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Vocabulary flashcards covering the anatomy, layers, vessels, and conduction system of the heart based on Lab 12 materials.
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Pericardium
A double layer sac surrounding the heart composed of a fibrous and a serous pericardial membrane.
Fibrous pericardium
The outermost layer of the pericardium made of tough, dense irregular connective tissue; it protects the heart, prevents overfilling, and anchors it to the diaphragm.
Serous pericardium
A thin double-layered membrane deep to the fibrous pericardium composed of a parietal layer and a visceral layer.
Parietal layer of serous pericardium
The simple squamous epithelium that lines the internal surface of the fibrous pericardium.
Parietal layer of serous pericardium
The simple squamous epithelium that lines the internal surface of the fibrous pericardium.
Pericardial cavity
contains serous fluid to reduce friction during heart contractions.
Pericardial cavity
contains serous fluid to reduce friction during heart contractions.
Epicardium
The outermost layer, consisting mostly of mesothelium and adipose tissue; it contains blood vessels and nerves.
Endocardium
The innermost layer, composed of simple cuboidal epithelium and loose connective tissue; it lines the chambers, valves, and base of great vessels.
Apex
The inferior tip of the left ventricle located posterior to the 5th intercostal space; it is the site for auscultation of the bicuspid valve closure.
Right atrium
receives deoxygenated blood from the superior and inferior vena cavae, the coronary sinus, and anterior cardiac veins.
Right auricle
increases its volume capacity during excessive venous return.
Pectinate muscles
increase atrial contraction force without increasing muscle mass.
Left atrium
receives oxygenated blood from the lungs via pulmonary veins.
Right ventricle
receives deoxygenated blood from the right atrium and pumps it to the lungs via the pulmonary trunk.
Papillary muscles
prevent valve inversion.
Chordae tendineae
prevent valve prolapse into the atria during ventricular systole.
Septomarginal trabeculae (moderator band)
it reinforces the wall and assists conduction.
Interatrial septum
divides the right and left atria to prevent the mixing of blood.
Interventricular septum
divides the right and left ventricles.
Right atrioventricular (tricuspid) valve
prevents backflow of blood.
Left atrioventricular (mitral / bicuspid) valve
prevents backflow during ventricular contraction.
Pulmonary semilunar valves
prevent blood backflow into the ventricle.
Aortic semilunar valve
prevent blood from flowing back into the ventricle.
Superior vena cava (SVC)
collects deoxygenated blood and drains it into the right atrium.
Inferior vena cava
collects deoxygenated blood from the body below the diaphragm and drains it into the right atrium.
Pulmonary trunk
carry deoxygenated blood to the lungs.
Pulmonary veins
carry oxygenated blood from the lungs to the posterior wall of the left atrium.
Aorta
carries oxygenated blood to the systemic circulation.
Coronary sinus
receives venous drainage from the great, middle, and small cardiac veins and drains into the right atrium.
Sinoatrial (SA) node
The pacemaker of the heart located in the posterior wall of the right atrium; it initiates the normal contractile rhythm.
Atrioventricular (AV) node
Conducting cells in the floor of the right atrium that transmit and slightly delay electrical impulses to allow the atria to empty before ventricular contraction.
Atrioventricular (AV) bundle
Conducting cells in the interventricular septum that receive impulses from the AV node and transmit them to the bundle branches.
Purkinje fibers
Conductive fibers that spread electrical impulses from the apex toward the atria within the ventricular walls.
Cardiac plexus
A network of nerves receiving axons from the Vagus nerves and sympathetic trunk that influences heart rate and contraction force.
Vagus nerve (CN X)
Provides parasympathetic axons to the heart to reduce heart rate, decrease contraction force, and cause vasoconstriction of coronary arteries.
Cardiac nerves
Postganglionic sympathetic fibers that increase heart rate, increase contraction force, and cause vasodilation of the coronary arteries.