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Hypertension Patho
increased peripheral resistance as a result of the narrowing of arterioles, increased pressure of blood/blood volume, primary/essential (unknown cause, 90-95% of cases; develops gradually over time), secondary (known cause, underlying condition)
Hypertension Risk Factors
genetics, obesity, diabetes, older age, dyslipidemia, stress, sedentary lifestyle, epigenetic factors (smoking, alcohol)
Hypertension S/S
asymptomatic frequently; but may include headache, elevated BP, CV and cerebral symptoms
Hypertension complications
stroke, heart attack, cardiac and neuro complications, renal failure
Hypertension Medical Diagnostics
measure BP at several different times, lipid profile, cardiac assessment
Hypertension Medical Treatment/Pharmaceuticals
prevention, modify risk factors, antihypertensive meds, physical activity, other lifestyle changes
Hypertension PT implications
education, consistent exercise, vitals
Myocardial infarction (heart attack) patho
myocardial cell death/heart attack due to prolonged ischemia/obstruction, type 1=from CAD caused by atherosclerosis, type 2=due to mismatch O2 supply and demand, secondary to something else.
STEMI(more serious, extends entire width of heart muscle) and non-STEMI (partial width of heart muscle)
Myocardial Infarction (Heart Attack) Risk Factors
stimulant drug use, smoking, mornings, thanksgiving-new years, middle age and up, angina, family history, metabolic syndrome, obesity, diabetes, LV most susceptible
Myocardial Infarction (Heart Attack) S/S
sudden onset of pressure, chest pain, left arm pain radiation, SOB, diaphoresis, heart burn, indigestion, extreme fatigue, N&V, women tend to go unnoticed with back pain and severe fatigue
Myocardial Infarction (Heart Attack) Complications
arrhythmias, pericarditis, sudden death/cardiac arrest
Myocardial Infarction (Heart Attack) Medical Diagnostics
clinical history, s/s, EKG, serum levels of cardiac enzymes, imaging
Myocardial Infarction (Heart Attack) PT implications
refer to cardiac rehab, vitals, use sternal precautions
Myocardial Infarction (Heart Attack) Medical Treatment and Pharmaceuticals
meds, reestablish blood flow, prevention of reoccurrence
Heart Failure Patho
unable to pump sufficient blood to body, structural (stiff and thick) or functional (saggy and floppy), LHF is caused by HTN and has pulmonary symptoms, RHF is caused by LHF and has systemic symptoms
Heart Failure Risk Factors
HTN, MI, CAD, heart valve disease, increases with age, diabetes, obesity, metabolic syndrome
Heart Failure S/S
psychological/neuro aspects: orthopnea; left has pulmonary symptoms and right has systemic symptoms
Heart Failure Complications
death, liver, spleen, speech, kidneys impaired, peripheral and pulmonary edema
Heart Failure Medical Diagnostics
EKG, blood tests, BMP lab test, imaging (echocardiogram)
Heart Failure PT implications
education, prevention, vitals, cardiac rehab, lifestyle changes, energy conservation
Heart Failure Medical Treatment and Pharmaceuticals
finding underlying cause and treating it, keep HF from progressing, lifestyle changes, surgery
Explain blood flow through the pulmonary circuit, including heart chambers and valves
Pulmonary Circuit:
Superior and inferior vena cava -> right atrium -> tricuspid valve -> right ventricle -> pulmonary valve -> pulmonary arteries -> lungs
Explain blood flow through the systemic circuit, including heart chambers and valves
Systemic Circuit:
Pulmonary veins -> left atrium -> mitral valve -> left ventricle -> aortic valve -> aorta -> body
What are the structures of the heart?
Pericardium: parietal and visceral
Layers of the wall:
Epicardium: visceral pericardium & loose connective tissue
Myocardium: cardiac muscle tissue
Endocardium: inner lining, endothelial tissue
Chambers of the heart: atrium and ventricles (right and left)
Valves of the heart: mitral (bicuspid) valve, tricuspid valve, aortic valve, pulmonary valve
What are the structures of the conduction system?
Conduction System:
Heart chambers coordinated (atrias contract while ventricles relax)
SA node: pacemaker
AV node: slows signal from atrium
AV bundle of His: sends AV signal to right and left bundle branches of ventricles
Purkinje fibers: contraction of ventricles
Pericarditis
Inflamed pericardium
Pericardial effusion
build up fluid in pericardial cavity
Cardiac tamponade
serious condition, fluid build up impedes function of heart
Endocarditis
inflammation of inside heart lining (endocardium, affect heart valves)
Pleural effusion
fluid in pleural cavity (lungs)
Pneumothorax
air buildup in pleural cavity, leads to collapsed lung
Dyslipidemia
atherosclerosis
coronary artery disease
heart attack, stroke
peripheral artery disease
Good Cholesterol
High density lipoprotein
bad cholesterol
LDL- low density lipoprotein
What is the mechanism of atherosclerosis?
Macrophages engulf LDL, forming foam cells →
Foam cells form fatty streaks → fibrous plaque
Stable angina
increased w/ activity, decrease w/ rest, predictable
Unstable angina
occurs at rest, increases with activity, pre-infarction state, unpredictable
increases the risk of orthostatic hypotension
Acute: medication, dehydration, poor nutrition, illness, pregnancy
Chronic: idiopathic or secondary to disorder
Angina
ischemia induced chest pain
PT response to orthostatic hypotension
monitor vitals during session
peripheral artery disease
Atherosclerosis in peripheral arteries narrows arteries
-Decreased blood flow
-Poor oxygen perfusion to tissue (usually legs)
-S/S: Increases w/ activity and skin changes
chronic venous insufficiency
Venous PVD in valves and walls of veins
-Difficult blood return, increased risk of deep vein thrombosis
-S/S: swelling in legs, varicose veins, skin changes
Stenosis
valve narrowing
Insufficiency
can't close or open effectively
What are the 3 cardiomyopathies?
Dilated: chambers dilate, muscle walls become thin and weak
→ systolic dysfunction
Hypertrophic: heart walls thick and stiff, non-compliant
→ diastolic dysfunction
Restrictive: heart muscle stiff and hard
→ diastolic dysfunction