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circadian rhythm
an organism’s 24 hr “internal clock” cycle of physical, mental, and behavioral changes
melatonin
the key sleep hormone released and suppressed at certain times during our circadian rhythm
sleep cycle stage 1: N1
transition stage
theta waves
hypnagogic sensations (vivid, dream-like sensations during the transition from wakefulness to sleep)
sleep cycle stage 2: N2
light sleep
sleep spindles (bursts of brainwave activity connected to memory consolidation)
sleep cycle stage 3: N3
deep sleep
delta waves (slow/large brainwaves)
decreased heart rate and blood pressure
physical development + strengthening of the immune system
sleep stage 4: REM
REM sleep
paradoxical stage (brain produces waves similar to wakefulness, but body is paralyzed neck down)
dreams
length of REM increases as night progresses
REM rebound
increased amount of REM needed when sleep deprived
leptin
the hormone responsible for metabolism and hunger
when sleep deprived, decreased hormone levels (leads to increased hunger)
EEG (electroencephalogram)
recording of how stimuli affect neural brain activity
sleep deprivation
fatigue
cognitive decline (poor memory and concentration)
depressed immune system
emotional irritability (disconnect between amygdala and prefrontal cortex)
decreased leptin hormone (increase hunger)
hypnagogic sensations
vivid, dream-like sensations during the transition from wakefulness to sleep
activation synthesis theory
sleep is the brain’s attempt to make sense of random neural activity in the brain stem during REM sleep
memory consolidation theory
sleep is useful for organizing memories and making short term —> long term memories
insomnia
difficulty falling or remaining asleep and persistent reawakening
treated w/ sedatives or sleep restrictions
narcolepsy
sudden and irresistible onsets of sleep during normal waking periods
wakefulness —> REM (lasts 10-20 mins)
treated w/ amphetamines
REM sleep behavior disorder
moving or acting out dreams during REM sleep
sleep apnea
frequent and temporary halt of breathing for 15-60 secs during sleep
treated w/ CPAP machine
somnambulism
sleepwalking during stage 3 (deep sleep)
absolute threshold
the lowest possible level of a stimulus that a person can detect (at least 50% of the time)
just noticeable difference (difference threshold)
the minimum difference a person can detect between 2 stimuli
weber’s law
just noticeable difference is proportional to the intensity of the original stimulus (ex. WILL notice a change in sound in a quiet room but WONT in an already loud room)
signal-detection theory
our ability to detect a stimulus depends on the strength of the stimulus and our psychological awareness
transduction
the process of converting a physical stimulus into a neuro-chemichal message sent to the brain
sensory interaction
McGurk affect - our vision can overpower most senses (ex hearing)
sensory adaptation
when we are exposed o stimulus for long enough, our receptors decrease their sensitivity to make them less noticeable
synesthesia
abnormal blending of the senses in which the stimulation of one sense produces sensation in other senses
retina
lining at the back of the surface of the eye
where transduction occurs
contains photo receptors to process light info
blind spot
part of retina with no photo receptor cells, where eye connects to the optic nerve
visual nerve
optic nerve
accommodation
curvature of lens adjusts to focus light and create a sharp image
rods
blurry, shapes, movement
low light
black and white
cones
sharp, detail
high light
colorful
trichromatic theory
process color by combining 3 types of signals
blue(short)
green(medium)
red(long)
opponent-process theory
ganglion cells process colors in pairs —> red-green, blue-yellow, black-white
—> explains after image effect with american flag
fovea
greatest point of visual activity on retina —> highest concentration of cone photo receptor cells
ganglion cells
next layer of cells that transfer messages from bipolar —> optic nerve
color blindness
dichromatism: see 2/3 primary colors
monochromatism: see 1/3 colors (red, green, blue)
Prosopagnosia
face blindness
blind-sight
damage to visual cortex causes blindness, but patients are able to subconsciously respond to visual stimuli
pitch
determined by the frequency/length
ex: short waves = higher pitch (and frequency)
loudness
determined by amplitude and frequency
place theory
different wavelengths trigger different “places” on basilar membrane
volley theory
group of neurons “take turns” firing rapidly, creating a volley to represent the highest-pitch sounds
frequency theory
the whole basilar membrane vibrates and brain measures intensity/frequency of neural impulses raveling up auditory nerve (explains low pitch)
sound localization
ability to perceive the location by processing differences in timing and intensity of sound waves
basilar membrane
hair cells bend on membrane as it ripples, triggering transduction, converting mechanical vibrations into neural impulses
conduction deafness
damage/blockage in the middle ear
sensorineural deafness
damage to the cochlea’s hair cells in the inner ear
vestibular sense
balance
kinesthetic sense
body position and movement awareness
gate control theory
perception of pain: the spinal cord contains a gate that blocks pain signals or allows them to pass on to the brain
closed by stimuli (rubbing a wound)
frontal lobe and prefrontal cortex
FL: problem solving, judgment, speaking
PFC: emotional traits, decision making, judgment
heritability
percent of variation among individuals in a group due to genes (not individuals)
contralateral functions
LEFT hemisphere (right visual field): language, speech, logical reasoning
can create explanations SO PPL COULD SAY WHAT SAW
RIGHT hemisphere (left visual field): spacial reasoning, perceptual awareness
COULD ONLY DRAW NOT SPEAK WHAT SAW
hypothalamus (hype to eat)
maintenance functions (homeostatic state)
thalamus (muscles control sensations)
sensory control center (directs and transmits sensory info)
temporal lobe (tempo)
auditory info processing, understanding language, hearing
occipital lobe (optic)
visual info
threshold
minimum stimulation needed to trigger a response