1/211
Looks like no tags are added yet.
Name | Mastery | Learn | Test | Matching | Spaced | Call with Kai |
|---|
No analytics yet
Send a link to your students to track their progress
labor
function by which the products of conception (fetus, amniotic fluid, placenta, membranes) are separated and expelled from the uterus through the vagina into the outside world
true labor
cervix softens, effaces and dilates and moves anteriorly
contractions become “longer, stronger and closer together”
regular contraction pattern
intensity increases with walking or standing
felt in lower back and radiate to lower abdomen
continue to increase in intensity despite comfort measure or position changes
false labor
usually little or no cervical change
cervix often stays in poster position
contractions have irregular pattern and intensity does not increase with time
contractions are easily interrupted with medications, walking or position change
felt in back or upper fundal area
labor initaition (patient)
uterine muscles stretch releasing prostagladins
pressure on cervix from fetus stimulates release of oxytocin (Ferguson reflex
estrogen/progesterone ratio shifts so that estrogen stimulates contractile response of uterus and drop of progesterone increases oxytocin
eestrogen= increased contractions
progesterone = relative decrease means oxytocin no longer inhibited meaning more contractions
dramatic increase of oxytocin during labor
labor initiation (fetal)
placental aging and deterioration
cortisol production
made in the fetal adrenal gland and stimulates labor onset by acting on placenta to reduce progesterone formation and increase prostaglandin
prostaglandin produced by fetal membranes and uterine decidua stimulate contractions
powers
uterine smooth muscle contractions with force generating capacity
frequency
duration
passengers
fetus; affected by size, presentation, position, presenting part, degree of flexion (attitude) and placenta
passage and position
bony boundaries of the pelvis and position of the patient in labor
psyche
patients emotional state determines their response to labor and influences physiological functioning
fear from past experiences
hospital as point of no return
unwanted pregnancy? high expectations?
stress hormones can stall labor
LISTEN EDUCATE SUPPORT
strength of contraction (powers)
pacemaker located in fundus because greater number of myometrial cells located there
normally 30-50mmHG/UC necessary for effective labor
adequate labor contractions= montevideo units (MVU’s) >200 mmHg total across contraction per 10 minutes (if IUPC)
desire no more than 5 UCs in 10 minutes (MVU’s <280 mmHG)
measuring powers
palpation
external tocodynamometer (gives the frequency and duration but not the intensity)
affected by patient habitus and proper placement
IUPC- measures MHG exerted by each contraction
associated with infection, uterine perforation, trauma
tachysystole
greater than 5 contractions in 10 minutes
hypertonus
contractions lasting longer than 2 minutes in duration
cephalopelvic disproportion
mismatch between size and/or shape of the fetal head and size and/or shape of the pelvis, making it difficult for the baby to pass through the birth canal
gynecoid
normal pelvis for individuals assigned female at birth with an incidence of 50%
round or transverse oval shape, all diameters are adequate
most ideal for space
android
pelvis for individuals assigned male at birth with an incidence of 20%
heart of wedge shaped
reduced in all diameters
arrest of labor is common
anthropoid
ape like pelvis with an incidence of 25%
long anteroposterior oval shape
OP presentation is common
platypelloid
flat with an incidence of 5%
transverse oval shape
delay of descent at inlet is common and c/s is common
fetal lie
reference to maternal spine
longitudinal
transverse
oblique
fetal presentation
cephalic: vertex, military, brow, face
breech: complete, frank, footling
shoulder
fetal attitude
flexion (vertex) - everything crossed in
extension- neck and arms pushed back
fetal position
occiput, sacrum, mentum (chin)
anterior
fontanelle that is closed by 18 months and is diamond
posterior
fontanelle that is closed by 6-8 weeks and is triangle
engagement
when the widest diameter of the presenting part has passed the inlet, usually 0 station
floating
when presenting part is entirely out of the pelvis and freely movable in the inlet
cardinal movements
engagement and descent
flexion
internal rotation
extension
restitution
external rotation
expulsion
5 P’s interventions
facilitate effective uterine contractions
minimize risk of infection/complication
facilitate maximum pelvic capacity
facilitate fetal cardinal movements with position changes
minimize anxiety and fear associated with labor
maximize coping strategies and knowledge and understanding
initial labor admission
review of systems
urinary protein and glucose
leopold’s maneuvers to determine fetal position
fundal height
signs of domestic abuse and support
signs of preeclampsia
Risk factors:
prior OB history
current pregnancy info
prenatal
Labor Sx and Fetal Status:
Oxygenation of patient and fetus
assessment of labor tolerance - patient and fetus
labor continued assessment
vitals checked every hour while in active labor
I/O every 4 hrs
contraction pattern - assess by palpation, even with IUPC- confirm uterine relaxation
cervical exam- dilation, effacement, fetal station
ROM- time,color, amount, odor
bleeding, discharge
hyperventilation
common with anxiety and occasionally occurs when utilizing relaxation breathing techniques
symptoms: lightheadedness, dizziness, tingling of fingers, spasms in hands or feet, circomoral numbness
Results: respiratory alkalosis
interventions:
replacement of bicarbonate ion by rebreathing CO2
platelets (labor)
decreased thought to be due to hemodilution
WBCs(labor)
increase to 30,000 considered normal
same response as strenuous exercise
first stage of labor
assist with positioning
monitor maternal/fetal status
integrate support persons into plan of care doulas, friends, family, partners
implement pain management techniques/meds
monitor I/O
assist with infection prevention- know your patient’s STI, GBS lab status
second stage of labor
encourage rest during latent phase
optimize oxygenation (open glottis pushing, side-lying position, push every other UC)
third stage of labor
facilitate bonding
observe for signs of separating placenta
monitor for and prevent increased bleeding
expectant vs active management (pitocin, cord traction, fundal massage)
inspect placenta for abnormalities
signs of placental separation
lengthening of cord
change in shape of fundus from discoid to globular
gush of blood
shiny schultz
fetal side of placenta
dirty duncan
maternal side of placenta
fourth stage of labor
newborn:
support trasition to extrauterine life
APGAR, initial assessment, meds, etc.
maternal:
monitor for signs of excessive bleeding
VS/fundal/lochia assessments- q15 ×4, q30×2 until stable
QBL- weigh pads (1gm=1ml)
oxytocin IV or IM
version
turning of fetus from one presentation to another (usually from breech/shoulder to vertex)
version prereqs
confirm >=37 weeks
ultrasound: placenta, cord, fluid, position, uterine anomalies
reactive NST to confirm fetal well-being
informed consent
tocolysis (terbutaline) to relax uterus
version postprocedure
assess for abruption, rupture, PTL, AFE, fetal distress
monitor FHR, contractions for 1hr or until stable
RhoGam if mom is Rh-
version contraindications
uterine anomalies (ex: bicornuate uterus)
labor contractions - can’t do with contracted uterus
3rd trimester bleeding (ex: previa)
multiple gestation
internal version can be done for 2nd twin
oligohydramnios
evidence of uteroplacental insufficiency
nuchal cord
prior c/s of significnat uterine surgery
obvious cephalopelvic disproprortion
1st stage pain
visceral
cervical dilation and effacement
ischemia of uterine muscles
stretching of LUS
pressure on pelvis, bowel, bladder
2nd stage pain
somatic pain
distention of vagina
distention and stretching of perineal tissue
ischemia of uterine muscles
pressure on pelvis, bowel and bladder
nonpharacologic pain management
cognitive: relaxation, patterened breathing, imagery, hypnosis
cutaneous: massage, warmth, hydrotherapy, positional assistance (birthing/peanut ball), rockinging/swaing
nitrous oxide
laughing gas
50% oxygen 50% nitrous oxide
commonly used in midwives and homebirths
narcotics- fentanyl and butorphanol
opioid agonist
increases pain threshold
may increase or decrease uterine contractions
may cause drowsiness
cross placental barrier—→ administer during contraction to minimize fetal effect
can cause sinusoidal appearing FHR pattern
IV fentanyl
advantages:
strong narcotic
decreases severe pain
short acting
effect worn off in less than 1 hr
cardiovascular stability
BP usually does NOT drop
moderate sedation
minimal nausea
minimal newborn effects
can be given right up to time of delivery (epidural form only not IV)
inexpensive
disadvantages:
strong narcotic
can cause respiratory depression narcan
short acting
needs to be repeated frequently
itching is common
agonist-antagonists
butorphanol tartrate (stadol) or nalbuphine hydrocholride (nubain)
good analgesia with less respiratory depression and N/V
agonists
promethazine (phenergan)
decrease anxiety and apprehension, increase sedation and reduce nausea and vomiting
may potentiate narcotic- help them work more effectively (morphine)
sedatives
zolpidem (ambien) or secobarbitol (seconal)
provide sedation or sleep and reduce tension and fear
cross placenta and may affect newborn up to 12-24 hours
pudendal block
perineal body is injected with local anesthesia (into nerve)
performed just before delivery at site of episiotomy
no fetal effect
mechanism of action
drugs: lidocaine, bupivicaine, chloroprocaine, tetracaine
inactivates voltage dependent sodium channels
epidural advantages
superior pain relief and position changes are less uncomfortable
provides sufficient relief for episiotomy repair
emergency c/s can occur more quickly
beneficial to women for whom general anesthesisa is a risk
epidural contraindications
coagulation disorders
maternal hypotension
allergy to local anesthetics
infection at injection site
increased ICP
vaginal epidural
T10 to S5 placement
c/s epidural
T8 to S1 placement
epidural
placed in epidural space
increased vascularity and sensitivity to local anesthetics present during in pregnancy
observe for signs of toxicity within 60 seconds after injection- indicates meds injected into vein
tingling, tinnitus, shivering
goal is pain relief, not numbness, want sensation of lower extremities to facilitate movement
epidural disadvantages
Sympathectomy-induced vasodilation
venous pooling resulting in decreased blood pressure
lower extremity weakness and numbness
may prolong the second stage of labor
potential for post-dural puncture headache
technically challenging with great potential for harm
needs to be done by an anesthesiologist
BP drop (epidural)
sympathetic blockade with resulting BP drop is a common side effect of regional anesthesia
fetal hypoxia can occur if maternal systolic blood pressure drops below 100 mmHG in healthy pregnant woman
concurrent infusion of 500-1000ml of crystalloid fluid may be used to prevent hypotension
Ephedrine: drug used to treat BP drop- increases BP by increasing cardiac output without causing general vasoconstriction
preserves uterine/placental blood flow
ephedrine
drug used to treat BP drop- increases BP by increasing cardiac output without causing general vasoconstriction
preserves uterine/placental blood flow
spinal anesthesia
anesthetic deposited inside the dural sheath and has direct contact with the nerves
stops or decreases impulses at spinal cord
variable numbness and weakness
combined spinal epidural advantages
complete pain relief established after only a few minutes
ambulation is possible during the early part of labor
epidural provides route for very effective pain relief that can be used throughout labor and c/s if necessary
general anesthesia
use:
more for emergency
may be used if a contraindiciton to spinal or epidural block
maternal risk:
mortality associated with aspiration
stomach displaced upward
slow GI emptying
uterine relaxation may cause hemorrhage
fetal risk:
at risk for receiving anesthetic if procedure takes longer
maternal pain management complications
effects last up to 6hrs post medication:
respiratory depression
BP drop
limits mobility
itching
fetal pain management complications
decrease in FHR variability
neonatal respiratory depression
med admin care
monitor:
vitals, mobility, LOC, perception of pain
bladder status (distended bladder, decreased sensation or inability to void may indicate catheter
fetal response to medications
change patients position every hour—→ encourage lateral rather than supine
vaginal assisted indications
maternal
progress of 2nd stage stops due to inadequate contraction strength, poor pushing efforts, excessive fetal size, non-vertex position
condition requiring short 2nd stage (cardiac/pulmonary disease)
extreme fatigue
fetal
distress that warrants shortened 2nd stage
forceps/vacuum prereqs
no cpd
head is engaged
membranes ruptured
cervix is 10 cm dilated
empty bladder
forceps/vacuum complications
bruising
cephalhematoma
facial nerve damage
maternal lacerations, hematoma
maternal abx indicated after operative vaginal birth to prevent infection
bishop score
rating system to measure the physiologic readiness of cervix
decide if can go directly to pitocin or start with cervical ripening approaches
labor induction more likely to be successful with a higher score (13 points possible)
9 or more for nulliparous patients
5 or more for multiparous patients
risk of macrosomia
not a ACOG approved medical indication for induction
induction (augmentation) indications
maternal conditions (DM, HTN)
chorioamnionitits/ prolonged ROM
IUGR
postdates
fetal death
Rh sensitization
induction (augementation) relative contraindications
unfavorable cervix
presenting part not engaged
abnormal presentation
polyhydramnios
hypertonic uterus
induction (augementation) absolute contraindications
cephalopelvic disproportion
fetal distress
complete previa or vasoprevia
prior uterine surgery (not including c/s)
active genital herpes
induction risks
hyperstimulation of uterus (tachysytole)
fetal distress
increased incidence of c/s
uterine rupture
increased postpartum
cervical ripening
makes cervix more favorable for dilation and effacement - physically softer
mechanical
catheters, laminaria
chemical
cervidil(dinoprostone) alpha prostaglandin
misoprostil (cytotec) alpha prostaglandin
stripping of membranes
amniotimy (breaking the water)
fetal indications for cervical ripening
gestational age established by early ultrasound test or measurement of appropriate parameters
acceptable lecithin/sphingomyelin (L/S) ratio (usually 2:1 or higher)
prescence of phosphatidylglycerol in amniotic fluid
laminaria
seaweed or synthetic dilators
ballon dilators (cooks)
soften cervix
change bishop score and facilitate amniotomy
*usually come out around 3-4 cm
tachysystole occurs less often than with use of pharmacologic methods
misoprostil (cytotec)
prostaglandin E1
ripens cervix, making it soften and causing it to begin to dilate and efface and stimulate uterine contractions
used for preinduction cervical ripening (bishop score <4) and to induce labor or abortion)
Not FDA approved for use in cervical ripening or labor induction (off labor use)
considerations
caution with asthma, glaucoma, renal, hepatic or cardiovascular disorders
pt. void prior to insertion
supine position w/tilt or side lying 30-40 min after insertion (don’t want it to fall out)
Initate Oxytocin NO SOONER than 4 hrs after last dose d/t onset 20-6hrs d/t risk of uterine hyperstimulation
adverse effects
tachysystole
N/V/D
fever
risk lower at lower doses
amniotomy
artifically rupturing amniotic sac
used to induce labor when cervix is ripe or augment labor if progress begins to slow
considerations
performed by provider
fetal head engaged (no prolapse) —→ Check FHR immediately after AROM
avoid if vaginal infection (genital herpes, HIV)
note: time, color, consistency, amount, odor
assess temperature every 2 hrs
assess for s/sx of infection- tender abdomen, chills fetal tachycardia
labor usually beings within 12 hrs
can decrease duration of labor by 2hrs without oxytocin
adverse effects
increased risk of intra-amniotic infection
variable decelerations as result of cord compression
cord prolapse or low AFI
pitocin
synthetic version of the hormone oxytocin
produced by the posterior pituitary gland
stimulates the uterus to contract
hormone responsible for milk ejection reflex
only pharmacologic agent FDA approved for induction of labor
stimulation UC by increasing myometrial cell membrane permeability to sodium ion
dosing
0.5-1 mU/min
increased no more than 1-2 mU/min every 30-60 mins
steady state reached after 40 minutes
increased only until active labor is established and may be turned off
titrated as needed to eliminate tachysytole
>5 contractions in a 10-minute strip averaged over 30 minutes
high alert medication
uterine effects (pitocin)
stimulates contractions by increasing frequency strength and duration
enhances cervical ripening indirectly by stimulating the active synthesis of prostaglandins in the decidua
cardiac effects (pitocin)
initially a slight increase in maternal blood pressure
increased cardiac output and stroke volume (use caution in preeclamptic patients)
maternal adverse effect of pitocin
uterine hyperstimulation
placental abruption
uterine rupture
anaphylactoid syndrome of pregnancy
soft tissue damage
cervical lacerations
perinaeal hematoms
rectal tears
increased incidence of c/s
increased PPH
water intoxication- leading to seizure and coma
s/sx H/A, N/V, decreased urinary output, confusion, hypotension and arrhythmias
fetal adverse effects of pitocin
hypoxemia and acidosis resulting from uterine tachysytole
late decelerations and minimal/absent baseline variability
pitocin nursing
vaginal exam PRIOR to beginning infusion (document cervical status and presentation of fetus)
prepare IV solution
infuse through closest port to patient
perform ongoing maternal and fetal assessments
interventions for tachysystole:
stop med
reposition
IV fluids
O2
terbutaline
may take up to 1hr to wear off
c/s indications
medical (ex: previa)
complications of labor (dystocia or malpositon)
maternal request
non-reassuring fetal status (cord prolapse)
active HSV/HIV +
c/s contraindiciations
thrombocytopenia severe enough to prohibit clotting at time of delivery
respiratory or cardiac instability
c/s maternal complications
increased mortality from anesthesia
surgical wound infections
thromboembolism
increased blood loss
previa, accreta, scar dehiscence and uterine rupture in future pregnancies
c/s fetal complications
increased respiratory complications and NICU admissions
skin lacerations and clavicular fractures
low transverse
most commonly used c/s
associated with lowest risk of fetal injury
less blood loss
causes fewer intra-abdominal adhesions
vaginal birth is possible in subsequent pregnancies
classical
faster method c/s
uterine wall weaker in subsequent pregnancies
requires future pregnancies be delivered by c/s
VBAC
increased to 14.2% (2% in 2021)
contraindications
classical incision
more than 2 prior uterine scars and no vaginal births
complications
uterine rupture incidence (0.7-0.13%)
hemorrhage
hysterectomy
neonatal hypoxemia
cerebral palsy
predictors of success
history of SVD
history of successful VBAC
cervical dilation >4 at admission
SROM at admission
Uterine rupture
nonsurgical disruption of uterine cavity
complete- endometrium, myometrium, serosa separated
incomplete- not all layers disrupted
risk factors:
preserve uterine incision
operative vaginal delivery
abdominal trauma
uterine manipulation
postpartum fever during previous c/s
nursing assessment/ symptoms
Cat II or III EFM tracing
maternal abd. pain
unterine tenderness
change in uterine shape
cessation of contraction
hematuria
signs of shock \
Prepare for emergency C/S
gynecoid, anthropoid
pelvic anatomy types that are favorable for a vaginal birth
fetal monitoring low risk (no oxytocin)
latent phase (<4cm)
at least hourly
latent phase (4-5cm)
30 minutes
Active phase (>6cm)
30 minutes
2nd stage (passive fetal descent)
15 minutes
2nd stage (active pushing)
15 minutes
fetal monitoring with oxytocin or risk factors
latent phase (<4cm)
15 minutes with oxytocin, 30 without
latent phase (4-5cm)
15 minutes
Active phase (>6cm)
15 minutes
2nd stage (passive fetal descent)
15 minutes
2nd stage (active pushing)
5 minutes