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Take in food and fluids
Begin digestive process
Propel feed into intestines
What are the primary functions of the UPPER GI tract?
Mouth
Esophagous
Stomach
What are the parts of the UPPER GI tract?
Food broken up and mixed wtih saliva, to start the digestion of carbs
What is the function of the mouth?
Mouth
Which part of the body is the most common for portal infections in young infants?
Ends in lower esophageal sphincter
Where does the esophagous end?
Risk for spit ups due to relaxed sphincter
What are infants at risk for if they have an immature esophagus?
Size and acid levels increase
Hydrochloric acid (protection) not adult levels until 6 months
What happens to the stomach as children age?
Digest and absorb nutrients
Detoxify and excrete waste
What is the primary function of the LOWER GI tract?
Small intestines
Large intestines
Liver
Pancreas
What are the parts of the LOWER GI tract?
Absorb nutrients and vitamins
What is the function of the small intestine?
Water reabsorption, synthesis of vitamin B and K
What is the function of the large intestine?
Bile production
Detoxification
Glycogen storage and breakdown
Vitamin storage
What is the function of the liver?
Digestive enzymes
Secretes insulin
What is the function of the pancreas?
Growth patterns (weight gain, growth chart)
Dietary concerns (what did they eat?)
Toilet training and bowel patterns (diarrhea, older kids: constipation)
Pt. hx (baseline)
Fam hx
What is the health hx assessment of a GI patient?
Ab Xray, US, CT
Endoscopy; UGI, colonscopy w/biopsy
Barium studies; swallow, UGI, enema
Esophageal pH probe
Stool studies; culture and sensitivity, occult blood
What is the dx testing of a GI patient?
Tube that goes into the nose to the bottom of the esophagous, right above the sphinchter
Meaures acidity (should be basic) and left for 24 hours
Determines GE REFLUX
What is the esophageal pH probe?
Inspection and observation
Ab size and shape (distended or tender)
Ascultation (before palpation)
Percussion
Palpation
What is the physical exam of a GI patient?
Cleft lip and palate
GERD
Appendicitis
Hypertrophic pyloric stenosis
Intssusception
Volvulus
Hirschsprung disease/ congenital aganglionic megacolon
Celiac
Short bowel syndrome
Dehydration
What are the GI issues of children?
Genetic or enivronmental
Mom smoking
Mom drinking
Medications
What is the CAUSE of cleft lip and palate?
Early developement; tissues do not fuse properly during development
Unilateral or bilateral
What is the PATHO for cleft lip and palate?
ASPIRATION, airway problems
Feeding difficulty (can’t attach to breast)
Altered dentation (teeth)
Ear infection (airway open with fluid)
Speech and language development difficulty
Issues with parent-infant bond
What are the COMPLICATIONS of cleft lip and palate?
Surgery at young age
2 separate surgeries
First lip, second palate (6-9 months)
Speech therapist (feeding)
What is the TX for cleft lip and palate?
Feeding management (growth, speech therapist)
Use of a special feeding device for cleft palate
CPR training (risk of aspiration)
Parent support and bonding
Pre and post op education
Long term plan and follow-up
What is the fam edu for cleft lip and palate?
C: calming techniques
L: lip suture line care (AB ointment, keep clean)
E: evaluate plan
F: feeding techniques
T: teaching
L: lip protection (Logan bow)
I: infection (suture line)
P: Protective devices (no-nos on arm and face 24/7)
CLEFT LIP
What is the post op nuring care for CLEFT LIP?
P: pain management
A: airway management (assess swelling, HOB up)
L: liquid diet (3 weeks for sutures to heal)
A: avoid hard foods and objects in mouth (no straws, sippy cups, pacifiers)
T: too hot, spicy, citrusy fluids and foods should be avoided
E: educate parents on signs of infection (foul odor, oozing, bleeding), use of arm restraints (no-nos), and advancing diet
What is the post op nursing care for CLEFT PALATE?
Failure to thrive/ weight loss
Respiratory problems/infection (silent aspiration)
Hungry/ irritability (can’t keep food down)
“Wet burps” or vomiting after feeds
Infant: pain with feeding/posturing (arching is abnormal)
Older: dental erosion
What is the health hx and S/S for GERD?
pH probe (mild, mod, severe)
What is the DX for GERD?
Alter feeding techniques (initially, may outgrow)
Meds
Nissen fundoplication
What is the nursing management for GERD?
Small, frequent feedings with frequent burping
Upright positioning for 30-45 mins after feeds
Thickened formula with rice cereal (1tsp/oz) (heavier)
What does alter feeding techniques mean for management of GERD?
H2 blockers (Block acidity, -dine)
PPIs (-zole)
Prokinetics (increase stomach motility to empty faster)
What are the MEDS used for GERD?
Surgical procedure for severe GERD by wrapping the upper part of the stomach around the lower end of the esophagus
Risk for vomiting
Still need medications, not a tx
What is nissen fundoplication?
Obstruction of the appendix (stool)
Increased pressure (trapped mucus)
Edema
Bacterial overgrowth (gets caught inside)
Perforation (overtime can rupture)
What is the PATHO of appendicitis?
Perotinitis
Sepsis
What are the COMPLICATIONS of appendicitis?
Periumbilical pain
Progressing in intensity and localizing to the RLQ
N/V
Fever
Assessment
McBurney’s point (where appendix is, RLQ)
Rebound tenderness (release after pressing creates pain)
Increased WBC
What is the health hx and S/S for appendicitis?
Labs
Increased WBCs
DX
CT or US
What are the LABS/DX for appendicitis?
Non-perforated (uncomplicated)
Perforated (complicated)
What are the TWO TYPES of appendicitis?
Appendectomy
Preop AB
F/E
Pain meds
Possible post op AB
What is the TX for non-perforated appendicitis?
Appendectomy
Drainage and irrigation of peritoneal cavity
IV AB 7-14 days
What is the TX for perforated appenditcitis?
Pylorus becomes hypertrophied
Stenosis of the pyloric sphincter
Leads to obstruction
What is the PATHO for hypertrophic pyloric stenosis?
What are the RISK FACTORS for hypertrophic pyloric stenosis?
NON-BILIOUS emesis 30-60 mins after feeding (deydrated → shock, iv fluids)
Hungry and crying despite having been fed
Progressive weight loss
Physical exam
“Olive shaped” mass in upper ab
Progressive dehydration
What are the health hx and S/S for hypertrophic pyloric stenosis?
SURGICAL: pyloromyotomy (cut through thick pyloric muscle, letting food get through)
F/E management
Parental education
Pre and post op care
Resume feedings
Resume 1-2 days after surgery
What is the management for hypertrophic pyloric stenosis?
Bowel telescopes into a distal segment (slide inside another)
Edema and impaired blood circulation (ischemic)
Partial or total bowel obstruction (food and stool can’t move)
What is the PATHO for intussusception?
Sudden onset intermittent pain
Bilious emesis
“Currant jelly” stools (damaged bowel lining begins to bleed and produces mucus)
Lethargy
Physical exam
“Sausage” shaped mass (palpate)
What is the health hx and S/S of intussusception?
Barium or air enema (push the telescope section back into normal position)
effective in 95% of cases
Surgical repair (if enema doesn’t work or ischemia)
Pre/post op care
Emotional support
What is the management of intussusception?
Malrotation or twisting of the bowel that results in bowel obstruction
What is volvulus?
Children with neurological impairment
Who may have an increased risk for volvulus?
Pain
Bilious vomiting
Other signs bowel obstruction
(similar to intussusception)
What are the S/S volvulus?
X ray (differentiate from intussusception)
What is the DX for volvulus?
Surgery to prevent bowel ischemia
Nursing care is similar to intussusception who requires surgical tx
What is the TX and management for volvulus?
Lack of ganglionic cells in the bowel which cause inadequate motility (can’t move stool)
What is the PATHO for hirschsprung disease/ congenital aganglionic megacolon?
Not passing meconium for first 24 HOURS
Male
What are the RISK FACTORS for hirschsprung disease/ congenital aganglionic megacolon?
Ab distension and pain
Constipation (palpate)
Vomiting
Poor weight gain
Slow growth (don’t feel like eating)
What are the S/S hirschsprung disease/ congenital aganglionic megacolon?
First give golytely
Rectal suction biopsy
What is the DX of hirschsprung disease/ congenital aganglionic megacolon?
Surgical resection of the bowel with reanastomosis; “pull through” procedure
Depending on age and severity of disease, surgery may be done in “parts” and the child may have a temporary or permanent colostomy
Edu parent/child pre and post op
What is the TX for hirschsprung disease/ congenital aganglionic megacolon?
Autoimmune DO in genetically susceptible people
Inability to digest gluten
Found in wheat, barley, rye, some oats
Damages villi in small intestine (whole area not absorbing)
Malabsorbtion leading to malnutrition
What is the PATHO for celic disease?
Ab distension
Thin extremities
Poor muscle tone
Anemia
Steatorrhea
Poor weight gain, failure to thrive
What is the health hx and S/S for celiac disease?
Screening
Blood test
DEFINITIVE DX
Small bowel biopsy (shows villi atrophy)
GENETIC TESTING
What is the DX for celiac disease?
IgA antitissue transglutaminase antibodies (tTG2)
Will be positive in about 98% of patients for who are on a gluten containing diet
What is the blood test screening for celiac disease?
For human leukocyte antigens (HLA-DQ2 and HLA-DQ8)
Those with celiac disease usually carry 1 or both genes
What is the genetic testing for celiac disease?
Dietary alterations
Strict gluten-free diet for life (rice is ok)
Vitamins if malnourished
Referral to dietician
Close monitoring of growth
What is the management for celiac disease?
Typically caused by congenital malformations of the GI tract or surgical resection of the small intestine at a young age (eg. premature infants with NEC); too little functional small intestine
What is short bowel syndrome?
Severe malabsorption
Inability to gain weight
Vitamin and mineral deficiencies
What are the S/S of short bowel syndrome?
NO CURE
IV total parenteral nutrition via central line and enteral feedings as tolerated
What is the TX for short bowel syndrome?
Infection
Liver disease (due to long term TPN administration)
What are the complications for short bowel syndrome?
Occurs more readily in infants and young children due to:
Greater proportion of body water in extracellular fluids, with increased sodium and chloride
Have a higher metabolic rate and experience greater insensible fluid losses through the skin and GI tract
Higher body surface area (BSA) in ratio to body mass
2-3x higher in infants
5x higher in premies
Immature renal system in infants, is less capable to concentrate urine like adults
What are the RISK FACTORS for dehydration in children?
Reduced fluid intake or fluid loss
Sudden, rapid ECF loss
Imbalace in electrolytes
Loss of ICF
Cellular dysfunction
Hypovolemic shock
Death
How does dehydration in children quickly lead to shock and death?
Vomiting
Diarrhea
Fever
Hyperventillation
Burns
Trauma/shock
Hemorrhage
Diabetes
What are the types of decreased fluid intake or increased fluid loss?
Restore fluid volume and prevent hypovolemia
What is the GOAL of dehydration in children?
Tachypnea and tachycardia, thready pulse
Hypotension
Peripheral vasoconstriction
Sunken fontanelle
Reduced LOC, lethargy
Parched
Dry mucous membranes
Reduced tissue turgor, tenting
Oliguria (low UOP)
Eyes sunken and tearless
Sudden weight loss
Reduced cap refill
Cold, cyanotic extremities
What are the S/S dehydration in children?
Mild to moderate: Oral replacement therapy (ORT)
Pedialyte, Rehydralyte, Enfalyte recommended
Severe: IV fluids management; 20 mL/kg with LR or NS
What is the TX for dehydration in children?
For mild to moderate dehyration if NO vomiting or lethargy
When do you give ORT?
Oral: 50-100 mL/kg or an oral rehydration solution + relacing continued losses
Given over 3-4 HOUR period
How do you give ORT?
Can give up to 3 boluses of fluids to stablize patient
Then can move to IV fluids and ORT when able
How to give LR or NS for severe dehydration?