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Chicken Pox-Varicella
Contagious disease caused by varicella zoster virus
Varicella presentation
Skin rash with small itchy blisters
Spreads over other areas of the body
Fever fatigue sore throat and headache- lasts 5-7 days
Varicella treatment
Acetaminophen
Avoid aspirin
Pruritis- calamine lotion, soothing oatmeal bath, antihistamine
Hydration
Mumps
Viral infection
Mumps Presentation
Swelling of salivary glands
Parotitis-swelling of the parotid glands
Mumps Diagnostic studies
RT-PCR viral culture
Measles- Rubeola: presentation
Initial- fever cough rhinorrhea conjunctivitis
Followed- small red spots with white centers (Koplik spots) in mouth
Distinctive rash across body, starts at hairline and goes downward
Airborne precautions - if hospitalized
Rubella (German Measles) Gen Info
caused by rubella virus
Usually milder illness than measles
Rubella (German Measles) S/S
low-grade fever
Rash
Lymphadenopathy : behind ears and neck
Prevented by MMR vaccine
Measles- more severe, highly contagious
Rubella- milder but dangerous in pregnancy
Immunizations BIRTH
B
Hep B
Immunizations 2 months
B-DR-HIP
Hep B
Dtap (for kids younger than 7 yrs)
Rotavirus
Hemophilia influenza type b
IPV Polio
PCV- pneumococcal conjugate
Immunizations 4 months
DR-HIP
D2: diphtheria, tetanus, pertussis
Rotavirus
Hemophilia Influenza type B
IPV- Polio virus
P-PCV: Pneumococcal Conjugate vaccine- streptococcus pneumonia
Immunizations- 6 Months
B-DR-HIP-IN
B- Hep b
D3: diphtheria, tetanus, pertussis
Rotavirus
Hemophilia Influenza type B
IPV- Polio virus
P-PCV: Pneumococcal Conjugate vaccine- streptococcus pneumonia
Influenza-yearly
Immunizations 1-1.5yrs
MAD-HPV
Measles, Mumps, Rubella(12-15months)
A- Hep A (1-2 years)
D4: Dtap
H-Hemophillia influenza
P- PCV pneumococcal conjugate vaccine
V-varicella
Immunizations 4-6 yrs
VERY-DIM
Very- Varicella
D5: Dtap, Diptheria-tetanus-pertussis
I-IPV polio vaccine
M-meningococcal
Immunizations 11-12 years
Tada!!
Human
Human Papilloma Virus
Men
Meningococcal
Immunizations 16-18 years
Men booster
Meningococcal booster at 16 yrs
Vaccine contraindications
Febrile
Anaphylactic reaction- hold specific vaccine
Risk vs benefit
Vaccine nursing admin considerations
OBTAIN CONSENT
If no consent, educate and document
Document date route, site, lot number, expiration date: in case of any reaction
atraumatic care: appropriate play/ distractions
Avoid aspirin with vaccines- why?
Reye’s Syndrome
Tetanus gen info
Cause: Bacteria
Transmission: Enters body through wounds
MUSCLE RIGIDITY
Tetanus s/s
Muscle stiffness- lockjaw
Painful muscle spasms
Difficulty swallowing
Seizures-severe cases
Tetanus nursing care
Tetanus vaccine
Tetanus immune globulin
Scarlet fever cause
Group A streptococcus infection
Scarlet Fever S/S
Sandpaper Ike rash on trunk
Strawberry tongue
Fever
Scarlet Fever tx
Antibiotics required
COVID-19
Multi system inflammatory syndrome (MIS-C)
Supportive care
VACCINE
Sepsis RF
burns
Invasive Catheters
Immunocompromised
Antibiotics - long term
Early Signs of Sepsis
fever AND hypothermia (very common in infants)
tachycardia
Tachypnea
WARM extremities
BOUNDING pulses
BRISK cap refill
NORMAL UOP
ALOC
Late signs of Sepsis
Hypotension
PROLONGED cap refill
Mottled COOL extremities
WEAK pulses
Progressive mental changes
DECREASED UOP
Fever or hypothermia
SHOCK to ARREST with no interventions
Sepsis Nursing Interventions
Pan Culture: blood, sputum, urine, skin/tissue
Vitals, O2
IV fluid resuscitation=
Labs- CBC, chem, coag, glucose
Blood transfusion
IV ABX
Nutrition
MONITOR!! Hemodynamic/cardiorespiratory
Cardiac Dysfunction/Anomalies gen info
Congenital or acquired
Congenital-any defect that develops during the first 8 weeks of gestation
Makes up 30% of infant deaths related to birth defects in first year of life
Cardiac Dysfunction and Anomalies: caused by
exposure to drugs, alcohol, smoking
Maternal infections: rubella
KEEP WRITING
Heart Defect Main Manifestation
Murmur: Blood flow is being shunted away from normal path of flow
Patent foramen ovale

Cardiac output compensation
Elevated heart rate
Chronic desaturation s/s
<85% O2 sat
Tripod position
Clubbing nails
Pale/cyanotic
Chest retraction
Nasal flaring
Defects with increased pulmonary blood flow
HIGHER pressure on LEFT side of heart
more blood to lungs
Increased vascular resistance
RIGHT ventricular HYPERtrophy
Blood is shunting from LEFT to right
CHF can occur
INCREASED pulmonary blood flow conditions
PDA
ASD
VSD
DECREASED pulmonary blood flow conditions
TOF
Pulmonary artesia
Mixed defects conditions
TGA
Truncus Arteriosis
OBSTRUCTED systemic blood flow conditions
CoA
AS
PDA- Patent Ductus Arteriosus
Connection between pulmonary artery and aorta stays open causing mixing of blood
PDA ligation if hole does not close on its own

PDA meds
NSAIDs:
Indomethacin
IV ibuprofen
ASD- Atrial Septal Defect
Opening between right and left atria- top two chambers of the heart
Closes in 4 years
Defects with DECREASED pulmonary blood flow
Obstructed blood flow from RIGHT to left
RIGHT TO LEFT shunting due to increased pressures if in presence of a septal defect
Decreased oxygenation →HYPOXEMIA and CYANOSIS
POLYCYTHEMIA(more RBC production) compensatory response
Hypercyanosis with change of postition, crying, feeding, activity
Pulmonary Atresia tx
TOTAL OCCLUSION of pulmonary valve
Tx- opening of PDA
Prostaglandins - PGE1
Balloon angioplasty

Tetralogy of Fallot
Overriding aorta, VSD, RVH, Pulmonary stenosis
Multiple surgeries/palliative measures

TGA-Transposition of the Great Arteries
Parallel circulation due to aorta out flue from BOTH right side of heart and pulmonary artery for left side
NEEDS SURGERY WITHIN 2 WEEKS OF LIFE
VSD-Ventricular Septal Defects
Opening between right and left ventricles - bottom two chambers of the heart
Usually closes on its own within 6 months

Cardiac Catheterization Post-Op
Check before feeding: Gag reflex + bowel movement
Check affected extremities pulse (where the PICC is), compare it to opposite extremity
Prevent bleeding: pressure and immobilize
Truncus Arteriosis
Fetal anatomy of single vessel serving for both pulmonary and systemic flow
Defects with Obstructed Systemic Blood Flow
STENOSIS leading to pressure overload in the LEFT side of the heart→ can yield DECEASED cardiac output
High pressure in ventricle can lead to DECREASED cardiac output→ shock
Aortic Artesia
OBSTRUCTION OF FLOW to systemic circulation, blood pools in left ventricle regurgitates to left atrium
PDA NECESSARY TO SUSTAIN LIFE (PEG1 to open)
Coarctation of the Aorta- CoA
Narrowing/constriction of descending aorta
CYANOSIS IN LOWER EXTREMITIES, pre/post duct saturations differ, pulse pressure narrowed in lower extremities
Tx: balloon angioplasty/stent/surgical repair
CHF
Can be due to defect INCREASING pulmonary blood flow/obstruction of systemic blood flow
Decreased contractility, edema, pulmonary congestion→ r/l heart failure can progress to bilateral
Acquired disease:
cardiomyopathy
Kawasaki disease
CHF- initial manifestations
tires easily with feeds
Weight loss
Diaphoresis
irritability
Dyspnea
Abdominal pain/distention
Peripheral edema
Exercise intolerance
Frequent respiratory infections
CHF late manifestation
Tachypnea Tachycardia
Pallor cyanosis
Nasal flaring, grunting, retractions, cough
Third heart sounds
Fluid volume overload
Periorbita;, facial edema
Hepatomegaly
JVD
Cardiomegaly- cyanosis/weak pulse/hypotension, can lead to shock
Cardiomyopathy
Heart muscle disorder effecting chamber size, wall thickness, contractions, systolic/diastolic function ]
Affects contractility → ventricles
Cardiomyopathy Dilated
Most common
overall dilation, poor pump, blood pools
Treatment same as CHF, need for transplant
Cardiomyopathy Hypertrophic
ventricular hypertrophy
Rigid walls, obstruction
Cardiomyopathy Restrictive
Not common in children
Restricted ventricular filling
Pulmonary Artery Hypertension
Increased pressure in pulmonary arteries
Right sided HF → CHF
Vasodilators, diuretics, ca channel blockers, ace inhibitors, anticoagulant
NO CURE not sustainable with life without ongoing therapies
Acquired Heart Diseases
Infective Endocarditis
Rheumatic fever
Kawasaki
Infective Endocarditis
Infectious disease of inner muscle of the heart and surrounding vessels
Common Causative bacteria:
staphylococcus aureus
Staphylococcus viridans
Staphylococcus pneumoniae
Commonly seen in pt w hx of congenital or acquired disease/ indwelling prosthetic material
Infective Endocarditis Manifestation
persistent low grade fever
Heart murmur
Splenomegaly
Cutaneous signs- petechiae
Infective Endocarditis Diagnostic test
Modified Dukes Critieria
multiple blood cultures
Ech0
Rheumatic fever
Abnormal immune response to Group A strep- GAS- infection, usually pharyngitis
ASO titter for strep exposure
Rheumatic Fever manifestation
Fever
Large joint pain
Involuntary movement of ms
Rheumatic Fever tx
Antibiotics
Anti inflammatory therapy
HF management
Antibiotic prophylaxis-ranging from 5 years to indefinitely
Kawasaki Disease
Acute febrile
Vasculitis
Vascular inflammatory disorder of midsize and coronary arteries
Leading acquired heart disease→ ANEURYSM
Kawasaki Stages
Stages
1- Acute: 1-2 weeks of high fever, irritability
2- Subacute: 2-4 weeks fissures, joint pain, cardiac disease
3- Convalescent 6-8 weeks resolving but with signs of inflammation
Kawasaki Treatment
8 weeks
IV immunoglobulin
Aspirin
alt- clopidogrel, dipyridamole
NO LIVE VIRUS VACCINES FOR 11 MONTHS!
Hypovolemic Showck
Inadequate perfusion due to inadequate volume
Need fluid resuscitation, transfusion
Distributive shock
Abnormal distribution of blood
to extremities not organs
Anaphylaxis sepsis SCI
Cardiogenic Shock
Impairment of function due to pump failure
Like hypovolemic
CHF develops, cardiorespiratory monitoring needed
Fifth Disease- Erythema Infectiosum
Droplet or contact transmission
SLAPPED CHEEK RASH
Fever, malaise, headache, myalgia, NVD
Kawasaki signs
38 C fever and antibiotics dont work
Conjunctivitis
Large cervical lymph nodes
Strawberry tongue
Peeling of skin around finger/toe nails