EMERGENCY NURSING (TRAUMA)

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Last updated 4:09 AM on 11/14/25
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111 Terms

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  • Blunt acceleration forces

  • Deceleration forces

  • Penetrating forces

Most common forces involved in head trauma are:

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Blunt acceleration forces

when the head is struck with an object

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Deceleration forces

when the head is moving and strikes a stationary object

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Penetrating forces

when an object enters the head

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  • Missed injuries

  • secondary neurologic injuries (i.e. injuries to the brain and brainstem)

— are common in emergency care settings.

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  • to identify any obvious signs of head trauma and underlying neurological injury

  • to provide baseline data which can be used to identify a developing neurological injury

Two fundamental goals for neurological assessment:

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  • intracranial hemorrhage

  • edema of the soft tissues of the brain

Problems with PERRLA are often the first signs of increased ICP due to, for example, an — or —, etc

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  • disconjugate gaze (deviation of one eye)

  • ptosis (drooping of the eyelid/s)

— are common signs of neurologic injury:

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Decorticate / flexion (where the patient's arms are drawn rigidly up against their chest)

-– affects vision, hearing, eye movement, motor control and consciousness

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Decerebrate / extension (where the patient's arms turn rigidly outwards against the sides of their body)

– affects coordination, sensation, facial expression, swallowing, breathing

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  • hypertension (a compensatory mechanism to maintain cerebral blood flow)

  • cardiac dysrhythmia (due to brainstem dysfunction)

  • hyperthermia (as cerebral dysfunction and metabolism increases)

Signs of a serious brain injury include:

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  • Hypertension

  • Widening pulse pressure (i.e. increasing distance between systolic and diastolic BPs)

  • Bradycardia

A patient with a severe late brain injury, where there is significant pressure on the brainstem, will often demonstrate signs known as 'Cushing's triad':

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  • a CT scan be undertaken if a patient has a GCS of 13 on initial presentation or a GCS of <15 2 hours after the injury

  • a suspected skull fracture

  • a post-traumatic seizure

  • any focal neurological deficit

  • more than 1 episode of vomiting

The National Institute of Health and Clinical Excellence's Head Injury: Assessment and Early Management (CG176) guideline recommends that:

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Scalp lacerations

  • — is highly vascularised and — often bleed profusely

  • typically managed by direct pressure to control initial hemorrhage, and subsequent wound repair using sutures, staples or clips

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  • linear

  • depressed

Skull fractures:

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linear skull fracture

  • the fracture is non-displaced, and there is no or minor neurological damage

  • usually only require supportive care (e.g. rest, pain management, etc.)

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depressed skull fracture

  • one side of the fracture displaces below the other, and there is moderate to severe neurological damage

  • often require surgical repair, including plating.

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  • hemotympanum (leakage of blood from the ears)

  • 'panda' or 'raccoon' eyes (circular bruising around the eyes)

  • leakage of cerebrospinal fluid from the ear/nose

  • Battle's sign (bruising behind the ears)

Signs of a suspected skull fracture:

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Contusion

a bruise on the scalp or on the surface of the brain

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  • Coup

  • Contrecoup

When acceleration-deceleration forces are involved in the injury, two contusions on the surface of the brain may result:

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Coup

the initial site of impact

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Contrecoup

the opposite side of the brain, as it rebounds inside the skull

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  • an altered LOC

  • nausea and or vomiting

  • visual disturbances

  • weakness

  • difficulties with their speech.

Patients with contusion typically present with:

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Subdural or epidural hematoma

bleeding beneath or between the skull and one of the layers of the dura mater or arachnoid mater

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  • severe headache

  • pupillary dilatation on the same side of the body to that which they sustained the traumatic injury

  • hemiparesis (i.e. one-sided weakness) on the opposite side

Patients with haematoma often present with:

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Concussion

mild traumatic brain injury that involves a loss of consciousness with associated disruptions to neurological functioning

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  • nausea/vomiting

  • temporary amnesia

  • minor confusion and disorientation

  • Headache

  • Dizziness

  • Drowsiness

  • Irritability

  • visual disturbances

People with concussion will display mild, transient neurological symptoms such as:

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  • non-narcotic analgesia

  • anti-emetics

  • fluid therapy

Care for a patient who has experienced a concussion involves regular observation to identify more serious brain injury, and symptomatic management —

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Diffuse axonal injury (DAI)

a severe traumatic brain injury that results in shearing of axons, key structures within the white matter of the brain

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  • extended loss of consciousness

  • flexion or extension posturing

  • dysfunction of the autonomic nervous system

Patients with DAI typically present with:

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  • cerebral edema

  • increase in cerebral blood flow (e.g. hemorrhage, acute hypertension, etc.)

Increased ICP Causes:

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  • decreased LOC

  • changes in vital signs (including Cushing's triad)

  • pupillary dilatation

  • decrease in motor function

  • severe headache

  • nausea/vomiting

Patients with increased ICP will present with:

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brain death

Where ICP is very high, pressure on the brainstem may result in — where brain function completely and irreversibly ceases.

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  • treating the greatest threat to life first (management of airway, breathing and circulation)

  • effectively managing patient’s pain (contributes to an increase in ICP)

  • use small but frequent doses of intravenous opioid

Providing care to a person with injuries resulting from head and/or neurologic trauma

Focus treatment on:

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Orthopedic Trauma

bones, surrounding soft tissue, and associated neurovascular structures including the nerves and blood vessels

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  • road traffic accidents

  • falls from height

  • assaults

  • sports and recreation accidents

  • general accidents at work or in the home

Most common mechanisms that cause orthopedic injuries are:

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  • the long bones

  • hemorrhage

  • shock and severe pain

Orthopaedic injuries typically require urgent (rather than immediate) care; however, some orthopaedic injuries - including those involving — which may result in — - may require immediate care.

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  • obvious deformity

  • lacerations

  • contusions

  • edema

  • abrasions

  • pain

Examine trauma site/s for obvious signs of orthopedic injury:

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  • Colour

  • Temperature

  • Pulses

  • Sensation

  • motor function in the affected limb/s

Do a focused neurovascular assessment, where any of these signs are identified: (orthopedic injury)

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Strains and sprains

  • involve minor damage to a muscle, usually at its point of attachment to a tendon

  • although painful, strains and sprains do not require urgent care

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  • support

  • ice

  • elevate the affected limb

  • manage pain using oral analgesia (e.g. paracetamol, ibuprofen)

  • avoid weight-bearing for 24 to 72 hours

Patients should be encouraged to: (strains and sprains)

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Fractures

any disruption or break in the bone

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  • closed

  • open compound

Fractures may be described as:

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closed

where the bone is broken but the skin is intact

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open compound

where the bone is open and protrudes through the skin

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  • obvious deformity (confirmed by X-ray)

  • pain (which may be severe)

  • swelling

  • ecchymosis in the affected region

Patients with fractures usually present with:

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  • Immobilization of the fracture

  • traction splint or an adjacent leg splint for fractures of the femur

  • vacuum splint for all other long bone fractures

  • temporary casts may also be used

  • Minor fractures may be reduced (i.e. realigned) and fixed in the emergency care setting

  • More severe fractures require surgical intervention

Fracture Management:

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  • broken bones may lacerate vital organs/arteries/nerves

  • fractures of the large bones may result in hemorrhage, etc.

Fracture Complications:

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Dislocations

occurs when a joint exceeds its normal range of motion, and the joint surfaces become disconnected

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  • shoulder

  • elbow

  • finger

  • hip

  • knee/patella

  • ankle and toe

Common points of dislocation:

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  • obvious deformity (confirmed by X-ray)

  • pain (which may be severe)

  • swelling

  • ecchymosis in the affected region

Patients with dislocation usually present with:

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Subluxation

– term used to describe a dislocation if there is only partial or incomplete displacement of the joint surfaces

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Traumatic Amputation

involves the removal of all or part of a digit, limb or other body structure such as foot, hand, ear, nose, etc.

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  • Resuscitation is usually required for larger amputations involving hemorrhage

  • Preservation of the stump (focus of care in the emergency setting)

  • irrigating with normal saline to remove gross contamination

  • moist dressing

  • elevation and prophylactic antibiotic administration

Traumatic Amputation Management

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Limb Replantation

  • a complex microsurgical procedure that allows patients to have severed limbs reattached or “replanted” to their body

  • Most patients need — within hours of experiencing traumatic injuries

  • It is important to highlight that this is not successful in all cases.

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Muscle injuries

including injuries to the rotator cuff (muscles in the shoulder) and meniscus (fibrocartilage in the knee)

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rotator cuff

muscles in the shoulder

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meniscus

fibrocartilage in the knee

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Crush Injury

occurs when part of the body, typically a digit or limb, is crushed for a prolonged period

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  • necrosis of the crushed body part

  • symptoms of 'systemic crush syndrome’

  • myoglobinuria [myoglobin is a muscle breakdown product]

  • acidosis – release of lactic acid

  • renal failure – free myoglobin are too big to cross the glomerulus, resulting to plugging of holes

  • cardiac disruption – release of potassium systemically

A patient with crush injury may present with:

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myoglobinuria

myoglobin is a muscle breakdown product

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acidosis

release of lactic acid

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renal failure

free myoglobin are too big to cross the glomerulus, resulting to plugging of holes

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cardiac disruption

release of potassium systemically

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Compartment Syndrome

occurs when excessive pressure builds up inside an enclosed muscle space or compartment in the body

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Compartment Syndrome

causes a decrease in perfusion and function of the tissues within that space

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Compartment Syndrome

usually develops between 6 and 8 hours after the primary injury, when the compartment pressure exceeds capillary pressure and becomes clinically evident when the compartment pressure exceeds venous pressure

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Compartment Syndrome

results in lack of outflow, which worsens the compartment pressure as blood and edema back up in the space.

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  • Pain

  • disproportionate to the injury

  • worse with passive stretching of the muscles in the compartment (e.g., dorsiflexion of the foot for a compartment syndrome of the calf)

  • Paresthesia

Compartment Syndrome (5 P’s) Early signs:

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  • Pallor

  • Paralysis

Compartment Syndrome (5 P’s) Late signs:

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Pulselessness – occurs when the pressure finally exceeds arterial pressure (results in the need for amputation because the nerves and muscles are dead)

Compartment Syndrome (5 P’s) Last sign:

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Fasciotomy

  • a surgery to relieve swelling and pressure in a compartment of the body

  • tissue that surrounds the area is cut open to relieve pressure

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  • partial or complete paralysis

  • loss of motor ability

  • loss of conscious function of body processes

  • life-threatening CNS dysfunction (problems with A,B,C)

Spinal Trauma
Damage to the spinal cord, depending on the location of the injury, may result in:

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  • road traffic accidents

  • falls from height

  • assaults

  • sports and recreation accidents

  • general accidents at work or in the home

Mechanisms of spinal injury:

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Neurogenic shock

  • occurs when a spinal cord injury is complete, and all sensation and motor function below the level of the injury immediately ceases

  • is generally irreversible

  • patients must be carefully managed using spinal immobilization techniques to prevent further injury to the unstable cord in the immediate post-injury period

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Autonomic Dysreflexia

a complication of spinal cord injury which occurs above the level of the T6 vertebrae

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Autonomic Dysreflexia

occurs when impairments in the functioning of the sympathetic nervous system lead to a massive, uncontrolled cardiovascular response

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Autonomic Dysreflexia

often triggered by simple causes such as a full bladder or bowel, and it can occur any time after the onset of a spinal injury

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  • a sudden severe headache

  • hypertension

  • bradycardia

  • anxiety

  • nausea/vomiting

  • a combination of profuse sweating above and coolness below the level of the injury

Patients with Autonomic Dysreflexia will present with:

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  • hemorrhage

  • edema

  • hypoperfusion of the spinal cord

  • endogenous biochemical responses

Secondary injuries to the spinal cord

Manifestations:

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  • cervical spine immobilization

  • Spinal board

Management of Spinal Injuries:

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cervical spine immobilization

– routinely done by paramedics for suspected or actual head injury

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Spinal board

– used for those who complain of altered sensation in their peripheries

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Thoracic Trauma

any traumatic injury affecting the chest area

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  • blunt forces (e.g. the sudden deceleration, compression and or direct blows sustained in road traffic accidents)

  • penetrating injuries (e.g. stabbings, gunshot wounds, etc.)

Thoracic Trauma Causes:

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  • hypoxemia (a lack of oxygen in the blood)

  • hypoventilation (a low respiratory rate)

Two main problems associated with actual or potential thoracic injuries:

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Rib fractures

may involve a single rib or multiple ribs, and most often occur in the fourth to the tenth rib

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flail injury

  • a section of the ribcage moves independently from the main ribcage during breathing

  • caused by severe rib fractures, those involving eight or more ribs

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  • Splinting

  • rest

  • pain management

— are all key interventions in the management of rib fracture.

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Pneumothorax

involves an accumulation of air in the pleural space around the lung/s, and the resultant 'collapse' of the lung/s

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  • chest pain

  • dyspnea

  • tachycardia

  • decreased or absent chest sounds on the side/s of the collapsed lung/s

  • tracheal deviation away from the side of the pneumothorax

Patients with pneumothorax typically present with:

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Hemothorax

is a similar condition where blood fills the pleural space

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Cardiac tamponade

occurs when there is a rapid accumulation of the blood in the pericardial sac, which surrounds the heart

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  • hypotension

  • muffled or indistinct heart sounds

  • distended neck veins

'Beck's triad’:

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  • increasing dyspnea

  • decreased level of consciousness (LOC)

  • eventual death

If Cardiac Tamponade is left untreated, the condition results in —

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acute circulatory dysfunction including cerebral hypoxia

As the pericardial sac is a closed space with a definite volume, blood in this space places pressure on the ventricles and prevents them from filling to capacity. This results in —

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  • blunt force trauma

  • penetrating forces may also be seen

  • road traffic accidents (including pedestrian-versus-vehicle accidents)

  • falls from height

  • assaults

Abdominal and Genitourinary Trauma

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  • injuries to the solid organs (kidneys, pancreas, spleen, liver)

  • injuries to the hollow organs (stomach, urinary bladder, intestines)

Abdominal and Genitourinary Trauma Two types:

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Lacerations to the solid organs

  • liver and spleen

The — are common sites of traumatic abdominal injuries. Because the liver holds up to 25% of the body's circulating blood at any given time, injuries to the liver are particularly significant, often resulting in major hemorrhage.

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Renal injuries

  • kidneys

The — are another solid organ which are commonly affected by traumatic abdominal injury.