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when the missed roll it in, you were sin
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evolutionary psychology
the study of how natural selection influences behavior
heredity (nature)
how genes affect behavior
environment (nurture)
how everything nongenetic influences behavior (school, upbringing, etc.)
twin / adoption studies
showed how identical twins have higher likelihood of developing the same disease (genetics), but they show differences if raised in different situations (environment)
central nervous system
brain and spinal cord
peripheral nervous system
the rest of the nervous system apart from CNS; relays info to the CNS
somatic nervous system
involved with voluntary movement; has sensory and motor neurons
autonomic nervous system
involved in involuntary organs (heart, lungs)
sympathetic nervous system
fight-or-flight; typically activates everything except for digestion
parasympathetic nervous system
rest and digest; typically inhibits everything except for digestion
neuron
basic cell of the nervous system
dendrites
part of the neuron that receives incoming neurotransmitters
axon
part of neuron that the action potential travels down
myelin sheath
covers axon to speed up AP transmission and protect the axon
synapse
the gap between neurons
sensory neurons
afferent; arrive at the brain; receives sense signals from the environment
motor neurons
efferent; exits the brain; sends signals to move
interneurons
neurons in the CNS that transmit messages between the other two and are responsible for the reflex arc
reflex arc
important stimuli that skips brain and routes through spinal cord for immediate reactions (get hand off of hot iron)
glia/glial cells
“glue” cells; provide the myelin for myelin sheaths; clean up waste around neurons; provide nutrients
action potential (AP)
neuron receives stimulus, causes inside of cell to become more positively charged than the outside; ions move across membrane; sends electrical charge down the axon; via depolarization
soma
cell body
nucleus
in cell body, controlling cell function and regulation
terminal sac
holds neurotransmitters in the terminal axon
nodes of ranvier
gaps in myelin sheath where axon is exposed to the cell
resting potential
unequal distribution of ions, where more sodium is outside the cell and more potassium is inside the cell, maintains a stable, negative charge of -70mv across the cell membrane when not actively transmitting signals
depolarization
the inside of the neuron becomes less negative compared to the outside because of an influx of + ions (sodium) through the membrane’s ion channels. the change in elec charge causes the firing of an AP
threshold (of depolarization)
level of stimulation required to trigger an AP in the neuron. minimum needed to produce a response
all-or-nothing principle
neuron will fire at full strength when it reaches its threshold; response will not increase in intensity or speed if the stimulus is stronger; below threshold means no AP at all
refractory period
brief period following AP; neuron can’t generate another AP, needs to rest and reset first. neuron’s sodium channels are temporarily deactivated and the cell membranes return to resting state
neurotransmitters
chemical messengers sent and received by neurons across synaptic gap; allows for nervous system communication
excitatory neurotransmitters
increases likelihood of firing an AP in receiving neuron; binds to receptor sites, prompting an AP
inhibitory neurotransmitters
decreases likelihood of firing an AP
GABA
major inhibitory NT; undersupply linked to seizures, tremors, and insomnia
glutamate
major excitatory NT involved in memory; oversupply overstimulates the brain, causing migraines and seizures
dopamine
both ex and in NT; influences movement, learning, attention, and emotion; short-term reward and fine movement; associated with addiction (found in hypothalamus); oversupply linked to schizophrenia; undersupply linked to tremors and decreased mobility (parkinson’s)
serotonin
long-term moods, emotion, sleep, hunger, arousal, found in amygdala; undersupply associated with depression (some drugs that raise serotonin levels are used to treat depression)
acetylcholine (ACH)
muscle action, learning, memory, in hippocampus; ACh-producing neurons deteriorate with Alzheimer’s disease
norepinephrine
controls alertness and arousal within the sympathetic nervous system; undersupply can depress mood/is linked to depression
endorphins
influences perception of pain or pleasure/decreases pain; oversupply via opioids can suppress body’s natural endorphin supply
substance p
pain regulation/perception and immune response; abnormal oversupply can lead to chronic pain, inflammation
agonist
drugs that mimic neurotransmitters
antagonist
drugs that block neurotransmitters
reuptake
process where unused neurotransmitters are taken back up into the sending neuron (antidepressants block reuptake [reuptake inhibition] to treat depression)
psychoactive drugs
chemical substances that alter brain; causes changes in perceptions and moods by stimulating, inhibiting, or mimicking neurotransmitter activity
depressants
reduce neural activity, slow body functions (alcohol, opioids, barbiturates [tranquilizers])
opioids
opium and its derivatives; depress neural activity, temp lessening of pain and anxiety
barbiturates
depress CNS activity; reduce anxiety but impair memory and judgement
stimulants
excite neural activity, speed up body functions (caffeine, cocaine, nicotine, methamphetamines, ecstasy)
hallucinogens
psychedelic drugs; distort perceptions, evoke sensory images w/o sensory input; hallucinations and altered perceptions (marijuana, LSD/acid, ecstasy)
opioids
opium and its derivatives; depress neural activity; pain and anxiety relief by mimicking endorphins (agonist) (heroin, morphine, fentanyl)
tolerance
needing more of a drug to experience the desired effects
addiction
need to have the drug to avoid withdrawal symptoms
withdrawal
unpleasant mental or physical reactions when attempting to suddenly end drug use
multiple sclerosis
destruction of myelin sheath; disrupts APs, causing impaired mobility, paralysis, and pain
myasthenia gravis
blocked acetylcholine; disrupts APs, causing poor motor control and paralysis
blindsight
lesions to primary visual cortex let people “see” (catch a ball) despite being blind; evidence of association areas
prosopagnosia
face blindness caused by damage to occipital and/or temporal lobe
broca’s aphasia
damage to broca’s area; stuttered speech; intelligence is intact and understanding is present, but finding the words is difficult
wernicke’s aphasia
damage to wernicke’s area; jumbled speech; easy speech, but the meanings are odd (dont align with prompts)
phantom limb pain
pain from a limb that is no longer there (amputated); caused by brain plasticity
epilepsy
seizures related to too much glutamate (excitatory) and/or too little GABA (inhibitory). so, too much excitement/too little calming means seizures
alzheimer’s
destruction of acetylcholine in the hippocampus, causing memory loss
electroencephalogram (EEG)
shows broad brain activity (nonspecific) by observing brain’s electrical output with electrodes
functional magnetic resonance imaging (fMRI)
shows brain activity in specific regions by measuring oxygen via blood flow; shows brain function and structure
lesion
brain tissue destruction; may be natural from disease or trauma, may be from surgery, may be experimental by destroying with electrodes
hindbrain
medulla, pons, cerebellum; essential survival functions (breathing, sleeping, wakefulness, coordination, balance)
midbrain
atop brainstem; connects hindbrain with forebrain, controls some motor movement, transmits auditory and visual info
forebrain
cerebral cortex, thalamus, hypothalamus; complex cognitive activities, sensory and associative functions, voluntary motor activities
cerebellum
the “little brain”; processes sensory input, coordinates movement output and balance, enables nonverbal learning and memory. movement, balance, coordination, procedural memory
brainstem
central core of brain, beginning where the spinal cord swells as it enters the skull; responsible for automatic survival functions
medulla
base of the brainstem; controls heartbeat and breathing
reticular activating system
governs the reticular formation; controls alertness, arousal, sleep, eye movement
reticular formation
nerve network that travels through brainstem into the thalamus; filters info, significant in controlling arousal
basal ganglia
deep brain structures involved in motor movement
limbic system
neural system located mostly in the forebrain, below central hemispheres. includes amygdala, hypothalamus, hippocampus, thalamus, and pituitary gland; associated with emotions and drives
amygdala
two lima-bean-sized neural clusters in limbic system; linked to emotions and fear
hypothalamus
lies below thalamus; directs maintenance activities like eating, drinking, and body temperature; helps govern endocrine system and is linked to emotion and reward/pleasure
hippocampus
helps process explicit (conscious) memories (episodic and semantic memory; memory of events and facts) for storage
thalamus
forebrain’s sensory control center, on top of brainstem; directs messages to sensory receiving areas in cortex; transmits replies to cerebellum and medulla. relay center for all but smell
cerebrum
two cerebral hemispheres forming 85% of brain’s weight; enables perceiving, thinking, speaking
cerebral cortex
covers cerebrum; intricate, interconnected neural cells covering cerebrum; ultimate control and info-processing center
pituitary gland
comms w/ endocrine system and hypothalamus; releases hormones; master endocrine gland
frontal lobes
part of cerebral cortex just behind forehead; linguistic processing, muscle movements, higher-order thinking, executive functioning (making plans, judgements), personality, language
parietal lobes
part of cerebral cortex at top of head toward rear; receives sensory input for touch and body position
occipital lobes
part of cerebral cortex at back of head; includes areas that receive visual field info
temporal lobes
part of cerebral cortex above ears; auditory areas (each receives info primarily from opposite ear), enables language processing, hearing, face recognition
motor cortex
cerebral cortex area at rear of frontal lobes; controls voluntary skeletal movements; map of motor receptors
somatosensory cortex
cerebral cortex area at front of parietal lobes; registers, processes body touch and movement sensations; map of touch receptors
prefrontal cortex
front part of frontal lobes; enables judgement, planning, social interactions, processing of new memories; executive function
association areas
cerebral cortex areas that are not involved in primary motor or sensory functions; instead, involved in higher mental functions; learning, remembering, thinking, speaking. receives and integrates info from multiple areas and lobes
corpus callosum
large band of neural fibers connecting the two brain hemispheres and carrying messages between them. sometimes severed in those with severe seizures → split-brain patients
(neuro) plasticity
brain’s ability to reorganize, adapt throughout life in response to experiences, learning, and environmental changes. the brain can rewire to recover old/create new functions
aphasia
damaged speech from damage to left hemisphere association areas
broca’s area
left hemisphere frontal lobe; speech production, language processing, grammar formation, speech muscle coordination
broca’s aphasia
inability to produce fluent speech (broca → broken speech)
wernicke’s area
left hemisphere temporal lobe; language comprehension, understanding spoken and written language, interpreting words + sentences
wernicke’s aphasia
cant comprehend written or spoken language (wernicke’s → what?)
split-brain experiments
images shown to the R eye are processed in the L hemisphere - the patient can say what they saw (L hemisphere language). images shown to L eye are processed in R hemisphere - patient cannot say what they saw (could point with left hand)
endocrine system
body’s “slow” chemical communication system; set of glands and fat tissue that secrete hormones into the bloodstream throughout the body