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do McKenzie and TBC work with chronic LBP?
no, best with acute/subacute
what do traditional models for LBP focus on
anatomy, biomechanics
how do clinicians aim to explain pain to patients
from a tissue perspective:
• contrasting healthy and injured tissues (pathoanatomy)
• highlighting a mechanical deviance from normal expected patterns of movement (biomechanics)
• a disease state such as degenerative changes (pathoanatomy)
bottom up approach
Painful stimulus = pain
Amount of tissue damage = intensity of pain
Yes = Clinical value in more acute phases of injury, surgical, or disease states
what is the problem with bottom up approach
Lacks the ability to explain complex pain issues:
• peripheral and central sensitization,
• neuroplasticity,
• facilitation and inhibition,
• immune and endocrine changes
All of these have been implicated in more complex and persistent pain states
Limited Efficacy of Biomedical Models with treatment for chronic
1. Limited efficacy in alleviating pain and disability
2. May even increase patient fears, anxiety, and stress, thus negatively impacted their intended outcomes
Persistent or Chronic pain
Leads to significant changes in brain, alters processing of pain and activation of catastrophization
• Sensory homonculus
• Motor homonculus
Many areas of the brain are involved in the pain experience
Smudging
brain areas normally devoted to different body parts/functions start to overlap → limits movement in motor area, or makes more areas sensitive on sensory area
• Key Point - IT CAN BE REVERSED WITH TRAINING
Central Sensitization
● Peripheral noxious inputs into CNS lead to increased excitability where the
response to normal inputs is enhanced
● CNS has become hyper-excitable to input received from periphery
● Hypersensitivity of CNS
how to identify if a patient fall into chronic pain
Subjective cues - Flags
Functional outcome scores
Pain Catastrophizing questionnaires
Depression screens
Fear Avoidance behaviors
Behavior related factors and LBP
1. Inactivity
2. Current or former smokers
3. Current or former alcohol drinkers
4. Extreme sleep duration habits
5. Obesity
types of flags
red
yellow
blue
black
yellow
emotional distress
hypervigilance
pain catasstrophizing
elevated fear-avoidance beliefs
low self-efficacy
misunderstanding anout the nature and likely impact of pain
misunderstanding about the best strategies for long-term success
blue flags
Employee
◦ Fear of re-injury
◦ High physical job demand
◦ Low expectation of resuming work
◦ Low job satisfaction
◦ Low social support or social dysfunction in workplace
◦ Perception of high job demand/‘stress’
Workplace
◦ Lack of job accommodations/modified work
◦ Lack of employer communication with employees
black flags
Misunderstandings and disagreements between key players (e.g. employee and employer, or with healthcare).
Financial and compensation problems
Process delays (e.g. due to mistakes, waiting lists, or claim acceptance).
Overreactions to sensationalist media reports.
Spouse or family member with negative expectations, fears or beliefs.
Social isolation, social dysfunction.
Unhelpful policies/procedures used by company
The goal of identifying Flags is to find factors that can be
used positively to facilitate recovery and prevent or reduce long-term disability and work loss
identifying
• unintentional learning or emotional barriers (such as fear ofpain)
• less common intentional barriers (malingering)
Considerations During Objective Evaluation if you are getting positives for yellow
Rule out serious pathology
Low tech exam
Asterisk or comparable sign??
Acknowledge nice movement / good muscle contraction
Encouragement
Questions associated with patient’s beliefs
Waddell’s Signs
1. Tenderness (widespread, non-anatomic)
2. Simulation tests (axial loading, rotation)
3. Distraction test (SLR supine vs sit)
4. Regional Disturbances
• weakness (non-anatomic, cogwheeling)
• sensory (stocking rather than dermatomal)
5. Overreaction (disproportionate expressions
what are Waddell signs assoicated with?
Poorer outcome (surg and non-surg), increased freq of visits, greater impairments, higher % psych problems
Increased with Chronic LBP, compensation
Two ways to effectively manage patients with chronic LBP
1. Key into how you talk to them about what is going on – our language matters
2. Interventions – how to approach activity, exercises and overall treatment
Pain Self-Efficacy
The belief in one’s ability to manage and complete a task despite their pain
increased self efficacy leads to
• Decreased disability
•Decreased disease activity
•Decreased depressive symptoms
•Decreased fatigue
•Decreased emotional distress
Adherence to physical activity and participation
Effective Interventions
1. Pain Neuroscience Education of Body response
2. Facilitating Mastery of Experience
3. Vicarious Experience
4. Verbal Persuasion
5. Graded Exercise
6. Graded Exposure
7. Group Exercise (Aerobic exercise)
Pain Neuroscience Education (PNE)
Explains biological and physiological processes involved
in pain experience
Defocus issues associated with anatomical structures
Teaches patients to re-think their pain.
How to implement a PNE
Build rapport with patients to get an idea of their belief system and
knowledge of pain, as that plays a role in their overall pain experience.
Use questionnaires to fill out during the first session with the patient.
1) Physical Readiness Activity Questionnaire (PAR-Q) (Daly 2006)
2) Fear Avoidance Behavior Questionnaire (FABQ) (Waddell 1993)
3) Pain Science Knowledge Questionnaire (O’Connelly 2013).
Identify patients with acute pain vs. chronic pain > 3 months (Rolf-
Detlef T 2015), as that will impact when and how you want to implement pain science education.
how does PNE improve MSK disorders
Pain ratings
Pain knowledge
Disability
Pain catastrophization
Fear-avoidance
Attitudes and behaviors regarding pain
Physical movement
Healthcare utilization
who is PNE appropriate for
Chronic low back pain
CRPS Type 1
Phantom Limb Pain
Brachial Plexus Injury
Chronic pain s/p stroke
Chronic neck pain
Frozen shoulder
Intervention Guidelines for PNE
Encourage the use of a confrontation approach in those that normally wouldn’t
◦ Addressing the way the patient thinks about low back pain itself and the consequences of low back pain
◦ Addressing the way the patient participates in rehabilitation protocols
Turn “avoiders” into “confronters”
dosing for PNE
30 minutes - 4 hours
1 session - multiple sessions
1-on-1 > group
Used pictures, PPT presentations, metaphors, drawings, examples, books
what works best for chronic LBP
PNE and exercise
Graded Exercise
based on operant principles
Reward patient for meeting activity goals
No reward for activity limitation due to pain
This is a quota based system
◦ Exercise progression based on meeting quota
◦ Pain does not normally figure in exercise progression
graded exposure
Gradual exposure to specific activities that produce fear (George
2009, JOSPT)
Fear of Daily Activities Questionnaire
group therapy
Set time each week
Social
Outside of home
Support
YMCA, senior centers
self help
Aerobic exercise
● increases Blood flow and oxygenation of muscles & nerves
● Regulates stress chemicals
● Boosts immune system
● Improves memory
● Decreases sleep disturbance
● Diaphragmatic breathing - 5 minutes
how common is LBP
affects 80% of us some time in our lives
one of the most frequent reasons that patients visit PCP and is 2nd most common reason for time taken off from work
in the US, estimates of direct medical costs attributable to back pain are as high as 25 billion annually
can occur at any age but is most prevalent during the third to sixth decades of life
muscles
strain
myofascial pain
spasms
bone
vertebrae
fractures
trauma
osteoprorsis
tumors/cancer
facet joints
arthritis
bone spurs
SI joint
discs
cushions or shock absorbers between vertebral
herniation
tear/rupture
spinal canal
the chamber that holds the spinal cord and nerves
stenosis
what can cause back pain
advanced age leading to arthritis
falls
auto accidents
poor body mechanics or posture
high impact sports (football)
poor nutrition, obesity, and pregnancy
smoking
inflammation
spasms
nerve pain
plain old pain
pain
ultram
vicodon
percocet
MS contin
duragesic
side effects/problems
tolerance, addiction, abuse potential, street value
meds for nerve pain
neurontin
lyrica
cymbalta
tricyclic
side effects of nerve pain
drowsiness, swelling in the legs, dry mouth
look art PMH, age and other meds
spasms
muscles will spasm aroud the spine
meds for spasms
0 flexaril
soma
skelaxin
robaxin
norflex
zanaflex
side effects of spasm meds
drowsiness
how does smoking impact LBP
smoking accleerates the thinning of vessels in vertebrae
meds for inflammation
motirn
aleve
mobic
celebrex
modrol dosepak
side effects of inflammatory meds
upset stomach, risk for patients w/ ulcers or kindey problems
narcotics
aren’t all bad; however they must be used cautionsly and responsibly
comprehensive pain program
pain med contract
urine testing
PT
acupuncture
massage therapy
mind-body therapies
integrative psychotherapy and psychiatry
nutritional support
natural medicines
trigger point therapy w/ homeopathic meds
are there limitations to treatment
yes
determining cause of pain
history
physical exam
imaging
X-ray
MRI
CT scan
nerve conduction studies/EMG
how to treat muscle strain, spasms, and myofascial pain
medications
anti-inflammatories
muscle relaxants
PT
education
ice, heat, massage
strength, ednurance and flexibility lead to improved stabilization and efficiancy
trigger poin injections
trigger point injection
injection anaesthetic solution sometimes w/ steroid into muscle
how to treat compression fractures
will usually cause pain only in the back
medication: narcotics
bracing and PT with extension-based exercises
vertebroplasty
kyphoplasty
vertebroplasty and kyphoplasty
takin introducer and inserting into the bone, then inject a cement
thoracic are more difficult to treat
how to treat facet joint straina nd arthritis
usually cause pain only in the back
meds: ant-inflammatories
PT
more pain in extension
facet joint injection
injection of local anesthetic and steroid into the facet joint capsule to reduce inflammation
procedure is one under X-ray guidance as an outpatient in a surgical center
success and duration of relief is variable and depends on extent of inflammation, as well as post-injection habits
injection technique
c-arm: takes live X-ray
facet medial branch blocks
injection of local anesthetic only on the nerve fibers that transmit pain signals from the joint
procedure is done under X-ray guidance as an outpatient in a surgical center
a successful injection is measured by the amount of temporary relief of back pain
it is usually done twice on 2 separate visits for confirmation purposes
injection on to the bone instead of joint
after 2 confirmatory blocks, the nerve fiber is destroyed w/ specialized radiofrequency needle
success of medial nerve block
variable; in theory it should be permeant; however, these nerve fibers can grow back; this procedure can be repeated in as soon as 6 months
SI joint strain and arthritis
will usually cause pain only in the low back and buttocks
meds: anti-inflammatories
PT
SI joint injection
injection of local anesthetic and striod into SI joint to reduce inflammation and pain
done under X-ray guidance
success and duration of relief is variale and depends on extent of inflammation as well as post-injection habits
hwo to treat disc herniation and spinal stenosis
will usually cause pain in the back w/ radiation into the leg because of irritation of the nerve root
meds
PT: including traction
epidural steroird injection
referrals for surgery
meds for disc hernation and spinal stenosis
anti-inflammatories
nerve meds
pain med
epidural steroid injection
injection of local anesthesic and steirod inot epidural space or along the nerve roots to reduce inflammation and pain
under X-ray guidance
success and duration depnds on extent of inflammation and post-injection habits
appraches to epidural steriod
interlaminar approach: between lamia
transforaminal approach: through the foramen
caudal approach: through tailbone region through sacral hiatus
how many injection can i have
there is no consensus on the number of injection given in a year, 3 injections in a 6-12 month period
what are the benefits to an injection
concentration of steroid delivered to the site of pathology is presumed to be proportional to its effectiveness
injected steroid is not dependent on local blood flow, which has shown to be compromised w/ compressive lesions