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Define Infection
Invasion of the body by a harmful organism or infections agent such as a virus, parasite, bacterium or fungus
Define Colonisation
Presence of microorganisms on a body surface (such as skin, mouth, intestines or airway) that does not cause disease in the person or signs of infection
Describe the chain of infection
Infectious Agents → Susceptible Host → Portal of Entry → Mode of Transmission → Portal of Exit → Reservoir → Infectious Agents
Describe patient placement in the context of IPC
Clear handover of information including symptoms of infection or known infections
May prompt instigation of isolation facilities
Describe the 5 moments of Hand Hygiene in the context of IPC
Clean hands before touching patient
Clean hands before any ascetic procedure
Clean hands after exposure risk to bodily fluids
Clean hands before touching patient and patient surroundings
Clean hands after touching patient and patient surroundings
Which coloured buckets are used for which areas?
Red - Toilets and showers
Blue - General areas
Green- Kitchen and dining areas
Yellow - Ambulances
Describe the three steps of decontaminating environments and equipment
Cleaning
Involves fluid and friction, usually detergent and water to physically remove organic matter
Disinfection
Process of eliminating or redacting harmful micro-organisms from surface, e.g. Clinel wipes
Sterilisation
Killing all microorganisms through physical or chemical means. Not undertaken by LAS.
Describe which waste to dispose of where
Domestic waste, recyclables and general waste - clear and black bags
Offensive waste (non infections waste, e.g. PPE, sanitary waste, dressings) - tiger striped bag
Infectious waste (known infections items) - orange bag
Cat A waste (HART only, hazardous Cat A waste) - yellow bag
Medicinal sharps - Yellow box
Cytotoxic and cytostatic waste (Syntometrine) - Purple lid
How do you bag infectious or soiled linens?
Infected or soiled linens should be placed in the red water-soluble bags first, then placed into clear bags before being placed in the red used linen bin
Describe the Hierarchy of IPC controls, from most effective to least effective
Elimination (physically remove hazard)
Substitution (replace hazard)
Engineering controls (isolate people from the hazard)
Administrative controls (change the way people work)
PPE (protect worker with PPE)
What are some examples of micro-organisms spread by a contact route, and what PPE would you need?
MRSA, CDIFF, VRE, CPE
Gloves, Apron, Eye protection
What are some examples of micro-organisms spread by droplet transmission, and what PPE would you need?
Influenza, Meningococcal meningitis, Bodetella pertussis
Droplet mask, gloves, apron, eye protection (+FFP3 mask if aerosol generation procedures commenced)
What are some examples of micro-organisms spread by airborne transmission, and what PPE would you need?
TB, Chickenpox, Rubella, Measles
FFP3, gloves, gown, eye protection
What is the process for managing a sharps injury?
Bleed it
Wash it
Cover it
Report it
Describe the approach for Auscultation
Auscultation is the listening of sounds in the body, most commonly with a stethoscope.
You should auscultate working left to right to left again, back and forth in 6 spots on both the front and back. Press firmly and compare either side. Landmarks on the back include 6 on either side of the spine, and 2 under the shoulder blades. Landmarks on the front include 1 above each of the clavicles, three spots down along the chest and one on either side of the lateral portion of either side of the body.
What are the three categories of breathlessness?
Acute: suddenly or in minutes Subacute: over hours or days Chronic: weeks/months
How do you take a Resp rate and what is the normal adult resp rate?
Watch for chest rise and fall, measured over one minute. Can assist by placing a hand on their back or chest. Try to avoid telling your patient as they may subconsciously alter their breathing.
Normal adult resp rate range is between 12-20.
What is the ideal range for oxygen saturations in a normal healthy patient
Above 94%
What is the normal O2 saturation range for a pat with COPD?
88-92%
What is peak flow expiratory flow rate (PEFR) and how is it measured?
Peak flow is a measurement of how quickly you can breathe air out of your lungs, measured using a peak flow meter.
Explain the procedure, set to zero, get pat to hold level and exhale forcefully and quickly as much as they can. take 3 measurements and record the best. Compare on JRCALC with average for height and age. Asthma/COPD pat's may know their normal PEFR.
What is COPD, how is it diagnosed and what are the two most common diseases related to COPD?
Chronic Obstructive Pulmonary Disease (COPD) includes a range of illnesses that result in airflow obstruction. It is progressive and not fully reversible.
The two most common are chronic bronchitis and emphysema.
Diagnosed by cough and sputum production for at least 3 months in 2 consecutive years.
Describe Chronic Bronchitis, its signs and symptoms, and management.
Inflammation and increased mucus production in the bronchi/bronchioles narrows the airways, caused from long term infection and lung damage. Smoking is a huge factor affecting chronic bronchitis.
Signs and symptoms: productive cough, dyspnoea, cyanosis, tachycardia/pnoea, accessory muscle use, high BP, reduced 02, wheeze on auscultation, slow capillary refill, oedema, reduced level of consciousness/hypoxia (extreme cases)
Management: Full history, Resp assessment, Limit O2 to 6 mins on/6 mins off, position for comfort, ask about individual care plan, baseline obs and monitor
Describe Emphysema, its signs and symptoms, and management
Distension and destruction of alveoli by enzymes called proteases, which are normally kept under control by ant-proteases enzymes but smoking for example upsets this balance. Alveoli collapse, obstructing airway, and blister like air pockets can form (bullae).
Signs and symptoms: barrel chest (air trapped due to obstruction and ribs expand), pursed lips on expiration, productive cough, dyspnoea, tachycardia/pnoea, accessory muscle use, high BP, reduced 02, wheeze on auscultation, slow capillary refill, oedema, reduced level of consciousness/hypoxia (extreme cases)
Management: Full history, Resp assessment, Limit O2 to 6 mins on/6 mins off, position for comfort, ask about individual care plan, baseline obs and monitor
Describe chest infections, signs and symptoms, and management
An infection of the lungs. Main types are Acute Bronchitis and Pneumonia.
Most bronchitis cases are caused by viruses. Most pneumonia cases are caused by bacteria. Can cause fluid buildup in lungs (consolidation), sounds like crackles
Susceptible people include the extremes of age, overweight, pregnant, immunocompromised, smokers, and others with long term health conditions
Signs and symptoms: Persistent cough, yellow/green phlegm or haemoptysis, breathlessness, dyspnoea, febrile, tachycardia, pleuritic cp or tightness, confusion or disorientation
Management: full resp assessment, establish airway, if red flags pre alert, abs, 12 lead, consider o2
Describe Asthma, its signs and symptoms, and management
Caused by irritation of lower airway and immune response producing mucus, tightening muscles around bronchi, and swelling bronchi. Obstruction overcome by inspiration but expiration delayed by narrowing of the airway. Categorised as mild, moderate, severe and life threatening. Triggers may be cold, allergies, particulates in air, exercise.
Signs and symptoms: Wheeze on expiration, tachypnoea, tachycardia, cyanoses, exhaustion, altered level of cons, arrhythmia, hypotension, dib, cannot complete full sentence, accessory muscle use, silent chest
Management: full history (prev ED or ICU attendance, annual asthma review, med compliance), analysis of triggers, full respiratory assessment, oxygen treatment, bronchodilators, PEFR (if mild-moderate).
Describe pulmonary embolism, its signs and symptoms, and management
Obstruction of pulmonary vessels causing lung tissue death. Most common cause is a DVT, where a clot breaks off and travels via femoral and iliac veins through heart and into pulmonary vessels. Commonly misdiagnosed as panic attack.
High risk factors: surgery, trauma, disabilities affecting mobility, long flights, bed bound pats.
Signs and symptoms: Dys/tachypnoea, Tachycardia, Pleuritic CP, TLOC/syncope, cough/haemoptysis, pain worse on inhalation/stabbing pain, pain/thrombus in calf,
Differentials: pleurisy, pneumothorax, cardiac CP
Management: swift transport to hospital, ABCs, position of comfort, provide O2 of required, be prepared for cardiac arrest
Describe Coronary Artery Disease (I.e. CAD or ischaemic heart disease) and the process of atherosclerosis
CAD almost always caused by atherosclerosis of coronary arteries. Tears in the Tunica intima create build up in lipids and cholesterol in Tunica media (plaques). White blood cells create fibrous caps around the plaques. Causes narrowing of artery lumen (stenosis) leading to ischaemia.
Describe Stable Angina include signs, symptoms and management
Stable Angina: insufficient oxygen reaching myocardium from significant stenosis of at east one coronary artery due to coronary artery disease.
Signs/symptoms: Chest pain (can radiate) relieved at rest, dib, sob, increased WOB, indigestion like, belching, nausea, sweating
Management: Will usually self administer own glyceryl trinitrate (GTN). Place pat at rest, avoid walking moving excessively, analgesia as required, ECG.
Describe Acute Coronary Syndrome (ACS) including how it occurs and the three conditions
Includes: Unstable Angina, non-ST-segment elevation myocardial infarction (NSTEMI), ST-segment elevation myocardial infarction (STEMI)
Above process exacerbated by vasoconstricting substances associated with platelet aggregation, constricting and narrowing vessels.
Describe ACS signs, symptoms and management
Signs and symptoms: Chest pain not relieved at rest, nausea and vomiting, sweating, dib, pallor, impending sense of doom. 1/3 pats present atypically - watch out for sob, abdo pain and collapse/faint especially in the young (25-40), elderly (>75), unwell diabetics and unwell female pats
Management: 12 lead ASAP, correction of major ABC problems, administer medication (O2, entonox), rapid transport to nearest appropriate hospital
Explain the Pathophysiology of Heart Failure
Abnormality in cardiac structure or function, meaning the heart cannot deliver O2 at the rate the body needs. Can be acute or chronic (stable in last month).
Can be caused by previous MI, chronic hypertension, arrhythmias, genetic heart conditions.
L Ventricular Failure: Inefficient pumping of blood out of the heart to the body causes a backlog in the L ventricle, Mitral valve, L Atrium and pulmonary vein to the lungs.
R Ventricular Failure: Blood continues to back up from the lungs, pulmonary artery, R ventricle, R atrium, Vena cava, rest of body.
Describe Heart Failure signs, symptoms and management
Signs and symptoms include SOB, DIB, Tachypnoea, Tachycardia, Crackles, Fatigue, Haemoptysis, Congestion, Peripheral oedema
Management: Management of ABCs, pat at rest, full set of obs, o2 if required (<94%), ECG
Describe Sickle Cell, signs and symptoms, and management
Genetic disease where red blood cells are shaped like sickles. Reduced oxygen carrying capacity and can create blockages leading to hypoxia and pain.
Pats usually have UCPs with treatment plans and specific hospitals.
Signs and symptoms: Pyrexia, DIB, low saturations, coughs chest pain, pallor, fatigue. Dehydration, headache, priapism
Triggers for crisis: Exercise, altitude, infection, pregnancy, dehydration, stress, cold
Management: follow UCP, pain relief, repeat obs, ECG, consider fluids, put pat at rest and use chair, convey to specialist unit unless primary survey problems take precedence (e.g. HAC, HASU)
Describe Dementia
Disease that affects memory, thinking and daily life
1/14 over age of 65 have it
Risk groups are age, genetics, lifestyle
Controlled by medication but no known cure
Includes Alzheimer’s
Describe Parkinson’s Disease
Causes parts of brain to become damaged over many years
1/500 in UK
Men slightly more at risk of developing, though unclear what causes condition to occur
Treated with medication and physiotherapy
Describe Multiple Sclerosis (MS)
Affects brain and spinal cord, stopping nerve impulses from travelling correctly
Over 130000 in UK
Affects mainly woman and common disability in younger adults
Managed by medication but no cure
Describe Ataxia
Group of disorders affecting balance, coordination, speech
Mainly hereditary but can develop later in life
No cure, responds well to physiotherapy and medication
Describe paralysis
Defined as partial or complete loss of muscles function
Other terms include
-plegia (paralysis)
-paresis (muscle weakness or incomplete loss of muscle function)
Terms used to identify areas affected
Monoplegia - one limb
Hemiplegia - one side of body
Quadriplegia/tetrapelgia - all 4 limbs
Paraplegia - both lower limbs
What is a CVE and describe the different types and how they are differentiated
Cerebrovascular Event (CVE)
Stroke or TIA
Risk factors include Age, Sex, Ethnicity, Family Hx, Smoking, AF, Diabetes, Obesity, previous CVE or MI, Cholesterol, Diet, Hypertension
Stroke
Blood supply to brain interrupted
Signs and symptoms develop rapidly, last at least 24 hours
Bleed or blockage
TIA
Signs and symptoms of stroke but resolves within 24hrs
Anyone with continuing neurological signs during management - treat as stroke
Signs and symptoms
Numbness
Weakness or paralysis
Slurred speech
Blurred vision
Confusion/inability to communicate
Severe headaches
Management
FAST
If major ABC - nearest ED
If not but FAST+ HASU (If >10
Describe types of convulsions, fitting disorders and how they are managed
Convulsions are involuntary contractions and retractions of the muscles.
Generalised convulsion
Affect large part of cerebral cortex
Bilateral Tonic-Clonic seizures (BTCS)
Tonic phase - body wide rigidity, falls, crying out, tongue biting, jaw clenching
Clonic phase - regular jerking movements which eventually slow and stop - sometimes accompanied by incontinence
Prolonged period of confusion, disorientation, sleepiness or unconsciousness after convulsion called Post-Ictal phase
Absent convulsion
Staring/daydreaming/absent period
May be accompanied by repetitive motions
Last a few seconds with immediate recovery
Partial convulsion
Affects localised part of cerebral cortex, though may progress into generalised tonic clonic
Patient may be conscious or altered level of consciousness
May be changes in behaviour or mood
May be localised tonic clonic activity
Epilepsy
Uncontrolled firing of neurons in brain
Some well managed by medication, other struggle to manage
Can significantly limit cognitive development in children
Generally tonic clonic
Can progress to Status Epilepticus
Continuous convulsion lasting over 5mins
Series of convulsions (3 or more in an hour) close together with no recovery period in between
Non-epileptic seizures
FND (Functional Neurological Disorder) and PNES (Psychogenic non-epileptic seizures)
Usually last a lot longer
Usually triggered by psychological trauma
No tonic clonic period
Generally does not affect breathing or circulation
Other causes for non epileptic seizures
Hypoglycaemia
Kidney failure
Tumour
Infection
Alcohol
Eclampsia
Stroke
Management using the Primary Survey
R - if actively consulting, put on high-flow O2 and call for paramedic backup. If reduced LOC protect from injury and recovery position
A - Jaw may be clenched (trismus), may have bitten tongue, may be impossible to assess. Do NOT use OP if actively convulsing. Attempt NP if possible.
B - administer high flow, consider Capnogrophy if ventilating
C - oximetry may be difficult but can assess skin colour, tone, cap refill
D - reassess AVPU, any improvement? Convulsion caused by head injury? Check BM now! Could be Hypoglycaemia
E - look for medic alert bracelet, signs of underlying injection, obvious injury, protect pat from injury and protect dignity
Time critical if:
Major ABCD problem
Serious head injury
Status EP
Underlying infection
Remember when attending seizures
First, worst or longest should attend hospital
Describe meningococcal disease, symptoms of meningitis and septicaemia, and management
Bacterial infection of meninges caused by meningococcus bacteria. Presents as Meningitis, Septicaemia or combination of both. Can be fatal.
Meningitis symptoms:
Fever, vomiting, severe headache, rash (particularly non blanching rash), stiff neck, photophobia, sleepy/vacant, confusion/delirium, seizures
Septicaemia symptoms:
Fever/vomiting, limb/joint pain, cold and shivering, DIB, rash, sleepy/vacant, confusion/delirium
Management
Standard primary survey approach, look particularly for rash in E (however absence of rash does not exclude meningitis)
Time critical if
any major ABCD problem
Non-blanching rash
Any suspicion of meningitis/septicaemia
Describe coma and management
Coma is described as absence or loss of consciousness
GCS of 8 or below
May not always find cause, management is supportive
Causes
Stroke, convulsion, inadequate airway, inadequate breathing, hyper/hypoglycaemia, OD, meningitis, arrhythmias, carbon monoxide etc
Time critical if any major ABCD problem
Management
comprehensive obs, GCS, pupils
Thorough secondary survey
12 lead
Reassess
Scene for clues, med alerts, needle marks, consider trauma
Can you definitively diagnose causes of abdominal pain pre-hospitaly?
No. Can pin-point where pain is, how it is, timeline etc but cannot find cause
What are the four quadrants of the abdomen? (For assessment)
4 Quadrants
Left upper (contains some of liver, most of stomach, spleen, pancreas,
Right upper (contains majority of liver, gall bladder, some of stomach, first part of duodenum, right kidney)
Right lower (small and large intestine, appendix)
Left lower (small and large intestine)
What are the advantages of dividing the abdomen into quadrants for assessment?
Supports diagnosis
Highlights red flags
Aids documentation
What are the 9 quadrants of the abdomen? (For assessment)

What are some elements of primary survey you may pick up when someone has abdominal pain?
Airway - may have problems with vomit, saliva, blood
Breathing - rate may increase because of pain, may be shallow to avoid causing pain
Circulation - rate may increase because of pain, BP may be very high (pain) or very low and weak pulse (dehydration from D&V)
Disability - AVPU may be affected by low BP (dehydration)
What elements of your history taking are specific to abdo pain? (Using SAMLPE)
S - how they feel, any changes, ROS (resp, circ, abdomen of course)
A - allergies to medication food, any sensitivities, what normally happens when have thing you’re allergic to? (E.g. normally abdominal pain)
M - prescribed, OTC (over counter), recreational - any changes? Side effects?
P - medical and surgical, work back in time - any tests, scans, GP visits, hospital appointments? Chronic - eating disorders, alcoholic, IBD, Crohns
L - ins and outs - BO (bowls open), PU (Passing urine), D&V, diet and fluid intake
E - What led to calling 999, progression of symptoms, self care, progress
What should you consider in your review of symptoms for abdominal pain?
Headache, visual disturbances, appetite change, nausea/vomiting, dehydration, indigestion, belching
SOB (?due to pain), coughing anything up, pain
Pain, diarrhoea, constipation, urine output, BO/PU position related pain/relief
Swollen ankles, rashes, itching, weakness, pallor, sensation
What elements of the SOCRATES should you be particularly considerate of for abdo pain?
Site - get them to point specifically
Radiates - does it move anywhere? E.g. upper flank to wards bladder may be UTI, referred pain in shoulder (may be appendicitis)
What is Acute Abdomen?
Acute abdominal pains less than 7 days duration
Non-specific abdominal pain after examinations and investigations
What is a bowl obstruction, signs and symptoms, causes and key questions to ask?
Partial or complete obstruction of small or large intestine
Causes include
Adhesions
Hernias
IBD
Tumours
Diverticulitis
Signs and symptoms
Bile stained vomit
Abdsolute constipation
Absence of flatus/wind
Faecal vomiting
Abdominal pain
Nausea/vomiting
‘Currant jelly stool’
Cause death from lack of nutrients, back up of stool leading to infection
Key questions to ask
When did last open bowls? When did last pass wind?
Describe Abdominal Aortic Aneurism (AAA) and signs and symptoms
How it occurs:
Artery wall weakens and stretches
Aneurism expand, aorta dilates
Increases risk of rupture
Overall mortality is 85%
Signs an symptoms
Pulsating mass in 50% of patients
Low BP (faint, dizzy, collapse)
Abdominal pain, chest pain, radiating to loin or groin
Pallor, tachycardia
What is Aortic Dissection?
Result of AAA, tear in tunica intima of aorta allows blood flow into tunica media - blood then flows into ‘false’ lumen. Bleeding into abdominal cavity and die.
Describe Gastro-intestinal bleeds, causes, signs and symptoms and key questions to ask?
Divided into:
Upper GI bleed (higher than the duodenum) - caused by peptic ulcers, gastritis, oesophageal varices, tumours
Haematemesis
Melaena (dark, tarry stool)
Lower GI bleed (lower than the duodenum) - caused by covert ocular disease, IBD, Bowel Disease, Haemorrhoids, Tumours
Bleeding Per Rectum (BPR) - bright red or dark blood clots
Key Questions to ask:
When did bleeding begin?
Taking aspirin/NSAIDs?
Taking iron tablets?
Food containing dye/beetroot?
Beta blockers/calcium channel blockers?
Anti coats/anti platelet therapy?
Bleeding disorders/liver disease/abdo surgery/alcohol abuse/syncope?
Describe appendicitis, signs and symptoms and management?
Inflammation/infection of appendix
Usually causes pain in lower right side of abdomen
Signs and symptoms
Nausea, vomiting, loss of appetite, constipation, temperature, diarrhoea, facial flushing, dry tongue, tachycardia
Needs urgent treatment in hospital
Describe peptic ulcer and signs and symptoms
Erosion of lining of stomach or small intestine forming ulcer
Pain
Presents in upper part of abdomen
May feel like burning
Often worse at night
Describe diverticulitis and signs and symptoms
Inflammation/infection of diverticular (pouches that form in large intestine over time)
LLQ pain (sometimes LRQ)
Pain ++ after eating
Pain eases after passing wind or easing bowls
Constipation, Diarrhoea
Blood in stool
Bloating
Describe Hernias
Internal part of body pushes through abdominal wall
Becomes strangulated (Cut off from blood supply)
May lead to ischaemic bowl
Causes death of that tissue
Described ulcerative colitis and signs and symptoms
Comes under IBD
Chronic condition where colon and rectum become inflamed
Small ulcers develop on colon’s lining can bleed and produce pus
Signs and symptoms
Diarrhoea
Possible pus, mucus, blood
Abdominal pain
Bowel opening frequency
Fatigue
Loss of appetite
Weight loss
Painful joints
Mouth ulcers
Describe Crohn’s disease and signs and symptoms
Comes under IBD
Chronic condition that causes gut inflammation
Managed not cured
Remember, ulcers are always at risk of bursting
Signs and symptoms
Diarrhoea
Blood or mucus in stool
Abdominal pain
Rectal/anal pain
Weight loss
Fatigue
Loss of appetite
Describe gastroenteritis
common condition
Causes D&V
Usually bacterial or viral
D (5-7 days)
V (1-2 days)
Obtain full history, difficult to exclude serious pathology
What are three important assessment aspects to consider when dealing with abdominal pain?
Inspect, Auscultation, Palpation
Inspect
Skin colour, bruising, masses, pulsing, pallor, scars (PSHx)
Auscultation
LRQ to confirm present of BS
Palpation
Not recommended
Not over areas of pain
Very light Palpation only
Senior clinician only
What conditions could be associated with pain in each of the abdominal quadrants?

Describe the difference between the cause of Type 1 and Type 2 diabetes
Type 1: ‘not producing insulin’
As not producing insulin, glucose in blood cannot access cells causing increase in blood glucose
Causing
Increase in urination due to increased BS
Blurred vision as glucose build up in eyes
Slow healing as glucose in blood feeding bacteria
Increase occurrence of thrush, UTIs
Type 2: ‘receptor sites mutates or blocked up by fatty deposits, cannot ‘unlock’ lock’
Similar symptoms and also increasing blood glucose
Not all blocked so some can go in
What are the functions of Insulin and Glucagon
Insulin is the “key” to the lock of receptors on cells which allow glucose to enter cells
Glucagon releases glycogen stores in the liver which is turned into glucose
What are treatments for Type 1 and 2 diabetes?
Type 1: Insulin pens/injectors
Type 2: Lifestyle, metformin/glycoside, Insulin pens/injectors
What are Blood Glucose Levels measured as and what are normal values for diabetic and non diabetics?
Measured as millimoles per litre (mmol/L).
Normal values:
Non diabetic
3.0 to 5.6 mmol/L
Diabetic
4.0 to 7.0 mmol/L before meal
10 mmol/L one and half hours after meals
Around 8 mmol/L at bedtime
Describe Hypoglycaemia common causes, signs and symptoms, and management.
Common causes:
OD insulin or oral hypoglycaemic agents
Hepatic or Renal dysfunction
Other illnesses including infection
GI disturbances
Inadequate food intake
Increased physical activity
Alcohol intake
Pregnancy and breastfeeding
Elderly
Signs and symptoms:
Usually rapid onset
Skin can appear pale and sweaty
Normal or shallow respirations
Rapid and full pulse
Irritable, confused, uncoordinated, aggressive or anti-social behaviour
Hunger, Headache, Dizziness, Seizures (in late stages)
Management:
Is dependent on severity
Mild (able to swallow, cooperative and alert)
Encourage to eat fast acting carbohydrate (e.g. sugar water, sweets [not chocolate])
Monitor till BM >4mmol/L
Then encourage long acting carbohydrate (e.g. biscuits, toast, meal if due)
Continually reassess
Moderate (may be combative or unable to swallow)
As per Mild, however if not able to swallow or combative - escalate to senior clinician to administer glucose gel etc.
Severe (unconscious, fitting, severely combative or risk of choking/aspiration)
Manage ABCs
Escalate to senior clinician
Describe Hyperglycaemia common causes, signs and symptoms, and management
Common causes:
Undiagnosed diabetes
Illness
Infection
MI
Medication including steroids
Failure to administer diabetic medications
Signs and symptoms:
Usually gradual progression (hours to days)
Skin is dry
Deep and sighing respiration (Kussmals respiration)
Rapid and weak pulse (decompensating BP)
Restless, drowsy or lethargic behaviour
Sweet, fruity breath (Ketone breath)
Abdominal pain
Nausea
Polyuria
Polydipsia
Polyphagia
Rapid weight loss
Management:
Correct ABC problems
Measure and record BM
ECG
Administer O2 of hypoxaemic
Pre-alert
Explain the process for measuring blood glucose
Clean pat finger with alcohol swab
Once finger dry, obtain blood sample suing capillary lancet
Wipe away first drop of blood
Obtain fresh test strip and insert black wide end into meter
Wait for droplet to appear on screen
Insert end of test strip to blood sample
Wait approx 5 secs for rereading to appear
Press ejector button over clinical waste bag
What are some common post operative complications?
Fever
Wound infections
Embolisms
Deep vein thrombosis
What sort of complications might you expect 0-2 days post surgery?
Post operative fever
Superficial infection relating to the surgery
Haematoma
What sort of complications might you expect 3-5 days post surgery?
Pneumonia
Sepsis
Wound infection
DVT
Phlebitis
What sort of complications might you expect 5+ days after surgery?
Wound infection
Urinary Tract Infection
Chest infection
DVT
PE
How would you manage post operative complications?
Complete full set of obs
Senior clinician to complete differential diagnosis on presenting signs, symptoms and observations
Senior clinician to identify appropriate care pathway OR convey to nearest ED if appropriate
Describe allergic reactions
Mild symptoms in reaction to an allergen, e.g. localised urticaria, runny nose,
triggers include food, drugs, venom
Describe anaphylaxis and how it can develop
Severe life threatening generalised or systematic hypersensitivity reaction
Rapidly developing life-threatening airway/breathing/circulation problems
How develops?
First exposure to allergen, body creates antibodies
Next time exposed, body recognises/responds quicker
Disproportionate immune response (histamine and WBCs)
Histamine gives symptoms
Bronchospasm and vasodilation (anaphylaxis symptoms)
What are some symptoms of anaphylaxis?
Hypotension
Swelling of tissues (BP crashes causes fluid to leak into tissues from blood)
DIB wheezing due to bronchospasm
Hypoxia (confusion)
Tachypnoea and Tachycardia
Nausea and vomiting
Sweating
Diarrhoea
Decreased cardiac output (low BP)
How can allergens enter the body?
Injection
Ingestion
Absorption (skin contact)
Inhalation
What could be a presentation of early anaphylaxis?
Itchy rash
Swelling around eyes
Prickly lips
Dry non-productive cough
Restlessness
What could be a presentation of acute anaphylaxis?
Flushed skin
Tachycardia
Chest tightness
Tongue and throat swelling
Wheeze
Hoarseness
Dyspnoea
Strider
Cyanosis
Vomiting
Diarrhoea
How would you treat anaphylaxis?
Oxygen 15l using non-rebreather (continued until vital signs normal)
Paramedic intervention:
Salbutamol and Adrenaline
Advanced airway
IV fluids
If time for hotel backup is too long, consider rapid transport if have EMT
Continually monitor and pre-alert to hospital
Define poisoning
Exposure to a substance that may result in mortality or morbidity
What are the ways in which poisons may enter the body?
Inhalation
Ingestion
Injection
Absorption
Common types of poison
Household products
Pharmaceuticals/recreational substances
Plants/Fungi
Alcohol
Chemicals
Cosmetics
What are key considerations when assessing a poisoning patient (using medical model)?
Using Primary survey
Danger - consider cause of poisoning. - ?attack ?suicidal/mh crisis
Response - overdoses can alter loc
Airway - Overdose can cause nausea/vomiting - ensure airway clear of vomit if unresponsive
Breathing - Some overdose can cause respiratory depression -may need to oxygenate and bag
Circulation Some overdoses can cause low BP - may not be able to feel radial pulse - Elevate limbs or fluids
Disability - reassess AVPU
Evaluate - reevaluate and rapid extrication if time critical patient
History taking
Global overview - tablet packets all around? Drug paraphernalia?
If OD - ?staggered or all at once (PCHx)
Look at prescription history and if susceptible from PMHx
Toxbase
What should your management principles be for poisoning?
Identification of poison
Specific treatment for specific poisons
Rapid access to hospital
When should you consider carbon monoxide poisoning and how do we manage this?
Consider if patient is in a confined space with impaired ventilation or defective boiler
Could by hypoxic from CO affinity to Haemoglobin
Disorientation
SOB/DIB
Ataxia
Cyanosis
Dizziness
Nausea
Lethargy
Visual problems
Consider COMA
Cohabitants/companion - does anyone else feel unwell? Pets behaving abnormally?
Outdoors - Do you feel better if you go outside or away from property for a long time?
Maintenance - Are any heating appliances properly maintained?
Alarms - Do you have a working CO alarm? Has it activated?
Remove patient from source
Administer high flow O2 continuously (SPO2 monitoring has no value here)
Describe Urinary Retention, causes, signs and symptoms
Inability to empty bladder - can. Be acute or chronic
Acute urinary retention is a medical emergency
Causes
Prostate enlargement
Urethral stricture from surgery
Urinary tract stones - blocking opening to urethra
Nerve problems - vaginal childbirth, spinal cord injury, pelvic injury/trauma
Aging - inability to empty bladder completely
Signs and symptoms
Severe lower abdo pain
Unable to pass urine
Reduced flow
Swelling of bladder
Without prompt treatment, urine may pass back up into the kidneys, which may damage them
Describe Urinary Tract infections (UTIs), causes and signs and symptoms
Develops when part of urinary tract becomes infected, usually bacterial
Mainly occur in women
Are far less common in me and for that reason need investigating
Narrowing of urethra (a stricture)
Previous STI
Bladder stones
Problem with prostate gland
Can be lower (bladder and urethra) or upper (kidney and ureters)
Upper UTIs are potentially more serious than lower UTIs because of risk of kidney damage
Signs and symptoms
Pain/burning sensation when urinating
Cramping pain in lower abdomen or back
Dysuria - burning sensation
Increased frequency of urination
Confusion in the elderly
Strong smelling urine
Haematuria - cloudy urine or frank blood
? Kidney infection: temp >38, rigors, vomiting
Nausea and vomiting
Describe Kidney Stones (renal calculi), causes, signs and symptoms
Stone-like lumps that can develop in one or both kidneys
Move down ureters and stretches the wall
Small stones may be passed out painlessly in urine and go undetected
Fairly common for stone to block part of urinary system
Signs and symptoms
Vomiting, nausea
Persistent ache in lower back/groin/side of abdo
Periods of intense pain in back/side of abdomen, lasting minute or hours (renal colic)
Feeling restless and unable to lie still
Need to urinate more often
Pain when urinating
Blood in urine
Can lead to UTI
Describe Acute Kidney Injury (AKI), causes, signs and symptoms and management
Sudden episode of kidney failure or damage that happens within a few hours or days
Range from minor loss of kidney function to complete kidney failure
AKI causes a build-up of wast products in blood making it hard for kidneys to maintain right balance of fluid within the body
Essential that AKI is detected early and treated promptly. Without quick treatment, abnormal levels of salts and chemicals can build up in body, affects ability of other organs to work properly
Signs and symptoms
Nausea/vomiting
Less than 100ml urine excretion in 24hrs
Diarrhoea
Dehydration
Hypertension
Confusion
Drowsiness
Abdominal pain/back ache
Causes
Reduced blood flow caused by low blood volume, heart pumping less blood than normal, problems with vessels, certain medicines
AKI can be caused by problems with kidney itself or result of blockage affecting drainage of kidneys such as:
Enlarged prostate
Tumour in pelvis
Kidney stones
Management (Depends on severity and cause)
Increasing intake of fluids
Antibiotics if infection
Stop taking certain medicines
Urinary catheter if bladder blockage
Serious complications:
High potassium in blood
Fluid in lungs
Acidic blood
Describe Chronic Renal Failure/Chronic Kidney Disease (CKD), causes, signs and symptoms and management
Long term condition where kidneys don’t function normally
Common with elderly
Signs and symptoms
Tiredness
Swollen ankles, feet or hands
SOB
Nausea
Haematuria
Vomiting Hypertension
Caused by
Hypertension
Diabetes
High cholesterol
Kidney infections
Glomeluronephritis
Polycystic kidney disease
Blockages in flow of urine
Long-term regular use of certain medicines
Treatment depends on cause and severity
Lifestyle changes
Medicines to control associated problems
Dialysis
Kidney transplant
Management
Oxygen if indicated
Entonox to relieve pain
Constant Obs
Consider sepsis guidelines
12 lead
Transfer to further care
Define Sepsis and how it may present
Dysregulated host response to infection, causing life-threatening organ dysfunction
Can have an identifiable source of infection (Hx)
Causes BP to dilate, BP drops, organs cannot get oxygen → causes organ dysfunction
Hypertension also causes tachycardia and tachypnoea, high temperature as part of immune response
Dilation of vessels causes them to become ‘leaky’ - fluid leaking in tissues
Hypoxia can cause confusion
As organs start to die, blood becomes acidic
What is suspected sepsis?
Signs and symptoms, observations are indicative of sepsis but cannot identify source
What population are you more likely to see sepsis in?
Elderly
Immunocompromised
Chemotherapy treatments
Diuretics, Ace inhibitors, Metformin, NSAIDs (DAMN drugs)
Surgery in past 6 weeks
Lines/catherters
IV drug use
Breach of skin (burns, blisters, skin infection)
Pregnant women (DONT use NEWS2)
Recent birth
Children under 1
What is septic shock?
When BP crashes and rapid decline in condition
What is Disseminated Intravascular Coagulation (DIC)
Inflammation causes abnormal clotting in vessels (DIC)
Over active clotting blocks blood flow, causes organ damage
Platelets used up, meaning body cannot stop bleeding, presenting as ‘petechia’ rash
How should we treat sepsis?
15l of 02
Fluids
Rapid transport