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Parkinson’s Disease Epidemiology
Males > females
1.5x more than
ages 4070
progressive is faster with later diagnosis
Parkinson’s Disease
Loss of dopamine-producing brain cells
Decreased dopamine in the brain → inhibits acetylcholine
Parkinson’s Disease Clinical Manifestations
Resting tremors (mostly in hands. seen in asymmetrical motion)
Muscle rigidity
Slowness of movement (bradykinesia) or loss of movement (akinesia)
Postural instability (impaired balance and frequent falls)
Mask like face: not a lot of animation
Stooped postureL curve to shoulders
Slow, shuffling gait, defragmented turns
Parkinson’s Disease Diagnosis
Two or more cardinal symptoms with asymmetrical presentations\
No diagnostic test yet
Parkinson’s Disease Medications
Anticholinergics: “dried you up”, helping with oral secretion/drooling, helps decrease risk of aspiration
Dopamine-receptor agonists: 1st line treatment, Side effects: drowsiness, nausea
Parkinson’s Disease Nursing Diagnosis
Risk for falls
Fisk for constipation (bc of anticholinergic)
Powerlessness (loss of control, difficult to process)
Parkinson’s Disease Assessments
Tremors, rigidity, bradykinesia
Gag and swallow → risk for aspiration, speech therapist for further evaluation
Mobility (PT helps)
Bowel and bladder function (decrease in muscle control=incontinence)
Parkinson’s Disease Actions
Administer medications
Implement safety precautions (falls)
Facilitate nutritional intake (prevent aspirations: on a puree diet)
Elevate HOB: Helps with oral secretion to prevent aspiration
Suction equipment as bedside (“yanker/yonker”)
Encourage patients to participate in self-car activities
Facilitate interprofessional collaboration (nutritionalist PT, speech therapist)
Communication strategies (for late stages, not being able to talk anymore)
Parkinson’s Disease Teaching
Medication compliance: involve family/care givers. might not be able to take their own medications.
Safety precautions
Psychosocial support
Parkinson’s Disease Evaluating Care Outcomes
Prevention of falls
Can’t stop disease but can slow it down
Encourage independent living
Alzheimer’s Epidemiology
7.0 million Americans
7.4 million older than 65
Women > Men
Black, Hispanic populations
Dementia
Progressive cognitive decline that affects a person’s social and occupational functioning
Most common is Alzheimer’s dementia
Parkinson’s is a cause of dementia
Alzheimer’s Risk Factors
Family history
Alzheimer’s
No proven cause (think it is dying nerve cells accumulating in brain)
Decrease in neurotransmitters
No diagnosis test
Autopsy is the only definite confirmation
Alzheimer’s Clinical Manifestations
Forgetfulness: short term memory loss
Difficulty with language
Agnosia: inability to process sensory information
Emotional lability
Personality changes: typically opposite from before diagnosis
Loss of cognitive skills
Loss in ability to complete task
Difficultly problem solving
Alzheimer’s Diagnosis
No diagnostic test
History and physical
Neuro Exam: test cognitive function and physical exam
Dementia screening for 65+
Cerebral spinal fluid
MRI and CT
Alzheimer’s Treatment
Symptom management
Physical and emotional support
Donepezil, Rivastigmine, Galantamine, Memantine: increase AcH inhibiting Acetylcholinesterase
Alzheimer’s Nursing Diagnoses
Acute confusion
Caregiver role strain
Ineffective coping
Alzheimer’s Assessments
Weight (may forget to eat or drink)
Intake and Output
Bowel and bladder function (incontinence)
Skin: Pericare after bathroom
ADLs
Environment and safety: cooking and flight risks
Coping
Alzheimer’s Actions
Encourage/assist with feeding
Implement safety measures
Implement routine toileting practices
Provide clock and calendar
Speak calmly
Provide diversionary activites
Provide emotional support
Alzheimer’s Teaching
Teach families how to provide care
Label dangerous substances
Monitoring systems
Referral assistance
Alzheimer’s Evaluating Care Outcomes
Enhanced quality of life
Preparation for end of life