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Blacks classification
Class 1: occlusal + buccal cusps posterior
Class 2: interprox posterior
Class 3: ant interprox anterior
Class 4: ant incisal edge
Class 5: ant cervical margin
Erosion
shiny surface, proud restorations, fractures off incisal edge, shadowy depressions
Intrinsic: bulimia, pregnancy, alcoholism
Extrinsic: acidic foods and drinks, pickling farms
Abrasion
Pathological wearing away tooth structure resulting from repetitive mechanical process or habit
Tb, whitening toothpaste
signs - exposed root surface, wear facets at cervical margin
Attrition
Loss of tooth substance due to mastication or occlusal contact between the teeth
Bruxism: strong flexural force
Pathological: worn down mamelons + occlusal cusps, exposed dentine may be dark brown
Abfraction
V shaped at cervical margin due to biomechanical loading force resulting in flexing and failure of enamel and dentine
Treatment for NCTSL
mouth gaurd - for bruxism
Refer to GDP: for bulimia erosion, discuss diet (extrinsic + intrinsic), if medication discuss with GP different options
OHI:
erosion wait 30mins before brushing teeth
Abrasion: heavy handed with tb: med rough brush, show demonstration
Restore exposed dentine if causing sensitivity or risk of pulpal exposure
Flouride varnish or px HSFT
Constituents of amalgam
mercury
Zinc
Tin
Copper
Silver
Why is amalgam getting phased out
minimata convention - environment concerns re mercury
No amalgam <15 yrs or pregnancy as passes through placenta
Tooth preservation - amalgam needs more removal for retention
Health concerns (mercury vapour)
What are the types of amalgam
conventional 6% copper → Gamma 2 phase is most corrodible but can be inhibited by phosphate in saliva
Most creep phase - overhangs and deficiency at gingival margin
high copper amalgam (30% copper)
G2 replaced with copper tin phase
Copper provides strength and more corrosion resistance
What are the classifications of composite
flowable and conventional
How it’s set - hear, chemical and light
Particle size - nano hybrid, macro and micro
Nano hybrid - stronger, more wear resistant, harder to polish, opaque and less shrinkage
Hydrocolloid
reversible - agar
Irreversible - alginate
Xerostomia
caused by reduced saliva flow eg smoking, meds, sjogrens
increases risk of periodontal gram -ve bacteria
increase risk of caries: strep. mutants
reduced buffer protection:
Mirror sticks to mucosa and can be uncomfortable for pt
Stephan’s curve
Describes how the pH in the mouth changes over time after eating
X axis represents time
Y axis represents pH Levels
the normal resting pH of plaque is around 7.5, but it starts at around 8 on the graph
when we consume fermentable carbohydrates, oral bacteria metabolise them and produce acid which drops pH
the critical pH for enamel demineralisation is 5.5 - once the pH falls below this, tooth enamel starts dissolving
the pH reaches its lowest point within minutes and then gradually rises as saliva buffers the acids
it takes around 40 mins to 1 hr for the pH to return to it's pre-meal level, allowing remineralisation to occur
remineralisation doesn't occur if interuppted by frequent sugar exposure.
What’s sealant restoration
You’re removing caries but then sealant over - not placing a full restoration
its minimally invasive, preserving tooth structure
Indications
non - cavitated or early cavitated lesions
What are the 4 zones of WSL
Four Zones of an Early Carious Lesion (Enamel Caries)
Surface Zone → Intact enamel (due to saliva & fluoride), highly mineralised
Body of the Lesion → Largest part, most demineralised
this is the zone seen on the radiographs
3. Dark Zone → Some remineralisation happening.
mineral re-deposition, showing an attempt at natural repair
Translucent Zone → Earliest sign of demineralisation.
this is the first carious change
apatite has been dissolved by acid
Zones of dentine lesion
Advancing Front - zone nearest to the pulp (affected dentine)
· demineralisation caused by acids diffusing ahead of bacterial invasion
· no bacteria are present here, it's predominantly chemical damage
Zone of bacterial invasion (infected dentine)
· bacteria begin to penetrate this zone
· tubules become colonised by bacteria, leading to structural weakening
zone of destruction - most advanced part of lesion
· enzymatic degradation occurs as bacteria digest dentine
· cavitation and significant structural damage are present
Treatment plan
· Emergency care – pain, infection
· Prevention – OHI, diet, advice, F toothpaste
· Stabilisation – prevent current disease getting worse by perio therapy, XLA
· Restorative + rehab – perio then rest, start worst one first
· Maintenance – establish recall period
· Referrals
What does etch do
create porosities and micromechanical tags
type 1 - removes the core and leaves the periphery intact OR
type 2 - removes the periphery pf the enamel prism and leaver core intact OR
type 3 - haphazardly and does both all over the place
Dentine conditioning - removes smear layer
What does prime do
tooth surface is hydrophobic + bond has a lot of solvent so water base for bond is incompatible. Primer changes the tooth surface to become hydrophillic.
Bond
glue to hold composite in place
How do you diagnose caries
Visual inspection
wsl
brown/dark discoloration
cavitation
translumination
fiber optic translumination - shining light though a tooth, dark areas indicate decay
digital imaging
caries detecting dyes that stain demineralised areas
radiographs
bws
PA
OPG
electrical conductivity & laser flurescence
electical - measures porosity; more porosity = more decay
laser fluorescence - detects early caries by measuring fluorescence
brielle probe
How is caries classified
Location
position - anterior or posterior
specific location on the tooth - occlusal caries, interproximal caries, cervical caries, root caries
radiographic appearance
enamel caries
dentine caries (D1, D2, D3, P)
pulpal involvement
severity
status - secondary or primary
activity
active caries
arrested caries
DMFT
ICDAS (International caries detection and access system)
Risk factors for caries
Factors that increase caries risk
past caries history (DMFT score - decayed, missing, filled teeth)
poor OH (high plaque levels)
diet
low fluoride exposure
socioeconomic status
medical history (medication that cause dry mouth or contain sugar)
smoking and alcohol (reduce saliva)
family history
patient cooperation
Types of bonding systems
Three-Step (Total-Etch) – Most effective, longest-lasting.
Two-Step (Self-Etch or Etch & Rinse) – Moderate effectiveness.
One-Step (All-in-One Bonding) – Quick but least effective.
Pulp cap / treatments
Indirect Pulp Cap: Close but not exposed. Line with Ca(OH)₂ or Biodentine.
Direct Pulp Cap: Tiny mechanical exposure, clean site, control bleeding, MTA/Biodentine.
Stepwise Excavation: For deep lesions—partial removal, seal, reassess later.
Significant exposure: Usually → RCT or referral. But keep isolated and seek advise from dentist
Describe composite advantages and disadvantages
aesthetic
Binds to tooth
Good longevity
Preserves more tooth structure
Good wear resistance
Command set - more working time
Disadvantages
highly sensitive in dry fields
High technique sensitivity and moisture
Post op sensitivity common
More expensive compared to amalgam
Amalgam advantages and disadvantages
Advantages
less sensitive to moisture
Easy to place
Traditionally longer lasting - be careful cause can start debate
Excellent wear resistance
cheap
Disadvantages
Less aesthetic
Mechanical retention only
May require more tooth removal
Contains mercury (minimata concerns)
Less working time
What’s inside a local anaesthetic cartridge
anaesthetic
Vasoconstrictor
Ringer solution (water and salt)
Preservative
Buffer
What does the vasoconstrictor in LA do?
prolongs anaesthesia - reduces blood flow and keeps the anaesthetic in the area for longer
Minimises bleeding
Enhances moisture control due to less bleeding
Why is rubber dam the best form of moisture control
complete isolation
Prevents aspiration
Protects soft tissues
Improves efficiency
Better pt experience
Minimises cross contamination
Psychological benefit - some pts feel distracted from procedure
What a side effect of anti - anxiety meds (antidepressants)
Dry mouth
Why do we give diet diary to patients
hidden sugars and acids
Frequency of sugar
Amount of sugar
How do we know that sugar causes caries
Studies that have been done
vipeholm
Turku
Inuits
Tristan da cuhna
Hopewood house
Different ways to diagnose caries
clinically
Translumination
Radiographically
Fluorescence