Nutr 484 actual exam 1

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122 Terms

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Exercise
a planned structure of repetitive physical activity
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Physiology
The study of processes and functions of living organisms
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Together
The study of how and why the body responds to exercise

\-effective and efficient exercising

\-results body comp

\-minimize risk of injury
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Health components of physical fitness
Cardio respiratory endurance

Body comp

Muscular strength

Muscular endurance

Flexibility
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Skill components
Agility

Coordination

Balance

Power

Reaction time

Speed
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Types of exercise
Aerobic and anaerobic
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Aerobic
Endurance exercise such as cardio running cycling swimming etc
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Anaerobic exercise
Springing resistance training (concentric vs eccentric) plyometric training
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SAID principle
Specific

Adaptations to

Imposed

Demands

Basically adapt to how you train
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How much exercise ?
150 minutes per week of moderate intensity

Or

75 minutes per week of vigorous intensity
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Health benefits
Children and adults

\-increased muscular fitness

\-improved bone health

\-favorable changes in body comp

\-improved mood and mental health

Adults

\-reduced risk of premature death

\-Reduced risk of CVD risk factors and diseases

\-enhanced weight management

\-Reduced risk of cancer
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Prescription of exercise
FITT-VP

Frequency

Intensity

Time

Type

Volume

Progression
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Smooth muscle
Non striated

Autononomic nervous system

\-parasympathetic branch
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Cardiac muscle
Striated

Autonomic nervous system

\-parasympathetic brand -inhibits

\-sympathetic branch - excites
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Skeletal muscle
Striated

Somatic nervous system

\
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Epimysium
Outer layer
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Peri my sim
Middle layer of connective tissue surrounding the vascular
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Endomysium
Inner most layer surrounding the individuals muscle fibers
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Myofibril
Myosin filament - thick and dark

Actin filament - thin and light
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Sarcomere
Functional unit of a muscle fiber
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Z line
Beginning and of the sarcomerer

One z line to the next
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M line
Through the middle of myosin filament
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H zone
The region between two different actins
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A band
Runs the length of the myosin filament

Gives striated effect
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I band
Region from one myosin filament to the next
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Muscle contracting
Z lines come together

Z I H all shrink

Length of actin and myosin doesn’t change just slides
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Sliding filament theory
Action potential starts in pre central gurus (motor cortex)

Through pyramidal tracts

Through ventral horn

At neuromuscular junction

* Releases acetylcholine


* binds to the receptor which initiated action potential
* ACHE into the synaptic cleft , degrades the ACH and reopens the receptors for next stimulus
* Calcium into sarcoplasm
* Calcium binds to troponin (on actin covered by troop-myosin)
* Contract is going to occur

Synapses with lower motor neuron

* 85% cross over
* ADP and P on myosin head cause swivel and contraction process
* Tropnin and tropomyosin complex shift back if no more action potential, muscles relax
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Type 1 Slow twitch oxidative (SO)
Slow contraction

Weak force production

Fatigue resistant
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Type 2A fast twitch oxidative glycolysis (FOG)
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Type 2X Fast twitch (FG)
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Factors affecting force production
Size of muscle fibers

Number of motor units

Type of motor unit

Rate coding

Speed of muscle contraction

Angle of pull

Initial muscle length

Sally Never Touched Randys Spoon and Igloos
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All or nothing principle
Must hit threshold for response

Once threshold is hit it is a FULL response
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Fatigue
During prolonged exercise when physical performance starts to decline
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Central fatigue
Proximal to the neuromuscular junction

Insufficient binding of ACH
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Peripheral fatigue
Distal to the neuromuscular junction

In the muscle itself

Accumulation of metabolic byproducts

* Hydrogen
* Inorganic phosphate

Decreases pH and ability to produce force
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Accumulation hypothesis
30 seconds to three Minutes

Build up of metabolic byproducts

Lactate is not that bad

\-Does accumulate, but does not cause this

Hydrogen lower the pH

\-When it gets to 6.9 inhibits glycolysis

How to help

\-Bicarbonate loading

— reduce acidity in cellular environment

— favors diffusion of lactate
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Depletion hypothesis
Prolonged exercise

No fuel source to produce ATP

\-phosphagen

\-glycogen (2000 stored)

How to help

\-Carbohydrate loading 70% diet

— go to increase muscle glycogen stores before competition

In combination with tapering
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Blood
55% plasma

\-Mainly water with a little protein

Buffy coat

\-White blood cells

\-Platelets

Red blood cells erythrocytes 45%

\-hemoglobin, carrying oxygen

\-clotting and healing factors
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Cardiac output
The amount of blood pumped by the heart per minute

\-At rest 5000 mL

\-during exercise 25,000 mL

\-Heart rate and stroke volume affect

\-HR x SV
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Sinoatrial node (SA)
Pacemaker of the heart

Conduction starts in the snowed

60-100 beats per minute
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P-wave
Contraction or depolarization of the atria
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QRS complex
Ventricular depolarization contraction of the ventricles
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T-wave
Ventricle repolarization back to resting state
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First sound lub
Closing of the valves

Systolic
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Second sound dub
Opening of the valves

Diastolic
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Blood pressure
Systolic top number contraction of the ventricles, 120

Diastolic bottom number, relaxation of the ventricles to allow blood to refill 80

End, diastolic volume-the amount of blood left in the ventricles after contractions

End systolic volume-amount of blood left in the ventricles before contractions
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Stroke, volume
Amount of blood per contraction of the heart

End diastolic-and systolic usually 50 to 60%

Preload-stretching of the walls of the heart

Greater stretching equals greater contractility

More blood left in the heart more blood expelled
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Heart rate
Increases with exercise

Even Krisa is a little before beginning an exercise
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Cardiovascular drift
Prolonged exercise at a low threshold

Increases in heart rate without changing in work rate
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Stroke, volume
Decreases, so cardiac output can stay the same

To offset the heart rate increase

Increases to 40 to 60% max effort
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Cardiac output
Stays the same

Only way to increase cardiac output is increase heart rate
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Excess post exercise oxygen consumption (oxygen debt)
You’re in recovery and O2 remains elevated

Needed to replenish the stores that you used

Temperature regulation

Helps clear waste products
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Aerobic threshold
Heart rate at about 150

Energy metabolism becomes mainly anaerobic
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Anaerobic threshold
This is the ability to remove lactate from the area
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Lactate threshold
Same point of anaerobic threshold

Accumulation exceeds removal
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Ventilatory threshold
Idk but breathing
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Onset blood lactate accumulation(OBLA)
Specific point of four mmol of blood
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Oxygen, hemoglobin, disassociation curve
98% of O2 bound hemoglobin

2% plasma

Bohr effect -rightward shift in curve

\-Get oxygen to working muscles more efficiently

Normal to leftward shift is rare
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Steady state, VO2
Plateau of O2, during exercise at two minutes below threshold

02 deficit
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Slow component VO2
Two minutes above threshold, VO2 never steady states

Continuous increase after two minutes above
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Drift VO2
Continuous increase at below threshold
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Max VO2
The ability to consume transport and utilize oxygen
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VO2
Q xA-V VO2 diff

\-flicks equation

\-Ficks law-rate of diffusion is proportional to surface area of a difference in partial pressure of gases (directly related)

\-Inversely related to the thickness of the barrier of the wall

\-Q equals cardiac output

\-Difference in oxygen Contant, an arterial and venous blood
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Absolute VO2
L/min
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Relative VO2
Ml/kg/min

Anytime kilograms is present it is relative to their body weight

Relative = absolute x 1000 / BW (in kg)

Girls 40-45

Boys 45-50
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Factors affecting vo2 max
Mode of exercise

\-bike or treadmill

\-higher vo2 on treadmill compared to bike

—more muscle being used

Genetics

\-proportional of slow to fast twitch muscle fibers

Training

\-25%

Gender

\-women’s vo2 max is 15-32% lower

Body size and composition

\-lower % body fat - higher vo2

Age

\-peak at about 25-30 years

\-35 begins decline
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Nervous system
Sensations

Movement

Proprioreception

\-where body is in space

Posture

Balance

Involuntary activities of living
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Neuron
Starts at dendrite through axon to the nerve ending
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Divisions
Anatomical

Functional
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Central division
Brain and spinal cord
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Peripheral
Any other nerves outside of the brain and spinal cord

Innervates muscles and glands
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Parasympathetic
Slows

Inactivity

Rest and digest
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Sympathetic
Excite

Fight or flight

Increase heart rate s
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Somatic
Afferent neurons - sensory

Efferent neurons -motor
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Proprioception vestibular receptors
In semicircular canals

Endolymoh

Disturbs the crista through 8th cranial nerve

Rotational information

* acceleration
* Deceleration
* Twisting

Otoliths
* Linear accelerations
* Where we are in space
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Kinesthetic receptors
Movement and position of specific body parts in space re
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Reflexes
Involuntary movements to a sensory stimuli s
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Spinal reflex

\
Minimum 2 neurons
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Myostatic (stretch) reflex
2 neurons
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Flexion reflex
3 neurons
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Reciprocal inhibition
Idk
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Crossed extensor reflex
Activation of contra lateral
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Muscle spindle directly on the belly parallel to extra frusta fibers
Respons to stretch
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Golgi tendon not parallel just lies in a series

\
Allows to be activated by passive stretching And active shortening of the muscle or tendon

Only a sensor neuron
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Fundamental principles
Stimulating structural and functional adaptations to improve performance pr
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Progressive overload
Achieving appropriate overload requires manipulating training
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Anaerobic training
Resistance training

Plyometrics

Speed and agility

Interval training
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Neural factors
Learning affect

Motor unit adaptations

\-increases requirements

\-increased firing rate

\-synchronization

Adaptations at the neuromuscular junction

\-increased surface area

\-dispersed irregular shaped synapses and length of nerve terminal

\-increased end plate perimeter length

Enhanced reflex responses

\-enhance magnitude and rate of force development

\-muscle spindles

Attenuated inhibition

\-golgi tendon organs
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Hypertrophy
Increase in cell size

Skeletal muscle remodels sits internal architecture which could change external

Remodeling leads to increase in cross sectional area

Increase in size of muscle fibers

\-type 2> type 1

Increase in contractile protein
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Increase in protein synthesis
Testosterone

Growth hormone

Insulin

Insulin like growth factor 1 s
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Structural changes
Increased myofibril volume

\-more sarcomeres added

Increased angle of pennation

Reduced mitochondrial density

Satellite cells between basal laminate and sarcomeres
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Muscle cells remodeling
Skeletal muscles represent dynamic tissues

Muscle fibers undergo regeneration and remodeling to alter their phenotypic profile

\-stimulation of myogenic stem cells situated under a muscle fibers basement memebrane

Specific training can transform muscle type

\-become more efficient

Satellite cells incorporate into existing muscle fibers

Signals determine myofilament proteins to be made
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MTOR pathways

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Resistance training for protein synthesis
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MAPK
Resistance training for transcription factors for hypertrophic gene expression
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Absolute amount of hypertrophy represents the primary difference

\
Men experience greater absolute change in muscle size from their larger initial size

Enlargement as a percentage basis remains similar between sexes
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Strength vs hypertrophy
Strength is proportion to CSA

S 1 to 6 reps technique improved

Neural

\-Golgi tendon bodies

\-Recruitment patterns

Training specific

H 6 to 12 or 15 reps over 65%

Goal is to increase training volume

Must go to fatigue

Greater volume training

\-Requires less weight

\-No difference to strength if the volume is matched

Requires volitional fatigue
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Connective tissue
Mechanical force is Alyssa adaptations proportional to intensity

Increase in

\-Collagen, fibers diameter

\-Covalent crosslines between fibers

\-Collagen fibers

\-Density of collagen fibrils

Adapt, much more slowly than muscle
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Bone density
Positive relationship between strength and bone mineral density

Strength and power activities have much more bone mass than endurance athletes

Linear relation exists between increase bone density, and total strength training program

Trabecular response, faster than cortical

Minimal essential strain approximately 1/10 fracture force
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Long term aerobic training increases I heart mass and volume
Greater left ventricle end diastolic volume during rest and exercise E
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Eccentric hypertrophy n
Elongated cardiomyocyte

Adding sarcomeres in series similar