State Exam Posters 2025 – Core Vocabulary

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Essential vocabulary flashcards covering assessment tools, developmental theories, acid–base balance, transfusion reactions, pharmacology, fluid–electrolyte concepts, respiratory and cardiac pathologies for the 2025 state exam.

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53 Terms

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ADPIE

Five–step nursing process: Assessment, Diagnosis, Planning, Intervention, Evaluation.

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ISBAR

Structured clinical hand-over tool: Identification, Situation, Background, Assessment, Recommendation.

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SOAPIE

Problem-oriented progress note format: Subjective, Objective, Assessment, Plan, Intervention, Evaluation.

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Te Whare Tapa Whā

Holistic Māori health model encompassing spiritual, mental/emotional, family/social and physical dimensions.

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Ottawa Charter (1986)

WHO framework listing five action areas for health promotion: public policy, supportive environments, community action, personal skills, re-oriented services.

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Erikson’s Psychosocial Theory

Eight life stages where resolving specific conflicts (e.g., Trust vs Mistrust) shapes personality development.

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Piaget’s Cognitive Theory

Four developmental stages (sensorimotor, pre-operational, concrete operational, formal operational) describing how children acquire knowledge.

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Freud’s Psychosexual Theory

Five stages (oral, anal, phallic, latency, genital) where libido focuses on different body zones driving personality formation.

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Bronfenbrenner’s Ecological Transition

Model describing how life changes (e.g., starting school) create stress that may alter behaviour and development.

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Arterial Blood Gas (ABG)

Laboratory test measuring pH, PaCO₂, PaO₂, HCO₃⁻ and SaO₂ to assess oxygenation and acid–base status.

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PaCO₂

Partial pressure of carbon dioxide in arterial blood; respiratory component of acid–base balance (35–45 mm Hg).

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HCO₃⁻

Bicarbonate level (22–26 mEq/L); metabolic base regulating blood pH.

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Respiratory Acidosis

Acid–base disorder caused by CO₂ retention; characterised by low pH and high PaCO₂.

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Metabolic Alkalosis

Alkalotic state from loss of acid or gain of base; high pH with elevated HCO₃⁻.

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Renal Compensation

Kidneys adjust bicarbonate to correct pH; slow (hours–days).

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Respiratory Compensation

Lungs alter CO₂ (ventilation) to normalise pH; rapid (minutes).

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Packed Red Blood Cells

Blood component transfused to correct anaemia and improve tissue oxygenation.

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Platelet Concentrate

Blood product given to restore haemostasis and assist clot formation.

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Febrile Non-Hemolytic Reaction

Common transfusion adverse event marked by temperature ≥38 °C without hemolysis.

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Opioid Mechanism of Action

Bind CNS μ, κ, δ receptors, inhibit adenylate cyclase and neurotransmitter release, dampening pain transmission.

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Naloxone

Opioid antagonist that competitively blocks receptors and reverses respiratory depression.

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Intracellular Fluid (ICF)

Two-thirds of body fluid; potassium is predominant cation.

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Extracellular Fluid (ECF)

One-third of body fluid, including plasma and interstitial fluid; sodium is main cation.

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Hypotonic Solution

Less concentrated than plasma (e.g., 0.45 % NaCl); shifts water into cells.

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Isotonic Solution

Same tonicity as plasma (e.g., 0.9 % NaCl); expands ECF without cell size change.

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Hypertonic Solution

More concentrated than plasma (e.g., 3 % NaCl, D5NS); draws water out of cells.

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Hypovolaemia

Loss of water and electrolytes producing low blood volume, hypotension and tachycardia.

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Hypervolaemia

Fluid overload causing hypertension, oedema, crackles and weight gain.

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Hypernatraemia

Serum sodium >145 mmol/L; presents with thirst, fluid retention, restlessness.

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Hyponatraemia

Serum sodium <135 mmol/L; leads to headache, confusion, seizures.

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Hyperkalaemia

Serum potassium >5.2 mmol/L; ECG shows tall T waves and widened QRS.

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Hypokalaemia

Serum potassium <3.5 mmol/L; causes muscle weakness, U waves, arrhythmias.

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Asthma

Chronic inflammatory airway disease with reversible bronchoconstriction and mucus hypersecretion.

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β₂-Agonist (Salbutamol)

Inhaled bronchodilator that relaxes airway smooth muscle via β₂-receptor stimulation.

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Corticosteroid (Prednisone)

Anti-inflammatory agent that suppresses immune response, reduces airway oedema and mucus.

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Muscarinic Antagonist (Ipratropium)

Anticholinergic bronchodilator blocking M receptors to decrease vagal airway tone.

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Chronic Obstructive Pulmonary Disease (COPD)

Progressive airflow limitation due to emphysema and chronic bronchitis.

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Emphysema

Destruction of alveolar walls leading to hyperinflated lungs and reduced gas-exchange surface.

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Cystic Fibrosis

Genetic disorder causing defective CFTR protein, thick mucus, recurrent infections and malabsorption.

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Pneumonia

Infection-induced lung inflammation with alveolar exudate reducing gas exchange.

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Tension Pneumothorax

Trapped pleural air causing mediastinal shift, respiratory distress and circulatory collapse.

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Tuberculosis

Mycobacterium tuberculosis infection forming caseating granulomas; symptoms include night sweats, haemoptysis.

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Nasal Cannula

Low-flow oxygen device delivering 1–6 L/min (FiO₂ up to ~40 %).

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Venturi Mask

High-precision oxygen mask with colour-coded valves delivering fixed FiO₂ (24–60 %).

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Continuous Positive Airway Pressure (CPAP)

Device providing constant airway pressure to keep alveoli open and improve oxygenation.

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Rheumatic Heart Disease

Autoimmune valve damage following untreated Group A streptococcal pharyngitis.

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Mitral Regurgitation

Backward flow of blood from left ventricle to atrium due to damaged mitral valve.

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Acute Glomerulonephritis

Immune complex-mediated inflammation of glomeruli leading to renal dysfunction after strep infection.

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Congestive Heart Failure (CHF)

Inability of heart to pump or fill adequately, causing tissue hypoperfusion and fluid retention.

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Systolic Heart Failure

Reduced ejection fraction due to weakened ventricular contraction.

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Diastolic Heart Failure

Preserved ejection fraction with impaired ventricular relaxation and filling.

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Left-Sided Heart Failure

Ventricular dysfunction leading to pulmonary congestion, dyspnoea and crackles.

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Pulmonary Oedema

Fluid accumulation in lung interstitium/alveoli impairing gas exchange; hallmark of left HF.