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Female sexual interest/ arousal disorder
Lack of or significantly reduced sexual interest/arousal
Typically manifesting in
Reduced sexual interest
Reduced sexual activity
Fewer sexual thoughts
Reduced arousal to sexual cues
Reduced pleasure or sensations during almost all sexual encounters
And causes significant distress
Research suggests 7-46% of women experience low sexual desire
Male hypoactive sexual desire disorder
Little or no interest in any type of sexual activity
Masturbation, sexual fantasies, & intercourse are rare
Affects approximately 5% of men
As w/ other disorders: Not better explained by something else
Genito-pelvic pain/penetration disorder
FEMALE-SPECIFIC
In females, difficulty w/ vaginal penetration during intercourse, associated w/ one or more of the following
Pain during intercourse or penetration attempts
fear/anxiety about pain during sexual activity
Tensing of pelvic floor muscles in anticipated of sexual activity
Note:
Can be → Actual pain or Anxiety regarding Sex
Can also happen w/ pelvic muscle spasms involuntary
Erectile disorder
MALE-SPECIFIC
Difficulty achieving or maintaining an erection
Sexual desire is usually intact
The most common problem for which men seek treatment
Prevalence increases w/ age
50% of those over 60 experience erectile dysfunction
Only 2% under age 40
outside factors such as severe relationship stress that could be accounting for symptoms
if there is Relationship stress
if there is a Divorce
Any other outside reasons that could explain less sexual interest
reasons why healthcare professionals may be less inclined to bring up sexual health in an appointment with a patient/client.
Why would providers not bring it up?
Minimally education
they don’t feel competent in addressing these problems
Time crunch/constraint -> @ most 15-minute window
Address other/bigger issues
Personal factors
psychological disorders that may make someone more vulnerable to sexual dysfunction
Anxiety regarding Sex
Depression can cause less sexual interest
Substance Abuse can lead to sexual disfunction
social/cultural influences on sexual functioning
Learned negative attitudes toward sex & sexuality (Erotophobia)
Negative sexual experiences
Relationship challenges; dissatisfaction w/in romantic relationships
how physical disorders/disability can impact sexual functioning
Physical disability often increases the likelihood of sexual functioning problems
People w/ more severe physical disability report on average lower sexual satisfaction
treatment options for sexual dysfunction.
Education
Masters & Johnson’s Psychosocial Intervention
Sensate focus & non-demand pleasuring
Sexual activity w/ the goal of focusing on sensation w/out trying to achieve orgasm
Use of dilators to help women w/ painful intercourse
Exposure to erotic material for problems w/ low sexual desire
Medications (oral, injectable), vacuum-pump devices for ED
Referral to appropriate medical professionals (ex PCP, PT)
distinguish between substance use and abuse
Substance abuse -> Use in a way that is dangerous and substantial impairment (e.g. affecting job or relationships)
DIFF - ABUSE is dangerous use affecting you & your functioning (job) while USE is moderate amounts but doesn’t affect functioning
Substance use -> taking moderate amounts of substance in a way that doesn’t interfere with functioning
distinguish between tolerance and withdrawal
Tolerance -> needing more of a substance to the same effect/reduced effect from the same amount
DIFF - Tolerance is needing a higher dosage to get the same effects @ the beginning & Withdrawal is physical discomfort after stopping/not regularly use
Withdrawal -> physical symptom reaction when a substance is discontinued after regular use
5 main categories of substances common features of each, and examples of drugs that belong to each category
Depressants
Behavioral sedation -> alcohol, sedative, anxiolytic drugs
Depress CNS -> reduce anxiety & make us sleepy
Stimulants
Speed up body systems
Increase alertness & elevate mood -> cocaine, nicotine, caffeine
Opiates
Produced analgesia (masking of pain) and euphoria
heroin, morphine, codeine, oxycodone
Opioids, narcotics, pain killers -> same category
Hallucinogens
Alter sensory perception
LSD, Marijuana
Other drugs of abuse
Include inhalants, anabolic steroids, medications
clinical features of substance use disorders
Pattern of substance use leading to significant impairment & distress
Symptoms (need 2+ w/in year)
{side note: 2-3 Mild, 4-5 Moderate, 6+Severe}
Taking more of substance than indented
Desire to cut down use
Excessive time spent using/ acquiring/recovering
Craving for the substance
Took the place of legal problems related to substance
Role disruption (eg can’t perform @ work
Interpersonal problems
Reduction of important activities
Use in physically hazardous situations
common comorbid diagnoses with substance use disorders
Approx 75% of ppl in addition to treatment meet the criteria for at least 1 other psychiatric disorder
Mood disorder (40% or more)
Anxiety disorder & PTSD (25% or more)
When other psychiatric symptoms occur in the context of active substance use, clinicians must proceed carefully w/the diagnosis
long-term effects of heavy drinking.
Tremors
In hands
Withdrawal
Nausea/vomiting
Part of withdrawal
Hallucinations
Agitation
Insomnia
Seizures
Delirium tremens (DTs) in severe cases
Small %’s -> 3-5% w/ severe hx
Involves disorientation
Mental confusion
Hallucinations
The body starts shutting down
Effect regulation
HR & respiration
Liver disease
Cirrhosis of the liver
Permanente scarring of the liver
Pancreatitis
Cardiovascular disorders
Depression
Certain cancers
Esophageal
Colon
Breast
Brain damage
Dementia
Cognitive decline
Wernicke-Korsakoff Syndrome
Describe what delirium tremens (DTs) are.
the most severe form of alcohol withdrawal. It causes your sympathetic system to go into overdrive which then causes an irregular cardiovascular system. As a result people get really bad confusion, shaking, high blood pressure, fever, and hallucinations. This can be fatal.
what counts as a standard drink for beer, wine, and liquor.
12 fl oz of regular beer
Assuming 5% proof
5 fl oz of table wine
Assuming 12% proof
1.5 fl oz shot of distilled spirits (liquor)
gin, rum, tequila, vodka, whiskey, etc.
Assuming 40% proof
what is meant by moderate, heavy drinking, and binge drinking for men and women.
Moderate
no more than 2 drinks/day (men)
No more than 1 drink/day (women)
Some ppl (e.g pregnant women) should not drink
Heavy drinking according to NIAAA
For MEN consuming > 4 drinks on any day or more than 14/week
For WOMEN consuming > 3 drinks on any day or more than 7/week
Binge-drinking
4+ drinks for women
5+ for men w/in 2 hr period
Amt of alcohol blood level gets to 0.80
questions asked on the CAGE alcohol use disorder screening tool
Cutting down
Ex: have you ever felt that you should CUT down on your drinking
Annoyed
Ex: Have you ever become ANNYOYED by criticisms of your drinking
Guilty
Ex: Have you ever felt GUILTY about your drinking
Eye opener
Ex: Have you ever had a morning EYE OPENER to get rid of a hangover
what is meant by prescription drug misuse/abuse
Taking medication in a way that it was not originally intended
Describe how opioids have their effect
Opioid drugs bind to opioid receptors in the CNS
Bind to receptors
Inhibit production of GABA
GABA -> neurotransmitter -> helps regulate amt of dopamine
Allows more dopamine to be available in the brain
Feel good, pleasure, ect (when it’s active in body)
why someone would be at high risk of overdose from a relapse after a period of abstinence
They lose tolerance -> become too much (dose)
relevant biological treatments (like Naltrexone and Methadone) for opioid use disorder and alcohol use disorder
Naltrexone
FDA-approved for opioid & alcohol use disorders
Blocks pleasant effects of drugs; reduces cravings
Pt must complete the detox (medically managed withdrawal) prior to initiating
Methadone
FDA-approved for opioid use disorders
Blocks pleasant effects of drugs; reduces cravings
Potentially addictive
Suboxone
FDA-approved for opioid use disorders
Blocks pleasant effects of drugs; reduces craving
Potentially addictive
some of the positive outcomes of medication-assisted treatment for opioid use disorder
Lower the risk of fatal overdoses by approx 50%
Lower risk of non-fatal overdoses
Reduce drug-injecting
Reduce HIV transmission
Reduce criminal activity by opioid users
general nature of personality disorders (PDs)
The nature of personality disorders
Enduring, inflexible predispositions
Maladaptive, causing distress and/or impairment
High comorbidity
Poorer prognosis
Don’t tend to show up for treatment on own or stick with
More likely to show from other paths (ordered, fam)
3 personality clusters and be able to identify PDs belonging to each cluster
Cluster A
Personality disorders
Odd or eccentric cluster
includes paranoid, schizoid, schizotypal
Cluster B
personality disorders
Intense, unpredictable
dramatic, emotional, erratic cluster
Includes antisocial, borderline, histrionic, narcissistic
Cluster C
Personality disorders
Diff than anxiety -> stable across time ingrained personality
Fearful or anxious cluster
Includes avoidant, dependent, obsessive-compulsive
Ob-comp -> diff than OCD
common focus of treatment for Cluster A PDs.
Focus on interpersonal skills
Using cognitive strategy to combat
What's the evidence
Building trust where paranoia is a factor
Address comorbid conditions
Targeting interpersonal, and social skills, basically trying to reduces paranoia
CBT - focus on positive behaviors & reduce anxiety
clinical features of antisocial PD.
Antisocial PD -> CLUSTER B
Failure to comply w/ social norms
Violation of the rights of others
irresponsible, impulsive, and deceitful
Lack of a conscience, empathy, & remorse
Lack of remorse -> Engage in deceitful but don’t feel guilty
May be very charming, interpersonally manipulative
Likable -> until known
Used car sales men (idea -> want the sale)
Most often diagnosed in males
common features of early/family history of antisocial PD
Common of Early & Fam hx
Relation w/ early behavior prob & conduct
Early hx of behavioral problems, including conduct d/o
“Callous-unemotional” type of conduct disorder more likely to evolve into antisocial PD
Youth-> set fire, hurt animals -> NEED TO HAVE EVIDENCE CONDUCT DISORDER BEFORE AGE 18 -> to meet criteria to have antisocial personality disorder
Families w/ inconsistent parental discipline & support
Families often have hx of criminal & violent behavior
Can run on families
clinical features of borderline PD
Borderline PD - CLUSTER B
More women than men have/been diagnosed
Unstable moods and relationships
Have a pattern of rocky short-lived relationships
impulsivity, fear of abandonment, poor self-image
Instability in moods (affect)
Think of walking on eggshells
Chronically feeling of emptiness
Mood disorders -> MDD common (comorbidity)
Self-mutilation & suicidal gestures
treatment options for borderline PD
Antidepressant medications provide some short-term relief
Dialectical behavioral therapy
Marsha Linehnd - orig for Borderline PD
4 main models -> can be done as a group or individually
Mindfulness: the practice of being fully aware and present
Distress Tolerance: how to tolerate pain in different situations
How can I ride a wave of intense emotion w/out being harmful (to others or myself)
Interpersonal Effectiveness: how to ask for what you want & say no while maintaining self-respect and relationships with others
Learn skills to more effectively relate w/ other ppl
Emotion regulation: how to decrease vulnerability to painful emotions & change emotions that you want to change
How can help ppl stay grounded & stable
clinical features of obsessive-compulsive PD and how it differs from OCD.
Obsessive-Compulsive PD -> CLUSTER C
Excessive & rigid fixation on doing things the right way
This is the way it should be done and will always be done
Highly perfectionistic and orderly
Obsessions and compulsions are rare
DIFF -> OCPD -> NOT HAVING OBSESSIONS & COMPULSIONS
DSM-5 criteria for schizophrenia
A. Two (or more)
(1) delusions
(2) hallucinations
(3) disorganized speech
(4) grossly disorganized or catatonic behavior
(5) negative symptoms
B. level of functioning in one or more major areas, such as work, interpersonal relations, or self-care, is markedly below the level achieved prior to the onset.
C. at least six months.
what hallucinations are
Hallucination
Experience of sensory events w/out environmental input
Can involve all senses
Tasting something when not eating
Having skin sensations when not being touched
Most common: Auditory
types of hallucinations
Auditory - hearing things that aren’t there
Subtype: command hallucinations -> more safety risk -> voice telling to do something
Feeding negative beliefs
Running commentary
Narration of daily behaviors
Visual - seeing things that aren’t there
Animals or people
Olfactory - smelling things that aren’t there
Smelling rotten eggs (common)
Tactile - feeling things that aren't there
A sensation of bugs crawling under the skin (common)
what delusions are and be able to recognize examples of delusions of grandeur and persecution
Delusions
Gross misrepresentations of reality
Most common
Delusions of grandeur
having this special skill, talent, etc.
I am the king of England
I can speak directly to God
Delusions of persecution
Out to harm and get them
“CIA is tracking me”
positive symptom clusters in schizophrenia
Hallucination
Experience of sensory events w/out environmental input
Can involve all senses
Tasting something when not eating
Having skin sensations when not being touched
Most common: Auditory
Delusions
Delusions of grandeur
having this special skill, talent, etc.
I am the king of England
I can speak directly to God
Delusions of persecution
Out to harm and get them
“CIA is tracking me”
negative symptom clusters in schizophrenia
The negative symptoms
Absence or insufficiency of normal behavior
Spectrum of negative symptoms
Avolition (or apathy)
lack of initiation and persistence
Alogia
relative absence of speech
Anhedonia
lack of pleasure or indifference
Affective flattening
little expressed emotion
disorganized symptom clusters in schizophrenia
Disorganized speech
Tangentiality - “going off on a tangent”
Start in one place & ends at another
Loose associations - a conversation in unrelated directions
Blocking Speech -> halting, interrupted speech -> Robot reading, minor 2 pauses between thoughts
Disorganized affect
Inappropriate emotional behavior
someone says they feel one way but the face says something else
Disorganized behavior
Includes a variety of unusual behaviors
Pacing, inappropriate eye contact
Catalonia
clinical descriptions for schizophreniform
Psychotic symps lasting btwn 1 & 6 months
Need 2+ symp
Delusions, hallucinations, disorganized speech or catatonic behavior, neg symptoms
Note: Can develop into schizophrenia -> if symptoms keep occurring 6+months
clinical descriptions for brief psychotic disorder
Psychotic symptoms lasting less than 1 month
Need 1+ symptoms(s)
Delusions, hallucinations, disorganized speech, disorganized or catatonic behavior
Note: Some ppl can recover and never have another, others not so much
clinical descriptions for schizoaffective disorder
Note: mood disorder [depression type] meets psychotic symptoms [schizophrenia] & Timeline & order matters
NEED TO HAVE STANDALONE PSYCHOTIC SYMPTOMS
Psychotic symptoms need to happen outside the mood disturbance
Prognosis symptoms must also occur outside the mood disturbance
Such persons don’t tend to get better on their own
clinical descriptions for catatonia
Unusual motor responses, particularly immobility or agitation, and odd mannerisms
Tends to be severe & quite rare
May be present in psychotic disorders or diagnosed alone
May include
Stupor, mutism, or maintaining the same pose of hours
Opposition to lack of response to instruction
Repetitive, meaningless motor behaviors
Mimicking others’ speech or movement
differential diagnoses for schizophrenia (disorders we would need to rule out because they share similar clinical features)
Personality disorders
Cluster A
Substance use disorder
Bipolar disorder
Type I -> more severe
Major Depression Disorder
Might have psychotic symptoms during a severe episode
Medical condition
Parkinson’s Disease
Lewy Body Dementia
prevalence rate of schizophrenia, demographic patterns in the age of onset, and the relationship between schizophrenia and life expectancy
prevalence of schizophrenia worldwide
Abt 1% of population
Often in early adulthood
Demographic
Males -> early 20s
Females -> (bimodal) -> late 20s & mid 40s
Lower life expectancy
Due to increased risk of suicide, accidents, & poorer self-care
what is meant by the “prodromal” phase of schizophrenia.
Is the stage before diagnosis when there are psychotic behaviors that have developed but not enough to warrant a psychological disorder
alternative:
This is a phase of schizophrenia where individuals have intense mood swings, trouble with memory, depression, thoughts of suicide. This is basically a lot of the aspects of schizophrenia but no psychosis or hallucinations.
causal factors and correlates of schizophrenia
Genetics vs environment
Pretty high genetic -> risk increase depending on how close they are (mom vs cousin, mom - the child has higher risk)
BUT a healthy environment is a protective factor
Dopamine hypothesis
Schizophrenia is partially caused by overactive dopamine
Drugs that increase dopamine (agonists) result in schizophrenic-like behavior
Drugs that decrease dopamine (antagonists) reduce schizophrenic-like behavior
Neurobiological
structural & functional abnormalities in the brain
treatment options for schizophrenia and describe what “tardive dyskinesia” is
antipsychotic medication + psychosocial interventions
Social skills/living skills training
Family therapy
Vocational rehab
Noncompliance w/ meds is common issue
Meds have major & permanent side effects
Tardive dyskinesia -> a drug-induced side effect that causes involuntary muscle movements
inattentive symptoms of ADHD
Not giving close attention to details
Difficulty maintaining attention
Trouble w/ follow-through
Avoids tasks that require sustained mental effort
Often lose things necessary for tasks/activities
Easily distracted &/or forgetful
hyperactive/impulsive symptoms of ADHD
Fidgeting in seat
Often unable to engage in leisure activities quietly
“Driven by a motor”
Running/climbing in situations where inappropriate (or feelings of restlessness in adults)
Blurting out answers before questions in completed
Difficulty waiting their turn
recognize someone with ADHD, predominately inattentive type
Inattentive type of ADHD is when a person has the symptoms
ex: Trouble focusing, Trouble with time management, ect.
recognize someone with ADHD, predominately hyperactive/impulsive type
Hyperactive/impulsive ADHD type is when only hyperactive symptoms are present
ex: “Driven by a motor”, blurting out answers, ect.
recognize someone with ADHD, combined type
When both inattentive and hyperactive/ impulsive symptoms are present
ex. losing things & fideting ing seat
other diagnostic features of ADHD
Several sympt must be present before age 12
If older -> psych asks abt childhood
Symptoms present in 2 or more settings
Significant distress or impairment
Not better explained by another condition
prevalence rates/trends for ADHD
This occurs in approx 5% of school-aged children
Symptoms are usually present around the age of 3-4
Children w/ ADHD have similar probs as adults
Same criteria except hyper activity (restlessness)
Gender diff
Boys outnumber girls 3:1
Childhood prevalence by Race or Ethnicity, according to large national sample
White, non-hispanic 78.8%
Hispanic 9.1%
Black 6.0%
Asian 3.2%
Other 3.0%
prevalence rates/trends autism spectrum disorder (ASD)
Prevalence
1 in 44 children in US meet criteria
More commonly in males
4x more common
IQ interaction
Approx. 35% show intellectual disabilities
Most are fine
Worldwide prevalence = 1%
main areas of impairment in ASD and be able to recognize examples
Communication & social interaction
Restricted, repetitive patterns of behavior, interests, or activities
Be familiar with relationship between ASD and intellectual disability.
Those w/ ASD can have intellectual disability bc of their slower development
Be familiar with risk factors of ADHD
ADHD seems to run in the family
DAT1- Dopamine transporter gene has been implicated
Some ADHD drugs work by inhibiting DAT1
Neurobiological correlates of ADHD
Smaller brain volume
Hippocampus
Amigdla
Inactivity of the frontal cortex and basal ganglia (movement- voluntary- coordination)
Abnormal frontal lobe development & functioning
Decision making
Organization
Planning
Be familiar with risk factors of ASD
Biological dimensions of autism
Significant genetic componet
Familial component: if 1 child w/ austism, the 2nd w/ autism is 20% greater
100x greater risk than general population
Possible link btwn autism & levels of oxytocin (feel good/cuddle) receptor genes
Older pairent associated w/ increased risk
Particularly fathers @ time of conception
Neurobiological influences
Amygdala - fear response
Larger size @ birth = higher anxiety, fear
Elevated cortisol
Neuronal damage in the amygdala results from high stress, whch may affect processing social situations
Oxytocin
Lower levels
Bonding hormone -> might explain why might not need ppl in same sense as avg person
Vaccinations do not increase risk od autism
Mercury in some vaccinations was rumored to increase ausitm risk
Large-scale studies do not support this
High rates of vaccinations do not increase the risk for autism in the community at large
The health risk of not vaccinating is substantial
Be familiar with treatment options for ADHD
Biological Treat.
Stimulant medications
Currently prescribed for approximately 4 million American kids
Low doses of stimulants improve focusing abilities
Ex include
Ritalin, Dexedrine, Adderall
Behavioral treatment
Reinforcement programs
To increase appropriate behaviors/decrease inappropriate behaviors
Positive reinforcement -> token for good behavior
If bad -> token gets taken away (neg reinforcement for bad)
May also involve parent training
Combined bio-psycho-social treat
Often recommended
Maybe superior to medication or behavioral treatments alone
Be familiar with treatment options for ASD
Psychological
Behavioral approaches
Skill building
Reduce problem behaviors
Communication & language training
Increase socialization
Early intervention is critical - may “normalize” the functioning of the developing brain
Biological treat
Medical intervention has had little positive impact on core dysfunction
Sometimes in conjunction w/ psych-social
Manage symptoms -> if the child is causing harm to self -> banging head
Some drugs decrease agitation
Tranquilizers
SSRIs
Indicators or good prognosis
High IQ, good language ability