Oral motor Exam 3

0.0(0)
studied byStudied by 0 people
learnLearn
examPractice Test
spaced repetitionSpaced Repetition
heart puzzleMatch
flashcardsFlashcards
Card Sorting

1/143

encourage image

There's no tags or description

Looks like no tags are added yet.

Study Analytics
Name
Mastery
Learn
Test
Matching
Spaced

No study sessions yet.

144 Terms

1
New cards

_______ is the absence of speech, not necessarily sensorimotor, that has many possible etiologies.

neurogenic mutism

2
New cards

List the etiologies of neurogenic mutism.

severe dysarthria, laryngectomy, apraxia of speech, aphasia, disorders of arousal or consciousness, or postictal (after seizure)

3
New cards

____ is speechlessness resulting from severe loss of neuromuscular control over speech. It will present when AoS or dysarthria is in its most severe form.

Anarthria

4
New cards

Anarthria may be the end stage of dysarthria for _____.

degenerative disease

5
New cards

What are the three types of dysarthria that will most likely lead to anarthria?

spastic, hypokinetic, and mixed

6
New cards

_____ will present with mutism, severe dysphagia, preserved consciousness, vertical eye movements or blinking, and quadriplegia. What type of dysarthria will these individuals present with?

locked-in syndrome; severe spastic or mixed spastic-flaccid

7
New cards

Apraxia of speech with mutism only lasts ______ if stroke is the etiology, but will usually have concomitant non-verbal oral apraxia.

a few days

8
New cards

Aphasia with mutism may be _____, but usually transforms into ______. Prosody will be ____ for these individuals.

present initially; transcortical motor aphasia; flat

9
New cards

_____ presents as unarousable unresponsiveness and absence of sleep/wake cycles on EEG. It is typically caused by diffuse bilateral cerebral hemisphere damage, brainstem, injury, or both. These individuals have no _____, only reflexes.

Coma; purposeful movements

10
New cards

_____ presents as wakeful unawareness often associated with severe bilateral cerebral hemisphere involvement without brainstem involvement. ____ are preserved.

vegetative state; sleep/wake cycles

11
New cards

__ have a degree of awareness and responsiveness and may not be entirely mute. They may be able to ____, smile, cry, or reach for things.

minimally conscious state; visual tract

12
New cards

_____ individuals have reduced motivation (_____) to speak, difficulty initiating and sustaining the cognitive and motor effort required for speech, and it is caused by massive bifrontal lobe damage.

Akinetic mutism; abulia

13
New cards

The three types of etiology-specific neurologic mutism are _____ (following seizure), ____ (organ transplant, cancer treatment), and ______ (not very common).

speech arrest; drug-induced mutism; mutism after corpus callosotomy

14
New cards

Neurogenic mutism may be from severe dysarthria (______) and commonly results from ______ or ____ neurologic damage.

anarthria; bilateral or diffuse

15
New cards

_____ is dysfluent speech acquired as a direct result of neurologic disease.

Acquired neurogenic stuttering

16
New cards

List the etiologies of acquired neurogenic stuttering.

stroke and closed head injury (most common); Parkinson's disease; progressive supranuclear palsy syndrome (PSPS); MS; dementia; corticobasal syndrome; multiple system atrophy; seizure disorders; dialysis dementia; brain tumor; anoxia; bilateral thalamotomy; thalamic or globus pallidus deep brain stimulation; drug toxicity or abuse

17
New cards

The characteristics of acquired neurogenic stuttering include: _____ repetitions, prolongations, and blocking/hesitation, may not be _______, can occur with ___ and ___ words, ____ of dysfluencies but _____________, and they may not demonstrate _________ with choral reading or singing.

sound/syllable repetitions, prolongations, and blocking/hesitation; may not be restricted to initial syllables; can occur within content and function words; awareness of dysfluencies but without significant anxiety or secondary struggle behavior; may not demonstrate an adaptation effect or improvement with choral reading or singing

18
New cards

Possible associated deficits with acquired neurogenic stuttering include _____, ______, and _____.

aphasia, apraxia of speech, dysarthrias (hypokinetic more than other types)

19
New cards

_____ are compulsive repetition of words and phrases.

palilalia

20
New cards

List the etiologies of palilalia.

Parkinson's disease/parkinsonism, Alzheimer's disease and other dementias, progressive supranuclear palsy syndrome, closed head injury, stroke, tumor, multiple sclerosis, Tourette's syndrome, post thalamotomy, bilateral basal ganglia pathology

21
New cards

The characteristics of palilalia include: ______ of words and phrases, ____ rate and ____ loudness with successive repetitions, most prominent during ____ and ____ speech (tends to be reduced during reading, repetition, and automatic speech tasks), most common toward ______, awareness of deficit is possible but no ______, ____ can be inhibited temporarily with effort.

repetitions of words and phrases; increased rate and decreased loudness with successive repetitions; most prominent during spontaneous and elicited speech; tends to be reduced during reading, repetition, and automatic speech tasks; most common toward end of utterances but can occur anywhere; awareness of deficit is possible but no anxiety or secondary struggle; reiterations can be inhibited temporarily, with effort

22
New cards

True or false: Adaptation effect in people with palilalia is common.

False; it is uncommon

23
New cards

____ is unsolicited repetition of another's utterances.

echolalia

24
New cards

The characteristics of echolalia include: ____ of others' utterances, _____/____ quality, repetition may be ____ or ____, sometimes with spontaneous correction of syntax.

unsolicited repetition of others' utterances; compulsive, parrot-like quality; repetition may be complete or partial, sometimes with spontaneous correction of syntax

25
New cards

What are the types of echolalia? (4)

mitigated (only some words, helping comprehension); ambient (portions of speech from other conversation); effortful (self generated response, repeat phrases before other person gets response out); silent, simultaneous (quietly echoes)

26
New cards

List the etiologies of echolalia.

Stroke, Alzheimer's disease, Pick's disease and other dementias, PSPS, corticobasal syndrome, carbon monoxide poisoning, Tourette's syndrome, status epilepticus, schizophrenia, mental retardation, ASD, post emergence from coma

27
New cards

True or false: echolalia can co-occur with aphasia and diffuse cognitive deficits.

true

28
New cards

______ presents with reduced speed of verbal responding, reduced linguistic and cognitive complexity of content, reduced vocal loudness and incomplete phonation, and flattened prosody. Associated deficits include ______ impairments and ______ associated with postintubation, psychogenic. This is caused by closed head injury.

attenuation of speech; cognitive/affective; dysphonia/aphonia

29
New cards

____ are involuntary productions (verbal or vocal tics, repetitive grunting, groaning, humming, or lip smacking), associated with diffuse cognitive impairment. The etiologies include ____ and ____.

disinhibited vocalizations; Alzheimer's disease and Tourette's syndrome

30
New cards

____ is a neurologic speech disorder in which articulatory and prosodic characteristics are perceived as a foreign accent (_______). There is _____ among listeners regarding the specific ____ perceived.

foreign accent syndrome; pseudoforeign accent; unreliability; accent

31
New cards

The deviant speech characteristics of foreign accent syndrome include: _______( diphthongization, distortions and prolongations, ____ of unstressed vowels, _____), _____ (alterations in voicing, place, and manner features, leading to perception of ______), and ______ (alterations in stress, rhythm, and intonation).

vowel changes (diphthongization, distortions and prolongations, omissions of unstressed vowels, epenthesis)

consonant changes (alterations in voicing, place, and manner features, leading to perception of substitutions)

prosodic changes (alterations in stress, rhythm, and intonation)

32
New cards

List the etiologies and associated deficits of foreign accent syndrome.

Stroke, CHI, MS, brain tumor, dementia, and primary progressive aphasia

33
New cards

_____ are disturbances in the prosodic components of speech that are tied to the expression of attitudes, emotion, and emphasis. It required an ability to manipulate speech planning, programming, and monitoring for pragmatic/social purposes.

aprosodia

34
New cards

Patient complaints of aprosodia include: voice does not _______, altered ____(either higher or lower), reduced ____ and ____.

convey felt emotions; pitch; pitch range; loudness

35
New cards

The perceptual characteristics of aprosodia include: _____, ____ like spontaneous prosody, reduced ___ and ____ variation, reduced or abnormal _____, reduced ___, ____, and ___, tendency to equalize ____, poor expression of ___ and ____, poor projection of ____, lack of emphasis, and abnormal quality to emotional crying and laughter.

flattened, robot like spontaneous prosody; reduced pitch and loudness variation; reduced or abnormal intonational range; reduced affect, expression, emotion (indifferent); tendency to equalize stress; poor expression of irony and sarcasm; poor projection of voice; lack of emphasis; abnormal quality to emotional crying and laughter

36
New cards

List the accompanying deficits of aprosodia.

paucity of spontaneous emotional and propositional gestures; left-sided neglect; visuoperceptual disturbances; cognitive-communication deficits; left central facial weakness; dysarthria (UUMN); left hemiparesis

37
New cards

True or false: stroke is the most common etiology of aprosodia and can include any process that damage right or nondominant hemisphere.

True

38
New cards

____% of all patients with acquired communication disorders present with functional speech disorders. Associated conditions include ____ and ____.

4.9%; conversion disorder, somatization disorder

39
New cards

List the types of functional speech disorders/how they present.

Stuttering-like dysfluencies; prosodic abnormalities; articulation abnormalities; voice; language/cognitive abnormalities; breathing abnormalities

40
New cards

Individuals with functional speech disorders, stuttering-like dysfluencies can includes excessive ______ (long stretches of fluent with brief severe stuttering or vice verse), excessive _____ (stuttering on every other sound or syllable), speech ______, and absence of ____, ____, and ____.

variability; consistency; speech struggle behavior; absence of aphasia, AoS, and dysarthria

41
New cards

_____ involves physical symptoms that suggest a medical or neurologic cause but for which there is no demonstrable organic cause. There is ___ or _____ of volitional muscle control or sensation that is not consciously motivated.

Conversion disorder; loss or alteration

42
New cards

Patient characteristics of conversion disorder include: may occur in _____ when in a stressful situation, _______ frequently present, may have history of _____, ____, or _____. It will effect more ___ than ____.

healthy individuals when in a stressful situation; psychological disorder are frequently present; may have history of drug/alcohol, sexual abuse or poor relationships; affect more women than men

43
New cards

__ is a chronic illness characterized by recurrent, multiple physical complaints and a belief that one is ill. It usually develops ______ and may be accompanied by _____, ____, and _____. There is a lack of acute conflict, stress, or anxiety.

somatization disorder; before age 25; interpersonal problems, impulsivity, and antisocial personality traits

44
New cards

List the questions to ask yourself when diagnosing a functional speech disorder.

Can the speech disorder be classified neurologically?

Are oral mech exam findings consistent with the speech disorder and patterns of abnormality found n neurologic disease?

Is the speech deficit consistent? suggestible? susceptible to distractibility?

Does speech fatigue in a lawful manner?

45
New cards

____ can be congenital or acquired. There are different types depending on etiology.

childhood dysarthria

46
New cards

True or false: cerebral palsy type will always correspond with the dysarthria type.

false

47
New cards

What are the 3 types of cerebral palsy? ____ is the most common, may affect different body parts, muscles affected by ____, and may retain primitive reflexes. ____ is the next most common, affects the entire body, present with dystonia (uncontrollable muscle contractions) and choreoathetosis (involuntary twitching or writhing). ____ is the least common, affects the entire body, and hypo- or hypertonia, movement overshoot.

spastic; spasticity; dyskinetic; ataxic

48
New cards

What are the 2 types of functional motor classification systems for CP? ____ describes functional gross motor skills and is used for ages 6-12 years. ____ describes fine motor skills and is used for ages 4-18 years.

Gross motor function classification system (GMFCS); Manual Ability classification system

49
New cards

Impairments that may accompany cerebral palsy include: _____, _____, _____, _____, _____, and ______.

musculo-skeletal problems, seizure disorders, sensory impairments, intellectual disability, saliva management, feeding disorders

50
New cards

Speech characteristics in spastic cerebral palsy include ____, ____/____, and ____.

breathy, monopitch/monoloudness, hypernasality

51
New cards

Speech characteristics in dyskinetic cerebral palsy include ____, ___, and ____.

slow rate, inappropriate voice stoppages, dysrhythmia, reduced stress

52
New cards

Speech characteristics in ataxic cerebral palsy include ___ and ___.

dysrhythmia, irregular articulatory breakdowns

53
New cards

True or false: language and/or cognitive deficits may be present with all CP types.

true

54
New cards

List some other etiologies of childhood dysarthria (not including CP).

degenerative disease; infectious disease; fragile X; TBI; stroke; types of dysarthria (depending on site of lesions)

55
New cards

True or false: Cerebral palsy is the most common cause of childhood apraxia of speech.

false; childhood dysarthria

56
New cards

_____ is a disorder of motor programming/planning and is difficult to diagnose before the age of 3. It accounts for ___% of all childhood speech sound disorders.

childhood apraxia of speech; 5%

57
New cards

True or false: there is no gold standard of speech characteristics of childhood apraxia of speech.

true

58
New cards

True or false: non-verbal oral apraxia is always present in CAS

false; may or may not be present

59
New cards

True or false: CAS is relatively common and overdiagnosed.

false; relatively uncommon and overdiagnosed

60
New cards

True or false: CAS is higher in males than females (2-3:1)

true

61
New cards

What are the red flags for CAS?

little to no babbling; very reduced number of sounds; lack of differentiating vowels

62
New cards

Why can diagnosis of CAS before age 3 be difficult?

presence of developmental disabilities or co-morbid conditions; no single list of diagnostic features; some primary characteristics of CAS are characteristic of emerging speech in typically developing children; lack of sufficient speech sample size; inability versus unwillingness; changes in speech occurring during the first 3 years may alter the diagnosis

63
New cards

___ feature list for CAS includes inconsistent errors on consonants and vowels in repeated productions of syllables or words, lengthened and disrupted coarticulatory transitions between sounds and syllables, and inappropriate prosody, especially in the realization of lexical or phrasal stress.

ASHA consensus-based feature list

64
New cards

______ checklist includes difficulty achieving initial articulatory configurations and transitions into vowels, syllable segregation, lexical stress errors or equal stress, vowel or consonant distortions including distorted substitutions, groping (nonspeech), intrusive schwa, voicing errors, slow rate, slow DDK rate, and increased difficulty with longer or more phonetically complex words.

Strand's 10-point checklist

65
New cards

CAS assessment should include _____, _____, ______ (% consonants correct, syllable segregation, and lexical stress problems), and _____. Be sure to assess ____ and/or ______, sequences of ____, articulation of ____ and ___, spontaneous speech, and prosody.

hearing screening, language screening/assessment, multisyllabic words picture naming test (% consonants correct, syllable segregation, and lexical stress problems) and oral motor exam including diadochokinesis; praxis, orofacial movements, orofacial movements, syllables and phonemes

66
New cards

List some examples of CAS assessment protocols.

Diagnostic Evaluation of Articulation and Phonology (DEAP); Verbal motor production assessment for children; dynamic evaluation of motor speech skill; the orofacial praxis test; kaufman speech praxis test for children; madison speech assessment protocol

67
New cards

____ is a cultural consideration of CAS and may have an increased impact on intelligibility and error frequency.

tonal languages

68
New cards

List the cultural considerations of CAS assessment and diagnosis.

errors may be manifested differently in each language; may rely on earlier mastered sounds across all languages spoken; may appear to favor or use one language over another (but difference may be due to the relative ease of the phonemic inventory and word structure in that language rather than an indication of language choice or dominance)

69
New cards

True or false: Response to treatment will not help differentially diagnose CAS.

false; may help differentially diagnose CAS

70
New cards

Managing motor speech disorders may involve improving ____, ______, ____, ____, and ____.

speech intelligibility; comprehensibility; efficiency; naturalness; AAC

71
New cards

True or false: Onset of management of MSDs doesn't matter.

False; management should start early

72
New cards

True or false: Baseline data and organization of sessions are important.

true; baseline data is important for establishing goals and measuring change

73
New cards

The goals of MSD management are to be as ____, ____, and ____ as possible. You can achieve this by _____, ____, or _____. ____ a disorder might not be realistic if there is an entire loss of function. _____ promotes use of residual function. _____ a disorder reorganizes the environment or responsibilities (these adjustments might be temporary or permanent).

effective, efficient, and natural as possible; restoring, compensating, or adjusting; Restoring; compensation; adjusting

74
New cards

List the factors that influence decision making for management of MSDs.

medical prognosis; impairment, limitation, and restrictions; environment and communication partners; motivation and needs; associated problems; and healthcare system

75
New cards

True or false: Treatment duration and intensity is determined by: course of the disease, severity of deficits, specific goals of management, prognosis, motivation, communication needs, resources, and healthcare coverage.

true

76
New cards

What are the broad approaches to management of MSDs?

Medical (pharmacological-meds, botox; surgical); Prosthetic (assistive devices); Behavioral: speech oriented- increasing physiologic support or making mx use of residual abilities or communication oriented- environmental manipulations, educating frequent communication partners

77
New cards

True or false: AAC includes gestures, symbols, and aids.

true

78
New cards

Principles of motor learning are ____ number of trails, ____ practice, ____ practice, ____ practice, and ____ practice.

large number of trials; distributed practice; variable practice; random practice; complex practice

79
New cards

When referring to neuroplasticity, ____ areas must assume function. Changes supporting recovery are similar to those ______ in typical brains.

undamaged; underlying learning

80
New cards

True or false: Neuroplasticity role of some changes remains controversial.

true

81
New cards

Compensatory behaviors that induce brain changes may be ______ and _____ or may be ______.

adaptive and improve function; maladaptive (development of seizures)

82
New cards

List the principles of exercise-dependent neuroplasticity.

use it or lose it, use it and improve it, specificity, repetition matters, intensity matters, time matters, salience matters, age matters, transference, interference

83
New cards

What are the 5 considerations you should consider when organizing treatment sessions?

frequency, task ordering, error rates, fatigue, individual vs group therapy

84
New cards

True or false: All people with a MSD are a candidate for therapy.

false; not all people with a MSD are a candidate for therapy

85
New cards

True or false: Not all approaches are appropriate for all dysarthria types and some may be contraindicated.

true

86
New cards

True or false: Speaker-oriented and communication-oriented approaches are not appropriate and are not necessary for many individuals with dysarthria.

false; both speaker-oriented and communication-oriented approaches are appropriate and necessary for many individuals with dysarthria

87
New cards

True or false: If an individual has adequate loudness and appropriate breathing for speech, you should continue to focus on respiration.

false; don't focus on respiration

88
New cards

Referring to speaker-oriented treatment for dysarthria, how can you increase respiratory support?

required 5cm water pressure for 5 sec, controlled exhalation, pushing/pulling/bearing down, inspiratory and expiratory muscle strength training (allow for larger/faster inspirations, specifically for individuals with hypokinetic dysarthria)

89
New cards

List the types of prosthetics used for respiration speaker-oriented treatment for dysarthria.

abdominal trussing for posture and support: binding/corset, supervised and limited, individuals with spinal cord injury or paralysis of abdominal muscles

abdominal force for increased expiratory strength (lean or push on something)

90
New cards

Respiration speaker-oriented behavior treatment for dysarthria includes: ____ before speech, ____ per expiration (establish optimal breath group and advance as tolerated), and _______ + _______.

deep breath before speech, limit breaths per expiration, speech breathing treatment + Lee Silverman Voice Therapy (LSVT)

91
New cards

Speaker-oriented surgical phonation treatment for dysarthria includes _________ (medialization type I thyroplasty, arytenoid adduction surgery, or surgical anastomosis) and _______ (recurrent laryngeal nerve resection, botox injection, laryngeal adductor denervation-reinnervation, lateralization type II thyroplasty).

vocal fold paralysis or bowing; adductor spasmodic dysphonia

92
New cards

List the prosthetics for phonation speaker-oriented treatment for dysarthria.

SpeechVive for increasing loudness in individuals with Parkinson's disease, ambulatory voice monitoring and feedback devices; portable voice amplifier

93
New cards

Phonation speaker-oriented behavior treatment for dysarthria includes _____ techniques, ______ at the beginning of exhalation, ____ head, _____ manipulation, and ______.

effort closure techniques; initiate phonation at the beginning of exhalation; turning head; lateral digital manipulation; LSVT

94
New cards

True or false: Phonation speaker-oriented behavioral treatment for dysarthria is not effective for strained vocal quality.

true

95
New cards

True or false: Resonance speaker-oriented medical/surgical treatment has limited data.

true

96
New cards

_____ is a type of resonance speaker-oriented medical/surgical treatment for dysarthria that gets injected into pharyngeal walls that have a gap in pharyngeal wall and soft palate.

injection pharyngoplasty

97
New cards

List the resonance speaker-oriented prosthetic treatment types.

palatal lift prothesis, nasal obturator or one-way nasal speaking valve (occludes any airflow coming out)

98
New cards

Resonance speaker-oriented behavioral treatment for dysarthria includes modifying _____, _____ (CPAP), _____, and ______ movement (usually not effective).

the pattern of speaking, resistance training, feedback, and nonspeech velopharyngeal movement

99
New cards

True or false: Neural anastomosis, which is an articulation speaker-oriented medical/surgical treatment for dysarthria, is not effective for speech and is for cosmetics/functionality.

true

100
New cards

True or false: A botox injection can reduce tremor or dystonia for individuals with dysarthria, specifically focusing on articulation.

true