3.1 primary motives HUNGER

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3.1 primary motives

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HUNGER

  • prevents energetic deficits

  • maintain body weight

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Two models of hunger

  • Short-term appetite (glucostatic hypothesis).

  • Long-term energy balance (Lipostatic hypothesis)

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Glucostatic hypothesis - short-term appetite

  • Accounts for the onset & termination of hunger & eating.

  • short-term hunger cues regulate the initiation of meals, their size & termination.

  • The glucostatic hypothesis states that blood-sugar levels are critical to hunger (low bloodsugar - people feel hungry)

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PHYSIOLOGY OF HUNGER (two types)

  • SATIETY

  • HUNGER

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What brain structure regulates hunger & satiety

The hypothalamus — specifically, the lateral hypothalamus & ventromedial hypothalamus.

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What is the function of the lateral hypothalamus?

It is the appetite’s drive center — it stimulates hunger when activated.

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What is the function of the ventromedial hypothalamus?

It acts as the negative feedback’s appetite center — it signals satiety & stops eating.

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lipostatic hypothesis - long-term energy balance 

  • stored energy (fat mass) is available & used as a resource to supplement glucose-monitored energy regulation

  • fat produce energy

  • when the mass of fat stored drops below its homeostatic balance, adipose tissue secretes hormones into the bloodstream to promote weight gain (hormone → ghrelin)

  • when the mass of fat stored increases above its homeostatic balance, adipose tissue secretes hormones into the bloodstream to reduce food intake & promote weight loss motivation (hormone → leptin).

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when the mass of fat stored drops below its homeostatic balance

  • adipose tissue secretes hormones into the bloodstream to promote weight gain

  • (hormone → ghrelin)

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when the mass of fat stored increases above its homeostatic balance

  • adipose tissue secretes hormones into the bloodstream to reduce food intake & promote weight loss motivation

  • (hormone → leptin).

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Which physiological factors influence the hypothalamus in regulating hunger?

  • Liver (glucose levels)

  • Stomach dimensions (fullness)

  • Body temperature

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What happens when glucose levels in the liver are low?

  • The lateral hypothalamus is activated, triggering feelings of hunger.

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What happens when the stomach is full & glucose levels rise?

  • The ventromedial hypothalamus is activated, producing satiety signals.

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How does body temperature affect hunger?

  • Lower body temperature tends to increase hunger,

  • while higher body temperature decreases hunger.

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What are the two opposing centers in the hypothalamus controlling hunger & satiety?

  • Lateral hypothalamus – hunger center (LH)

  • Ventromedial hypothalamus – satiety center (VMH)

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Set point theory

  • body defends a fixed weight

  • linked to physiology, genetics & molecular biology.

  • adipose tissue intake & expenditure via a set point encoded in the brain

  • consistent with biological factors & energy balance

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if the set point changes in response to social influence on obesity

  • not a set point 

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Settling point theory

  • linked to psychology & nutrition 

  • replace the set point 

  • focus → human behaviour & environment 

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Settling point theory → body weight

  • influenced by a variety of factors

  • settles based on the interaction between biological, environmental & behavioural influence

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food available settling point theory

  • we have low expenditure.

  • our body fat settles at a higher point.

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Environmental influences affect eating behaviour

  • time of day

  • stress

  • sight

  • smell

  • appearance

  • taste of food

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Eating behavior increases when 

  • There is a variety of foods/flavours

  • large portions 

  • are in the presence of others 

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Restraint-release situations of hunger

  • Dieting & fasting can interfere with physiological guides.

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Dieting

  • Individual brings eating behaviour under cognitive rather than under physiological control

  • dieting causes subsequent bingeing.

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Counterregulation

  • a paradoxical pattern displayed by dieters

  • eat very little when just nibbling but eat very much after consuming a large, high-calorie “preload”

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Binge-eating leads to 

  • Conditions that threaten ego

  • High-calorie food

  • Alcohol

  • Anxiety & depression

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Cycle of binge eating / diet - illustration

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WHO - overweight + obesity

  • abnormal excessive accumulation of fat that can harm health

  • obesity has tripled since 1975

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overweight - BMI

by a body mass index greater than equal to 25 and obesity by a BMI greater than or equal to 30.

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how to prevent overweight

  • Decrease eating through self-regulation strategies.

  • Exercise motivation (increasing physical activity to expend fat stores).

  • Mindfulness over one’s environmental influences.