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Essential vocabulary flashcards covering assessment tools, developmental theories, acid–base balance, transfusion reactions, pharmacology, fluid–electrolyte concepts, respiratory and cardiac pathologies for the 2025 state exam.
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ADPIE
Five–step nursing process: Assessment, Diagnosis, Planning, Intervention, Evaluation.
ISBAR
Structured clinical hand-over tool: Identification, Situation, Background, Assessment, Recommendation.
SOAPIE
Problem-oriented progress note format: Subjective, Objective, Assessment, Plan, Intervention, Evaluation.
Te Whare Tapa Whā
Holistic Māori health model encompassing spiritual, mental/emotional, family/social and physical dimensions.
Ottawa Charter (1986)
WHO framework listing five action areas for health promotion: public policy, supportive environments, community action, personal skills, re-oriented services.
Erikson’s Psychosocial Theory
Eight life stages where resolving specific conflicts (e.g., Trust vs Mistrust) shapes personality development.
Piaget’s Cognitive Theory
Four developmental stages (sensorimotor, pre-operational, concrete operational, formal operational) describing how children acquire knowledge.
Freud’s Psychosexual Theory
Five stages (oral, anal, phallic, latency, genital) where libido focuses on different body zones driving personality formation.
Bronfenbrenner’s Ecological Transition
Model describing how life changes (e.g., starting school) create stress that may alter behaviour and development.
Arterial Blood Gas (ABG)
Laboratory test measuring pH, PaCO₂, PaO₂, HCO₃⁻ and SaO₂ to assess oxygenation and acid–base status.
PaCO₂
Partial pressure of carbon dioxide in arterial blood; respiratory component of acid–base balance (35–45 mm Hg).
HCO₃⁻
Bicarbonate level (22–26 mEq/L); metabolic base regulating blood pH.
Respiratory Acidosis
Acid–base disorder caused by CO₂ retention; characterised by low pH and high PaCO₂.
Metabolic Alkalosis
Alkalotic state from loss of acid or gain of base; high pH with elevated HCO₃⁻.
Renal Compensation
Kidneys adjust bicarbonate to correct pH; slow (hours–days).
Respiratory Compensation
Lungs alter CO₂ (ventilation) to normalise pH; rapid (minutes).
Packed Red Blood Cells
Blood component transfused to correct anaemia and improve tissue oxygenation.
Platelet Concentrate
Blood product given to restore haemostasis and assist clot formation.
Febrile Non-Hemolytic Reaction
Common transfusion adverse event marked by temperature ≥38 °C without hemolysis.
Opioid Mechanism of Action
Bind CNS μ, κ, δ receptors, inhibit adenylate cyclase and neurotransmitter release, dampening pain transmission.
Naloxone
Opioid antagonist that competitively blocks receptors and reverses respiratory depression.
Intracellular Fluid (ICF)
Two-thirds of body fluid; potassium is predominant cation.
Extracellular Fluid (ECF)
One-third of body fluid, including plasma and interstitial fluid; sodium is main cation.
Hypotonic Solution
Less concentrated than plasma (e.g., 0.45 % NaCl); shifts water into cells.
Isotonic Solution
Same tonicity as plasma (e.g., 0.9 % NaCl); expands ECF without cell size change.
Hypertonic Solution
More concentrated than plasma (e.g., 3 % NaCl, D5NS); draws water out of cells.
Hypovolaemia
Loss of water and electrolytes producing low blood volume, hypotension and tachycardia.
Hypervolaemia
Fluid overload causing hypertension, oedema, crackles and weight gain.
Hypernatraemia
Serum sodium >145 mmol/L; presents with thirst, fluid retention, restlessness.
Hyponatraemia
Serum sodium <135 mmol/L; leads to headache, confusion, seizures.
Hyperkalaemia
Serum potassium >5.2 mmol/L; ECG shows tall T waves and widened QRS.
Hypokalaemia
Serum potassium <3.5 mmol/L; causes muscle weakness, U waves, arrhythmias.
Asthma
Chronic inflammatory airway disease with reversible bronchoconstriction and mucus hypersecretion.
β₂-Agonist (Salbutamol)
Inhaled bronchodilator that relaxes airway smooth muscle via β₂-receptor stimulation.
Corticosteroid (Prednisone)
Anti-inflammatory agent that suppresses immune response, reduces airway oedema and mucus.
Muscarinic Antagonist (Ipratropium)
Anticholinergic bronchodilator blocking M receptors to decrease vagal airway tone.
Chronic Obstructive Pulmonary Disease (COPD)
Progressive airflow limitation due to emphysema and chronic bronchitis.
Emphysema
Destruction of alveolar walls leading to hyperinflated lungs and reduced gas-exchange surface.
Cystic Fibrosis
Genetic disorder causing defective CFTR protein, thick mucus, recurrent infections and malabsorption.
Pneumonia
Infection-induced lung inflammation with alveolar exudate reducing gas exchange.
Tension Pneumothorax
Trapped pleural air causing mediastinal shift, respiratory distress and circulatory collapse.
Tuberculosis
Mycobacterium tuberculosis infection forming caseating granulomas; symptoms include night sweats, haemoptysis.
Nasal Cannula
Low-flow oxygen device delivering 1–6 L/min (FiO₂ up to ~40 %).
Venturi Mask
High-precision oxygen mask with colour-coded valves delivering fixed FiO₂ (24–60 %).
Continuous Positive Airway Pressure (CPAP)
Device providing constant airway pressure to keep alveoli open and improve oxygenation.
Rheumatic Heart Disease
Autoimmune valve damage following untreated Group A streptococcal pharyngitis.
Mitral Regurgitation
Backward flow of blood from left ventricle to atrium due to damaged mitral valve.
Acute Glomerulonephritis
Immune complex-mediated inflammation of glomeruli leading to renal dysfunction after strep infection.
Congestive Heart Failure (CHF)
Inability of heart to pump or fill adequately, causing tissue hypoperfusion and fluid retention.
Systolic Heart Failure
Reduced ejection fraction due to weakened ventricular contraction.
Diastolic Heart Failure
Preserved ejection fraction with impaired ventricular relaxation and filling.
Left-Sided Heart Failure
Ventricular dysfunction leading to pulmonary congestion, dyspnoea and crackles.
Pulmonary Oedema
Fluid accumulation in lung interstitium/alveoli impairing gas exchange; hallmark of left HF.