psyc3010

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Last updated 5:28 AM on 6/5/23
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95 Terms

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RM variable
________ treated as multiple DVs and combined /weighted to maximise difference between levels of other variables.
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covariate
________ is continuous control variable known to be associated with DV.
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ANOVA
________ cannot detect non- linear relationships, reduces power.
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rho
________ or radj is population coefficient, compared to r.
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moderator
________ enhances or attenuates relationship between IV /predictor and DV /criterion.
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levels of WPF
Variance and covariance are the same at all ________ (often violated)
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block
________ (trial) and group are both fixed factors; participant is not fixed.
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error used for any effect in RM ANOVA is = to
the interaction between that effect and the effect of participants (applies to main and simple effects and their follow up tests)
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DV
Interaction: effect of A on ________ changes over levels of B.
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heteroscedasticity
________: variance of Y values are consistent across yhat values (homogeneity of variance)
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Semipartial variation
________ (spr2)= proportion of variance in DV uniquely explained by IV.
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participant reacts
Contrast- previous treatment sets standard to which ________ (more broad than sensitisation)
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regression
Moderated ________ asks if XY interaction significantly contributes to prediction of Y.
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Orthogonal contrasts
________: variance is partitioned without overlap, use different portions of variance to avoid inflating t1 error.
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main effect
are the means of the population corresponding to each level of the factor different
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variance
spread of scores around mean
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MSerror
pooled within-cell variance (SSerror/DFerror)
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E(MSerror)
long term average of variance in each sample = population variance
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orthogonal contrasts
variance is partitioned without overlap, use different portions of variance to avoid inflating t1 error
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block 1
predict DV from IV (IV entered)
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contrast
previous treatment sets standard to which participant reacts (more broad than sensitisation)
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adaptation
adjustment to previous treatment changes reaction to next (more broad than habituation)
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direct carry-over
learn something in previous trials that is applied to latter ones
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within participant variability
(variance of participant outcome over IV trials)
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interaction
effect of A on DV changes over levels of B
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regression intx
relationship between X and Y varies over values of Z (moderator)
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error used for any effect in RM ANOVA is = to
the interaction between that effect and theĀ  effect of participants (applies to main and simple effects and their follow up tests)
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one-way factorial design
are the means from each level of the factor different from the grand mean (each other)

independent t test or one way ANOVA
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two-way factorial design
combine 2 one-way designs, every factor is crossed

is there a main effect of factor 1 or 2, or an interaction

2 way ANOVA
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structural model of one-way ANOVA
DV score= grand mean + effect of a treatment factor (j) + error for i person in treatment (j)
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structural model of two-way ANOVA
DV score = grand mean + effect of treatment (j) at factor (A) + effect of treatment (k) at factor (B) + effect of differences in factor A treatments at different levels of factor B treatments + error for (i) person in j and k treatments
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assumptions of ANOVA
population normally distributed with homogeneity of variance

samples are random and independent

interval/ratio data
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eta squared (Ī·2)
proportion of variance in sample DV accounted for by effect

easy to interpret, most common

biased estimate of true variance in population

sseffect (e.g. AxB) /sstotal
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omega squared (Ω²)
proportion of variance in population accounted for by effect

less biased, more conservative

however usually same or similar to eta squared depending on sample size (if n = >20)

larger error variance, smaller sample size = bigger diff. between eta and omega sq

(sseffect-(dfeffectxMSerror))/sstotal+MSerror
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partial eta squared (pĪ·2)
proportion of total variance (residual variance) in sample accounted for by investigated effect

model removes (controls for) variance attributable to other effects + interaction

compare variable A to variance attributable to A + error

inflated, gives larger effect size

values for each effect are not comparable (and may add up to >1), cannot make meaningful comparisons

useful only when only have controls and 1 focal IV but often still report n2
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simple simple comparisons
follow up significant simple simple effects with >2 levels

t test and linear contrasts (+- 1SD from mean)
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2 way interaction
the effect of one factor changes depending on the level of another factor (averaging over levels of a third factor)

AKA simple effects not the same
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3 way interaction
the two-way interaction between two factors changes depending on the level of the third factor
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SALE - reduce error, increase power
increase **s**ample, increase ***a*** level, **l**arger effects, decrease **e**rror variance
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4 ways to reduce error
improve operationalisation of variables

improve measurement of variables

improve design

improve methods of analyses
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Cohen’s d
measures effect size (overlap of distributions between groups)

closely related to power
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concomitant variable
control variable (interval) closely associated with DV, increases power by removing error variance
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blocking design set up
divide people into groups (blocks) according to control variable (e.g. IQ) known to be associated with DV

people within blocks each randomly assigned to different levels of IV (stratified random assignment)
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blocking design positives/drawbacks
* benefits
* may equate treatment groups better (assuming equal n for blocking levels)
* more power
* can check interactions of treatment and blocks
* limits
* more expensive
* loss of power if blocking variable is not correlated with DV (fewer df error, higher fcrit)
* artificial grouping due to arbitrary levels of blocking IV may result in loss of information (e.g. IQ hi, med, lo)
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covariance
multiply together cross products of deviation scores

is scale dependant, need scale info to answer questions on association
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pearson’s *r*
relationship between 2 variables in terms of stdevs (average cross product of standardised scores)

standardised but biased to sample
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*rho* or radj
population coefficient, compared to *r*
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*r*2
standardised, tends to be overly liberal (high) with small samples
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regression
estimating scores on variable on the basis of scores of a predictor variable

ā€œregress DV on IVā€
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linear regression equ.
y = ab+x

y = mx+c

Y^ = š‘1š‘„ + š‘0

Y^ = š‘X + š‘Ž
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standardised linear regression equ.
zY = β1š‘§X + β0
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standardised Beta
unstandardised beta change from units to standard deviations

B = Zscore change in Y predicted by 1 SD increase in X
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standard error of estimate
bigger correlation = smaller

how scores are expected to cluster relative to line

x percent of people expected to be within \~ x SDs of regression line

underestimated for small samples
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ANCOVA design
adding an extra IV (post hoc) that is known to account for changes in DV in a study where there is a large amount of unexplained variance (reduce error)

covariance is tendency of two scores to vary together

covariate is continuous control variable known to be associated with DV

can be applied to any ANOVA design
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difference between ANCOVA and blocking
blocking at design level

ANCOVA post-hoc error term adjustment
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ANCOVA test conducted
effects of covariate are subtracted from error term, then treatment means are adjusted to account for (remove) differences in covariate

if there isn’t same mean on covariate, it is a confound and removed from data (partial out/control for effects of covariate from focal IV and error term)
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issue with ANCOVA
only done on the assumption there should be no differences in groups due to random assignment

if covariate unrelated to DV, error term is reduced and power not increased (increases t2 error)
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assumptions of ANCOVA
DV is normally distributed in population, homogeneity of variance

random sample

linear relationship between DV and covariate, overall and within each group

homogeneity of regression slopes

* no IV x covariate interaction
* relationship between DV and covariate is same in each group
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strengths of ANCOVA
ability to analyse continuous variables

splitting into groups causes loss of info and increase in error

better than blocking if variables are continuous and is applied correctly
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multivariate regression
variation as function of multiple predictors acting together

predictors are correlated and contribution overlaps

have to remove overlap to accurately understand effect of variance
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chronbach’s A
indicator of internal consistency for 2 items on continuous scale

how well items hang together

scales should have A > .7 to reduce error
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collinearities
intercorrelations among predictors

smaller is better, less error

to maximise R2 should have high validities, low collinearities
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SMR
all variables entered simultaneously

each predictor evaluated in terms of what it uniquely adds to prediction (unique variance)

model r2 evaluated in 1 step
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HMR
predictors are entered sequentially in prespecified order based on logic/theory (a priori, predictors can be entered singly or in blocks

SPSS outputs r2 and r2 change for each step

fuller model = variables added, reduced model = without added variables

*each* predictor evaluated on what it adds to prediction at point of entry, model r2 assessed in multiple steps
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assumptions of multiple regression
population normally distributed

heteroscedasticity: variance of Y values are consistent across yhat values (homogeneity of variance)

no linear relationship between yhat and errors of prediction

independence of errors
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moderator
enhances or attenuates relationship between IV/predictor and DV/criterion
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mediator
indirect relationships between IV and DV via 3rd variable

accounts for the underlying mechanism or process between those variables
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bootstrapping
take sample and assume it was taken from population without bias

create new samples based on that data

report results with larger set of data

uses CI, not p
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assumptions of mediation
SMR shows IV is related to mediator (path A)

IV must be associated with DV (path C, though not necessary for indirect effect to be present, esp. with suppression models)
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assumptions of WP ANOVA
sample is randomly drawn from population (often violated due to convenience samples)

DV scores are normally distributed in population

compound symmetry
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sphericity
determines where variance and covariance is roughly equal – is sig. if assumptions are violated

often not sig. when assumptions are violated (not robust)

if sphericity is violated, F is positively biased (t1 error)
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when does sphericity not matter
between-participants (as unrelated treatments)

when within-participants designs only have 2 levels

only 1 covariance
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epsilon adjustment
change Fcrit by adjusting df'

e is no. by which Fcrit is multiplied

is 1 when sphericity is not violated

smaller = more conservative

greenhouse-geisser is most common e adjustment
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MANOVA
creates linear composite of DVs

RM variable treated as multiple DVs and combined/weighted to maximise difference between levels of other variables

creates predicted DV score that maximises difference across levels of IV

uncommon, tends towards t1 error, very specific use case
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advantages/disadvantages of MANOVA
advantages

* very powerful, more sensitive
* simplifies procedure (less people)

disadvantages of within-participants

* restrictive assumptions
* sequencing effects (counterbalancing required)
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mixed model ANOVA (split-plot ANOVA)
best way to do WP ANOVA

has a between participants and within participants factor

can use non-experimental variables

observations are independent, can avoid carry-over and sequencing effects
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assumptions of mixed model ANOVA
homogeneity of variance between groups and levels of factor

DV is normally distributed in population

homogeneity of interaction between RM factor and p factor at all levels of WPF (error for RM factor doesn’t change between groups)

variance-covariance matrix consistency (variance and covariance are the same at all levels of WPF (often violated), solved via epsilon adjustments)

pooled variance-covariance matrix has sphericity
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mixed model ANOVA error terms
1 required for p within g factor (between factor)

1 for within participants factor and interaction (block x p within g)

all df terms add up to dftotal

following up sig. intx:

* between participants, use error term from between participants main effect (MS Ps within G)


* within participants, separate error term as in MANOVA (MS B \* Ps within G)

\
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mixed model ANOVA simple effects
one-way ANOVA of WPF at BPF

repeat for each block of trials

each simple effect has own unique error term

average of simple effect error terms is same as that of MS bxps within g intx

or pooled error term (MS ps within cell)
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ANOVA
as opposed to correlational design, can infer causality

due to random assignment to IV levels

interaction: effect of A on DV changes over levels of B
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design methods for increasing power in ANOVA
blocking

improving measurement

remove individual differences (within ps or RM design)

include covariate (ANOVA)
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design methods for increasing power in regression
use of covariate

improved measurement
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one-way ANOVA error term: main effect of A
MSw
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between-participants two-way ANOVA error term: main effect of A
MSw
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between-participants two-way ANOVA error term: AxB interaction
MSw
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between-participants two-way ANOVA error term: simple effect of A at B1
MSw
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within-participants one-way ANOVA error term: main effect of A
MSa\*p
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within-participants two-way ANOVA error term: main effect of A
MSa\*p
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within-participants two-way ANOVA error term: interaction
MSa\*b\*p
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within-participants two-way ANOVA error term: simple effect of A at B1
MSa\*b1\*p
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mixed model two-way ANOVA error term: main effect of the BPF
MS(Ps within BPF Gs)
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mixed model two-way ANOVA error term: main effect of the WPF
MS(WPF \* Ps within BPF Gs)
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mixed model two-way ANOVA error term: WPF \* BPF interaction
MS(WPF \* Ps within BPF Gs)
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mixed model two-way ANOVA error term: simple effect of WSF at BPF1
MS(WSF \* Ps within BPF1)
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mixed model two-way ANOVA error term: simple effect of BPF at WSF1
MS(Ps within BPF at WSF1) or MS(within cell)