueing
Use of rhythm
Immediate feedback
What cueing techniques should be used for AOS?
Tactile
Simultaneous production by the clinician and client
Clinician modeling followed immediately by client imitation
Delayed imitation
Carrier phrases
Singing
Phonetic contrasts
What is Sound Production Treatment?
A therapy approach for AOS. Emphasizes teaching articulation of words with minimal contrast.
What is dysarthria?
Neurological based speech disorder characterized by abnormal strength, speed, range, steadiness, tone and accuracy. Affects all aspects of speech production, including respiration, phonation, articulation, prosody and resonance.
What causes dysarthria?
non progressive conditions (stroke, infections, TBI, etc), degenerative neurological conditions (parkinsons, ALS, MS), trauma, infections (AIDS), toxic-metabolic (drugs, encephalopathy, etc)
Where is the lesion for ataxic dysarthria?
Damage to the cerebellar system. So, coordination of muscle movements is difficult.
What speech deficits do you see in ataxic dysarthria?
Respiration
-exaggerated and paradoxical movement during speech
Phonation
-monopitch, mono loudness, harshness
Resonance
-hyponasality (only in some)
Articulation
-imprecise production. irregular breakdowns. distortion.
Prosody
-excessive and even stress. prolonged phonemes and intervals between words/syllables. slow rate of speech
Other
-gait disturbances: instability of trunk and head, tremors and rocking, rotated head posture, hypotonia
-movement: over and undershooting. uncoordinated, slow, imprecise movements
Where is the lesion for flaccid dysarthria?
Damage to lower motor neurons. These are the connections that go from spinal and cranial nerves to muscles. It may only affect one muscle or a group of muscles.
What neurological conditions may cause flaccid dysarthria?
ALS, MSA, myasthenia gravis, demyelinating diseases, trauma, infections, strokes, etc.
What speech deficits do you seen in flaccid dysarthria?
Respiration
-reduced subglottic air pressure, weak inhalation
Phonation
-breathy voice, audible inspiration, short phrases
Resonance
-hypernasality, nasal emissions
Articulation
-imprecise consonants
Prosody
Other
-weakness, hyptonia, atrophy, diminished reflexes, fasciculations (which are isolated twitches of resting muscles), rapid weakness of muscle
Where is the lesion for hyperkinetic dysarthria?
Damage to the basal ganglia. It may affect muscle tone and involuntary movements. Mainly prosody.
What speech deficits do you see in hyperkinetic dysarthria?
Respiration
-Audible inspiration or forced and sudden inspiration/expiration.
Phonation
-Voice tremor, intermittently strained, vocal noise, harsh, loudness variations
Resonance
-Sometimes hyper nasality, but mild
Articulation
-Imprecise consonant productions, distorted vowels, slower
Prosody
-Prolonged inter-word intervals, silent periods, phoneme prolongations, excess and equal stress, mono pitch, monoludness, reduced stress, short phrases
What is/are ______________
-orofacial dyskinesia
-myoclonus
-tics
-chorea
-athetosis
-dystonia
-tremor
All associated with hyperkinetic dysarthria
-orofacial dyskinesia: abnormal, involuntary movements of the orofacial muscles
-myoclonus: involuntary, rapid jerks of body parts. like hiccups
-tics: commonly of face and shoulders. typically patterned, rapid and stereotyped.
-chorea: purposeless, random involuntary movements of body parts
-athetosis: slow, writhing, purposeless movements.
-dystonia: contractions of muscles that cause abnormal posture.
What is spasmodic torticollis? blepharospasm?
spasms of the neck muscles
forceful and involuntary closure of eyes
Where is the lesion for hypokinetic dysarthria?
basal ganglia. most common cause is Parkinson's.
What speech deficits do you see in hypokinetic dysarthria?
Respiration
-Reduced vital capacity, irregular breathing
Phonation
-Monopitch, low pitch, monoloudness, harsh, breathy
Resonance
-Hypernasality in some
Articulation
-imprecise or distorted consonants
Prosody
-reduced stress silent intervals, short rushes of speech, variable and increased rate in segments, short phrases
Other
-Tremors, but diminished when moving voluntarily
-Mask-like-face
-Micrographic writing
-Walking disorders (slow initiation, rapid, shuffling steps)
-Postural disturbances characterized by difficult changing positions
-Decreased swallowing and accumulation of saliva in mouth
Where is the lesion for spastic dysarthria?
BILATERAL damage to UMN.
What speech deficits do you see in spastic dysarthria?
Respiration
Phonation
-Hyperadduction of VF
-Breathy
-Harsh
-Pitch breaks
-Strained/ strangled
Resonance
-Hypernasality
Articulation
-Imprecise production
Prosody
-Excess and equal stress, mono, reduced stress
Other
-Spasticity and weakness of face
-Reduced range
-Increased muscle tone
What type of dysarthria is commonly associated with ALS?
Mixed flaccid-spastic
What type of dysarthria is commonly associated with MS?
ataxic-spastic
Where is the lesion in unilateral upper motor neuron dysarthria?
Upper motor neurons that supply cranial or spinal nerves.
What are speech deficits associated with UUMN dysarthria?
Respiration
Phonation
-harsh voice, reduced loudness, strained harshness, wet hoarseness, breathiness
Resonance
-hypernasality
Articulation
Prosody
-slow rate, excess and equal stress, mono pitch, mono loudness
Other
-unliateral lower face weakness, tongue weakness, palatal weakness
-hemiplegia
How do you asses dysarthria?
Complete case history
-Examine medical records
-Interview patient/family
-Record a conversation and reading sample
-Imitation tasks
-Sustained vowel
-Diadochokinetic rates
-Assess speech production mechanisms, respiratory problems, phonatory problems, articulation, prosody, resonance, speech intelligibly, muscle strength, range, accuracy, tone and steadiness
-Use standardized measure like Frenchay Dysarthria Assessment
How do you assess the speech production mechanism during a dysarthria assessment?
Observe facial symmetry, tone, tension, droopiness, expression, etc
Observe facial structures as the patient puffs cheeks, retracts and rounds lips, bites lower lip, etc
Note jaw movements and deviations
Observe VP mechanism
Assess nasal airflow by holding a mirror at the nares as the patient prolongs the vowel /i/
Assess laryngeal function by asking the patient to cough
How do you assess respiration during a dysarthria assessment?
Observe patients posture and breathing habits during rest and speech
How do you assess phonation during a dysarthria assessment?
ah prolongation task
take note of pitch, breaks, diplophonia, loudness
take note of voice tremors
take note of quality
How do you modify respiration when treating dysarthria?
Teach consistent production of subglottic air pressure
Teach maximum vowel prolongation
Teach controlled exhalation
Teach silent to push, pull during speech tasks
Modify posture
How do you modify phonation when treating dysarthria?
Using biofeedback
Portable amplification
Artificial larynx
How do you modify resonance when treating dysarthria?
Mirror
Open mouth wider
Nose clip
How do you modify articulation when treating dysarthria?
Bite block for jaw control
phonetic placement
instruction -> demonstration -> modeling -> shaping -> immediate feedback
compensatory skills
How do you modify speech rate when treating dysarthria?
delayed auditory feedback
pacing board
metronome
hand tapping
How do you modify pitch when treating dysarthria?
Visi-Pitch