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Four processes of pain
transduction
transmission
perception
modulation
what are pain receptors
nociceptors
transduction
activation of the nociceptors
transmission
nerve signals carried via A-delta and C-fibers
nociceptors are found basically everywhere besides…..
the brain
a delta
myelinated fibers producing sharp pain
c fibers
unmyelinated fibers producing dull pain
perception
brain becomes aware of pain
modulation
body alters pain signals
NSAIDS effect which pain process
transduction
opioids effect which pain process (s)
percpetion/modulation
gate control theory
pain signas can be modulated by non-painful input
emphasizes emotions and distractions
Spinothalamic tract
2nd order neurons travel up to ST tract → thalamus —> somatosensory cortex
in charge of the localization of pain
spinoreticular tract
2nd order neurons —> reticular formation → thalamus, hypothalamus, and the cortex
response for the emotional aspect of pain
face pain signals
trigeminal nerve
types of pain
acute
chronic
cancer
phantom
referred
radicular
acute pain
rapid in onset, last less than 6 months
varies in intensity and duration
protective in nature
chronic pain
limited, intermittent, or persistent longer than 6 months
last beyond the normal healing periods
periods of remission
cancer pain
acute, chronic, or both
phantom pain
pain coming from a body part that is no longer there
refferred
pain that is felt from a part of the body that is not the actual source of problem
radicular pain
irritation or compression of the nerve root
ex: sciatica
Eitologies of pain
nociceptive - tissue injury
neuropathic
inflammatory
psychogenic
mixed/cancer
somatic pain
skin, muscles, joints
visceral pain
internal organs
neuropathic pain
due to nerve damage
ex: diabetic neuropathy
inflammatory pain
response to immune response
psychogenic pain
originating from psychological distress
patient responses to pain
physiologic
behavioral
emotional
physiologic responses
increased HR, BP, RR, pupil dilation
behavioral responses
crying, guarding, grimacing
emotional responses
anxiety, fear, hopeless
describing pain
quality
severity
periodicity
factors influencing pain
cultural
gender and age
anxiety and stress
past experiences
subjective data pain assessment
onset, duration, location, quality, and intensity
objective pain assessment
non-verbal cues, vital signs, physical assessment
Numeric Rating Scale (NRS)
For people who understand 0-10
FLACC
Behavioral pain assessment for infants to children age 7 years old
PAINAID
Dementia patients, patients who can’t express or tell pain
NIPS
neonatal pain scale
Wong-banker Pain rating
Children who can’t tell us a number 0-10
Children should avoid being prescribed ____ in regards to pain
aspirin
Older adults may…..
underreport pain, atered drug metabolism
ATC
around the clock
give meds until not needed
PRN
take as needed
four types of administration methods
oral
IV
topical
epidural
PCA
patient controlled analgesia
Numeric Sedation Scale (NSS)
Used to monitor opioid side effects
What are the phases of the NSS
S
1
2
3
4
S on NSS scale
sleep, easy to arouse
no action necessary
1 on NSS scale
awake and alert
no action necessary
2 on NSS scale
occasionally drowsy, but easy to arouse,
no action necessary
3 on NSS scale
frequently drowsy, drifts off to sleep during conversation,
reduce dosage
4 on NSS scale
Somnolent with minimal or no response to stimuli, discontinue opioid,
action:consider of naloxone